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1 ed with nuts, or a control diet (advice on a low-fat diet).
2 t compared with normal littermates even on a low fat diet.
3          Clps-/- mice had normal intake on a low fat diet.
4  rats upon feeding them a high carbohydrate, low fat diet.
5 eficient in linoleate in all subjects on the low fat diet.
6 und, become obese on a high fat but not on a low fat diet.
7 red to striatal slices taken from rats fed a low fat diet.
8  in the two soybean oils, coconut oil, and a low-fat diet.
9 A- or MUFA-enriched high-fat diets (HFDs) or low-fat diet.
10 n Bf(-/-)/Ldlr(-/-) and Ldlr(-/-) mice fed a low-fat diet.
11 celerates AT accumulation even in mice fed a low-fat diet.
12 patitis (NASH) and HCC on a high-fat but not low-fat diet.
13 eflects a positive response to statins and a low-fat diet.
14 e lacking MGAT2 have a normal phenotype on a low-fat diet.
15 reas no effects were seen with a traditional low-fat diet.
16 high-fat diet and those consuming a standard low-fat diet.
17 olesterol concentration improved more on the low-fat diet.
18  intake, as an alternative to a conventional low-fat diet.
19 tal and LDL cholesterol-lowering effect of a low-fat diet.
20 d fat, or for 1 week on a single high-fat or low-fat diet.
21 ificantly (1.53 kg; P < 0.001) only with the low-fat diet.
22 erol and higher HDL cholesterol) than does a low-fat diet.
23 with the low-carbohydrate diet than with the low-fat diet.
24 ces insulin responsiveness on a high-starch, low-fat diet.
25 t reduced risk of colon cancer if they eat a low-fat diet.
26 terol concentrations in subjects consuming a low-fat diet.
27 and significantly overestimated for the very-low-fat diet.
28 rcentage) in the lower-fat diet and the very-low-fat diet.
29 sceptibility to induction of this trait by a low-fat diet.
30 igh-fat diet converted to phenotype B with a low-fat diet.
31 a high-fat diet compared with controls fed a low-fat diet.
32 ot alter any of the parameters in mice fed a low-fat diet.
33 o 11.0 mg/dL}]; P < 0.001) than those on the low-fat diet.
34 ardiovascular risk factor reduction than the low-fat diet.
35 ss was observed with any low-carbohydrate or low-fat diet.
36 n diet (WD) fatty acid profile or a standard low-fat diet.
37 lower after the high-fat diet than after the low-fat diet.
38 nflammation on a high-fat diet compared to a low-fat diet.
39  responses of different MNS blood types to a low-fat diet.
40 duals after the HSF-DHA diet relative to the low-fat diet.
41 ficant larger decreases for TT carriers on a low-fat diet.
42 ns in plasma glucose and insulin only on the low-fat diet.
43  experienced adverse glycemic effects of the low-fat diet.
44 n omega-3-acid ethyl esters in patients on a low-fat diet.
45 genotype by choosing a high-carbohydrate and low-fat diet.
46 cells is evident in eNos(-/-) mice even on a low-fat diet.
47 recent reports questioned the superiority of low-fat diets.
48 n some people who exercise regularly and eat low-fat diets.
49 alance methods in animals fed both high- and low-fat diets.
50        HDL cholesterol decreased during both low-fat diets.
51 L subclass phenotype A during both high- and low-fat diets.
52  decrease in HDL cholesterol associated with low-fat diets.
53  livers of ethanol-treated rats fed high- or low-fat diets.
54 rate) diet or a calorie- and fat-restricted (low-fat) diet.
55 or gene main effects and interactions with a low-fat diet (20% from energy) compared with a high-fat
56 0-46% fat) show less lipoprotein benefits of low-fat diets (20-24% fat) than do men with a high-risk
57  that differed in macronutrient composition (low-fat diet: 20-25% fat, 15% protein, and 60-65% carboh
58 rolled consumption of a high-fructose (HFr), low-fat diet (24% of calories).
