ライフサイエンスコーパス: loss-of-function mutant

1 vity, phenotypes not observed with the eIF4G loss of function mutant.

2 to photoperiod in a manner similar to a cdf loss-of-function mutant.

3 activated by NaCl are absent from the Atann1 loss-of-function mutant.

4 l in vivo, as they complemented a yeast GLXI loss-of-function mutant.

5 tion mutant of PLN, whereas R14del is a mild loss-of-function mutant.

6 truncated rhBARF1 from clone 16 rhLCV was a loss-of-function mutant.

7 (BL)-insensitive phenotype similar to a bri1 loss-of-function mutant.

8 an avirulent bacterial pathogen in the bap1 loss-of-function mutant.

9 utant than to either wild-type I domain or a loss-of-function mutant.

10 ity phenotype was similar to that of the AS2 loss-of-function mutant.

11 he striking abnormalities of lin-4 and let-7 loss of function mutants.

12 e Dlx1/2 mutant phenotypes, we made compound loss-of-function mutants.

13 nted the observation of strong phenotypes in loss-of-function mutants.

14 enient and effective approach for generating loss-of-function mutants.

15 altered in MPK6 overexpression lines or mpk6 loss-of-function mutants.

16 ctase were increased in rfd1 and the AUX/IAA loss-of-function mutants.

17 ly predicts root growth, as observed in bri1 loss-of-function mutants.

18 ns, R205X and E239X, were shown to be Dyrk1a loss-of-function mutants.

19 d trunk mesoderm, and closely resemble nodal loss-of-function mutants.

20 redundancy was confirmed through analysis of loss-of-function mutants.

21 ted nutations were also eliminated in etr1-7 loss-of-function mutants.

22 s a permissive temperature for wild-type and loss-of-function mutants.

23 ethality that often occurs in both gain- and loss-of-function mutants.

24 what E2F actually does based on analyses of loss-of-function mutants.

25 ina can often complicate the analysis of CBP loss-of-function mutants.

26 f and floral organ production in stm partial loss-of-function mutants.

27 9F, and Y744F), but not in the ADA and W650R loss-of-function mutants.

28 ressed "kinase-dead" ILK fully rescues pat-4 loss-of-function mutants.

29 parable in expressivity to the corresponding loss-of-function mutants.

30 th pathways was confirmed by the analysis of loss-of-function mutants.

31 creased cell migration beyond canonical PTEN loss-of-function mutants.

32 naling, and this effect is blocked in usp-46 loss-of-function mutants.

33 e may be of interest to restore transport in loss-of-function mutants.

34 show an increase in Lys acetylation in srt2 loss-of-function mutants.

35 ark respiration was observed in the eIFiso4G loss of function mutant, a reduction in chlorophyll leve

36 as a promoter, may be mutagenized to isolate loss-of-function mutants able to survive under selection

37 ality of AtCDA in vivo was demonstrated with loss-of-function mutants accumulating high amounts of cy

38 A previously described loss-of-function mutant affecting both isoforms, sr45-1,

39 r show that removing MUL1 in PINK1 or parkin loss-of-function mutant aggravates phenotypes caused by

40 istant locomotory behavior, resembling slo-1 loss-of-function mutants, albeit to a lesser extent.

41 We show that the partial loss-of-function mutant alix-1 displays reduced vacuolar

42 on during neural development, we generated a loss-of-function mutant allele of m-numb.

43 Seeds of a loss-of-function mutant allele, imb1, show impaired coty

44 Biallelic loss-of-function mutant alleles underlie several differe

45 However, the NAD286G loss-of-function mutant also associated with the host fa

46 Through a combination of loss-of-function mutant analyses, genetic mapping, and t

47 A loss-of-function mutant analysis also revealed that sing

48 A combination of loss-of-function mutant analysis and protein interaction

49 of violaxanthin de-epoxidase in the eIFiso4G loss of function mutant and an increase in its xanthophy

50 With the combination of two reagents, an HC loss-of function mutant and the green fluorescent protei

51 Experiments using a C5a peptidase loss-of-function mutant and an intranasal infection mode

52 catula development by isolating the lfl/wox3 loss-of-function mutant and performing genetic crosses w

53 q544) allele behaves genetically as a strong loss-of-function mutant and putative null.

