ライフサイエンスコーパス: lower limb

1 different body sites (face/arms, trunk, and lower limbs).

2 sunburns at other body sites (face/arms and lower limbs).

3 remote ischemia was induced to the upper or lower limb.

4 rings and gastrocnemius are extensors of the lower limb.

5 quantify collateral arterial function of the lower limb.

6 e performed due to critical ischemia of left lower limb.

7 n both the superficial and deep veins in the lower limb.

8 (31%); these changes were more common in the lower limb.

9 us to quadriceps motor neurones of the human lower limb.

10 as a dominant source of blood supply to the lower limb.

11 mal stimulus to the ipsilateral hind paw and lower limb.

12 ferent areas of the ipsilateral hind paw and lower limb.

13 It also exhibits a humanlike foot and lower limb.

14 with a minimum length of 10 cm in 1 of their lower limbs.

15 r who have deep vein thrombosis (DVT) of the lower limbs.

16 f complex natural movements in the upper and lower limbs.

17 rized by abnormal gait and spasticity of the lower limbs.

18 18-29 years and for counts of the trunk and lower limbs.

19 active hand vector was co-ordinated with the lower limbs.

20 to severe sensory and motor deficits in the lower limbs.

21 erreflexia and progressive spasticity of the lower limbs.

22 l and central somatosensory signals from the lower limbs.

23 or neurons and progressive spasticity of the lower limbs.

24 ne oxygenation with neurologic injury of the lower limbs.

25 undergone previous revascularization of the lower limbs.

26 me with symptoms predominantly affecting the lower limbs.

27 : the intentional and systematic breaking of lower limbs.

28 -section multidetector CT angiography of the lower limb (0.625-mm collimation, intravenous administra

29 [36%]) ulcerative (805 [66%]) lesion of the lower limb (733 [60%]).

30 Somatotopy (upper versus lower limb: 93% accuracy for both conditions) was also d

31 e objective was to determine whether reduced lower limb activity alters IMAT in healthy young adults

32 althy proximal muscle of patients undergoing lower limb amputation (n = 3), were analyzed for capilla

33 Nine "painless" patients with lower limb amputation and nine control subjects (sex- an

34 the extent of cortical reorganization after lower limb amputation in patients with nonpainful phanto

35 ings suggest that motor reorganization after lower-limb amputation occurs predominately at the cortic

36 s associated with high risk of infection and lower-limb amputation.

37 reorganization in 16 subjects with traumatic lower-limb amputation.

38 of 13 cases of carotid endarterectomy and 16 lower limb amputations and in the thoracic aorta of 4 of

39 mens obtained from carotid endarterectomies, lower limb amputations, and thoracic aortas from autopsi

40 Secondary outcomes included rates of lower limb amputations, bypass surgical procedures, and

41 In this study of 7 patients with lower limb amputations, inclusion of EMG signals and tem

42 In the United States, approximately 280 lower-limb amputations for ischemic disease are performe

43 ions of diabetes resulting in >70,000 annual lower-limb amputations in the United States alone.

44 imination and reaction time) on 12 upper and lower limb amputees and found that consistently reported

45 and humans become mechanically coupled when lower limb amputees walk with powered prostheses, but th

46 demonstrate a functional remapping of S1 in lower limb amputees.

47 Here we describe the lower limb anatomy of Au. sediba and hypothesize that th

48 ombosis DVT, pain, oedema and rubor of right lower limb and drug abuse.

49 peripheral gangrene, and 1 patient required lower limb and finger amputations.

50 The authors report a case where proximal lower limb and genital swelling in a 23-yr-old man was i

51 are successful in emptying deep veins of the lower limb and preventing stasis in a variety of subject

52 ultrasonography, including 20 (57.1%) in the lower limbs and 24 (68.6%) related to central venous cat

53 erized by a predominance of paralysis at the lower limbs and an early adulthood onset.

54 lar veins are subdermal veins located in the lower limbs and are mainly associated with aesthetic com

55 que isometric and isokinetic strength of the lower limbs and handgrip strength.

56 DVT affects most frequently the lower limbs and hence DVT of the leg will be the focus o

57 eczema, and persistent hyperreflexia of the lower limbs and with nonsignificantly increased rates of

58 shortening of proximal segments of upper and lower limbs) and delayed collagen folding.

59 ived RIPost (induced by ischemia to upper or lower limb), and a third was the control group.

