ライフサイエンスコーパス: low-grade

1 and headache; 27% had fever (short-term and low-grade).

2 nal adverse events, which were predominantly low grade.

3 = 26) compared to those who continued to be low grade (1.99 +/- 0.82; n = 17); however, this did not

4 ns], WHO III: 17, WHO IV: 13, without biopsy low-grade: 1, high-grade: 1) were investigated with a hy

5 ) ovarian stroma, benign ovarian tumors (3), low grade (4) and high grade (5) serous tumors, and endo

6 t to cervical cytology for the management of low grade abnormal cytology, and in a test of cure.

7 These irAEs are often low grade and manageable, but severe irAEs may lead to p

8 from anaemia, toxicities with olaparib were low grade and manageable.

9 undetermined significance (ASCUS); 2173 with low-grade and 1282 with high-grade squamous intraepithel

10 FC1, a new candidate susceptibility gene for low-grade and borderline serous EOC.

11 able methodology for differentiating between low-grade and high-grade DCIS.

12 e to distinguish between normal and squamous low-grade and high-grade dyskaryosis, and between normal

13 examine the mechanistic differences between low-grade and high-grade inflammation induced by E. coli

14 High low-risk HPV DNA loads were found in low-grade and high-grade lesions, while high high-risk H

15 ompared it to non-transformed and neoplastic low-grade and high-grade prostate epithelial tissue from

16 wing that DACH1 was higher in normal breast, low-grade and luminal-type cancer in comparison with bre

17 Es with nivolumab monotherapy were primarily low grade, and most resolved with established safety gui

18 d 45 minutes after injection, there was only low-grade background activity in the majority of analyze

19 arditis (IE) mostly occurs after spontaneous low-grade bacteremia.

20 ion syndrome are prone to the development of low-grade brain tumors (gliomas) within the optic pathwa

21 ears) with residual or progressive benign or low-grade brain tumors at a single center between April

22 s with residual and/or progressive benign or low-grade brain tumors requiring radiotherapy for long-t

23 with neurofibromatosis type 1 (NF1) develop low-grade brain tumors throughout the optic pathway.

24 s with residual and/or progressive benign or low-grade brain tumors treated with SCRT and ConvRT tech

25 nd a three-class classification into normal, low grade cancer, and high grade cancer.

26 ting high- or intermediate-grade tumors from low-grade cancers.

27 sed in normal breast epithelial cells and in low-grade cancers.

28 , including germ cell tumors, high-grade and low-grade carcinomas and benign tissues.

29 could be associated with the development of low-grade carcinomas.

30 Tyr705-phosphorylated STAT3 increased from low-grade cervical intraepithelial neoplasia (CIN1) to p

31 es impaired intestinal barrier function with low grade chronic inflammation, hyperinsulinemia, and in

32 rload and calorie excess during obesity is a low grade chronic inflammatory state with diminished abi

33 l microRNA profiles, immunosenescence, and a low-grade chronic inflammation (inflammaging).

34 gar-sweetened beverage (SSB) consumption and low-grade chronic inflammation are both independently as

35 Osteoarthritis (OA) is a low-grade chronic inflammatory joint disease.

36 Studies of long-term survivors show low-grade chronic inflammatory, fibrotic, and microvascu

37 sumed over 8 d did not differentially affect low-grade chronic systemic inflammation in normal-weight

38 Low-grade, chronic inflammation has been associated with

39 Prespecified outcome measures included low-grade CIN (grade 1 [CIN1]) and high-grade CIN (grade

40 btype indicated that the ER(+)/PR(+)/HER2(-)/low-grade clinical subtype was preferentially responsive

41 in unexpected genes; permitted detection of low-grade constitutional, somatic, and revertant mosaici

42 ed ultrasound was judged semiquantitatively; low-grade contrast enhancement (CE) suggested its absenc

43 90% for genital warts, approximately 45% for low-grade cytological cervical abnormalities, and approx

44 image-read LBC screening with HPV triage of low-grade cytology ('LBC screening'), (ii) HPV screening

45 te, 0.53 per thousand), and 182 diagnoses of low-grade DCIS (detection rate, 0.25 per thousand).

