ライフサイエンスコーパス: low-flow ischemia

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1 deficiency (G4H-/-) were subjected to global low-flow ischemia.

2 ylproprionic acid derivative (RSR13), during low-flow ischemia.

3 o the sarcolemma occurs in vivo during acute low-flow ischemia.

4 PC could protect against injury arising from low-flow ischemia.

5 ntractile dysfunction that is apparent after low-flow ischemia.

6 -NMR isotopomer analysis after 30 minutes of low-flow ischemia (0.3 mL/min) and 60 minutes of reperfu

7 Hearts had 60 minutes of low-flow ischemia (10% of baseline coronary flow) and 30

8 MAPK activation was markedly reduced during low-flow ischemia (2.3- versus 7-fold in wild-type heart

9 During low-flow ischemia [3-13C]alanine enrichment was higher,

10 Similarly, after 45 minutes of low-flow ischemia, after diastolic pressure had increase

11 RSR13 given after the onset of low-flow ischemia also improved cardiac PCr/ATP ratios a

12 urine hearts were subjected to 60 minutes of low-flow ischemia and 120 minutes of reperfusion.

13 in left ventricle) rat and rabbit hearts to low-flow ischemia and increased extracellular calcium (f

14 Our goals were to (1) simulate the degree of low-flow ischemia and mixed anaerobic and aerobic metabo

15 red-cell perfused rabbit hearts by imposing low-flow ischemia and pacing tachycardia.

16 nstrumented canine myocardium after bouts of low-flow ischemia and persists after reperfusion.

17 tion of glucose uptake and glycolysis during low-flow ischemia and plays an important protective role

18 During low-flow ischemia and postischemic reperfusion in vitro,

19 We tested the effects of low-flow ischemia and reperfusion on the ratio of tracer

20 flow, 15 mins of global ischemia, 5 mins of low flow ischemia, and 30 mins of reperfusion.

21 rat hearts were perfused during preischemia, low-flow ischemia, and reperfusion, using (3)H-substrate

22 2 mL/min per gram wet wt), (D) 90 minutes of low-flow ischemia at 10% of baseline coronary flow (0.29

23 5 minutes of reperfusion), (C) 90 minutes of low-flow ischemia at 10% of baseline coronary flow (0.31

24 nditions, then subjected to 50 min of severe low flow ischemia followed by 60 min of reperfusion.

25 uced by coronary artery occlusion and global low-flow ischemia in isolated hearts.

26 hearts, the LV stiffened more rapidly during low-flow ischemia in the old hearts than in the adults,

27 Thus during sustained regional low-flow ischemia in vivo, the ischemic myocardium incre

28 ng that if activation of PKC occurred during low-flow ischemia, it was not protective.

29 However, whether low-flow ischemia leads to the translocation of glucose

30 During low-flow ischemia, MyBP-C is dephosphorylated, and the n

31 In a canine model of regional low-flow ischemia (n = 9), serial IPPA SPECT images (2 m

32 ine triphosphate content falls slowly during low-flow ischemia, PKC may be activated and translocated

33 In the isolated working rat heart, low-flow ischemia rapidly activated AMPKK activity when

34 respectively; P<.05) but not resulting from low-flow ischemia (recovery of developed pressure was 40

35 Low-flow ischemia resulted in a 270% increase (P:<0.05)

36 During low-flow ischemia simulating an acute myocardial infarct

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