59 ts and lifestyle changes, a more traditional low-fat diet (25% fat kcals) with flaxseed supplementati
60 d 50% of energy from carbohydrates) and 2) a low-fat diet (25% of energy from fat and 62% of energy f
61 in diet, participants consumed an isocaloric low-fat diet (60% of energy from carbohydrate, 20% from
62  5.33 to 9.25 kg] at 12-month follow-up) and low-fat diets (7.99 kg [95% CI, 6.01 to 9.92 kg] at 6-mo
63 ilon study) in which they were assigned to a low-fat diet, a high-fat high-SFA (HSF) diet, and the HS
64                              Compared with a low-fat diet, a low-carbohydrate diet program had better
65                              Compared with a low-fat diet, a Mediterranean diet enriched with nuts co
66 , and 10 d of eating a constant high-fat and low-fat diet administered in a crossover design.
67      We then put these mice back on a normal low-fat diet, after which the mice exhibited normal body
68 hese disorders includes a high-carbohydrate, low-fat diet and avoidance of those events that promote
69                     Lifestyle modifications (low-fat diet and exercise) were associated with improvem
70 perfusion from 10 to 26 IU/g/h in rats fed a low-fat diet and from 17 to 34 IU/g/h in rats fed a high
71 , have impaired glucose tolerance when fed a low-fat diet and independent of body weight.
72                               Two diets, the Low-Fat diet and the Low-Fat Plus diet, designed to be i
73  in the intestine may sensitize the gut to a low-fat diet and to potential accompanying food-borne to
74               This finding was specific to a low-fat diet and was unrelated to other dietary variable
75  not eating snacks, following low-calorie or low-fat diets, and limiting portion sizes.
76                           High carbohydrate, low fat diets appear to have a more favorable impact on
77 feeding a fat-free diet, which suggests that low-fat diets are likely to be beneficial in lipodystrop
78 rs for cardiovascular disease exists because low-fat diets are typically recommended.
79 ercentage (-2.6% vs -0.9%; P = .03) than the low-fat diet at 18 months.
80 tes may be more effective than a high-GI and low-fat diet at reducing body weight and controlling glu
81 bohydrate diets are at least as effective as low-fat diets at reducing weight and improving metabolic
82 ental LDL-subclass patterns when consuming a low-fat diet: AxA mating (9 families with 19 children),
83 esterol-lowering diet may be preferable to a low-fat diet because of more favorable effects on the CV
84 glyceride levels significantly in rats fed a low-fat diet, but increased values about 2.4-fold in rat
85                                              Low-fat diets can increase plasma triacylglycerol and re
86                                   Ad libitum low-fat diets cause weight loss in nondiabetic populatio
87                               The ad libitum low-fat diet caused 6 kg weight loss and decreased highl
88 ectedly, a high-fat, Western-type (but not a low-fat) diet caused death in two-thirds of the beta3-/-
89 previously fasted animals high-carbohydrate, low-fat diets causes a dramatic induction of enzymes-suc
90 ancer rate was 0.42 in those assigned to the low-fat diet compared with 0.46 in the control group (HR
91 insulin resistance, with particular focus on low-fat diets compared with the currently popular low-ca
92  findings, we propose that, especially under low-fat diet conditions, adipose tissue-resident iNKT ce
93  response to feeding of a high carbohydrate, low fat diet, conditions that favor enhanced lipogenesis
94 We showed that iNKT cell-deficient mice on a low-fat diet, considered a normal diet for mice, display
95                                            A low-fat diet consisted of limited energy intake (1200 to
96 g: 1) a normal low fat (13% kcal) diet, 2) a low fat diet containing n-3 PUFAs, 3) a high fat (41% kc
97 pharmacological inhibition of ACAT2 were fed low fat diets containing various amounts of cholesterol
98 5 weeks of age, groups of animals were fed a low-fat diet containing 5% corn oil (LFCO).