46 tio, 1.11; P = .016) and not significant for low-grade DCIS (P = .10).

47 unds, cancer detection rates were lowest for low-grade DCIS (range, 0.11 [58 of 508 817 patients] to

48 h the prevalence round; conversely, rates of low-grade DCIS and, less markedly, intermediate-grade DC

49 f MR imaging versus conventional imaging for low-grade DCIS components was 74.0% (20 of 27) versus 40

50 val benefit of performing breast surgery for low-grade DCIS was lower than that for intermediate- or

51 Detection rates for low-grade DCIS were significantly lower in the first (od

52 testing, RT only for cohort 2; (3) no RT for low-grade DCIS, test for intermediate- and high-grade DC

53 ed) to distinguish high-, intermediate-, and low-grade DCIS.

54 more aggressive invasive breast cancer than low-grade DCIS.

55 etween the surgery and nonsurgery groups for low-grade DCIS.

56 of active surveillance for the management of low-grade DCIS.

57 us contrast enhancement of vertebral bodies, low-grade destruction of vertebral bodies, hyperintense/

58 patients with T1N0 breast cancer was 10% for low-grade disease, 13% for moderate-grade disease, and 1

59 containing EAC, high grade dysplasia (HGD), low grade dysplasia (LGD), Barrett's esophagus (BE), col

60 adenoma, n = 36; tubulovillous adenoma with low grade dysplasia, n = 27; sessile serrated adenoma, n

61 00 person-years among patients with baseline low-grade dysplasia (95% CI 1.5-7.2), and 7.3 per 100 pe

62 00 person-years among patients with baseline low-grade dysplasia (95% CI, 4.9-14.0), and 13.5 per 100

63 nts with inflammatory bowel disease and flat low-grade dysplasia (fLGD) in the colon.

64 Barrett's esophagus (BE) with low-grade dysplasia (LGD) can progress to high-grade dys

65 rinciples in the diagnosis and management of low-grade dysplasia (LGD) in Barrett's esophagus patient

66 intestinal pathologists, in the diagnosis of low-grade dysplasia (LGD) in patients with Barrett's eso

67 intestinal pathologists, in the diagnosis of low-grade dysplasia (LGD) in patients with Barrett's eso

68 s with Barrett's esophagus, the diagnosis of low-grade dysplasia (LGD) is subjective, and reported ou

69 s with Barrett's esophagus, the diagnosis of low-grade dysplasia (LGD) is subjective, and reported ou

70 to be distinguished from regions containing low-grade dysplasia (LGD), high-grade dysplasia (HGD) or

71 Notably, those suspected of low-grade dysplasia have p53 mutations or undergo amplif

72 splasia-containing AGWs of HIV+ MSM harbored low-grade dysplasia in 6 cases (16%), high-grade dysplas

73 ale sex, smoking, length of BE, and baseline low-grade dysplasia that identified patients with BE at

74 r that could be used to assign patients with low-grade dysplasia to a low- or high-risk group.

75 P = .02), and patients with either high- or low-grade dysplasia were more likely to present with ana

76 moking, length of BE, and baseline-confirmed low-grade dysplasia were significantly associated with p

77 th BE (baseline nondysplastic BE +/- BE with low-grade dysplasia) and at least a 3-year follow-up per

78 ong adults with nondysplastic BE (or BE with low-grade dysplasia) at their index endoscopy and at lea

79 erived from patients with Lynch syndrome--78 low-grade dysplastic adenomas, 57 high-grade dysplastic

80 isease recurrence in women with early-stage, low-grade endometrial carcinoma.

81 orbidity in women with presumed early-stage, low-grade endometrial carcinoma?

82 entire course of a study, including chronic low-grade events.

