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1 rine model of colitis relative to mice fed a low salt diet.
2 tubule isolated from Dahl S and R rats fed a low salt diet.
3 olitis on either a high salt diet (HSD) or a low salt diet.
4  relative to corresponding control rats on a low salt diet.
5 and activity of ENaC compared with feeding a low-salt diet.
6 develops albuminuria early in life even on a low-salt diet.
7 recessive S strain; the population was fed a low-salt diet.
8 different from that seen in dogs ingesting a low-salt diet.
9 mmHg; P < 0.05) on the high-salt than on the low-salt diet.
10 mmHg; P < 0.01) on the high-salt than on the low-salt diet.
11 nd CsA + L-NAME (3.5 mg/kg) for 28 days on a low-salt diet.
12 1 mg/kg), or CsA+RAPA for 28 days while on a low-salt diet.
13 nt and wild-type mice on control or high- or low-salt diets.
14 s who were on carefully controlled high- and low-salt diets.
15                     Mice were administered a low-salt diet (0.01% sodium) for 1 wk followed by CsA tr
16 ion exchange greater than that produced by a low-salt diet alone.
17                         Prss8(KO) mice fed a low-salt diet also exhibited sodium loss through feces a
18 anagement of Meniere's disease can involve a low-salt diet and a diuretic.
19                                  Recommended low-salt diet and triple antihypertensive drug regimens
20 ved mainly in preparations from rabbits on a low-salt diet and were completely inhibited by either a
21                    All patients maintained a low-salt diet and were examined at 1 month after the pro
22  peripheral edema, treated with diuretics, a low-salt diet, and fluid restriction.
23 on of renin-producing cells in response to a low-salt diet combined with an ACE inhibitor.
24 15 mg/kg/day) for 5 and 7 weeks to rats on a low salt diet (CSA group).
25 with controls, Prss8(KO) mice fed regular or low-salt diets exhibited significantly reduced PDamil in
26                               Feeding rats a low-salt diet for 14 days induced a significant increase
27                                              Low-salt diet greatly increased the expression of both C
28                Control mice fed a regular or low-salt diet had a significantly higher amiloride-sensi
29                                Compared with low-salt diet, HS significantly (P < 0.005) increased re
30 ism, the adverse effects of short-term, very-low-salt diets in high-risk subjects suggest that contin
31               However, Scnn1a(KO) mice fed a low-salt diet lost significant amounts of sodium in thei
32                               Rats placed on low salt diet (LSD) or normal salt diet (NSD) were treat
33  either a high salt diet containing 8 % or a low salt diet of 0.4 % sodium chloride for a period of 4
34 ncentration and the effects of a high- and a low-salt diet on plasma renin concentration were similar
35 s NCC and SPAK phosphorylation promoted by a low-salt diet or AngII infusion, suggesting that AngII a
36                                              Low-salt diet or infusion of AngII for 4 d induced phosp
37                      By every criterion, the low-salt diet reduced expression of guanylin to 30%-40%
38                                              Low salt diet resulted in a strong increase in beta and
39 and p-Ncc protein that did not change with a low-salt diet that increased p-Spak.
40 low in healthy human volunteers studied on a low salt diet to activate the renin system than did ACE
41 condition of teeth; little or no exercise; a low-salt diet; trouble chewing meat; self-reported prote
42          Sprague-Dawley rats maintained on a low-salt diet underwent uninephrectomy and contralateral
43 e on both low- and high-salt diets, and on a low-salt diet VENIRKO mice showed insulin resistance.
44                The aldosterone response to a low-salt diet was blunted in Oat3-null mice, but baselin
45                          Cyp4a10-/- mice fed low-salt diets were normotensive but became hypertensive
46 alt (400 mEq/d) diet, and the third during a low-salt diet, whereas in the other 3 subjects, the sequ

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