ライフサイエンスコーパス: low-protein

1 ] carbohydrate, and 0.4 g/[kg/h] lipid) or a low protein (0.4 g/[kg/h] protein, 2.2 g/[kg/h] carbohyd

2 Canada goose goslings fed low-protein (10%) diets were on average 44% lighter in b

3 diets containing 5% of energy from protein (low protein), 15% (normal protein), or 25% (high protein

4 state (0-3 h), who were fed half-hourly with low-protein (2% of energy, 3-6 h) and isoenergetic highe

5 or 24 h each and included the following: (1) low protein (3%), (2) standard (50% carbohydrate, 20% pr

6 A low-protein (6.25 g) mixed macronutrient beverage can be

7 n (7.21 +/- 3.08 MJ/d) condition than in the low-protein (9.33 +/- 3.52 MJ/d) and normal-protein (9.6

8 had only a single peptide match, indicating low protein abundance and/or false-positive peptide matc

9 in thousands of individuals with high versus low protein abundance.

10 cient to target firefly luciferase (LUC) for low protein accumulation equivalent to that observed pre

11 n the necessary cis-acting element to confer low protein accumulation onto LUC, while a fusion protei

12 ts original host, R. eutropha, revealed only low protein amounts.

13 oth loss and the proportion of patients with low protein and caloric intake (P = 0.02 and 0.01, respe

14 d pressure parameters in male offspring from low protein and control-fed dams measured simultaneously

15 with low N content and artificial diets with low protein and high carbohydrate content.

16 g mainly of poorly bioavailable lignin, with low protein and lipid content, the carbohydrates from fa

17 balance to Deltanitrogen intake between the low-protein and higher-protein periods, was 0.68 +/- 0.0

18 The benefits of low protein (and phosphorus) diets include the ameliorat

19 Low-protein (and phosphorus) diets can also ameliorate u

20 r metabolic diseases who were treated with a low-protein, and consequently, low-lactose diet.

21 blood pressure and heart rate in control and low protein animals, ruling out an effect of enhanced pr

22 onitis, upper gastrointestinal bleeding, and low-protein ascites with associated poor liver function.

23 This suggests that low protein availability in fruits influences current le

24 id not inhibit the P450 3A4 isozyme, and had low protein binding (18.22% for 23) and a desirable log

25 ife of 5-7 h in three species and moderately low protein binding in both mouse (69%) and human (63%)

26 microbiological potency, low clearance, and low protein binding that can result in lower efficacious

27 ny favorable pharmacological traits, such as low protein binding, minimal human serum effect on anti-

28 was discovered for designing molecules with low protein binding.

29 ith high dependence on tubular secretion but low protein-binding affinity.

30 sually involves efficient manufacturing, but low protein bioavailability resulting in higher doses co

31 Persistently low protein C levels are related to poor prognosis.

32 Low protein C levels were associated with prolongation o

33 High thrombomodulin and low protein C levels were significantly associated with

34 ociated with high soluble thrombomodulin and low protein C levels.

35 ta demonstrate that C. parvum transcripts of low protein-coding potential are selectively delivered i

36 dentified a panel of RNA transcripts of very low protein-coding potential in C. parvum.

37 The low protein-coding potential of meRNAs suggests that man

38 ntransparent samples, to use samples of very low protein concentration (< or = 0.3 mg/ml), and to stu

39 le-resonance assignment strategy tailored to low protein concentration (0.2 mM) and poor chemical shi

40 Folding of equine cytochrome c at a low protein concentration (26 microM) eliminated a slow

41 The first occurs at low protein concentration and is characterized by an inc

42 presented that facilitates NMR studies at a low protein concentration of approximately 20 micoM.

43 the site-specific torsional relaxation at a low protein concentration under physiological conditions

44 For low protein concentration, no direct method is currently

45 At low protein concentration, the NBDs display an initial s

46 ngle stable complex with the gRNA gA6[14] at low protein concentration, while at higher protein conce

47 degradation rate compared to those formed at low protein concentration.

48 neutral pH reversibility is observed only at low protein concentrations (<0.5 mg/ml).

49 ains its structure and function at extremely low protein concentrations (<10(-)12 M).