83 tion of high efficiency and selectivity from low-grade feedstocks can be achieved by engineering the

84 d with the transformation and progression of low-grade fibrillary astrocytoma to high-grade anaplasti

85 practice, and the survival of patients with low-grade FL has improved.

86 it as a treatment strategy for patients with low-grade FL, our understanding of the biology of the di

87 mendation for a newly diagnosed patient with low-grade FL?

88 in tumor cells from high-grade compared with low-grade follicular lymphoma patients.

89 We conclude that parainflammation, a low-grade form of inflammation, is widely prevalent in h

90 gative disease is heterogeneous and includes low-grade forms driven by distinct sets of genetic alter

91 Low-grade germinal matrix-intraventricular hemorrhage (G

92 ratio to distinguish between high-grade and low-grade glial tumors.

93 rized RCAS/Ntv-a mouse model of PDGFB-driven low grade glioma.

94 lastoma (4.4%), head and neck cancer (1.0%), low-grade glioma (1.5%), lung adenocarcinoma (1.6%), lun

95 rformance for IDH gene mutation detection in low-grade glioma (AUC, 0.818) and MTI in high-grade glio

96 sion kurtosis imaging (DKI) to differentiate low-grade glioma (LGG) (World Health Organization [WHO]

97 -toxicity profile in patients with pediatric low-grade glioma (PLGG) who experienced treatment failur

98 Outcome of low-grade glioma (WHO grade II) is highly variable, refl

99 rescence-guided resection of microscopic and low-grade glioma brain tumors with invasive or diffusive

100 ival in the three recently defined molecular low-grade glioma subgroups (IDHmt, with or without 1p/19

101 n progression-free survival in patients with low-grade glioma when treated with either radiotherapy a

102 le with a history of metastatic tectal plate low-grade glioma who was diagnosed at age 2.8 years tran

103 CMRO2 was decreased (P = .037) in low-grade glioma with a mutated IDH gene, and MTI was si

104 1 osteosarcoma, 1 sarcoma, 1 astrocytoma, 1 low-grade glioma, and 2 preinvasive breast cancers [duct

105 y temozolomide chemotherapy in patients with low-grade glioma, and assessed progression-free survival

106 ates with significantly inferior survival in low-grade glioma.

107 Infiltrating low grade gliomas (LGGs) are heterogeneous in their beha

108 These pediatric low-grade gliomas (LGGs) are fundamentally different fro

109 tation is the earliest genetic alteration in low-grade gliomas (LGGs), but its role in tumor recurren

110 Pediatric low-grade gliomas (PLGGs) are commonly associated with B

111 Angiocentric gliomas are pediatric low-grade gliomas (PLGGs) without known recurrent geneti

112 ed to update response criteria for high- and low-grade gliomas and to address such issues as pseudore

113 children with NF1 are at risk for developing low-grade gliomas of the optic pathway and brainstem, in

114 Low-grade gliomas with favorable characteristics are slo

115 isms underlying the malignant progression of low-grade gliomas with mutations in IDH1 (encoding isoci

116 ognostic outcome of certain cancers, such as low-grade gliomas, acute myeloid leukaemia, and chondros

117 nonneoplastic brain tissue, particularly for low-grade gliomas.

118 , in bladder and renal carcinomas as well as low-grade gliomas.

119 lower CBFi and CMRO2i in ELGA neonates with low-grade GM-IVH compared to neonates without hemorrhage

120 premature brain, yet the long-term impact of low-grade GM-IVH on cerebral blood flow and neuronal hea

121 gestational age (ELGA) neonates (seven with low-grade GM-IVH) and monitored them weekly until they r

122 owever, this association was not observed in low-grade (grade 1 and 2) breast cancers.

123 that discriminates high-grade (>/=GS7) from low-grade (GS6) cancer and benign disease.

