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1 discovered more than 340 yr ago in the human louse.
2  egg production and reproduction in the book louse.
3 be greatest in the cooler temperature of the louse.
4 poE is significantly upregulated at the body louse (28 degrees C) versus the human host (37 degrees C
5 ere, we present genome sequences of the body louse and its primary bacterial endosymbiont Candidatus
6  Relapsing fever (RF) is caused by tick- and louse-borne Borrelia spp., is characterized by recurrent
7 ission, it is possible that we are missing a louse-borne branch of the alphaviruses.
8 tralis, R. rickettsii, and R. akari with the louse-borne R. prowazekii.
9 odies will allow serological confirmation of louse-borne relapsing fever and determination of disease
10  The hallmark of disease caused by tick- and louse-borne relapsing fever due to Borrelia infection is
11                                        Human louse-borne relapsing fever occurs in sporadic outbreaks
12 serum samples obtained from 42 patients with louse-borne relapsing fever were tested with an indirect
13 ponent of Borrelia recurrentis, the agent of louse-borne relapsing fever.
14  commissions identified that the disease was louse-borne.
15 erium, Tenacibaculum, Pseudomonas) with high louse burdens.
16 enate, an essential vitamin deficient in the louse diet.
17                 The genome of the obligatory louse endosymbiont Candidatus Riesia pediculicola encode
18  (hbpC and hbpB) predominates in a simulated louse environment (high heme), and subgroup II (hbpA, hb
19 tana transcriptional program within the body louse environment.
20 ize matters, we measured three components of louse fitness: attachment, feeding, and escape from host
21 n the intention-to-treat population who were louse-free 1 day after treatment (day 2) and remained so
22 than patients receiving vehicle control were louse-free on day 2 (94.9% vs. 31.3%), day 8 (85.2% vs.
23 Compared with other insect genomes, the body louse genome contains significantly fewer genes associat
24                               Thus, the body louse genome project offers unique information and tools
25 ents of the Pediculus humanus corporis (body louse) gut and the human vasculature.
26                                     The body louse has the smallest known insect genome, spanning 108
27                                         Head-louse infestation remains a public health problem.
28 matic review first identified trials on head-louse infestation; 49 were selected and their methodolog
29 omplicates the public health problem of head-louse infestations and drives the need for continuing de
30 ive than vehicle control in eliminating head-louse infestations at 1, 7, and 14 days after treatment.
31 e efficient than placebo at eliminating body louse infestations by day 14; however, this difference w
32                               The human body louse, its primary endosymbiont, and the bacterial patho
33 n hbpC transcript levels in response to the "louse-like" temperature of 30 degrees C.
34 Furthermore, when this strain is grown at a "louse-like" temperature, an inversion of the transcript
35 nscription of irr (a >5-fold decrease) at a "louse-like" temperature, suggesting that Irr may functio
36 rcular mitochondrial chromosomes of the body louse may be linked to the loss of the gene encoding the
37 louse Pediculus humanus capitis and the body louse P. h. humanus.
38 s gambiae and Culex quinquefasciatus, a body louse Pediculus humanus and a tick species Ixodes scapul
39 ction of the mycetomic bacterium of the head louse Pediculus humanus capitis and the body louse P. h.
40 s an obligatory parasite of humans, the body louse (Pediculus humanus humanus) is an important vector
41 uefasciatus), tick (Ixodes scapularis), body louse (Pediculus humanus), kissing bug (Rhodnius prolixu
42 ganisms, and another compared the human body louse, Pediculus humanus humanus, against itself and sel
43 mimetabolous insects, the genome of the body louse thus provides a reference for studies of holometab
44 d optimal designs to standardize future head-louse treatment trials, thereby obtaining valid conclusi
45 o stressors that are encountered in the body louse vector environment, a decreased temperature and an
46  has adapted to both the human host and body louse vector niches, producing persistent infection with
47 ted human bloodstream to the hemin-rich body louse vector.
48 e transition from the human host to the body louse vector.
49  the pathogen in the human host and the body louse vector; e.g., we observed a dramatic (>100-fold) i

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