54 Accordingly, cells behave like a rad53 loss-of-function mutant and show reduced NHEJ efficiency

55 ta roles of Pht1;1 and Pht1;4, we identified loss-of-function mutants and also created a double mutan

56 Loss-of-function mutants and ectopic expression assays r

57 We utilize both gain- and loss-of-function mutants and gene expression patterns to

58 The stabilization of loss-of-function mutants and insensitivity of gain-of-fu

59 GR3, named CGR2, and evaluated the effect of loss-of-function mutants and over-expression lines of CG

60 With the enhanced ability to generate loss-of-function mutants and overexpression lines, the f

61 a Drosophila fragile X syndrome model using loss-of-function mutants and overexpression of the FMR1

62 Analysis of atprx71 loss-of-function mutants and plants overexpressing AtPRX

63 The combined use of cytochrome c (CYTc) loss-of-function mutants and respiratory complex III inh

64 validate genetic experiments performed with loss-of-function mutants and RNA interference (RNAi) lin

65 l fate and polarity, through the analysis of loss-of-function mutants and transgenic plants that ecto

66 riants conferring resistance to malaria are 'loss-of-function' mutants and appear to be recent polymo

67 isolated dominant-negative mutants, unc-108 loss-of-function mutant animals are defective in locomot

68 In Lrrk loss-of-function mutants, anterograde movement of GOPs w

69 n oncogene and neuroprotective protein whose loss-of-function mutants are associated with certain typ

70 nd phenotypes of blh6, knat7, and blh6 knat7 loss-of-function mutants are consistent with the existen

71 Because BOB1 loss-of-function mutants are embryo lethal, we used a pa

72 d as having highly branched phenotypes, ccd1 loss-of-function mutants are indistinguishable from wild

73 pment of the trophectoderm lineage and Eomes loss-of-function mutants arrest at implantation.

74 present study, several gain-of-function and loss-of-function mutants as well as engineered mutants o

75 CYTc loss-of-function mutants, as well as the d-LDH mutants,

76 Strong loss-of-function mutants assemble apolar intersecting mi

77 The method involves generation of partial loss-of-function mutants, at either buried or functional

78 eritability of Cas9 activity in heterozygous loss-of-function mutant backgrounds, to rapidly evaluate

79 er Myc mutants, MycDeltaMBIV is not a simple loss-of-function mutant because it is hyperactive for G2

80 We find that in dfer loss-of-function mutants beta-catenin is hypophosphoryla

81 n of an AWC(ON) marker is initiated in nsy-7 loss-of-function mutants, but subsequently lost, so that

82 n wild-type maize and in the isogenic mop1-1 loss-of-function mutant by using Illumina's sequencing-b

83 action, we circumvented the lethality of keg loss-of-function mutants by silencing KEG using an artif

84 In DSAS-6 loss-of-function mutants, centrioles failed to close and

85 We report here characterization of a C-tail loss-of-function mutant, CF327A, and a related suppresso

86 have reduced FLC expression, but, unlike clf loss-of-function mutants, clf-59 mutants do not display

87 , although stomata of the Arabidopsis syp121 loss-of-function mutant close normally in ABA and high C

88 rs1-2 ers2-3 and triple etr2-3 ers2-3 ein4-4 loss-of-function mutants constitutively nutated in air.

89 ucose-fed conditions, the Arabidopsis sr45-1 loss-of-function mutant contains higher amounts of the e

90 Ddok loss-of-function mutant (Ddok(PG155)) germ-line clones p

91 The dominant negative loss-of-function mutant Delta(N)117 was co-immunoprecipi

92 ci appear to be "Myddosomes." The MyD88 S34Y loss-of-function mutant demonstrates how proper cellular

93 Here, we show that single luh loss-of-function mutants develop normal flowers, but lug

94 Zebrafish rad51 loss-of-function mutants developed key features of FA, i

95 mutants, although it was reported that etr1 loss-of-function mutants display a growth defect limitin

96 demonstrated to be critical in vivo as pux1 loss-of-function mutants display accelerated growth rela

97 We show that partial loss-of-function mutants display temperature-sensitive (

98 A cdf-2 loss-of-function mutant displayed impaired growth and re

99 bt2 loss-of-function mutants displayed a hypersensitive resp

100 The weak loss-of-function mutant dse1 exhibits pleiotropic phenot

101 activity, but they all appear to manifest as loss-of-function mutants due to defects in solubility, a

102 Here we show that in mef2ca(b1086) loss of function mutant embryos and early larvae, develo

103 Biochemical analysis of single FPGS loss-of-function mutants established that folate polyglu

104 -function ET receptor mutant, etr1-3, or the loss-of-function mutants etr1-7 and ers1-3 and the wild

105 Loss-of-function mutants exhibit bouton overgrowth at la

106 Conversely, wdr-20 and wdr-48 loss-of-function mutants exhibit changes in locomotion b

107 associated with vascular tissues, while vup1 loss-of-function mutants exhibit collapsed morphology of

108 ted deletion of the cmpy locus and find that loss-of-function mutants exhibit excessive NMJ growth.