60 cle mass, (iii) redistributed muscle mass to lower limbs, and (iv) decreased relative mass of skin du

61 ildhood hypotonia, progressive spasticity of lower limbs, and abnormal craniofacial features in adult

62 ess, atherosclerotic plaques in the aorta or lower limbs, and diabetes mellitus.

63 y progressive spasticity and weakness of the lower limbs, and pathologically by retrograde axonal deg

64 mproved our understanding of consequences of lower limb angular deformities on knees.

65 calf contraction even when the veins of the lower limb are distended due to the presence of a physio

66 Transcranial magnetic stimulation of the lower limb area of the contralateral motor cortex decrea

67 Our data show that lower limb arterial blood flow is sustained when venous

68 d to 85 (62%) of 138 (P = 0.006) and that of lower-limb arterial incompressibility increased to 24 (1

69 rwent treatment for stenosis or occlusion of lower limb arteries at the Department of Radiology of th

70 of galectin-3 in atherosclerotic carotid and lower limb arteries compared with that of umbilical cord

71 ision to perform angiographic examination of lower limb arteries was made.

72 1 ABI value < or =0.9, indicating obstructed lower limb arteries, increased from 18 (9.2%) of 191 pat

73 d atherosclerosis, particularly extensive in lower limb arteries, was admitted to the Department of A

74 rosclerosis by assessment of the carotid and lower-limb arteries in a group of people aged around 70

75 nd 17 (8%) of 219 had > or =1 incompressible lower-limb artery (ABI >1.3).

76 Lower-limb artery obstruction (ABI < or =0.9) was not as

77 ity for detecting infections associated with lower limb arthroplasty and is more accurate for detecti

78 r the detection of infection associated with lower limb arthroplasty.

79 vealed that patients assimilated the virtual lower limbs as if they were their own legs.

80 ight patients with inflammatory nodules on a lower limb associated with locoregional anatomical chang

81 , painful and debilitating neuropathy of the lower limbs associated with weight loss, and we think it

82 m the community underwent assessment of both lower limbs at baseline and at an 18-month followup.

83 Upper limb ataxia was evident in 75%, lower limb ataxia in 90% and gait ataxia in 100% of pati

84 y, intellectual impairment, weakness in both lower limbs, ataxia and abnormal facies and diagnosed on

85 which are richer in foam cells, than in the lower limb atherosclerotic lesions, which are more fibro

86 r 3 generations displayed mild and quiescent lower-limb axonal sensorimotor neuropathy.

87 s to review the major surgical approaches to lower-limb bone tumors and their impact on pediatric pat

88 ines in mediolateral bending strength of the lower limb bones and strength of the humerus are much sm

89 trends in relative strength of the upper and lower limb bones in a sample of 1,842 individuals from a

90 n upper right third molar (RM(3)); and EQH3, lower limb bones of a young Neandertal male.

91 with an anencephalic head, spine, upper and lower limb buds.

92 e aim of this study was to determine whether lower limb (calf) sequential compression devices (SCDs)

93 erves as a result of damage to the upper and lower limbs caused by gunshot wounds and fragments of ar

94 balance control and also that the upper and lower limbs co-ordinate for an appropriate whole-body sw

95 trabecular density throughout the upper and lower limbs compared with other primate taxa and (ii) th

96 d by the Neuropathy Impairment Score for the Lower Limbs, compared between the 2 groups.

97 Fat fraction correlated with the lower limb components of the inclusion body myositis fun

98 at spike-timing-dependent protocols modulate lower limb cortical circuitry during walking in a phasic

99 ation of chronic venous insufficiency of the lower limbs (CVI), with an outline of their history, use

100 Although well-established for suspected lower limb deep venous thrombosis, an algorithm combinin

101 The pulmonary embolism risk was increased by lower-limb deep venous thrombosis (odds ratio 4.0; 95% c

102 e with DNA from 253 patients with congenital lower limb disorders.

103 ght to determine the frequency of DVT in the lower limb during long-haul economy-class air travel and

104 istry of patients (n = 473) with symptomatic lower limb DVT, results of 312 urokinase infusions in 30

105 Gait ataxia and lower limb dysaesthesiae were the most frequent presenti

106 Premorbid lower limb dysfunction was associated with increased ris

107 13-63), beginning with chorea in 50%, focal lower limb dystonia in 42.5% and parkinsonism in 7.5%.

