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1 rine model of colitis relative to mice fed a low salt diet.
2 tubule isolated from Dahl S and R rats fed a low salt diet.
3 olitis on either a high salt diet (HSD) or a low salt diet.
4 relative to corresponding control rats on a low salt diet.
5 and activity of ENaC compared with feeding a low-salt diet.
6 develops albuminuria early in life even on a low-salt diet.
7 recessive S strain; the population was fed a low-salt diet.
8 different from that seen in dogs ingesting a low-salt diet.
9 mmHg; P < 0.05) on the high-salt than on the low-salt diet.
10 mmHg; P < 0.01) on the high-salt than on the low-salt diet.
11 nd CsA + L-NAME (3.5 mg/kg) for 28 days on a low-salt diet.
12 1 mg/kg), or CsA+RAPA for 28 days while on a low-salt diet.
13 nt and wild-type mice on control or high- or low-salt diets.
14 s who were on carefully controlled high- and low-salt diets.
20 ved mainly in preparations from rabbits on a low-salt diet and were completely inhibited by either a
25 with controls, Prss8(KO) mice fed regular or low-salt diets exhibited significantly reduced PDamil in
30 ism, the adverse effects of short-term, very-low-salt diets in high-risk subjects suggest that contin
33 either a high salt diet containing 8 % or a low salt diet of 0.4 % sodium chloride for a period of 4
34 ncentration and the effects of a high- and a low-salt diet on plasma renin concentration were similar
35 s NCC and SPAK phosphorylation promoted by a low-salt diet or AngII infusion, suggesting that AngII a
40 low in healthy human volunteers studied on a low salt diet to activate the renin system than did ACE
41 condition of teeth; little or no exercise; a low-salt diet; trouble chewing meat; self-reported prote
43 e on both low- and high-salt diets, and on a low-salt diet VENIRKO mice showed insulin resistance.
46 alt (400 mEq/d) diet, and the third during a low-salt diet, whereas in the other 3 subjects, the sequ
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