109 At elevated temperatures, dao loss-of-function mutants exhibit seizures associated wit

110 Frazzled loss of function mutants exhibited similar defects in sy

111 eIFiso4G loss of function mutants exhibited smaller cell, leaf, p

112 sleepless (sss) loss-of-function mutants exhibited altered Shaker locali

113 the dominant act-5 mutation, or a recessive loss of function mutant, exhibited normal morphology and

114 similar to glh-1/glh-4(RNAi), the kgb-1(um3) loss-of-function mutant exhibits germline over-prolifera

115 root growth after Al exposure, because alt2 loss-of-function mutants fail to halt root growth after

116 at4, an At4 loss-of-function mutant fails to redistribute Pi to the

117 In a loss of function mutant for genes encoding PHR1 and PHL1

118 onstrated that the alpha 8 G22R isoform is a loss-of-function mutant for alpha 8, as well as a domina

119 Here, the Arabidopsis loss-of-function mutant for annexin1 (Atann1) was found

120 In this study, we used loss-of-function mutants for each receptor isoform to de

121 This method can provide homozygous mammalian loss-of-function mutants for forward genetic application

122 In gain- and loss-of-function mutants for Gsx1 and Olig1/2, we observ

123 planta by feeding [(3)H]serine to the WT and loss-of-function mutants for OAS-TLs in the cytosol, pla

124 onfers increased Al tolerance similar to the loss-of-function mutants for the cell cycle checkpoint g

125 te this issue, we produced a complete set of loss-of-function mutants for the three annotated Arabido

126 n297, Ala301, Phe307, and Tyr308 represented loss-of-function mutants; furthermore, the measurable re

127 l(2)tn, along with loss-of-function mutants generated for tn, showed no rel

128 d endoreplicating tissues in Drosophila gatA loss-of-function mutants grow slowly and never achieve w

129 UPR loss-of-function mutants, hac1Delta and ire1Delta, are a

130 Consistently, zat12 loss-of-function mutants had higher Fe content than the

131 The cpr1-2 (cpr30-1) loss-of-function mutant has constitutive defense respons

132 ment from fertilization, the cyp78a8 cyp78a9 loss-of-function mutant has reduced seed set due to oute

133 Chloroplasts prepared from fro7 loss-of-function mutants have 75% less Fe(III) chelate r

134 Loss-of-function mutants have a pleiotropic phenotype in

135 Both NUA and AtMAD2 loss-of-function mutants have a shorter primary root and

136 rte1 loss-of-function mutants have an enhanced ethylene respo

137 tory molecules is wide-ranging, however, few loss-of-function mutants have been identified in miRNA g

138 Because no loss-of-function mutants have been identified, the impor

139 ate earlier than the wild type, whereas etr2 loss-of-function mutants have increased sensitivity to A

140 Large-scale screens for loss-of-function mutants have played a significant role

141 msca1 loss-of-function mutants have reduced meristem size and

142 Instead, etr1 loss-of-function mutants have reduced sensitivity to abs

143 eproductive and social behaviors, yet rodent loss-of-function mutants have relatively subtle behavior

144 ession phenotypes closely resembles those of loss of function mutants in the para-encoded sodium chan

145 recordings identified the R215H mutant as a loss-of-function mutant in inducing GABAergic synaptogen

146 A loss-of-function mutant in Kif20b, magoo, was found in a

147 We report here a loss-of-function mutant in the Arabidopsis homolog of RA

148 By screening for loss-of-function mutants in a Caenorhabditis elegans kil

149 natural ELF3 polyQ variants phenocopied elf3 loss-of-function mutants in a common reference backgroun

150 All three loss-of-function mutants in Arabidopsis (Arabidopsis tha

151 toperiod-dependent flowering in plants, with loss-of-function mutants in barley (Hordeum vulgare), le