108 early-onset pyramidal tract signs and marked lower limb dystonia.

109 The main symptoms are bilateral lower limb edema, serosal effusions, and vitamin D malab

110 botic actuators, including a custom-designed lower limb exoskeleton capable of delivering tactile fee

111 f the quadriceps musculature in closed chain lower limb extension may be more important than is typic

112 o the motor control strategies available for lower limb extension.

113 unknown mechanisms and this in turn acts to lower limb flow velocity.

114 on; 3.RIPC+IR: 6 cycles of 4x4 min IR of the lower limb followed by IR group procedure; (4) 1H-[1,2,4

115 Furthermore, they demonstrate that upper and lower-limb forces are co-ordinated to produce an appropr

116 Upper and distal lower limb fracture rates were increased mainly in women

117 bitual levels and patterns of loading on the lower limbs from body mass, proportions, and locomotion.

118 minimal shoes, and may protect the feet and lower limbs from some of the impact-related injuries now

119 th spinal cord injury had impaired upper and lower limb function bilaterally, a 30% reduced cord area

120 , education, measures of cognitive function, lower limb function, chronic illness, smoking, alcohol,

121 Thoracic cord GM areas were correlated with lower limb function.

122 than RMS and correlates with disability and lower limb function.

123 specifically required for balance and upper/lower limb functional integrity.

124 s lymphadenitis about 40 days after onset of lower limb gangrene.

125 ssive disorder led to severe weakness of the lower limbs, generalized in the oldest subject (aged 57

126 including Neuropathy Impairment Score of the Lower Limbs, hand grip strength, and evaluation of veget

127 onance (BOLD-CMR) to assess perfusion in the lower limb has been hampered by poor reproducibility and

128 es with distinctly preserved strength of the lower limbs), hearing loss, optic atrophy and respirator

129 e region of onset was bulbar in 17 patients, lower limb in 31 patients and upper limb in 28 patients.

130 Lesions predominantly affected the lower limbs in an asymmetric manner and were characteriz

131 lordosis stabilizes the upper body over the lower limbs in bipeds by positioning the trunk's centre

132 ecrease tissue perfusion causing ischemia to lower limbs in patients with peripheral arterial disease

133 foot deformity, decreased muscle mass of the lower limbs, inability to walk, and growth retardation.

134 dynamic simulations were performed on seven lower limb (including superficial femoral, deep femoral

135 d the specificity was 81.1% for detection of lower limb infections.

136 Lower-limb infections in diabetic patients are most ofte

137 Upper and lower limb intermuscular coherence was measured during a

138 linical items as asymmetry and predominantly lower limb involvement.

139 Ischemia of the myocardium and lower limbs is a common consequence of arterial disease

140 y used imaging tests for symptomatic chronic lower limb ischemia, for which the administration of iod

141 oning was induced by four 5-minute cycles of lower limb ischemia.

142 walking economy, stride characteristics and lower limb joint angles were measured.

143 ominins, in having an enlarged body size and lower limb joint surfaces in combination with a relative

144 in hand (first dorsal interosseus, FDI) and lower limb (leg anterior compartment, LEG) muscles.

145 index (AAI), ratio of interventional/control lower limb length (LLI), and leg length difference (LLD)

146 the amount of body weight support (BWS); and lower limb loading.

147 l aging process and traumatic events such as lower-limb loss can alter the human ability to control s

148 dds ratio for atherosclerotic disease in the lower limbs (low ABPI) was highest in people with the lo

149 condition characterized by pubertal onset of lower limb lymphedema and an aberrant second row of eyel

150 Lower limb lymphoscintigraphy showed features of an unus

151 S1pr2(-/-) mice, whereas family DEM4154 has lower limb malformations in addition to hearing loss.

152 Other lower-limb malformations, including patellar hypoplasia,

153 left-sided weakness involving the upper and lower limbs (medical research council graded muscle stre

154 sted the hypothesis that the excitability of lower limb motor cortex assessed during walking is incre

155 scranial magnetic stimulation (TMS) of human lower limb motor cortex paired with common peroneal nerv

156 Can lower limb motor function be improved after a spinal cor

157 This allows a much greater preservation of lower limb motor function in the parturient, with a subs

158 ecovery was paralleled by the reemergence of lower limb motor imagery at cortical level.

