ライフサイエンスコーパス: low-frequency stimulation

1 gh frequency tetanic stimulation, but not by low frequency stimulation.

2 were found in NMDA-dependent LTD induced by low-frequency stimulation.

3 F-PC synaptic transmission is reduced during low-frequency stimulation.

4 imulation or long-term depression induced by low-frequency stimulation.

5 F facilitate long-term depression induced by low-frequency stimulation.

6 th those of sham controls that received only low-frequency stimulation.

7 excitation but is difficult to activate with low-frequency stimulation.

8 scrib synapse behaves relatively normally at low-frequency stimulation.

9 ked inhibitory synaptic currents (34%) after low-frequency stimulation.

10 high probabilities of release in response to low-frequency stimulation.

11 ffect depotentiation, the reversal of LTP by low-frequency stimulation.

12 eated and untreated slices when initiated by low frequency stimulation (0.2 Hz).

13 rom the iLF, cLF, cIML and DH in response to low-frequency stimulation (0.03-0.1 Hz) were sensitive t

14 m depression (LTD) was elicited by prolonged low frequency stimulation (1 Hz, 15 min).

15 The LTD induced by low-frequency stimulation (1 Hz) protocols was found to

16 Strikingly, low-frequency stimulation (1 Hz, 15 min) of the glutamat

17 ve of synaptic strength induced by prolonged low-frequency stimulation (1-5 Hz) is systematically up-

18 Transmitter output during low-frequency stimulation (2-5 Hz) is maintained entirel

19 This LTP required relatively low-frequency stimulation (20 Hz) and simultaneous activ

20 Sustained low-frequency stimulation (5 Hz) of primary afferent fib

21 At low-frequency stimulation, 5-CT markedly reduces charge

22 We show that at low-frequency stimulation, a filamentous-actin cell cort

23 We show that at low-frequency stimulations, a first sub-pool is graduall

24 hip14 mutants show exocytic defects at low frequency stimulation and a nearly complete loss of

25 it long-term depression (LTD) in response to low-frequency stimulation and modest depolarization.

26 responses were observed for 56 (25%) of the low-frequency stimulations and for 76 (50%) of the high-

27 strate CA1-region long-term depression after low-frequency stimulation, and AC8 KO mice also fail to

28 1 hour), input-specific, depotentiates with low-frequency stimulation, and is blocked by N-methyl-D-

29 Evoked inhibition was larger under low-frequency stimulation, and, importantly, this change

30 ontaneous or evoked synaptic currents during low-frequency stimulation at 0.05 Hz in these Xenopus cu

31 Previous studies in slices have shown that low-frequency stimulation at 5 Hz, i.e., theta pulse sti

32 sion was extremely frequency dependent, with low-frequency stimulation being largely ineffective.

33 rm of long-term depression (LTD) produced by low-frequency stimulation combined with glutamate transp

34 Low-frequency stimulation given shortly after LTP induct

35 tetanus [post-tetanic potentiation (PTP)] or low-frequency stimulation [homosynaptic depression (HSD)

36 How such low frequency stimulation in turn elicits sufficient cat

37 Chronic low-frequency stimulation increased the levels of slow-t

38 Chronic low-frequency stimulation increased the myosin heavy cha

39 Low frequency stimulation induced long-term depression (

40 Low-frequency stimulation induced a transient depression

41 tively detect proBDNF or mBDNF, we show that low-frequency stimulation induced predominant proBDNF se

42 e and high levels of PE persistently reduced low-frequency stimulation-induced eCB-LTD.

43 c core, TLR4.KO animals exhibit a deficit in low-frequency stimulation-induced NMDAR-dependent long-t

44 ity suppresses epileptiform activity and (2) low frequency stimulation is an effective stimulation pr

45 e induction of long-term depression (LTD) by low-frequency stimulation is accompanied by a marked shr

46 diaphragm fatigue resulting from repetitive low-frequency stimulation is associated with lipid perox

47 sults of this preliminary study suggest that low-frequency stimulation is tolerable and reduces epile

48 Low-frequency stimulation known to induce long-term depr

49 We find that ACh release evoked by low-frequency stimulation leads to biphasic excitatory-i

50 In the presence of dynasore, low-frequency stimulation led to sustained accumulation

51 In control conditions (0.5 mM EGTA) low frequency stimulation (LFS; 200 stimuli) delivered t

52 he induction, but not maintenance, of LTD by low-frequency stimulation (LFS) (1 Hz/15 min) without af

53 Prolonged low-frequency stimulation (LFS) (900 pulses/1 Hz) of the

54 magnitude with that obtained with prolonged low-frequency stimulation (LFS) but requires far fewer s

55 tablished the efficacy of cell type-specific low-frequency stimulation (LFS) in controlling ictogenes

56 Low-frequency stimulation (LFS) is an alternative tool f

57 Low-frequency stimulation (LFS) of non-nociceptive affer

58 Like LTP, spine expansion was reversed by low-frequency stimulation (LFS) via a phosphatase-depend

59 Here, we use low-frequency stimulation (LFS), a protocol shown to ind

60 e in synaptic strength induced by subsequent low-frequency stimulation (LFS).

