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1 ed with nuts, or a control diet (advice on a low-fat diet).
2 t compared with normal littermates even on a low fat diet.
3 Clps-/- mice had normal intake on a low fat diet.
4 rats upon feeding them a high carbohydrate, low fat diet.
5 eficient in linoleate in all subjects on the low fat diet.
6 und, become obese on a high fat but not on a low fat diet.
7 red to striatal slices taken from rats fed a low fat diet.
8 in the two soybean oils, coconut oil, and a low-fat diet.
9 A- or MUFA-enriched high-fat diets (HFDs) or low-fat diet.
10 n Bf(-/-)/Ldlr(-/-) and Ldlr(-/-) mice fed a low-fat diet.
11 celerates AT accumulation even in mice fed a low-fat diet.
12 patitis (NASH) and HCC on a high-fat but not low-fat diet.
13 eflects a positive response to statins and a low-fat diet.
14 e lacking MGAT2 have a normal phenotype on a low-fat diet.
15 reas no effects were seen with a traditional low-fat diet.
16 high-fat diet and those consuming a standard low-fat diet.
17 olesterol concentration improved more on the low-fat diet.
18 intake, as an alternative to a conventional low-fat diet.
19 tal and LDL cholesterol-lowering effect of a low-fat diet.
20 d fat, or for 1 week on a single high-fat or low-fat diet.
21 ificantly (1.53 kg; P < 0.001) only with the low-fat diet.
22 erol and higher HDL cholesterol) than does a low-fat diet.
23 with the low-carbohydrate diet than with the low-fat diet.
24 ces insulin responsiveness on a high-starch, low-fat diet.
25 t reduced risk of colon cancer if they eat a low-fat diet.
26 terol concentrations in subjects consuming a low-fat diet.
27 and significantly overestimated for the very-low-fat diet.
28 rcentage) in the lower-fat diet and the very-low-fat diet.
29 sceptibility to induction of this trait by a low-fat diet.
30 igh-fat diet converted to phenotype B with a low-fat diet.
31 a high-fat diet compared with controls fed a low-fat diet.
32 ot alter any of the parameters in mice fed a low-fat diet.
33 o 11.0 mg/dL}]; P < 0.001) than those on the low-fat diet.
34 ardiovascular risk factor reduction than the low-fat diet.
35 ss was observed with any low-carbohydrate or low-fat diet.
36 n diet (WD) fatty acid profile or a standard low-fat diet.
37 lower after the high-fat diet than after the low-fat diet.
38 nflammation on a high-fat diet compared to a low-fat diet.
39 responses of different MNS blood types to a low-fat diet.
40 duals after the HSF-DHA diet relative to the low-fat diet.
41 ficant larger decreases for TT carriers on a low-fat diet.
42 ns in plasma glucose and insulin only on the low-fat diet.
43 experienced adverse glycemic effects of the low-fat diet.
44 n omega-3-acid ethyl esters in patients on a low-fat diet.
45 genotype by choosing a high-carbohydrate and low-fat diet.
46 cells is evident in eNos(-/-) mice even on a low-fat diet.
47 recent reports questioned the superiority of low-fat diets.
48 n some people who exercise regularly and eat low-fat diets.
49 alance methods in animals fed both high- and low-fat diets.
50 HDL cholesterol decreased during both low-fat diets.
51 L subclass phenotype A during both high- and low-fat diets.
52 decrease in HDL cholesterol associated with low-fat diets.
53 livers of ethanol-treated rats fed high- or low-fat diets.
54 rate) diet or a calorie- and fat-restricted (low-fat) diet.