159 teristics and phase dependent differences in lower limb movement pattern between the two conditions w

160 Lower limb MRI scans of patients with LGMD2C-2F, ranging

161 The extent of lesions on lower limb MRI showed a high correlation with overall cl

162 d with lumbar intrathecal fentanyl impairing lower limb muscle afferent feedback (FENT).

163 e investigated the influence of group III/IV lower limb muscle afferents on the development of supras

164 g feedback from mu-opioid receptor-sensitive lower limb muscle afferents.

165 readmission at 1 year, including measures of lower limb muscle as part of a larger clinical trial.

166 We hypothesized that signaling from lower limb muscle group III/IV sensory afferents to the

167 pelvic and lower limb muscle MRI scans of 269 symptomatic individua

168 Corticospinal drive to upper and lower limb muscle shows developmental changes with an in

169 r extremity-predominant, is characterized by lower limb muscle weakness and wasting, associated with

170 Typically, the illness presents with lower limb muscle weakness slowly spreading to involve t

171 arly old age, statin use was associated with lower limb muscle-related outcomes, and some were only a

172 Latencies in lower limb muscles were longer.

173 nucleotide injections into single peripheral lower limb muscles, whereas a viable therapy will need a

174 over, none of them examined the behaviour of lower limb muscles.

175 modulate motor system excitability of other lower limb muscles.

176 reliable tool for assessing perfusion in the lower limb musculature and merits further investigation

177 MRI magnetisation transfer ratios (MTRs) in lower-limb musculature as markers of pathology in periph

178 MRI of lower-limb musculature can be used to detect minimal sig

179 Magnetic resonance imaging (MRI) of lower-limb musculature systematically showed fatty atrop

180 Traumatic lower-limb musculoskeletal injuries are pervasive amongs

181 muscle contraction with validated models of lower limb musculotendon paths and segment dynamics.

182 large insertions in the upper limb (n = 21), lower limb (n = 27), and spine (n = 4) from 60 cadavers.

183 Patients had inflammatory nodules on a lower limb (n = 6) that were unilateral (n = 6) and had

184 bined locoregional anatomical changes in the lower limbs (n = 5).

185 type, and Neuropathy Impairment Score in the Lower Limbs (NIS-LL) were evaluated.

186 f neurological deficits (impairments) of the lower limbs (NIS[LL]) and adds to this transformed numbe

187 re of the Neuropathy Impairment Score of the lower limbs [NIS(LL)-W] was significantly worse at 3, 6,

188 eatment of choice for reticular veins in the lower limbs, no consensus has been reached regarding to

189 IE-large and small fibres polyneuropathy and lower limbs oedema.

190 Subsequent remineralization in the lower limb of the AMOC, between subpolar and subtropical

191 mal stimulus to the ipsilateral hind paw and lower limb of the rat.

192 he function of conduit arteries in upper and lower limbs of patients late after successful coarctatio

193 ity to perform mechanical work in the plegic lower limbs of persons with hemiplegia.

194 tern subpolar North Atlantic, both upper and lower limbs of the AMOC transport high-DOC waters.

195 sensory loss that predominantly affects the lower limbs, often preceded by hyperpathia and spontaneo

196 system region defined as bulbar, upper limb, lower limb or diaphragmatic), diagnosis, functional invo

197 d 3.64 for amputations of all or part of the lower limb (P<0.001 for both).

198 High heels are a major source of chronic lower limb pain.

199 hetic devices is essential to restore a full lower limb perceptual experience in spinal cord injury (

200 However, the effect of ischemic lower limb peripheral arteries on hemodynamics remains u

201 Lower limb peripheral artery disease is a prevalent chro

202 ffected individuals, weakness and wasting is lower-limb predominant, and typically involves both prox

203 -years-old girl reported a lump in the right lower limb present for approximately 2 years, which was

204 -knee amputations for ischemic ulcers of the lower limbs, presented at our clinic due to a sudden los

205 resiensis is that of a biped, but the unique lower-limb proportions and surprising combination of der

206 rval, -3.9%-6.2%) and the lesions of painful lower limb prostheses (+4.03% +/- 11.32%; n = 25; P > 0.

207 Robotic lower limb prostheses can improve the quality of life fo

208 residual-limb soft-tissue envelope change in lower-limb prosthetics is precise and can be used to det

209 es, ESA therapy did not increase the risk of lower limb proximal deep venous thrombosis (RR 0.97, 95%

210 mortality without an increase in the rate of lower limb proximal deep venous thrombosis.