50 nanoparticles conjugated with antibodies at low protein concentrations (<40 mug/mL) display self-ass

51 and (15)N-{(1)H} NOE data were collected at low protein concentrations (<or=100 microM) and at two f

52 zing system, especially in studies done with low protein concentrations (0.1 microM), and at elevated

53 ta protein fibrils that formed at relatively low protein concentrations and exhibited remarkably high

54 d of NF-LH transition to an isotropic gel at low protein concentrations as a function of increasing m

55 Low protein concentrations in these experiments prevente

56 A lag phase at low protein concentrations indicates that fibril formati

57 Protection at low protein concentrations is more pronounced and more e

58 unlabelled protein, it is not limited to the low protein concentrations normally required for single-

59 of the NCM complex assembled in vivo and at low protein concentrations so that self-association of t

60 At low protein concentrations the structural rearrangements

61 41 +/- 3 ns when measured at low proton and low protein concentrations to minimize protein aggregati

62 The fluorescence enhancement at low protein concentrations was buffer-dependent and requ

63 Dimerization constants calculated at low protein concentrations were 265, 750, 1212, and 7879

64 ibril formation is accelerated at relatively low protein concentrations, and the ability to seed the

65 At low protein concentrations, ATP promotes dimerization of

66 cular autoreduction process is implicated at low protein concentrations, but oligomerization decrease

67 ects with origin DNA and ssDNA especially at low protein concentrations, but only half were defective

68 d with Ni2+ and Cu2+ binding to H144*UreE at low protein concentrations, consistent with binding to s

69 uring static light scattering experiments at low protein concentrations, frequently the protein is as

70 The first phase, occurring at low protein concentrations, involves both electrostatic

71 By contrast, REGbetaDeltai exhibited, at low protein concentrations, reduced proteasome activatio

72 e-molecule screening in human serum at ultra-low protein concentrations.

73 ingle-stranded DNA and origin DNA binding at low protein concentrations.

74 rmal single-stranded DNA binding activity at low protein concentrations.

75 inge model for noncooperative HMG binding at low protein concentrations.

76 lding is reversible and two-state for EP3 at low protein concentrations.

77 his same Bcd domain plays a positive role at low protein concentrations.

78 lysis of MIF after chemical cross-linking at low protein concentrations.

79 high protein concentrations and monomers at low protein concentrations.

80 nstants, and for performing experiments with low protein concentrations.

81 At relatively low-protein concentrations, XPA formed a complex with DN

82 terleukin (IL)-8 was selectively released at low-protein concentrations.

83 -based reporter displacement assay with very low protein consumption was developed to enable the larg

84 ddition of amaranth flour, with a relatively low protein content (16.45%), did not mitigate acrylamid

85 , it is shown that they are small cells with low protein content suggestive of G0.

86 with 79% protein and a starchy product with low protein content.

87 omposed of neutral and acidic sugars, with a low protein content.

88 DDGS extracts had relatively low protein contents (14-44.9%, w/w), regardless of the

89 sponse to androgen exposure, were grown in a low protein-defined media under androgen-stimulated (A+)

90 otein aggregates have so far been limited to low protein densities in either vesicular or bilayer mor

91 are not pairwise additive, for sufficiently low protein density, thermodynamic properties depend onl

92 es improves agreement with experiment when a low protein dielectric constant is assumed; (3) despite

93 are more accurate than those computed with a low protein dielectric constant; (4) the pKa shifts in r

94 Patients were prescribed a very low protein diet (0.3 g/kg) plus supplemental essential

95 - 3.9 micromol kg-1 h-1, HP + W) but not the low protein diet (51.1 +/- 5.9 micromol kg-1 h-1, LP + G

96 protein diet (18% casein; NPD) or isocaloric low protein diet (9% casein; LPD) restricted to one ovul

97 We studied maternal low protein diet (LPD) during pregnancy/lactation in mic

98 Maternal low protein diet (LPD) fed during only the preimplantati

99 The impaired NO release in the MA of low protein diet dams was not accompanied by reduced eNO

100 We have shown maternal low protein diet exclusively during mouse preimplantatio

101 oduced significantly less weight gain in the low protein diet group (3.16 kg; 95% CI, 1.88-4.44 kg) c

102 The effect of maternal low protein diet in pregnancy on the function of offspri

103 n used to model the effects of exposure to a low protein diet in utero on adult blood pressure.