124 s in host-microbiota interactions that cause low-grade gut inflammation can promote colon carcinogene

125 tomic analysis showed that during protracted low-grade H(2)O(2) stress, Escherichia coli responds by

126 Optimum surgical intervention for low-grade haemorrhoids is unknown.

127 e-negative breast cancer, and, despite being low-grade, harbors the complex genomic landscape of usua

128 HE) is a promising technology for converting low grade heat to electricity.

129 d on these unique phase behaviors to convert low grade heat to work or electricity.

130 oling systems is being placed that can cycle low grade heat.

131 ll-scale TOEC systems to extract energy from low-grade heat and identifies key factors for performanc

132 age and membrane distillation (MD) utilizing low-grade heat as a separation stage.

133 s can more clearly identify the prospects of low-grade heat conversion and large-scale energy storage

134 Low-grade heat energy from sources below 100 degrees C i

135 he atmosphere at ambient conditions by using low-grade heat from natural sunlight at a flux of less t

136 rmoradiative cells have potential to harvest low-grade heat into electricity.

137 ess has the potential to harvest energy from low-grade heat sources by using a temperature difference

138 purify contaminated, acidic wastewater using low-grade heat sources, such as geothermal energy, witho

139 significantly higher for high-grade than for low-grade hemispheric (116 mL/min/100 g [interquartile r

140 a more favorable prognosis, and tumors with low-grade histology especially tend evolve slowly.

141 long-term changes in neuronal function, and low grade inflammation of the bowel have been hypothesiz

142 eration, leading to microbiota encroachment, low-grade inflammation (LGI), and metabolic syndrome.

143 justment for C-reactive protein, a marker of low-grade inflammation (mean difference in LTL = -0.3%,

144 adipose tissue (VAT) is the seat of chronic low-grade inflammation (metaflammation), but the mechani

145 s underlying the association between chronic low-grade inflammation and disruption of normal tissue f

146 Obesity is associated with low-grade inflammation and elevated levels of circulatin

147 abetes are associated with increased chronic low-grade inflammation and elevated plasma glucose level

148 We have completed an EWAS of chronic low-grade inflammation and identified many novel genetic

149 ed fasting that may in the long term lead to low-grade inflammation and impaired glucose homeostasis.

150 sue macrophages (ATMs) contribute to chronic low-grade inflammation and obesity-induced insulin resis

151 ymethylcellulose and polysorbate-80, induced low-grade inflammation and obesity/metabolic syndrome in

152 sity and type 2 diabetes are associated with low-grade inflammation and specific changes in gut micro

153 in school-aged children suggesting systemic low-grade inflammation as a phenotypic characteristic of

154 ed host-microbiota interactions resulting in low-grade inflammation can promote adiposity and its ass

155 and its disruption may lead to non-resolving low-grade inflammation conducive to atherosclerosis.

156 Systemic low-grade inflammation has been demonstrated in a range

157 etabolic disorder are accompanied by chronic low-grade inflammation has fundamentally changed our vie

158 challenge the common assumption that central low-grade inflammation in schizophrenia is mirrored by i

159 Recent findings suggest that low-grade inflammation in the intestine is promoted by c

160 e DNA methylation is associated with chronic low-grade inflammation may reveal novel pathways or ther

161 Sustained low-grade inflammation mediated by non-resolving inflamm

162 Obesity is associated with chronic low-grade inflammation of adipose tissue (AT) and an inc

163 Thus, microglia and low-grade inflammation of myelinated tracts emerged as t

164 ith adipocytes are essential for the chronic low-grade inflammation of obese adipose tissue.

165 all of these functions of WAT, together with low-grade inflammation of the tissue in obese individual

166 e underlying cause of catatonic signs is the low-grade inflammation of white matter tracts, which mar

167 Chronic low-grade inflammation reflects a subclinical immune res

168 PD) is a condition characterized by systemic low-grade inflammation that could increase the productio

169 Obesity promotes a state of low-grade inflammation that exacerbates chronic inflamma

170 Obesity is associated with low-grade inflammation that leads to insulin resistance

171 and glucose metabolism, satiety, and chronic low-grade inflammation to contribute to obesity and type

172 Chronic low-grade inflammation, a hallmark of obesity, involves

173 nd type 2 diabetes (T2D) are associated with low-grade inflammation, activation of immune cells, and

174 metabolic tissues, and contribute to chronic low-grade inflammation, and mediate insulin resistance a

175 Obesity is associated with chronic low-grade inflammation, and metabolic regulators linking

176 poproteins represent causal risk factors for low-grade inflammation, ASCVD, and all-cause mortality.

