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1 , and tricyclic antidepressants (for chronic low back pain).
2 r acute or chronic nonradicular or radicular low back pain.
3 int degeneration is a major cause of chronic low back pain.
4 uideline addressed pharmacologic options for low back pain.
5 effective treatment for chronic or recurrent low back pain.
6 ted the effectiveness of massage for chronic low back pain.
7 e lumbar facet joints has been implicated in low back pain.
8 osis, acute myocardial infarction, and acute low back pain.
9 ises following acute, experimentally induced low back pain.
10 Mr S, a 50-year-old man, has long-standing low back pain.
11 ach to using available treatment options for low back pain.
12 s associated with modest effects for chronic low back pain.
13 gic therapies are available for treatment of low back pain.
14 for chronic or subacute (>4 weeks' duration) low back pain.
15 l restoration are also effective for chronic low back pain.
16 fective primary care management strategy for low back pain.
17 e, examining the direct health care costs of low back pain.
18 e the most frequently prescribed therapy for low back pain.
19 en among older individuals with arthritis or low back pain.
20 to recovery in populations of patients with low back pain.
21 ollowing diskectomy for persistent radicular low back pain.
22 arch shows widespread use of acupuncture for low back pain.
23 Acupuncture effectively relieves chronic low back pain.
24 tients consulting primary care with subacute low back pain.
25 MRI changes are the strongest predictor of low back pain.
26 to improve decision making for patients with low back pain.
27 eplacement for radiographs for patients with low back pain.
28 ical outcomes for primary care patients with low back pain.
29 ing worse physical health than patients with low back pain.
30 xercise programs in the treatment of chronic low back pain.
31 bark and magnets, have marginal benefit for low back pain.
32 h reduced incidence of back injury claims or low back pain.
33 udes adults with acute, subacute, or chronic low back pain.
34 on our small group of subjects with chronic low back pain.
35 role in the development and perpetuation of low back pain.
36 ination for the effective treatment of acute low back pain.
37 r clinical results in patients with subacute low back pain.
38 ness and harms of SMT for acute (</=6 weeks) low back pain.
39 d spinal manipulation for some patients with low back pain.
40 damaged tissues in patients with nonspecific low back pain.
41 ve effectiveness and costs of treatments for low back pain.
42 ertebral disc in the pathogenesis of chronic low back pain.
43 ertebral disc in the pathogenesis of chronic low back pain.
44 follow published guidelines for treatment of low back pain.
45 g modalities in the evaluation of persistent low back pain.
46 ement for plain radiography in patients with low back pain.
47 hysicians concerning appropriate therapy for low back pain.
48 the index visit and no previous episodes of low back pain.
49 rstand the role of MBSR in the management of low back pain.
50 recommendations on noninvasive treatment of low back pain.
51 acologic and nonpharmacologic treatments for low back pain.
52 tebral disc degeneration (IVDD) is linked to low back pain.
53 efficacy and safety of MBSR in patients with low back pain.
54 bility as a primary outcome in patients with low back pain.
55 essed nonpharmacologic treatment options for low back pain.
56 d exercise program for patients with chronic low back pain.
57 or young and middle-aged adults with chronic low back pain.
58 e treatment option for patients with chronic low back pain.
59 f glial activation, in patients with chronic low back pain.
60 ation of muscle fat content in patients with low back pain.
61 ion and to ameliorate IVD-associated chronic low back pain.
62 r acute or chronic nonradicular or radicular low back pain.
63 d degeneration are believed to contribute to low back pain.
64 sed the relationships of BMI and height with low back pain.
65 cceptability of acupuncture to patients with low back pain.
66 ity, and they can be responsible for chronic low back pain.
67 tool as they considered prognosis studies of low back pain.
68 spinal injections of methylprednisolone for low back pain.
69 clinical care without immediate imaging for low-back pain.
70 transitional vertebra in young patients with low-back pain.
71 iations between allergies and depression and low-back pain.
72 nondrug treatments for patients with chronic low back pain?
75 prevalence of back and/or neck pain was 31% (low back pain: 34 million, neck pain: 9 million, both ba
76 2013, and October 24, 2014, and had chronic low back pain, a positive diagnostic block at the facet
77 egenerative disc disease often causes severe low-back pain, a public health problem with huge economi
80 am (SMP) on primary care patients with acute low back pain (ALBP) from low income, inner city neighbo
82 that acupuncture may be useful for headache, low back pain, alcohol dependence, and paralysis resulti
83 verity were divided according to symptoms of low back pain alone and symptoms of low back pain with o
84 pain and disability in patients with chronic low back pain, although this difference became nonsignif
85 s a major pathological process implicated in low back pain and is a prerequisite to disk herniation.