99 corn oil, whereas one group continued on the low-fat diet containing 5% corn oil.
100 weeks of age, groups of animals were fed the low-fat diet containing 5% corn oil.
101 y of evidence from RCTs to determine whether low-fat diets contribute to greater weight loss than par
102     Participants were randomly assigned to a low-fat diet control group or TMD intervention groups [t
103 ented with mixed nuts, or advice to follow a low-fat diet (control group).
104 compared with 10 monkeys fed a low-fructose, low-fat diet (control).
105 mpared to aged-matched control animals fed a low-fat diet, correlating with enhanced alloreactive T c
106 t a high sucrose content in a hypoenergetic, low-fat diet did not adversely affect weight loss, metab
107 le fractured knee joints of mice receiving a low-fat diet did not demonstrate significant differences
108      DIO mice underwent switch to ad libitum low-fat diet (DIO-switch) or caloric restriction (CR) fo
109 Importantly, reversing the maternal HFD to a low-fat diet during a subsequent pregnancy improved feta
110  topics in the field, obesity and the use of low-fat diets early in life, have been the subject of re
111  approximately 1 h/d), eating a low-calorie, low-fat diet, eating breakfast regularly, self-monitorin
112                Refeeding a high carbohydrate/low fat diet enhances fatty acid synthesis by 5- to 20-f
113  B, type I) and apolipoprotein E genes fed a low-fat diet exhibit a constellation of pathologies shar
114 lated from AFABP-KO and WT mice fed high- or low-fat diets exhibited similar rates of basal and norep
115 ith the low-glycemic load diet than with the low-fat diet, expressed in absolute terms (mean [SE], 96
116 In sedentary nondiabetic persons following a low-fat diet, FABP2 Thr54 carriers have lower glucose to
117 mpaired glucose and insulin tolerance in LF (low fat diet)-fed control (AhR(fl/fl)) mice but not in a
118 ice fed a high-fat diet, compared with their low-fat diet-fed counterparts.
119 riatal slices of rats adapted to high fat or low fat diet feeding for 3 days.
120 rol mice were placed on either a high fat or low fat diet for 3.5 months.
121 caused by HFD were rescued by switching to a low fat diet for one month, suggesting a functional role
122 nd non-obese twin pairs consumed recommended low fat diets for 6 weeks before they received a 6-week
123 e of four treatment groups after following a low-fat diet for > or = 8 wk.
124 effects of 10.2 g psyllium/d adjunctive to a low-fat diet for >/=8 wk in individuals with mild-to-mod
125 at feeding, but reduces after switching to a low-fat diet for 1 d.
126 nto two groups and consumed either a high-or low-fat diet for 16 wk.
127 thionein transgenic mice were fed a high- or low-fat diet for 5 months to induce obesity.
128     Psyllium offers a potential adjunct to a low-fat diet for the treatment of hypercholesterolemia i
129 h modified Lieber-DeCarli ethanol-containing low-fat diets for 4 weeks.
130                         The effectiveness of low-fat diets for long-term weight loss has been debated
131 ns with 8 animals of each sex fed a high- or low-fat diet from each strain.
132 eviously reported that during refeeding on a low-fat diet, glucose tolerance is normal but insulin-de
133 be randomised into either a Mediterranean or low fat diet group for a 3 month intervention period.
134  the low-carbohydrate diet group than in the low-fat diet group (mean change, -12.9% vs. -6.7%; P < 0
135 s in insulin and HOMA-IR was observed in the low-fat diet group (P=0.02 and P=0.04, respectively).
136  of the low-carbohydrate diet group than the low-fat diet group completed the study (76% vs. 57%; P =
137       An opposite effect was observed in the low-fat diet group, although in this group the T allele
138  not change during the 10-wk interval in the low-fat diet group.
139 ition in the high-fat diet group than in the low-fat diet group.
140 vels, and more adverse symptoms than did the low-fat diet group.