55 or gene main effects and interactions with a low-fat diet (20% from energy) compared with a high-fat
56 0-46% fat) show less lipoprotein benefits of low-fat diets (20-24% fat) than do men with a high-risk
57 that differed in macronutrient composition (low-fat diet: 20-25% fat, 15% protein, and 60-65% carboh
59 ts and lifestyle changes, a more traditional low-fat diet (25% fat kcals) with flaxseed supplementati
60 d 50% of energy from carbohydrates) and 2) a low-fat diet (25% of energy from fat and 62% of energy f
61 in diet, participants consumed an isocaloric low-fat diet (60% of energy from carbohydrate, 20% from
62 5.33 to 9.25 kg] at 12-month follow-up) and low-fat diets (7.99 kg [95% CI, 6.01 to 9.92 kg] at 6-mo
63 ilon study) in which they were assigned to a low-fat diet, a high-fat high-SFA (HSF) diet, and the HS
68 hese disorders includes a high-carbohydrate, low-fat diet and avoidance of those events that promote
70 perfusion from 10 to 26 IU/g/h in rats fed a low-fat diet and from 17 to 34 IU/g/h in rats fed a high
73 in the intestine may sensitize the gut to a low-fat diet and to potential accompanying food-borne to
77 feeding a fat-free diet, which suggests that low-fat diets are likely to be beneficial in lipodystrop
80 tes may be more effective than a high-GI and low-fat diet at reducing body weight and controlling glu
81 bohydrate diets are at least as effective as low-fat diets at reducing weight and improving metabolic
82 ental LDL-subclass patterns when consuming a low-fat diet: AxA mating (9 families with 19 children),
83 esterol-lowering diet may be preferable to a low-fat diet because of more favorable effects on the CV
84 glyceride levels significantly in rats fed a low-fat diet, but increased values about 2.4-fold in rat
88 ectedly, a high-fat, Western-type (but not a low-fat) diet caused death in two-thirds of the beta3-/-
89 previously fasted animals high-carbohydrate, low-fat diets causes a dramatic induction of enzymes-suc
90 ancer rate was 0.42 in those assigned to the low-fat diet compared with 0.46 in the control group (HR
91 insulin resistance, with particular focus on low-fat diets compared with the currently popular low-ca
92 findings, we propose that, especially under low-fat diet conditions, adipose tissue-resident iNKT ce
93 response to feeding of a high carbohydrate, low fat diet, conditions that favor enhanced lipogenesis
94 We showed that iNKT cell-deficient mice on a low-fat diet, considered a normal diet for mice, display
96 g: 1) a normal low fat (13% kcal) diet, 2) a low fat diet containing n-3 PUFAs, 3) a high fat (41% kc
97 pharmacological inhibition of ACAT2 were fed low fat diets containing various amounts of cholesterol
101 y of evidence from RCTs to determine whether low-fat diets contribute to greater weight loss than par
102 Participants were randomly assigned to a low-fat diet control group or TMD intervention groups [t
105 mpared to aged-matched control animals fed a low-fat diet, correlating with enhanced alloreactive T c
106 t a high sucrose content in a hypoenergetic, low-fat diet did not adversely affect weight loss, metab
107 le fractured knee joints of mice receiving a low-fat diet did not demonstrate significant differences
108 DIO mice underwent switch to ad libitum low-fat diet (DIO-switch) or caloric restriction (CR) fo
109 Importantly, reversing the maternal HFD to a low-fat diet during a subsequent pregnancy improved feta
110 topics in the field, obesity and the use of low-fat diets early in life, have been the subject of re
111 approximately 1 h/d), eating a low-calorie, low-fat diet, eating breakfast regularly, self-monitorin
113 B, type I) and apolipoprotein E genes fed a low-fat diet exhibit a constellation of pathologies shar
114 lated from AFABP-KO and WT mice fed high- or low-fat diets exhibited similar rates of basal and norep
115 ith the low-glycemic load diet than with the low-fat diet, expressed in absolute terms (mean [SE], 96
116 In sedentary nondiabetic persons following a low-fat diet, FABP2 Thr54 carriers have lower glucose to
117 mpaired glucose and insulin tolerance in LF (low fat diet)-fed control (AhR(fl/fl)) mice but not in a
121 caused by HFD were rescued by switching to a low fat diet for one month, suggesting a functional role
122 nd non-obese twin pairs consumed recommended low fat diets for 6 weeks before they received a 6-week
124 effects of 10.2 g psyllium/d adjunctive to a low-fat diet for >/=8 wk in individuals with mild-to-mod
128 Psyllium offers a potential adjunct to a low-fat diet for the treatment of hypercholesterolemia i
132 eviously reported that during refeeding on a low-fat diet, glucose tolerance is normal but insulin-de
133 be randomised into either a Mediterranean or low fat diet group for a 3 month intervention period.