104 With the low protein diet leucine oxidation rates during feeding

105 In maternal low protein diet rat models of programming, administrati

106 after infection, with those on the high fat/low protein diet showing 30% survival at 8 days, vs. alm

107 f the total visceral CO2 production during a low protein diet, this increase did not compensate entir

108 s affecting the urea cycle while consuming a low protein diet.

109 idney development and function by a maternal low protein diet.

110 did not increase during overfeeding with the low protein diet.

111 s were instructed and their adherence to the low-protein diet (0.6 g/kg of body weight per day) was e

112 of ketoanalogue-supplemented vegetarian very low-protein diet (KD) compared with conventional low-pro

113 we revealed that newborns of dams exposed to low-protein diet (LP0.5) throughout pregnancy exhibited

114 NPY-Y2R system is also activated by maternal low-protein diet (LPD) and linked to obesity in offsprin

115 protein diet (KD) compared with conventional low-protein diet (LPD).

116 ites in CRF may be a beneficial adjunct to a low-protein diet (LPD).

117 ng sheep were fed either a control (n=15) or low-protein diet (n=16, 17 vs. 8.7 g crude protein/MJ me

118 diet plus resistance training (n = 14) or a low-protein diet alone (n = 12) for 12 weeks.

119 her, these findings indicate that a maternal low-protein diet alters microRNA and mTOR expression in

120 ary calcium decreased significantly with the low-protein diet and increased significantly with the hi

121 to be effective against the catabolism of a low-protein diet and uremia in patients with renal failu

122 ion of renal disease in patients receiving a low-protein diet compared with patients receiving a usua

123 nd by day 14 1.6-2.7 times higher during the low-protein diet compared with the medium-protein diet.

124 In contrast, offspring of a low-protein diet displayed no deficits in operant learni

125 Offspring of males fed a low-protein diet exhibited elevated hepatic expression o

126 In the low-protein diet group whose excess energy was fat, the

127 SleepEE was unchanged by overfeeding in the low-protein diet group, and baseline surface area predic

128 tudies demonstrated that in mouse dams fed a low-protein diet hepatic expression of FOXA2 and FOXA3 m

129 hancing autophagy by exposure to a prolonged low-protein diet improved cardiac function in Python mic

130 e reported previously that rats exposed to a low-protein diet in utero and postnatal catch-up growth

131 usly, we demonstrated that rats exposed to a low-protein diet in utero that underwent postnatal catch

132 we hypothesized that exposure to a maternal low-protein diet increases glomerular Ang II AT1 recepto

133 promoted impulsivity, whereas exposure to a low-protein diet led to marked inattention.

134 of support for protein leverage effects on a low-protein diet may stem from the fact that protein int

135 tary manipulations in the daughters in a rat low-protein diet model.

136 They were randomly assigned to a low-protein diet plus resistance training (n = 14) or a

137 five studies of nondiabetic renal disease, a low-protein diet significantly reduced the risk for rena

138 es of insulin-dependent diabetes mellitus, a low-protein diet significantly slowed the increase in ur

139 hase protein intake was 13% higher after the low-protein diet than after the high-protein diet (253 +

140 Pigs fed a low-protein diet that were not vaccinated with SLT-IIe(E

141 Fractional calcium absorption after the low-protein diet was 0.19+/-0.03, which was significantl

142 ion model, in which animals are exposed to a low-protein diet while in utero and then are cross-foste

143 In addition, after the low-protein diet, food preferences for savory high-prote

144 nce of one or more cofactors, particularly a low-protein diet, thiamine deficiency, alcoholism, and h

145 day 4 of the high-protein compared with the low-protein diet.

146 ts of soy-protein intake resemble those of a low-protein diet.

147 s the ability of the patient to respond to a low-protein diet.

148 econdary hyperparathyroidism on day 4 of the low-protein diet.

149 rs in the initial IMCD (IMCD1) of rats fed a low-protein diet.

150 MA-B (P = 0.004) in participants consuming a low-protein diet.

151 ihydroxyvitamin D] in subjects consuming the low-protein diet.

152 changes in fat distribution in response to a low-protein diet.

