ライフサイエンスコーパス: low cardiac output

1 assist in the detection of low-flow states (low cardiac output).

2 gement of patients with ADHF presenting with low cardiac output.

3 wer limb SCDs because they produce a falsely low cardiac output.

4 iliary circulatory support in the setting of low cardiac output.

5 severe arterial unsaturation, and usually a low cardiac output.

6 ated with underfilling of the left heart and low cardiac output.

7 ospital mortality attributable to persisting low-cardiac output.

8 idence of perioperative infarction was 3.3%, low cardiac output 2.7%, stroke 2.2%, reoperation for bl

9 after tetralogy of Fallot repair results in low cardiac output and a prolonged stay in the intensive

10 erosclerosis, and the combined end points of low cardiac output and atrial fibrillation.

11 of sepsis and was only found in models with low cardiac output and decreased renal blood flow (p < 0

12 recognized that these patients present with low cardiac output and high peripheral resistance and th

13 use severe LA and LV enlargement, leading to low cardiac output and hydrops.

14 of age, P=0.06), with a parallel decline in low cardiac output and length of hospital stay.

15 Fluid overload, renal dysfunction, low cardiac output and neurological dysfunction remain m

16 Two patients developed low cardiac output, and 3 patients had pleural effusions

17 both ventricles accompanied by hypotension, low cardiac output, and high filling pressures occurring

18 erative support techniques and postoperative low cardiac output are associated with cerebral hypoperf

19 ion of the right ventricle (RV); hydrops and low cardiac output are often associated.

20 ocardiography is a useful tool for assessing low cardiac output AS.

21 During states of low cardiac output, cerebral blood flow and thus cerebra

22 on of mitral regurgitation (MR) results in a low cardiac output (CO) state because of an acute increa

23 In addition to "abnormal flow" through low cardiac output, dilated cardiac chambers and poor co

24 0.55), as well as persistent hypotension and low cardiac output (in 83 percent of the patients, vs. 1

25 rculation, which can be impaired either by a low cardiac output or arterial vasodilation, is an impor

26 ent, reflected the presence of an associated low cardiac output or low renal blood flow syndrome.

27 CI 1.1 to 1.7], p = 0.02), and postoperative low cardiac output (OR 3.0 [95% CI 1.7 to 5.2], p = 0.00

28 ed by elevated cardiac filling pressures and low cardiac output, or b) ongoing signs of hypoperfusion

29 acity in people with severe TR is related to low cardiac output reserve relative to metabolic needs,

30 common use of inotropes and vasodilators for low cardiac output septic shock associated with elevated

31 a significant reduction in the incidence of low cardiac output state (odds ratio, 0.22; 95% confiden

32 a significant reduction in the incidence of low cardiac output state and the need for inotropic supp

33 A low cardiac output state can complicate the postoperativ

34 ally challenging scenarios including AS with low cardiac output state or other structural heart disea

35 etry may be more accurate in patients with a low cardiac output state.

36 tant therapeutic option in children with the low cardiac output state.

37 l in origin, including small aortic calibre, low cardiac output states, high vasopressor requirements

38 of infection (12.9% vs 29.7%; p = 0.002) and low cardiac output syndrome (6.5% vs 26.6%; p = 0.002).

39 Low cardiac output syndrome (LCOS), affecting up to 25%

40 Low cardiac output syndrome after cardiac surgery is ass

41 own in small studies to prevent or treat the low cardiac output syndrome after cardiac surgery.

42 ped relationship was observed for stroke and low cardiac output syndrome but not for renal replacemen

43 entification and aggressive treatment of the low cardiac output syndrome peculiar to these patients.

44 Composite end point reflecting low cardiac output syndrome with need for a catecholamin

45 rge as a composite of myocardial infarction, low cardiac output syndrome, infection, stroke, or in-ho

46 Complications such as low cardiac output syndrome, prolonged ventilation and r

47 rative levosimendan to prevent postoperative low cardiac output syndrome.

48 atients after cardiac surgery because of the low cardiac output syndrome.

49 The primary outcome was the incidence of low cardiac output syndrome.

50 ive stroke rate unless it was accompanied by low cardiac output syndrome.

51 eplacement remained high, largely because of low cardiac output syndrome.

52 y of intervention, conversion to sternotomy, low cardiac output that required mechanical support, aor

53 tion died 7 weeks after surgery secondary to low cardiac output; the other 3 had resolution of effusi

54 y may potentially lead to an inappropriately low cardiac output, with a subsequent compromise of micr