ライフサイエンスコーパス: lower esophageal sphincter

1 with 2 sensors located 5 and 20 cm above the lower esophageal sphincter).

2 ness of a new magnetic device to augment the lower esophageal sphincter.

3 destruction of the abdominal segment of the lower esophageal sphincter.

4 junction, and electrical stimulation of the lower esophageal sphincter.

5 ecorded 3, 6, 9, 12, 15, and 18 cm above the lower esophageal sphincter.

6 orable outcomes (66%) after treatment of the lower esophageal sphincter.

7 ry showed a high prevalence of a hypotensive lower esophageal sphincter (55%) and impaired esophageal

8 haracterized by incomplete relaxation of the lower esophageal sphincter and a loss of normal peristal

9 isted of one or more bundles entering at the lower esophageal sphincter and coursing to the forestoma

10 crete esophagogastric junction contributors (lower esophageal sphincter and crural diaphragm) during

11 high-resolution manometry to determine UES, lower esophageal sphincter, and intraesophageal pressure

12 Lower esophageal sphincter augmentation through injectio

13 They have an inhibitory effect on the lower esophageal sphincter but an excitatory effect on t

14 arditis was associated with deterioration of lower esophageal sphincter characteristics and increased

15 disease were compared between groups (i.e., lower esophageal sphincter characteristics, esophageal a

16 utcracker esophagus at 2 and 10 cm above the lower esophageal sphincter during wet swallow.

17 Botulinum toxin injection into the lower esophageal sphincter is an established therapy for

18 and botulinum toxin (BOTOX) injection of the lower esophageal sphincter largely have replaced cardiom

19 Separation of the lower esophageal sphincter (LES) and crural diaphragm wa

20 etween those who believed in a morphological lower esophageal sphincter (LES) and those who did not.

21 ction of the esophagus and relaxation of the lower esophageal sphincter (LES) are mediated by nitric

22 In contrast, lower esophageal sphincter (LES) circular muscle maintai

23 amine the physiologic signal transduction of lower esophageal sphincter (LES) circular muscle.

24 The lower esophageal sphincter (LES) in patients with gastro

25 he SCJ was closer to the upper border of the lower esophageal sphincter (LES) in subjects with a larg

26 The motor control of the lower esophageal sphincter (LES) is critical for normal

27 identified adrenergic receptor sites in the lower esophageal sphincter (LES) of animals and humans.

28 istal esophageal acid exposure and increased lower esophageal sphincter (LES) pressure and length bas

29 d regurgitation after therapy have low or no lower esophageal sphincter (LES) pressure.

30 Isolated lower esophageal sphincter (LES) relaxation associated w

31 ater in the pharynx leads to a long-duration lower esophageal sphincter (LES) relaxation that is an i

32 been proposed that nitrergic nerves mediate lower esophageal sphincter (LES) relaxation with intramu

33 On the contrary, during transient lower esophageal sphincter (LES) relaxation, longitudina

34 r/water discrimination seen during transient lower esophageal sphincter (LES) relaxation-associated r

35 for its antireflux action is that it reduces lower esophageal sphincter (LES) relaxation.

36 baclofen reduces the incidence of transient lower esophageal sphincter (LES) relaxations.

37 both completeness and propagation velocity, lower esophageal sphincter (LES) resting pressure, LES r

38 In cat spontaneous lower esophageal sphincter (LES), tone is maintained by

39 magnetic sphincter augmentation (MSA) on the lower esophageal sphincter (LES).

40 ngeal, and hyoid muscles, esophagus, and the lower esophageal sphincter (LES).

41 to reveal the anatomical counterpart of the lower esophageal sphincter (LES).

42 nts, consisting of 24-hour esophageal pH and lower esophageal sphincter manometry.