134 the low-carbohydrate diet group than in the low-fat diet group (mean change, -12.9% vs. -6.7%; P < 0
135 s in insulin and HOMA-IR was observed in the low-fat diet group (P=0.02 and P=0.04, respectively).
136 of the low-carbohydrate diet group than the low-fat diet group completed the study (76% vs. 57%; P =
141 baseline (n = 3,375), those assigned to the low-fat diet had a breast cancer rate of 0.27 compared w
144 vious studies comparing low-carbohydrate and low-fat diets have not included a comprehensive behavior
147 (2)): 35.8 +/- 2.9] or a calorie-restricted, low-fat diet (High Carb; BMI: 36.7 +/- 4.6) for 6 wk.
148 ness of the Mediterranean diet to a standard low fat diet in terms of differences in insulin sensitiv
150 tolerated and safe when used adjunctive to a low-fat diet in individuals with mild-to-moderate hyperc
151 safety of psyllium when used adjunctive to a low-fat diet in men and women with hypercholesterolemia.
153 ed fat, is as efficacious as the traditional low-fat diet in producing weight loss and may be more be
154 ious than a conventional, energy-restricted, low-fat diet in reducing cardiovascular disease risk.
156 icles (subclass pattern B) can be induced by low-fat diets in healthy subjects with large LDL particl
157 the concept that both hyperinsulinemia and a low-fat diet increase DNL, and that DNL contributes to h
159 improvement in lipid profiles from long-term low-fat diet intake in the APOA5 rs964184 risk allele.
160 s in REE and TEE that were greatest with the low-fat diet, intermediate with the low-glycemic index d
161 from this study indicate that a high-fiber, low-fat diet intervention is associated with reduced ser
162 indings suggest that the long-term effect of low-fat diet intervention on bodyweight depends on the i
163 ning plant sterol esters incorporated into a low-fat diet is a beneficial adjunct in the dietary mana
166 domly divided into three groups: (1) control low-fat diet (LF-SED; 15% of calories from fat), (2) hig
167 7BL/6 mice were fed an HFD (60% fat kcal) or low-fat diet (LFD) (10% fat kcal) for 8 or 12 weeks.
168 ung (age 25 days) Sprague-Dawley rats with a low-fat diet (LFD) alone or with vitamin D depletion (LF
169 nd cholesterol esters (CEs) in response to a low-fat diet (LFD) and a moderate-fat diet (MFD) and ass
170 were fed with high-fat diet (HFD) or normal low-fat diet (LFD) and subjected to a protocol of ovalbu
172 iet (SFD), a high-trans-fat diet (TFD), or a low-fat diet (LFD) for 4 wk prior to mating, and remaine
173 We compared the effects of an LGD and a low-fat diet (LFD) on body composition and components of
174 from small intestine of C57BL/6J mice fed a low-fat diet (LFD) or high-fat diet (HFD) for 12 weeks.
