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1 were traded against a potential risk (i.e., loudness).
2 olute threshold corresponds to a fixed small loudness.
3 reshold corresponds to a fixed small partial loudness.
4 hanges, suggesting a perceptual dimension of loudness.
5 , negatively correlated with improvements in loudness.
6 had substantial beneficial effects on vocal loudness.
7 ong thought to be the primary determinate of loudness.
9 ral function can be associated with abnormal loudness adaptation and the magnitude of the adaptation
10 sorder had abnormally increased magnitude of loudness adaptation to both low (250 Hz) and high (8000
11 tested when afebrile for (i) psychophysical loudness adaptation to comfortably-loud sustained tones;
12 cts with auditory nerve disorders had normal loudness adaptation to low frequency tones; all but one
13 ort-term modifications in perceived tinnitus loudness after acoustic stimulation (residual inhibition
15 though the relationship between sound source loudness and power is well known when source distance is
16 tinnitus characteristics such as subjective loudness and the percent of time during which the tinnit
18 able to reliably report perceived intensity (loudness), and discriminate fine intensity differences,
19 sound frequency (pitch) and sound intensity (loudness), and thus suggest a resolution to a long-stand
20 ese modifications led to a finite calculated loudness at absolute threshold, which made it possible t
27 mations of the underlying neural signal from loudness data contradict estimations based on intensity
32 g targets by pitch (Experiments 1A and 2) or loudness (Experiment 1B) while ignoring previously prese
34 rallel, versions of the model for predicting loudness for hearing-impaired ears have been developed a
35 beyed a compressive function paralleling the loudness function up to sound pressure levels of at leas
36 was to test this prediction by examining the loudness functions in tinnitus ears (n = 124) compared w
39 te return (i.e., not considering the risk of loudness), however, DSL m[i/o] prescribed more outright
41 view is that sound intensity (subjectively, loudness) is encoded in spike rates, whereas sound frequ
43 el, featuring central adaptation to the mean loudness level and operating on the detection of maximum
47 e explanations include (a) the idea that the loudness of sound depends on its frequency, (b) the freq
48 For example, humans naturally regulate the loudness of speech in accord with a visual estimate of r
50 strate that males can dynamically adjust the loudness of their songs according to the distance to a f
51 ting in sympathetic vibrations that increase loudness, or at different frequencies, resulting in audi
52 n PC circuits in word recognition (P =.002), loudness (P =.003), overall liking (P =.001), aversivene
54 perating on the detection of maximum central-loudness rate of change, can account for the paradoxical
56 isentangle hypersensitivity (hyperacusis) to loudness recruitment, tinnitus and non-tinnitus ears wer
58 an be characterised by monotony of pitch and loudness, reduced stress, variable rate, imprecise conso
60 seems to depend more on the overall specific loudness than on the peripheral masking properties of th
61 ibration, nerve-fiber activity, or perceived loudness, the ear is most sensitive to small signals and
62 also modified to give predictions of partial loudness-the loudness of one sound in the presence of an
63 The nature of the neural codes for pitch and loudness, two basic auditory attributes, has been a key
66 auditory cortex is correlated with tinnitus loudness, we assessed resting-state source-localized EEG
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