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1 an blood pressure variability by oscillatory lower body negative pressure.
2 , during decreased cardiac filling by use of lower-body negative pressure (-15 and -30 mm Hg), and af
3 P < 0.001) plasma norepinephrine response to lower body negative pressure (3.0 +/- 0.3 vs. 2.0 +/- 0.
7 erformed while preload was manipulated using lower body negative pressure and rapid saline infusion t
8 onary capillary wedge pressure and SV during lower body negative pressure and saline loading in 7 men
9 ng curves were constructed during decreases (lower body negative pressure) and increases (saline infu
10 ring maneuvers, baseline, with two stages of lower body negative pressure, and repeat baseline with t
11 hocardiography at baseline, -15 and -30 mmHg lower-body negative pressure, and 15 and 30 ml kg(-1) sa
12 r mass (by echocardiography) at rest, during lower-body negative pressure, and after saline infusion
13 eline, during decreased cardiac filling with lower-body negative pressure, and increased filling with
16 slope of the line relating SV to PCWP during lower-body negative pressure characterized the steepness
18 sured for 10 min at rest and during 5 min of lower body negative pressure (LBNP) at -10 and -40 mmHg
19 -induced sympathetic activation, produced by lower body negative pressure (LBNP) at -40 mmHg, on cere
20 n sympathetic nerve activity (SNA) evoked by lower body negative pressure (LBNP) at rest and during m
21 (HG) and disengagement of baroreflexes with lower body negative pressure (LBNP) can engage the sympa
23 humans, reflex sympathetic activation during lower body negative pressure (LBNP) evoked decreases in
24 bnormal forearm vasodilator responses during lower body negative pressure (LBNP) in 21 non-obstructiv
25 rded in humans (n = 20) during a progressive lower body negative pressure (LBNP) protocol designed to
26 nse to either carotid sinus massage (CSM) or lower body negative pressure (LBNP) received Paxil (20 m
29 compared when a cold pressor test (CPT) and lower body negative pressure (LBNP) were superimposed up
33 pressure were recorded during nonhypotensive lower body negative pressure (LBNP; -10 mm Hg) and nonhy
35 easurements were also obtained during graded lower body negative pressure (LBNP; activates baroreflex
36 d while central blood volume was reduced via lower-body negative pressure (LBNP) during normothermia,
37 tic stress achieved by stepwise increases in lower-body negative pressure (LBNP) in 14 healthy young
38 FVR) during application of -20 and -30 mm Hg lower-body negative pressure (LBNP) in 24 patients with
40 4 years) participants underwent progressive lower-body negative pressure (LBNP) until pre-syncope; e
41 hostatic tolerance was assessed using graded lower-body negative pressure (LBNP) until the onset of s
42 nary capillary wedge pressure (PCWP), during lower-body negative pressure (LBNP) while subjects are n
45 lacebo (0.9% NaCl) on endogenously mediated (lower body negative pressure [LBNP]) and exogenously med
46 tolerance to hypotensive hypovolemic stress (lower body negative pressure [LBNP]) in healthy human ma
47 ng 5 different loading conditions: baseline, lower-body negative pressures of -15 and -30 mm Hg, and
49 ing blood pressure fluctuations (oscillatory lower body negative pressure, OLBNP) across a range of f
50 med in a 2x2 fashion with the combination of lower body negative pressure or not (normovolemia), and
55 ood pressure was associated with 23% greater lower body negative pressure tolerance using an active i
57 in human volunteers by applying progressive lower body negative pressure (under two experimental con
60 did not differ when a cold pressor test and lower body negative pressure were superimposed upon heat
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