141  baseline (n = 3,375), those assigned to the low-fat diet had a breast cancer rate of 0.27 compared w
142                            CCK-KO mice fed a low-fat diet had a reduced acute insulin response to glu
143                                An isocaloric low-fat diet has been shown to slow androgen-sensitive L
144 vious studies comparing low-carbohydrate and low-fat diets have not included a comprehensive behavior
145                         When maintained on a low-fat diet, HDL isolated from both strain C57BL/6J (B6
146                                          The low-fat "diet-heart hypothesis" has been controversial f
147 (2)): 35.8 +/- 2.9] or a calorie-restricted, low-fat diet (High Carb; BMI: 36.7 +/- 4.6) for 6 wk.
148 ness of the Mediterranean diet to a standard low fat diet in terms of differences in insulin sensitiv
149          The MED/LC diet was superior to the low-fat diet in decreasing intrahepatic, intrapericardia
150 tolerated and safe when used adjunctive to a low-fat diet in individuals with mild-to-moderate hyperc
151 safety of psyllium when used adjunctive to a low-fat diet in men and women with hypercholesterolemia.
152 ugment the cholesterol-lowering effects of a low-fat diet in persons with hypercholesterolemia.
153 ed fat, is as efficacious as the traditional low-fat diet in producing weight loss and may be more be
154 ious than a conventional, energy-restricted, low-fat diet in reducing cardiovascular disease risk.
155 er the HSF and HSF-DHA diets relative to the low-fat diet in the APOE3/E4 group (P < 0.015).
156 icles (subclass pattern B) can be induced by low-fat diets in healthy subjects with large LDL particl
157 the concept that both hyperinsulinemia and a low-fat diet increase DNL, and that DNL contributes to h
158                                              Low-fat diets increase plasma triacylglycerol and decrea
159 improvement in lipid profiles from long-term low-fat diet intake in the APOA5 rs964184 risk allele.
160 s in REE and TEE that were greatest with the low-fat diet, intermediate with the low-glycemic index d
161  from this study indicate that a high-fiber, low-fat diet intervention is associated with reduced ser
162 indings suggest that the long-term effect of low-fat diet intervention on bodyweight depends on the i
163 ning plant sterol esters incorporated into a low-fat diet is a beneficial adjunct in the dietary mana
164 ild-to-moderate hypercholesterolemia after a low-fat diet lead-in phase lasting >/=8 wk.
165                              In mice fed the low-fat diet, leptin activated STAT3 signaling when admi
166 domly divided into three groups: (1) control low-fat diet (LF-SED; 15% of calories from fat), (2) hig
167 7BL/6 mice were fed an HFD (60% fat kcal) or low-fat diet (LFD) (10% fat kcal) for 8 or 12 weeks.
168 ung (age 25 days) Sprague-Dawley rats with a low-fat diet (LFD) alone or with vitamin D depletion (LF
169 nd cholesterol esters (CEs) in response to a low-fat diet (LFD) and a moderate-fat diet (MFD) and ass
170  were fed with high-fat diet (HFD) or normal low-fat diet (LFD) and subjected to a protocol of ovalbu
171 Sprague-Dawley rats were fed either a HFD or low-fat diet (LFD) for 4 weeks.
172 iet (SFD), a high-trans-fat diet (TFD), or a low-fat diet (LFD) for 4 wk prior to mating, and remaine
173      We compared the effects of an LGD and a low-fat diet (LFD) on body composition and components of
174  from small intestine of C57BL/6J mice fed a low-fat diet (LFD) or high-fat diet (HFD) for 12 weeks.
175       As adults, offspring were fed either a low-fat diet (LFD) or high-fat diet (HFD) for 6 wk.
176 elevated fat mass and decreased lean mass on low-fat diet (LFD), accompanied by leptin resistance and
177 57BL/6J mice were divided into three groups: low-fat diet (LFD), high-fat diet (HFD) and HFD suppleme
178 high-fat diet (HFD-S), similar to mice fed a low-fat diet (LFD).