177 Obesity, through low-grade inflammation, can drive insulin resistance and

178 rotein (CRP), which is a sensitive marker of low-grade inflammation, in a large European population (

179 fully adjusted model that included age, BMI, low-grade inflammation, lifestyle factors, and morbiditi

180 y an epithelial cell dysfunction, leading to low-grade inflammation, macrophage recruitment, and coll

181 an atypical, metabolically induced, chronic low-grade inflammation, plays an important role in the d

182 This crosstalk results in chronic low-grade inflammation, thereby contributing to adiposit

183 A condition more common than IBD is low-grade inflammation, which correlates with altered gu

184 ly contributes to the development of chronic low-grade inflammation, which often accompanies obesity.

185 levels of metabolic endotoxemia and systemic low-grade inflammation, while transgenic conversion of t

186 ch lipoproteins are causally associated with low-grade inflammation.

187 issue function, adipocyte death, and chronic low-grade inflammation.

188 echanistic links between fructose intake and low-grade inflammation.

189 s of each monocyte subset in obesity-related low-grade inflammation.

190 which was strongly associated with systemic low-grade inflammation.

191 racterized by insulin resistance and chronic low-grade inflammation.

192 oxygen species (ROS) production and chronic low-grade inflammation.

193 increase in intestinal permeability leads to low-grade inflammation.

194 Histologic analysis showed absent to low-grade inflammatory infiltrates without cardiomyocyte

195 ficient in exosome secretion have a chronic, low-grade inflammatory phenotype characterized by elevat

196 is accompanied with a local amplification of low-grade inflammatory process implicating several pathw

197 whether TSPO imaging can accurately capture low-grade inflammatory processes such as those present i

198 syndrome (MetS) is associated with a chronic low-grade inflammatory state and may be affected by the

199 gard the effect of metabolic alterations and low-grade inflammatory state, clinically observed in obe

200 m those that arise as a result of short-term low-grade interactions with noxious thermal, chemical, o

201 levels in patients with active FPIES suggest low-grade intestinal mast cell activation or increased m

202 inous neoplasms (IPMN), 2 adenocarcinomas, 1 low-grade intraepithelial pancreatic neoplasia, and 1 ca

203 aniridia who experienced chronic, recurrent low-grade intraocular inflammation and irritation for mo

204 ve endophthalmitis in any case of recurrent, low-grade intraocular inflammation.

205 ing between aseptic prosthesis loosening and low-grade joint infection.

206 r the discrimination of aseptic loosening vs low-grade joint infection.

207 -risk types were found in 19 samples (50%; 1 low-grade lesion and 18 high-grade lesions), and only hi

208 or another site in the lungs than those with low-grade lesions (P = 0.03).

209 HPV-types were present in 11 samples (29%; 2 low-grade lesions and 9 high-grade lesions), low-risk an

210 itu might lead to a reclassification of some low-grade lesions as non-cancerous entities.

211 With the exception of 1 biopsy, all low-grade lesions were p16INK4a-negative, whereas 25 of

212 HPV-types were present in 8 samples (21%; 3 low-grade lesions, 4 high-grade lesion, and 1 cancer-con

213 the histologic and molecular features of the low-grade lesions.