43 We have previously used the normal lower esophageal sphincter (N-LES) of human organ donors

44 Botulinum toxin injection into the lower esophageal sphincter of patients with achalasia re

45 nce of 5 cm above the proximal border of the lower esophageal sphincter (PBLES) as appropriate to the

46 sturbance classically manifests as a reduced lower esophageal sphincter pressure (LESP) and loss of d

47 rway resistance, 5 cm H2O/L/sec), adjustable lower esophageal sphincter pressure (LESP) and simulated

48 eal manometry studies revealed a decrease in lower esophageal sphincter pressure (LESP) from 37 +/- 1

49 No significant changes in lower esophageal sphincter pressure and esophageal peris

50 ophageal acid exposure, esophagitis, resting lower esophageal sphincter pressure and medication use.

51 was a 36% decrease in baseline mean resting lower esophageal sphincter pressure in the gastric band

52 ensors extending 5.5 cm proximal to the peak lower esophageal sphincter pressure point was increased

53 urgitation, proton pump inhibitor (PPI) use, lower esophageal sphincter pressure, and patient satisfa

54 gery according to the size of hiatal hernia, lower esophageal sphincter pressure, Barrett esophagus,

55 ty of Life scores, esophageal acid exposure, lower esophageal sphincter pressure, number of beads (si

56 The lower esophageal sphincter pressure, the motility of the

57 staltic pressure, and a reduction in resting lower esophageal sphincter pressure.

58 e 2 techniques with respect to postoperative lower-esophageal sphincter pressure or postoperative dys

59 Lower esophageal sphincter pressures had also returned t

60 ores were reduced from 7 to 1 (P < .001) and lower esophageal sphincter pressures were reduced from 2

61 had significant improvements in postmyotomy lower esophageal sphincter profiles.

62 uences vulnerability to reflux and transient lower esophageal sphincter relaxation (tLESR) during gas

63 otility disorder characterized by incomplete lower esophageal sphincter relaxation and aperistalsis r

64 al reflux disease (e.g. vomiting, disordered lower esophageal sphincter relaxation and gastric accomm

65 Transient lower esophageal sphincter relaxation is central to the

66 Transient lower esophageal sphincter relaxation is the main mechan

67 The transient lower esophageal sphincter relaxation nadir also typical

68 The transient lower esophageal sphincter relaxation onset invariably p

69 ent for this patient population is transient lower esophageal sphincter relaxation reducers.

70 We assessed the relationship of transient lower esophageal sphincter relaxation to gastroesophagea

71 the cardinal feature of achalasia, impaired lower esophageal sphincter relaxation, can occur in seve

72 proximally for the duration of the transient lower esophageal sphincter relaxation.

73 cholinergic pathways in modulating transient lower esophageal sphincter relaxation.

74 tric junction (EGJ) opening during transient lower esophageal sphincter relaxations (tLESRs) using hi

75 Transient lower esophageal sphincter relaxations and hiatal hernia

76 an tension receptors in triggering transient lower esophageal sphincter relaxations and subsequent re

77 ics in GERD; (2) the mechanism for transient lower esophageal sphincter relaxations and their associa

78 Although there is evidence that transient lower esophageal sphincter relaxations are neurally medi

79 ted a dose-dependent inhibition of transient lower esophageal sphincter relaxations in a dog model.

80 Rather, transient lower esophageal sphincter relaxations must occur before

81 Termination of transient lower esophageal sphincter relaxations occurred about th

82 another school of thought is that transient lower esophageal sphincter relaxations result from gastr

83 se data refute the hypothesis that transient lower esophageal sphincter relaxations result from passi

84 Transient lower esophageal sphincter relaxations were not increase

85 value of compounds that can reduce transient lower esophageal sphincter relaxations.

86 The lower esophageal sphincter shows normalized pressures an

87 Recordings were obtained at 2 cm above the lower esophageal sphincter under 2 study conditions in n

88 A hypertensive lower esophageal sphincter was diagnosed in three patien

89 layed megaesophagus and achalasia, and their lower esophageal sphincter was resistant to nitric oxide

90 support treatments focused on disrupting the lower esophageal sphincter with pneumatic dilation (70%-