152 Generation of all major OAS-TL double loss-of-function mutants in combination with radiolabele

153 Loss-of-function mutants in dPIP4K show reduced body wei

154 In particular, pink1 and parkin loss-of-function mutants in Drosophila show similar phen

155 rgan defects and indeterminacy that resemble loss-of-function mutants in E-function floral organ spec

156 ies converge on similar causes for olfactory loss-of-function mutants in evolutionary transitions to

157 te that the phenotypes associated with novel loss-of-function mutants in EXO70, are entirely consiste

158 Here, we report single and higher-order loss-of-function mutants in members of the cation/proton

159 Loss-of-function mutants in one of the genes, designated

160 Loss-of-function mutants in PRC2 subunits initially deve

161 In this study, we isolated loss-of-function mutants in the Arabidopsis MIPS1 gene f

162 Loss-of-function mutants in the corresponding genes had

163 Here, by generating and analyzing loss-of-function mutants in the liquid facets-Related (l

164 ole of tomosyn in neurosecretion we analysed loss-of-function mutants in the single Caenorhabditis el

165 utant hearts of heartstrings (hst), the tbx5 loss-of-function mutants in zebrafish.

166 rate that the gonadogenesis defects of gon-2 loss-of-function mutants (including a null allele) can b

167 in addition to classical screens for simple loss-of-function mutants, including genetic modifier scr

168 psis resistance to Pst DC3000, whereas ADPG2 loss-of-function mutants increase the resistance to the

169 Analyses of BR-C, E74A, and E93 loss-of-function mutants indicate that these genes regul

170 tantly, the BIG signature was reduced in Bmp loss-of-function mutants, indicating Bmp-regulated targe

171 similar to those seen in ced-10;mig-2 double loss-of-function mutants, indicating that they interfere

172 sion was drastically reduced in beta-catenin loss-of-function mutants, indicating that Wnt signaling

173 The AtSAP18 loss- of-function mutant is more sensitive to NaCl, and

174 The ABA hypersensitive phenotype of FyPP loss-of-function mutants is ABI5 dependent, and the amou

175 osmotic stress, while the growth of erf5erf6 loss-of-function mutants is less affected by stress.

176 h and show that their derepression in miR164 loss-of-function mutants is likely to account for most o

177 The ability of two loss-of-function mutants, L31A and L31C, of phospholamba

178 n wild-type (WT) STAT1 cells, as well as the loss-of-function mutants L706S and Y701C.

179 While elt-7 loss-of-function mutants lack a discernible phenotype, s

180 een called "master control" proteins because loss-of-function mutants lack eyes and ectopic expressio

181 These C-terminal loss-of-function mutants lacked a dominant negative phen

182 phenotype of the etr1-6;etr2-3;ein4-4 triple loss-of-function mutant line was examined.

183 Genomic profiling of H3K27me3 in loss-of-function mutant lines for Maize Enhancer of zest

184 importance of Nisch, here we generated Nisch loss-of-function mutant mice and analyzed their metaboli

185 In contrast to loss-of-function mutants, moderate overexpression of BLF

186 Using a loss-of-function mutant mouse lacking the guanylate kina

187 al prefrontal cortex (n = 21); and 4) a Nrg3 loss-of-function mutant (n = 59) to functionally implica

188 d-type FIT2, whereas FIT1 and a FIT2 partial loss-of-function mutant, N80A, had significantly lower t

189 ll three alleles are similar to those of the loss-of-function mutants obtained by RNA interference or

190 Leaf reticulation in the re and rer3 loss-of-function mutants occurred, along with accumulati

191 etion mutant of FOF2 (FOF2DeltaF), or double loss of function mutant of FOF2 and FOL1 (FOF2-LIKE 1) p

192 Loss of function mutants of BGD2 are acyanogenic in leav

193 Loss of function mutants of NM1, Phe-1136 and dS2, that

194 (GBP) interact with the N terminus of GluTR Loss-of function mutants of ClpR2 and ClpC1 proteins sho

195 ants of AGL15, alone or when combined with a loss-of-function mutant of a closely related family memb

196 ressing bcl-xL, ced-9, and bcl-xL (G138A), a loss-of-function mutant of bcl-xL.