176 elevated fat mass and decreased lean mass on low-fat diet (LFD), accompanied by leptin resistance and
177 57BL/6J mice were divided into three groups: low-fat diet (LFD), high-fat diet (HFD) and HFD suppleme
180 nd VMH FA levels, rats were trained to eat a low-fat diet (LFD; 13.5%) or an HFD in 3 h/day and were
181 diet options (low-carbohydrate diet [LCD] or low-fat diet [LFD]) before choosing and were allowed to
182 session) and sedentary rats fed either chow (low-fat diet [LFD]; normal insulin sensitivity) or a hig
183 the presence of an up-titration regiment and low-fat diet, lomitapide is generally well tolerated and
186 n diet and lipid-lowering drugs or on strict low-fat diet (<10% of calories) without lipid drugs; and
187 rcise recommendation, and group meetings, or low-fat diet (<30% energy from fat, <300 mg of cholester
188 (</=45% of energy from carbohydrates) versus low-fat diets (</=30% of energy from fat) on metabolic r
189 r in monounsaturated fat, however, eucaloric low-fat diets may increase plasma triacylglycerol concen
190 rate diet lost more weight than those on the low-fat diet (mean [+/-SD], -5.8+/-8.6 kg vs. -1.9+/-4.2
191 e, the decrease in REE was greatest with the low-fat diet (mean [95% CI], -205 [-265 to -144] kcal/d)
193 wever, in hepatocytes from animals given the low-fat diet, most of the differences between the variou
194 /genotype) as well as from rats fed high- or low-fat diets (n=8/treatment) were analyzed in parallel
198 e long-term effects of physical exercise and low-fat diet on the progression of coronary artery disea
199 ethanol for dietary carbohydrate in high-and low-fat diets on energy expenditure and body composition
200 tested the long-term effects of high-fat and low-fat diets on males of two inbred strains of mice and
202 when they were maintained on either a basal low fat diet or a western-type high fat/high cholesterol
203 respectively) by hepatocytes from rats fed a low-fat diet or a diet supplemented with the correspondi
204 )-transduced regeneration tissues were fed a low-fat diet or a high-fat diet and treated with vehicle
205 KO and wild-type (WT) littermates were fed a low-fat diet or a high-fat diet to investigate the effec
206 mor biology can be altered by either a vegan low-fat diet or eliminating simple carbohydrates accompa
208 nephropathy, we treated 1) C57BL/6J mice on low-fat diet or high-fat diet with FXR agonists (GW4064
209 notype were at lower risk if they consumed a low-fat diet (OR, 0.2; 95% CI, 0.1-0.5) relative to thos
210 tensity, evidence from RCTs does not support low-fat diets over other dietary interventions for long-
217 dress these issues, we designed two diets: a low-fat diet providing approximately 20% of energy as fa
218 ated with the changes from baseline with the low-fat diet (r = -0.74, P = 0.01), as well as with the
221 ch diet of maternal milk, and substituting a low-fat diet reduced UCP2, restored mitochondrial coupli
222 atic overexpression of VLDLR combined with a low-fat diet regimen induces regression of advanced plaq
223 Treatment with HD-Ad-VLDLR together with a low-fat diet regimen resulted in reduced lesion size (cr
231 h dietary support including high-protein and low-fat diets supplemented with medium-chain triglycerid
233 urve after the test meal was lower after the low-fat diet than after the high-fat diet (P < 0.04).
234 e low-carbohydrate diet vs. -4.8 kg with the low-fat diet) than fat-free mass (change, -3.3 kg vs. -2
238 n1LKO mice are similar to control mice fed a low-fat diet, they are protected against insulin resista
239 n sedentary nondiabetic subjects following a low-fat diet, Thr54 FABP2 carriers have lower glucoregul
240 used GM-CSF-deficient (Csf2(-/-)) mice fed a low fat diet to test the hypothesis that adipose tissue
244 hat country, age, sex, smoking, alcohol use, low-fat diet, waist circumference, recent weight gain (>
248 (n = 122) aged 50-75 y who were following a low-fat diet were genotyped and underwent oral-glucose-t
249 several lipid lowering treatments including low fat diet when compared with LDL pattern A individual
250 11 transporter expression in comparison with low-fat diet, whereas liver-to-feces RCT was preserved a
251 = 0.01) in participants who were assigned to low-fat diets, whereas there was no significant genotype
252 with these in vitro findings, feeding mice a low fat diet with ethanol for 4 weeks resulted in a sign
253 icacy of 2 moderate-carbohydrate diets and a low-fat diet with different GIs on weight loss and the m
255 e to placebo in subjects already consuming a low-fat diet, with no effect on serum HDL or triacylglyc
256 ry greatly in their lipoprotein responses to low-fat diets, with some of this variation being attribu
257 owever, because it lowers HDL cholesterol, a low-fat diet without substantial weight loss may not be
258 , in type 2 diabetes patients, an ad libitum low-fat diet would cause greater weight loss than would
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