179 ched to HFD supplemented with 10% HPMC, or a low-fat diet (LFD).
180 nd VMH FA levels, rats were trained to eat a low-fat diet (LFD; 13.5%) or an HFD in 3 h/day and were
181 diet options (low-carbohydrate diet [LCD] or low-fat diet [LFD]) before choosing and were allowed to
182 session) and sedentary rats fed either chow (low-fat diet [LFD]; normal insulin sensitivity) or a hig
183 the presence of an up-titration regiment and low-fat diet, lomitapide is generally well tolerated and
184                              Rats received a low-fat diet (Low-Fat) or one enriched with either satur
185                    Both low-carbohydrate and low-fat diets lowered weight and improved metabolic risk
186 n diet and lipid-lowering drugs or on strict low-fat diet (&lt;10% of calories) without lipid drugs; and
187 rcise recommendation, and group meetings, or low-fat diet (&lt;30% energy from fat, <300 mg of cholester
188 (</=45% of energy from carbohydrates) versus low-fat diets (&lt;/=30% of energy from fat) on metabolic r
189 r in monounsaturated fat, however, eucaloric low-fat diets may increase plasma triacylglycerol concen
190 rate diet lost more weight than those on the low-fat diet (mean [+/-SD], -5.8+/-8.6 kg vs. -1.9+/-4.2
191 e, the decrease in REE was greatest with the low-fat diet (mean [95% CI], -205 [-265 to -144] kcal/d)
192                                 One received low-fat diet, medium-chain triglyceride supplementation,
193 wever, in hepatocytes from animals given the low-fat diet, most of the differences between the variou
194 /genotype) as well as from rats fed high- or low-fat diets (n=8/treatment) were analyzed in parallel
195 5) with the high-fat diet and to 7% with the low-fat diet (NS).
196 bic exercise or moderate lifestyle activity; low-fat diet of about 1200 kcal/d.
197 found no effect from a high vegetable/fruit, low-fat diet on breast cancer prognosis.
198 e long-term effects of physical exercise and low-fat diet on the progression of coronary artery disea
199 ethanol for dietary carbohydrate in high-and low-fat diets on energy expenditure and body composition
200 tested the long-term effects of high-fat and low-fat diets on males of two inbred strains of mice and
201 regardless of whether the animals were fed a low fat diet or a high fat/high cholesterol diet.
202  when they were maintained on either a basal low fat diet or a western-type high fat/high cholesterol
203 respectively) by hepatocytes from rats fed a low-fat diet or a diet supplemented with the correspondi
204 )-transduced regeneration tissues were fed a low-fat diet or a high-fat diet and treated with vehicle
205 KO and wild-type (WT) littermates were fed a low-fat diet or a high-fat diet to investigate the effec
206 mor biology can be altered by either a vegan low-fat diet or eliminating simple carbohydrates accompa
207 d 2, 8, and 18 months were fed 16 weeks of a low-fat diet or HFD.
208  nephropathy, we treated 1) C57BL/6J mice on low-fat diet or high-fat diet with FXR agonists (GW4064
209 notype were at lower risk if they consumed a low-fat diet (OR, 0.2; 95% CI, 0.1-0.5) relative to thos
210 tensity, evidence from RCTs does not support low-fat diets over other dietary interventions for long-
211 eported less hunger than those receiving the low-fat diet (P = .04).
212 uring the high-fat diet, and 3.2% during the low-fat diet (P = 0.4, 0.19, respectively).
213  diet and -0.19 mmol/L [-7.4 mg/dL] with the low-fat diet; P = 0.2).