197 t, a constitutively active Toll mutant and a loss-of-function mutant of Cactus, an I kappa B-like fac

198 rel map development using barrelless mice, a loss-of-function mutant of calcium/calmodulin-activated

199 e biological function of GIF1, we isolated a loss-of-function mutant of GIF1 and prepared transgenic

200 R9C is a complete loss-of-function mutant of PLN, whereas R14del is a mild

201 Co-expression of a loss-of-function mutant of polycystin-2 in CHO cells doe

202 We also studied the phenotypes of a loss-of-function mutant of Prosalpha6T generated by targ

203 The loss-of-function mutant of SR1IP1 is more susceptible to

204 multi-copy suppressors of a cold-sensitive, loss-of-function mutant of the cyclin-dependent kinase C

205 expression phenotypes in overexpression and loss-of-function mutants of 15 Arabidopsis A-type heat-s

206 Here we report that loss-of-function mutants of AGL15, alone or when combine

207 We also construct conditional loss-of-function mutants of AGO1 to allow transcript pro

208 , we generated double, triple, and quadruple loss-of-function mutants of all four members of the RWA

209 n content and vein density were increased in loss-of-function mutants of Arabidopsis MYC2, a suppress

210 scriptional machinery during leaf senescence Loss-of-function mutants of Arabidopsis thaliana were us

211 Loss-of-function mutants of AtCAND1 were resistant to si

212 Pheromone preparations from loss-of-function mutants of daf-22, a gene required for

213 Because loss-of-function mutants of ftsZ(Bbu) might be lethal, t

214 we investigated the binding of A1 to GOF and loss-of-function mutants of GP Ibalpha with mutations in

215 We found that loss-of-function mutants of LecRKA4.2 and LecRKA4.3 exhi

216 Loss-of-function mutants of LOX6 were more attractive to

217 ype A. nidulans versus temperature-sensitive loss-of-function mutants of nudA and nudF.

218 Loss-of-function mutants of PALM1 develop dissected leav

219 Using loss-of-function mutants of Ros and inducible epidermal

220 Loss-of-function mutants of rtp replicate this axonal pr

221 Using gain- and loss-of-function mutants of seven transcription factors

222 tants plcS and dsbA, we show that Drosophila loss-of-function mutants of Spatzle, the extracellular l

223 cal neurons and impaired root hair growth in loss-of-function mutants of the ATL1 ortholog rhd3 in th

224 Loss-of-function mutants of the gene goa-1, which codes

225 Here, we analyzed loss-of-function mutants of the unicellular cyanobacteri

226 Furthermore, the loss-of-function mutants of these genes in the fly cause

227 Although loss-of-function mutants of UBP27 do not show obvious ph

228 ysis of root phenotypes induced by gain- and loss-of-function mutants or in treatments with GLV-deriv

229 We also examined loss-of-function mutants ourselves, identifying new guid

230 In addition to this loss of function, mutant p53 can have a dominant negativ

231 In mrg-1 loss-of-function mutants, pairing is compromised specifi

232 The loss of function mutant phenotype and localization of an

233 comprehensive dataset of 2,400 genes with a loss-of-function mutant phenotype in Arabidopsis.

234 Generally the loss-of-function mutant phenotype is simplified from the

235 silencing in Drosophila embryos resulted in loss-of-function mutant phenotypes for 43 genes, which i

236 or neurons - where it recapitulates expected loss-of-function mutant phenotypes.

237 GATA17, and GATA17L (GATA17-LIKE), based on loss-of-function mutant phenotypes.

238 tor mutant, myb57-2, closely phenocopied the loss-of-function mutant pin6-2.

239 an phenotypes reported in the literature for loss-of-function mutant plants.

240 In unc-6/netrin and unc-5 loss-of-function mutants, presynaptic components misloca

241 In the loss-of-function mutant pro(DeltaGRAS), all examined GA

242 es as either a recessive gain-of-function or loss-of-function mutant protein, depending on signaling

243 NA (mtDNA) in Arabidopsis thaliana Gain- and loss-of-function mutants provided evidence for a role of

244 and continuously on vWF-A1 surface while the loss-of-function mutant, Q232V, showed fast, saltatory m

245 Progressive loss of KANADI activity in loss-of-function mutants results in progressive transfor

246 The RCAR7 loss-of-function mutant revealed no changes in ABA respo

247 Analysis of bhlh loss-of-function mutants revealed that the single bhlh m

248 fted onto PC synthase cad1-3 atpcs2-1 double loss-of-function mutant root tissues.