214 The overall evidence provides a low-grade level of the evidence supporting the efficacy

215 The drivers of neoplasia within low-grade luminal breast cancers remain undelineated.

216 sociations with tumour morphology within the low-grade luminal breast cancers, tubular and lobular (p

217 at BUB1, BUB3, and CDC42 are key kinases for low-grade luminal tumours whereas BUB1B and CDC2 kinases

218 e cohort used for MIPI-b development (German Low-Grade Lymphoma Study Group [GLSG] 1996 and GLSG2000)

219 munochemotherapy (151 patients from a German Low-Grade Lymphoma Study Group [GLSG] trial and 107 pati

220 Bronchoscopy with biopsy revealed a low-grade lymphoma with the following immunophenotype: C

221 mation was lower than that observed in other low-grade lymphomas.

222 are variant of biologically indeterminate or low-grade malignant melanocytic tumors in which the mole

223 n electrochemically active alloys to harvest low-grade mechanical energies from various low-frequency

224 toma (SN-HPC) is an uncommon, site-specific, low-grade mesenchymal neoplasm of probable perivascular

225 Lymphangioleiomyomatosis (LAM) is a rare, low-grade, metastasizing neoplasm that arises from an un

226 d disturbances in the intestinal microbiota; low-grade mucosal inflammation, immune activation, and a

227 in the olaparib and placebo groups included low-grade nausea (24 [75%] of 32 patients vs two [40%] o

228 The most frequent adverse events were low-grade nausea, vomiting, and diarrhea.

229 m by which subtle myelin abnormalities cause low-grade neuroinflammation and catatonic behavior.

230 mall lymphocytic lymphomas (CLL/SLLs), and 1 low-grade NHL not otherwise specified.

231 inary data seem to confirm their activity on low-grade NHL.

232 d that the adult mammalian heart undergoes a low grade of cardiomyocyte turnover.

233 wide range of environmental conditions using low grade or solar heating as a supplementary energy sou

234 tification accuracy and are unable to detect low-grade or diffuse infiltrating gliomas (DIGs).

235 eduled visits (50%) compared with those with low-grade or high-grade orthodema (52% and 68%, respecti

236 reast reconstruction are done for widespread low-grade or intermediate-grade ductal carcinoma in situ

237 employed commercially to recover metals from low grade ores, but the production efficiency remains to

238 atients had high-grade orthodema and 35% had low-grade orthodema.

239 sed on proteinuria (Absent(A) <150 mg/day or low-grade(P)150 mg-1 g/day) and blood pressure (Normoten

240 romotes both the initiation and expansion of low-grade pancreatic intraepithelial neoplasia (PanINs),

241 tor receptor 3 (FGFR3) occur in up to 80% of low-grade papillary urothelial carcinoma of the bladder

242 keratinocyte clones, derived from cells of a low-grade premalignant lesion naturally infected with th

243 Patients with untreated low-volume low-grade prostate cancer (clinical stage T2a or lower;

244 Low-grade proteinuria and systolic hypertension (SHT) ar

245 ing, cigarette smoke, inactivity, persistent low-grade pulmonary and systemic inflammation) and add t

246 oth (CMT) neuropathy, have demonstrated that low-grade secondary inflammation implicating phagocytosi

247 t-Marie-Tooth neuropathy have indicated that low-grade secondary inflammation involving phagocytosing

248 tissue from patients with high grade but not low grade serous ovarian cancer.

249 therapy exists for patients with metastatic low-grade serous (LGS) ovarian cancers.

250 pes of ovarian cancer, including clear cell, low-grade serous and endometrioid carcinomas.

251 al of clear cell carcinoma (Arid1a, Pik3ca), low-grade serous carcinoma (Braf), endometrioid (Ctnnb1)

252 Women age < 35 years with low-grade serous carcinoma and those with persistent dis

253 ed chemotherapy in women with stage II to IV low-grade serous carcinoma of the ovary or peritoneum.