153 dard diet; a high-carbohydrate, low-fat, and low-protein diet; or a low-carbohydrate, high-fat, and h

154 47, respectively) but not in subjects in the low-protein-diet group (P = 0.384 and 0.078, respectivel

155 reover, there is no evidence that the use of low protein diets (LPD) in the predialysis period result

156 ng most pronounced in female animals fed the low protein diets and the effects of protein reduction b

157 Low protein diets are commonly prescribed for patients w

158 core were significant only in animals on the low protein diets.

159 When low-protein diets are prescribed, patients should be clo

160 Low-protein diets cause a urinary concentrating defect i

161 Canada and snow goose goslings fed low-protein diets had reduced growth rates of the tarsus

162 searched for studies examining the effect of low-protein diets in humans with chronic renal disease.

163 muscle wasting, which may be exacerbated by low-protein diets prescribed to delay disease progressio

164 In rats with renal disease, low-protein diets slow the decline in renal function, hi

165 goose goslings were unable to survive on the low-protein diets, and those fed high- or medium-protein

166 Studies have compared high- and low-protein diets, but there are few data on carbohydrat

167 s by the gut compared with animals receiving low-protein diets.

168 construed as an argument against the use of low-protein diets.

169 due to maternal malnutrition via high-fat or low-protein diets.

170 ructural size to captive-reared goslings fed low-protein diets.

171 n Association for the Study of Diabetes, and low-protein diets.

172 Very low protein-DNA ratio was required if incubations were c

173 At a low-protein dose and over a similar range of plasma CEA,

174 primary tumor of unknown origin) underwent a low-protein-dose diagnostic study (0.3-2.6 mg of protein

175 to be fed an MFGM-supplemented, low-energy, low-protein experimental formula (EF) or a standard form

176 od consistently shows increased pressures in low protein exposed rodent offspring compared to control

177 As adults, the low-protein-exposed sheep had reduced glomerular number

178 HR for negative, 1.57; comparison of high v low protein expression HR, P = .049).

179 on of BCL2 transcripts commensurate with the low protein expression level.

180 M motif confer peroxisomal targeting even at low protein expression levels.

181 s that is capable of detecting both high and low protein expression levels.

182 tumors, and colorectal cancer patients with low protein expression of SIRT1 have a poor prognosis.

183 Low protein expression of the antioxidant PRDX2 gene was

184 Low protein expression, protein mislocalization, and red

185 in the apical membrane of initial IMCD from low-protein fed or hypercalcemic rats; (2) active urea r

186 Although low protein feeding increases the contribution of entera

187 We hypothesized that low protein feeding would lower the contribution of amin

188 suggests that degraded habitats with mostly low-protein forage may be able to support Canada gosling

189 Exposure to low-protein grand-maternal diet leads to decreased ovari

190 ight change was not independent from that of low protein in a multivariate model.

191 stinal calcium absorption explains, in part, low-protein-induced secondary hyperparathyroidism.

192 valuated the impact of habituation to either low protein intake (LOW PRO) or high protein intake (HIG

193 to derive a similar benefit from a maternal low protein intake as did GDM-exposed offspring.Overall,

194 the long-term developmental consequences of low protein intake in free-living populations remains li

195 In maintenance hemodialysis (MHD) patients, low protein intake is associated with protein-energy was

196 Low protein intake may have additional protective effect

197 with moderate to severe renal insufficiency, low protein intake may slow renal function decline.

198 It remains to be shown whether a relatively low protein intake would cause overeating or would be th

199 dation was substantially suppressed during a low protein intake.

200 ies examining 1) the effects of "high versus low" protein intake or 2) dietary protein's synergistic

201 Abdominal fat was highest with low protein intakes (<16% of energy), and midthigh fat w

202 ariable regression analysis assessed whether low protein intakes and the MST score were predictive of

203 Low protein intakes are unlikely to affect milk volume b

204 te the progression of renal disease and that low protein intakes have beneficial effects.

205 lta cells was unphosphorylated and exhibited low protein kinase activity.

206 be markedly decreased, suggesting that this low protein level produced the observed regulatory effec

207 the disulfide bond linkage patterns, at very low protein levels (<0.5 nmol), in two cysteine-rich pol

208 s might be expected, all the meals contained low protein levels (0.67-3.15 g/100 g) with the highest

209 strocytes and C6 cells: astrocytes expressed low protein levels of Hspa5 compared to C6 cells but acc

210 expression in melanocytes is inhibitory with low protein levels present in surviving cells, suggestin

211 observed Bicoid binding despite vanishingly low protein levels.