61 cke solution containing 100 microM choline), low-frequency stimulation (LFS, 3-5 Hz/15 min) of the pr

62 To produce LTD, prolonged low-frequency stimulation (LFS, 900 stimuli at 1 Hz) was

63 s preparation, we have tested the effects of low-frequency stimulation (LFS; 1 Hz for 15 min) on syna

64 In the hippocampal CA1 region, low-frequency stimulation (LFS; 200 pulses at 1 Hz) caus

65 t stimulation [TBS]) or LTD (900-pulse, 1-Hz low-frequency stimulation [LFS]) was induced in the DG o

66 tients with epilepsy and that suppression by low frequency stimulation may be mediated by long-term d

67 LTD was input-specific, i.e., low frequency stimulation of one pathway produced LTD of

68 nerve can elicit or inhibit micturition, and low frequency stimulation of the compound pudendal nerve

69 Low-intensity, low-frequency stimulation of a vagus nerve elicited pauc

70 In conclusion, low-frequency stimulation of C1 neurons activates pontin

71 Prolonged low-frequency stimulation of excitatory afferents to bas

72 ed rapidly and reversibly by brief trains of low-frequency stimulation of mossy fiber axons.

73 Low-frequency stimulation of parallel fibers facilitates

74 Here, we report that low-frequency stimulation of PP inputs to CA1 has no las

75 ia gelatinosa neurons that can be induced by low-frequency stimulation of primary afferent Adelta-fib

76 Low-frequency stimulation of the fornix activates the hi

77 Low-frequency stimulation of the fornix occurred in 4-ho

78 We tested the hypothesis that low-frequency stimulation of the fornix reduces interict

79 In current-clamp recordings, low-frequency stimulation of the MNTB led to a decline i

80 When LSO neurons were depolarized, low-frequency stimulation of the MNTB produced a signifi

81 This realization has led to low-frequency stimulation of the PPN for treating patien

82 the mechanisms of transmitter release during low-frequency stimulation of the Schaffer collaterals we

83 The differential effects of high- and low-frequency stimulation of this pathway seem most like

84 aintained in culture without or with chronic low frequency stimulation (one 5 s train of 5 Hz pulses

85 lease probability under conditions of either low-frequency stimulation or high-frequency augmentation

86 tion of LTP by high-frequency stimulation or low-frequency stimulation paired with postsynaptic depol

87 mutant with reduced TrkB by a depolarization-low-frequency stimulation pairing protocol that puts min

88 Low-frequency stimulation produced long-term depression

89 Secondary EPSPs evoked by low-frequency stimulation ranged from 0.5 to 10 mV in am

90 We also found that low-frequency stimulation reduced the amplitude of the p

91 Repetitive low frequency stimulation results in potentiation of twi

92 Moreover, high-frequency, but not low-frequency stimulation selectively induced the secret

93 In contrast, prolonged low-frequency stimulation shifts the equilibrium toward

94 We suggest that repeated low-frequency stimulation simultaneous with LTD inductio

95 Here we show that, during low-frequency stimulation, single-vesicle fusion leads t

96 rimental design independent LTP and control (low-frequency stimulation) sites were examined.

97 , and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-i

98 The stronger the afferent activation during low-frequency stimulation, the greater was the probabili

99 elicits larger spine calcium transients than low-frequency stimulation under all stimulus conditions,

100 For low-frequency stimulation under normal physiological con

101 np54p, long-term depression (LTD) induced by low-frequency stimulation was blocked in the mouse hippo

102 ause the induction of synaptic plasticity by low-frequency stimulation was enhanced at an unprimed sy

103 long-term depression evoked by paired-pulse low-frequency stimulation was modestly facilitated in th

104 evoked from the iLF, cLF, cIML and DH during low-frequency stimulation were reduced by NMDA receptor

105 ), synaptic strength recovered rapidly after low-frequency stimulation, whereas in another group of n

106 ptor activation, for instance in response to low-frequency stimulation, whereas LTP is induced by the

107 Conversely, low-frequency stimulation, which reduces the number of s

108 ORNs reliably follow low frequency stimulations with high fidelity by generat

109 otentiation was induced, however, by pairing low-frequency stimulation with direct depolarization of

110 est tremor may be significantly entrained by low frequency stimulation without stimulation timing-dep