86 lly significant difference in improvement in low back pain and lower extremity pain between groups.
87 with betamethasone and triamcinolone reduced low back pain and lower extremity pain, although there w
90 anxiety, musculoskeletal disorders including low back pain and neck pain, diabetes, and cirrhosis--in
91 A 20-year-old man presented with 1 week of low back pain and progressive lower extremity weakness.
92 ic images, intracranial pressure processing, low back pain and real-time tumour tracking; (3) outcome
93 in intensity and disability in patients with low back pain and sciatica after lumbar disc herniation.
94 nce (MR) imaging in patients with persistent low back pain and sciatica effectively demonstrates spin
95 ans should evaluate patients with persistent low back pain and signs or symptoms of radiculopathy or
96 on (SOE, moderate) are effective for chronic low back pain and strengthens previous findings regardin
97 the most important factor leading to chronic low back pain and subsequent disability after discectomy
98 s important to remember about rare causes of low back pain and to perform detailed physical examinati
101 e-sacral articulation of young patients with low-back pain and a lumbosacral transitional vertebra.
102 e; age range, 6-19 y; mean age, 15.7 y) with low-back pain and a lumbosacral transitional vertebra.
104 maging on clinical outcomes in patients with low-back pain and no indication of serious underlying co
105 r imaging in patients with acute or subacute low-back pain and without features suggesting a serious
106 ed by local DRG inflammation (a rat model of low back pain) and by a peripheral paw inflammation mode
107 h as imaging for patients with uncomplicated low back pain) and using the results for public reportin
108 nophen, skeletal muscle relaxants (for acute low back pain), and tricyclic antidepressants (for chron
109 nophen, skeletal muscle relaxants (for acute low back pain), and tricyclic antidepressants (for chron
111 served, duloxetine was effective for chronic low back pain, and benzodiazepines were ineffective for
112 that acetaminophen is ineffective for acute low back pain, and duloxetine is associated with modest
113 for improving function and reducing chronic low back pain, and the benefits persisted for at least s
115 harmacologic therapies for primarily chronic low back pain are associated with small to moderate, usu
116 of moderate efficacy for chronic or subacute low back pain are cognitive-behavioral therapy, exercise
117 ood evidence of short-term effectiveness for low back pain are NSAIDs, acetaminophen, skeletal muscle
119 s for plain radiographs in the evaluation of low back pain are too sensitive and expose patients unne
122 ent state of managing chronic (and subacute) low back pain as reflected in recently published guideli
123 ack disorders, with a particular emphasis on low back pain, as this area has been most represented in
126 d effect on troublesome subacute and chronic low-back pain at a low cost to the health-care provider.
127 in into 1 of 3 broad categories: nonspecific low back pain, back pain potentially associated with rad
128 sis and assessment of disease progression in low back pain, brain tumours and primary epilepsy; (2) e
131 and height are linked to the pathogenesis of low back pain, but evidence-based confirmation is lackin
132 ion is a commonly used treatment for chronic low back pain, but high-quality evidence for its effecti
133 iscs has been linked with the development of low back pain, but little is known about factors affecti
135 ga is effective for mild to moderate chronic low back pain (cLBP), but its comparative effectiveness
137 Recommendation 2: For patients with chronic low back pain, clinicians and patients should initially
139 clinically important improvement in chronic low back pain compared with a standardized exercise prog
142 in health centres respiratory insufficiency, low back pain, degree of physical function, presence of
143 arious musculoskeletal conditions, including low back pain, despite little scientific support for the
144 f different imaging methods, and duration of low-back pain did not affect the results, but analyses w
147 342 adults aged 20 to 70 years with chronic low back pain enrolled between September 2012 and April
149 s approximately 1.5), and a history of prior low back pain episodes and demographic variables were no
150 lated for prediction of persistent disabling low back pain for findings attainable during the clinica
151 thritis (OA), rheumatoid arthritis (RA), and low back pain from 12 community pharmacy sites responded
153 udies of patients with fewer than 8 weeks of low back pain from which likelihood ratios (LRs) were ca
156 en that most patients with acute or subacute low back pain improve over time regardless of treatment,
157 aluated included reduction or elimination of low back pain, improvement in back-specific and overall
159 al cost of diagnostic imaging for persistent low back pain in 1990 relative to 1987 was estimated at
160 al cost of diagnostic imaging for persistent low back pain in 1990 relative to 1987 was estimated.