212 owever, neuronal SNAREs do promote fusion at low protein/lipid ratios when triggered by higher concen

213 We hypothesized that long-term low protein, low calorie intake and endurance exercise a

214 BMI was lower in the low-protein, low-calorie diet (21.3 +/- 3.1) and runner

215 x hormone-binding globulin was higher in the low-protein, low-calorie diet and runner groups than in

216 ed adiposity, and long-term consumption of a low-protein, low-calorie diet are associated with low pl

217 21 sedentary subjects, who had been eating a low-protein, low-calorie diet for 4.4 +/- 2.8 y (x +/- S

218 I to IGF binding protein 3 were lower in the low-protein, low-calorie diet group (139 +/- 37 ng/mL an

219 l C. pneumoniae challenge, C57BL/6 mice on a low-protein/low-antioxidant diet, but not C57BL/6 mice o

220 and Fgf21-KO mice were placed on control and low protein (LP) diets to assess changes in energy expen

221 al lysine use by the PDV was not affected by low protein (LP) feeding (HP, 213 micromol/kg per h; LP,

222 d either a control (C, 18% casein n = 22) or low protein (LP, 9% casein n = 14) diet.

223 Young progenies in a rat model of maternal low-protein (LP) diet are normoglycemic despite collapse

224 haracterizing progeny of pregnant dams fed a low-protein (LP) diet.

225 was increased 10-fold in mice and rats fed a low-protein (LP) diet.

226 Both maternal malnutrition [notably a low-protein (LP) diet] and stress/glucocorticoid exposur

227 otein-energy malnutrition induced in mice by low-protein (LP) feeding has a detrimental impact on CD8

228 study, we demonstrated that splenocytes from low-protein (LP) guinea pigs vaccinated 6 weeks previous

229 Here we use our maternal low-protein (LP) rat model to determine the effect of ea

230 Conversely, low protein males were relatively hypertensive at 8 week

231 he highest cholesterol-to-protein ratio have low protein mass.

232 re the concentration of SCN(-) is relatively low, proteins may be the principal initial targets of HO

233 us uptake was greatly diminished in maternal low protein (MLP) livers, accounting for a major fractio

234 that is challenged by the tight stacking and low protein mobility in grana.

235 We used an established maternal low-protein model where animals are born small but under

236 EE during fasting, a smaller EE response to low-protein overfeeding, and a larger response to high-c

237 Under conditions of low protein oxidation (zero to three oxygen atoms added

238 new signal that manifests in late flowering, low protein oxidation during light stress, and enhanced

239 restriction during lactation (the postnatal low-protein [PLP] group).

240 sociated with diminished mRNA expression and low protein production.

241 Selecting soybean lines with low protein, protein plus oil, calcium, manganese, and b

242 sects encounter challenging diets containing low protein quantities, recalcitrant carbohydrate source

243 Furthermore, isolated low Protein S may be causally associated with hepatic ar

244 highest degree of resistance to mAbs showed low protein stability and high local dynamic motions.

245 hr-31 is a key determinant of the relatively low protein stability, thereby promoting apoLp-III to in

246 A low protein state also increased preferences for savory

247 areas (orbitofrontal cortex, striatum) in a low-protein state than in a high-protein state.

248 objective was to investigate the effect of a low protein status compared with a high protein status o

249 arm muscle area indicate muscle wasting and low protein stores.

250 mbranes and vesicles, we demonstrate that at low protein surface densities, binding of N-BAR domain p

251 Cells with low glucose uptake capacity and low protein synthesis rates were less ligand-sensitive,

252 ed by active niche signaling integration and low protein translation capacity.

253 reversibility of the inhibition and the very low protein turnover rate observed for the enzyme are pa

254 High total fruit and low-protein vegetable intakes were associated with a low

255 r adequate protein (AP; 18% protein) or very low protein (VLP; 2% protein) in an established murine m

256 ng either adequate protein (AP; 18%) or very low protein (VLP; 2%) in a mouse model.

257 f diabetes may benefit more from consuming a low-protein weight-loss diet in improving insulin resist

258 However, the low protein yields associated with these small samples p