249 e intestine and interneurons and that ehbp-1 loss-of-function mutants share with rab-10 mutants speci

250 Loss-of-function mutants show an elongated hypocotyl und

251 We report that both recN and recJ loss-of-function mutants show decreased DNA repair abili

252 AtSWEET2 sequesters sugars in root vacuoles; loss-of-function mutants show increased susceptibility t

253 Loss-of-function mutants show irregular, uncoordinated c

254 The recX loss-of-function mutant showed decreases in pilus phase

255 plants with defective CIA components, grxs17 loss-of-function mutants showed some degree of hypersens

256 scopy of isolated lipid droplets from cgi-58 loss-of-function mutants showed they contain triacylglyc

257 istent enhanced collagen binding whereas the loss-of-function mutants showed variable degrees of func

258 With the help of a mitochondrial SOD2 loss-of-function mutant, Sod2(n283), we measured the sen

259 Similar to unc-43, tir-1 and nsy-1 loss-of-function mutants, specific disruption of microtu

260 In sprouty loss-of-function mutants, splitting of gene expression d

261 on of a phoPR promoter-lacZ fusion in a scoC loss-of-function mutant strain grown in low-phosphate de

262 ant for Pan1p inhibition, and a pan1 partial loss-of-function mutant suppressed the temperature sensi

263 AURKC p.L49Wfs22 is a loss-of-function mutant that perturbs localization of th

264 netic screen in the Drosophila to search for loss-of-function mutants that are sensitive to low O2.

265 transferred into etr1-6;etr2-3;ein4-4 triple loss-of-function mutants that have constitutive growth i

266 In unc-89 loss-of-function mutants that lack the SH3 domain, param

267 We identified 18 presumed loss-of-function mutants that reduced the growth of the

268 Moreover, in such loss-of-function mutants the levels of acetylated H4K5,

269 We used the soc1ful loss of function mutant - the woodiest genotype known in

270 In syd-2 loss of function mutants, the normal polarized localizat

271 In rsp1 loss-of-function mutants, the eMTOC persists and organiz

272 alt2-1 and atr loss-of-function mutants, the latter of which affects th

273 In SAC gain- and loss-of-function mutants, the levels of PtdIns monophosp

274 In loss-of-function mutants, the zygote does not elongate p

275 Furthermore, we show that in cftr loss-of-function mutants, there is a deficiency of larva

276 ction of secondary roots are impaired in the loss-of-function mutant, thus identifying AtANN1 as a ke

277 We have used loss-of-function mutants to confirm a role for mes-3, -4

278 Here, we use loss-of-function mutants to show that RNA polymerase II

279 Two loss-of-function mutant transporters (Y335A and D79G) th

280 BRG1 loss-of-function mutant tumours respond to EZH2 inhibiti

281 ues the late-flowering phenotype of the gi-2 loss-of-function mutant under both short-day and long-da

282 erences in germination between etr1 and etr2 loss-of-function mutants under salt stress could not be

283 By analyzing an rdr2 loss-of-function mutant using two different parallel seq

284 A newly isolated ACD6 loss-of-function mutant was less responsive to BTH and u

285 Consistent with these results, a Col-0 rfo1 loss-of-function mutant was more susceptible to f. matth

286 An atfs-1 loss-of-function mutant was partially resistant to the e

287 The enhanced resistance in the loss-of-function mutants was associated with increased i

288 The phenotype displayed by etr1 loss-of-function mutants was rescued by treatment with a

289 Using gain- and loss-of-function mutants, we demonstrate that acetylatio

290 with green fluorescent protein in different loss-of-function mutants, we demonstrate that dynactin a

291 Through an extensive analysis of loss-of-function mutants, we demonstrate that the Arabid

292 ting them in the respective four Arabidopsis loss-of-function mutants, we experimentally proved that

293 Using RNA interference (RNAi) to generate loss-of-function mutants, we show dramatic defects in ce

294 pressed AtLTPI-4 Crown galls of the atltpI-4 loss-of-function mutant were much smaller compared with

295 cumulation in wild-type roots, whereas Atao1 loss-of-function mutants were unresponsive to the hormon

296 psis plants expressing HaRxL44 and in med19a loss-of-function mutants, whereas SATI is elevated in pl

297 s found in the arr1-3 arr10-5 arr12-1 triple loss-of-function mutant, which showed almost complete in

298 In contrast to the loss-of-function mutant with large embryo and small endo

299 The isolation of loss-of-function mutants with enhanced disease susceptib

300 this function was only apparent when double loss-of-function mutants with pp2ca-1/ahg3 were generate