214                Compared with participants on low-fat diets, persons on low-carbohydrate diets experie
215            After an additional 14 weeks on a low-fat diet, plasma cholesterol levels decreased from 2
216                                          The low-fat diet provided 18.8% of energy from fat with a ra
217 dress these issues, we designed two diets: a low-fat diet providing approximately 20% of energy as fa
218 ated with the changes from baseline with the low-fat diet (r = -0.74, P = 0.01), as well as with the
219              Compared with recipients of the low-fat diet, recipients of the low-carbohydrate diet ha
220                 With rising obesity, despite low-fat diet recommendations, there is an increased inte
221 ch diet of maternal milk, and substituting a low-fat diet reduced UCP2, restored mitochondrial coupli
222 atic overexpression of VLDLR combined with a low-fat diet regimen induces regression of advanced plaq
223   Treatment with HD-Ad-VLDLR together with a low-fat diet regimen resulted in reduced lesion size (cr
224 , -0.37, and -0.45 with the AAD, Step I, and low-fat diets, respectively (P < 0.0001).
225 48 +/- 0.18 mmol/L with the AAD, Step I, and low-fat diets, respectively.
226 72 +/- 0.11 mmol/L with the AAD, Step I, and low-fat diets, respectively.
227                Refeeding a high carbohydrate/low fat diet resulted in a 4- to 5-fold increase of nucl
228                    A switch from high-fat to low-fat diet reversed these outcomes, with switched C57B
229 ped" to match the FM of mice maintained on a low-fat diet (standard diet [SD] mice).
230                         When mice were fed a low-fat diet supplemented with taurocholic acid, but not
231 h dietary support including high-protein and low-fat diets supplemented with medium-chain triglycerid
232                                              Low-fat diets tend to improve low-density lipoprotein ch
233 urve after the test meal was lower after the low-fat diet than after the high-fat diet (P < 0.04).
234 e low-carbohydrate diet vs. -4.8 kg with the low-fat diet) than fat-free mass (change, -3.3 kg vs. -2
235                                         On a low fat diet, the Clps-/- mice did not have steatorrhea.
236                                         On a low fat diet, the Slc10a2-/- mice did not have steatorrh
237             In hepatocytes from rats fed the low-fat diet, the association of radioactivity with the
238 n1LKO mice are similar to control mice fed a low-fat diet, they are protected against insulin resista
239 n sedentary nondiabetic subjects following a low-fat diet, Thr54 FABP2 carriers have lower glucoregul
240 used GM-CSF-deficient (Csf2(-/-)) mice fed a low fat diet to test the hypothesis that adipose tissue
241     These findings do not support the use of low-fat diets to prevent weight gain.
242 ts with breast cancer randomly assigned to a low-fat diet versus control.
243                 The subjects consumed a very-low-fat diet (VLFD; 11% of energy as fat), a Step I diet
244 hat country, age, sex, smoking, alcohol use, low-fat diet, waist circumference, recent weight gain (>
245 igher than concentrations observed after the low-fat diet was consumed.
246                                            A low-fat diet was not significantly associated with adver
247                                          The Low-Fat diet was relatively typical of a low-fat U.S. di
248  (n = 122) aged 50-75 y who were following a low-fat diet were genotyped and underwent oral-glucose-t
249  several lipid lowering treatments including low fat diet when compared with LDL pattern A individual
250 11 transporter expression in comparison with low-fat diet, whereas liver-to-feces RCT was preserved a
251 = 0.01) in participants who were assigned to low-fat diets, whereas there was no significant genotype
252 with these in vitro findings, feeding mice a low fat diet with ethanol for 4 weeks resulted in a sign
253 icacy of 2 moderate-carbohydrate diets and a low-fat diet with different GIs on weight loss and the m
254                      Finally, feeding mice a low-fat diet with ethanol resulted in significantly redu
255 e to placebo in subjects already consuming a low-fat diet, with no effect on serum HDL or triacylglyc
256 ry greatly in their lipoprotein responses to low-fat diets, with some of this variation being attribu
257 owever, because it lowers HDL cholesterol, a low-fat diet without substantial weight loss may not be
258 , in type 2 diabetes patients, an ad libitum low-fat diet would cause greater weight loss than would

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