254 performed among patients with stage II to IV low-grade serous carcinoma treated with primary surgery

255 Conclusion Women with stage II to IV low-grade serous carcinoma who received HMT after primar

256 rline ovarian tumors (BOT) can progress into low-grade serous carcinomas and have relatively indolent

257 tors separated endometrioid, clear cell, and low-grade serous carcinomas from high-grade serous and m

258 clear cell, and seromucinous carcinomas; ii) low-grade serous carcinomas; and iii) mucinous carcinoma

259 Low-grade serous ovarian carcinomas (LGSC) are associate

260 istically nonsignificantly increased risk of low-grade serous tumors (pOR = 1.48, 95% CI: 0.92, 2.38)

261 patients with chronic pancreatitis, 13 with low-grade side-branch IPMNs, and 15 patients with PDAC;

262 The majority of the missed cases had low-grade smears.

263 sults, and also in HIV-infected women with a low-grade squamous intraepithelial lesion (LSIL; benchma

264 amous cells of undetermined significance and low-grade squamous intraepithelial lesion) and CIN1+ was

265 lls of undetermined significance (ASC-US) or low-grade squamous intraepithelial lesions (LSIL) who we

266 lls of undetermined significance (ASC-US) or low-grade squamous intraepithelial lesions (LSIL) who we

267 but this proportion increased through ASCUS, low-grade squamous intraepithelial lesions, CIN1, and CI

268 high-grade squamous intraepithelial lesion, low-grade squamous intraepithelial lesions, or high-grad

269 Chronic, low grade, sterile inflammation frequently accompanies a

270 RP, if any, thereby excluding this marker of low-grade systemic inflammation as a possible explanatio

271 The association between number of teeth and low-grade systemic inflammation deserves consideration w

272 rbated IL-6 production in this depot and the low-grade systemic inflammation typically associated wit

273 Obesity is characterized by chronic low-grade, systemic inflammation, altered gut microbiota

274 o design high efficient OHEs for recovery of low grade thermal energy to work or electricity.

275 However, abundant heat from these low-grade thermal streams cannot be harvested readily be

276 oangiogenic myeloid cells, and prevention of low-grade to high-grade transition.

277 witch necessary for malignant progression of low-grade to high-grade tumors.

278 ical examination but had retinal vasculitis, low-grade to moderate vitritis, and hypocyanescent lesio

279 Combination therapy was associated with low-grade toxic effects.

280 ancer, although it resulted in more frequent low-grade toxicities.

281 r indolent behavior, can be distinguished: a low-grade triple-negative breast neoplasia family, which

282 Among the less common low-grade triple-negative lesions, two large subgroups,

283 report a detailed study of a musician with a low-grade tumor in the right temporal lobe.

284 Results Low-grade tumors (grade 0, 1, or 2) demonstrated a favor

285 tem, individuals with NF2 typically manifest low-grade tumors affecting the cranial nerves (vestibula

286 ue) and high-grade tumors from other tissue (low-grade tumors or benign tissue).

287 ratio to discriminate between high-grade and low-grade tumors to be 2.21.

288 tric brain tumors display higher CBF than do low-grade tumors, and they may be accurately graded by u

289 mpared with levels in normal endometrium and low-grade tumors.

290 ble prognosticators, such as ER-positive and low-grade tumors.

291 d) high- or intermediate-grade tumors versus low-grade tumors.

292 d by decreased visual acuity, mild pain, and low-grade uveitis several weeks or months after intraocu

293 imens into normal versus lesional tissue and low-grade versus high-grade tumors with 100% accuracy.

294 Conversion of low-grade waste heat into electricity is an important en

295 (thermocells) are of interest for harvesting low-grade waste thermal energy because of their potentia

296 f merit (or ZT), which can directly converts low-grade wasted heat (400 to 500 K) into electricity, h

297 such as histologic grade (G1, G2, or G3, for low-grade [well differentiated], intermediate-grade [mod

298 ed patients aged 18 years or older who had a low-grade (WHO grade II) glioma (astrocytoma, oligoastro

299 ed patients aged 18 years or older who had a low-grade (WHO grade II) glioma (astrocytoma, oligoastro

300 Results Low-grade (WHO grade II) glioma showed areas with increa