165 Higher BMI was significantly associated with low back pain in males (for overweight, odds ratio = 1.0
170 carpal tunnel syndrome, 59 million have had low back pain in the past 3 months, and 30.1 million hav
171 ith DZ twins, equating to a heritability for low back pain in the range of 52-68% and for neck pain i
174 Subjects responded to questions regarding low-back pain in the past 12 months and history of asthm
176 spinal manipulation; for chronic or subacute low back pain, intensive interdisciplinary rehabilitatio
177 ical examination to help place patients with low back pain into 1 of 3 broad categories: nonspecific
190 niated disc (HD), one of the major causes of low back pain, is often resolved spontaneously without s
191 the afternoon in 47 subjects without current low back pain (IVDs = 230; age range, 20-71 years) after
192 llitus, and chronic pain conditions (chronic low back pain, knee osteoarthritis, and fibromyalgia).
202 program to adults with chronic or recurrent low back pain led to greater improvements in back functi
205 with the largest number of YLDs in 2010 were low back pain, major depressive disorder, other musculos
207 nal treatments for participants with chronic low back pain (Mint study) were conducted in 16 multidis
209 that acetaminophen was ineffective for acute low back pain, nonsteroidal anti-inflammatory drugs had
210 ry of any allergy were more likely to report low-back pain (odds ratio = 1.51; 95% confidence interva
211 ore likely to have both major depression and low-back pain (odds ratio = 3.03; 95% confidence interva
214 ostic imaging is indicated for patients with low back pain only if they have severe progressive neuro
216 es, women were 1.5 times more likely to have low back pain or symptoms of intervertebral disc herniat
217 ity were half as likely to have work-related low-back pain (OR=0.50, 95% CI 0.26-0.96) and nurses rep
218 heumatoid arthritis, osteoarthritis, chronic low back pain, or ischemic heart disease since 1995.
219 clinical trials of participants with chronic low back pain originating in the facet joints, sacroilia
221 asone recipients demonstrated improvement in low back pain (P = .04, Fisher exact test), whereas 55%
222 betamethasone recipients had improvement in low back pain (P = .26), whereas 49% of triamcinolone re
223 betamethasone recipients had improvement in low back pain (P = .38), whereas 52% of triamcinolone re
226 ated to a person's health status) indicating low back pain severity were divided according to symptom
229 logic therapy with proven benefits-for acute low back pain, spinal manipulation; for chronic or subac
231 iagnostic tests in patients with nonspecific low back pain (strong recommendation, moderate-quality e
232 fferent rheumatic conditions (eg, neck pain, low back pain, systemic lupus erythematosus, fibromyalgi
234 atory drugs had smaller benefits for chronic low back pain than previously observed, duloxetine was e
235 endorse triage to identify the rare cases of low back pain that are caused by medically serious patho
236 ive inception cohorts of patients with acute low back pain that measured fear of pain (often describe
238 e preceding medications for acute or chronic low back pain that reported pain outcomes, back-specific
239 The proportion of visits to specialists for low back pain that were new consultations increased from
242 studies varied from a low of 31% (n = 8) for low back pain to a high of 68% (n = 23) for fibromyalgia
244 e authors randomly selected 62 patients with low back pain to undergo either rapid MR imaging or plai
245 idemiologic studies report the prevalence of low back pain to vary from 7.6% to 37% in different popu
246 ic reviews and RCTs, for RCTs of adults with low back pain treated in ambulatory settings with SMT co
248 rm trials showing modest effects for chronic low back pain; trials were not designed to assess seriou
249 ard deviation]; age range, 20-79 years) with low back pain underwent standard 1.5-T MR imaging, which
250 include intense vulvar and vaginal itching, low back pain, uterine cramps, fetal distress, and prete
253 raphy by a sports medicine clinic because of low back pain were evaluated for the presence of asympto
254 mponents for predicting persistent disabling low back pain were maladaptive pain coping behaviors, no
255 adults with troublesome subacute or chronic low-back pain were recruited from 56 general practices a
256 nostic imaging and testing for patients with low back pain when severe or progressive neurologic defi
257 atment selection in patients with persistent low back pain who are suspected of having herniated nucl
258 Recommendation 3: In patients with chronic low back pain who have had an inadequate response to non
259 y with SPECT can help identify patients with low back pain who would benefit from facet joint injecti
260 ty-seven patients (23 men and 24 women) with low back pain, who were scheduled for facet joint inject
261 ptoms of low back pain alone and symptoms of low back pain with objective corroborating findings.
263 patients with evidence-based information on low back pain with regard to their expected course, advi
264 s is known about the epidemiology of chronic low-back pain with no associated work disability or comp
265 the preceding therapies for acute or chronic low back pain (with or without leg pain) that reported p
267 ive-month period because of an acute, severe low back pain, with sphincter dysfunction, partially res
268 eporting (OP-8) reduced MR imaging rates for low back pain without conservative therapy in either Med
269 ion of MR imaging examinations performed for low back pain without history of conservative therapy.
271 cians and American Pain Society guideline on low back pain, would provide better care to patients, im
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