211 ecline, parkinsonism, peripheral neuropathy, lower limb proximal muscle weakness, and autonomic dysfu

212 ecline, parkinsonism, peripheral neuropathy, lower-limb proximal muscle weakness, and autonomic dysfu

213 In contrast, posterior tibial FMD, NTG, and lower limb PWV were comparable.

214 ar signs, and the majority had myoclonus and lower limb pyramidal signs, with relatively preserved co

215 olving initially the proximal muscles of the lower limbs ranged from 17 to 40 years.

216 pective cohort study, 20 patients undergoing lower limb reconstruction and 10 age-matched controls we

217 such as older age of onset, preservation of lower limb reflexes and cardiac presentations.

218 reduced tendon reflexes, dysarthria, absent lower limb reflexes, and loss of position and vibration

219 Remote sub-lethal ischemic injury to both lower limbs results in cerebral protection from subseque

220 mmendations for antithrombotic therapy after lower limb revascularization are inconsistent and not al

221 d 3-D rotational imaging of five fresh human lower limbs revealed calcification in all main arteries.

222 ired for mediating the protective effects of lower limb RIPC on hepatic MBF in liver IR injury.

223 ed to assess the role of hepatic sGC-cGMP in lower limb RIPC-induced protection against liver IR inju

224 s protection is nitric oxide (NO) induced by lower limb RIPC.

225 using the Neuropathy Impairment Score of the Lower Limbs (rs = -0.58 [95% CI, -0.84 to -0.11]; P = .0

226 ld not be done during the inflation cycle of lower limb SCDs because they produce a falsely low cardi

227 muscular atrophy predominantly of the distal lower limbs, scoliosis, and mild distal sensory involvem

228 Enhanced lower limb segmental excitability had emerged in nearly

229 iated through the inhibition of group III/IV lower limb sensory muscle afferents.

230 nscriptional profiles in sets of human adult lower limb skeletal muscles.

231 We used microdialysis techniques in the lower-limb skeletal muscle to measure [Ca2+]o changes in

232 s with incomplete or complete SCI (affecting lower limbs' somatosensation), with respect to controls.

233 e demonstrate a new approach for reproducing lower limb somatosensory feedback in paraplegics by rema

234 ced unsteadiness is due to interference with lower limb somatosensory input, vestibular reflexes or c

235 somal-recessive neurodegenerative subtype of lower limb spastic paraparesis with additional diffuse s

236 egia (HSP) is a genetic disorder that causes lower limb spasticity and weakness and intellectual disa

237 inantly, but not exclusively, by progressive lower limb spasticity and weakness resulting from distal

238 hy of the corticospinal tracts, resulting in lower limb spasticity and weakness.

239 y classified as either pure with predominant lower limb spasticity, or complex where spastic parapleg

240 are clinically characterised by progressive lower limb spasticity.

241 s in the corticospinal tract and progressive lower limb spasticity.

242 gical disorders characterized by progressive lower-limb spasticity and weakness.

243 xtension of spontaneous, acute, symptomatic, lower-limb superficial-vein thrombosis (SVT) is debated.

244 nesthesia and postoperative analgesia of the lower limb surgery.

245 muscle fibres decreased following unilateral lower limb suspension (ULLS) and returned to normal afte

246 Unilateral lower limb suspension (ULLS) has been suggested as an al

247 ects were subjected to 3 weeks of unilateral lower limb suspension (ULLS), a widely used human model

248 e hypothesis that, in response to unilateral lower limb suspension (ULLS), the magnitude of tendon me

249 od, which was followed by 4 wk of unilateral lower limb suspension.

250 ientated trabeculae were more obvious in the lower limb than the upper limb (59% versus 29%).

251 s well as how they are co-ordinated with the lower limbs to maintain balance.

252 We studied human lower limbs to test the hypothesis that the application

253 f onset; the site of onset ascended from the lower limbs to the face as the mean age of onset increas

254 e if subjecting a distant organ, such as the lower limb, to a similar priming ischemic insult would r

255 n leg muscle function, we performed isolated lower-limb training in 17 patients with severe CHF.

256 ng-term follow-up of pediatric patients with lower-limb tumors is needed to (1) determine in a unifor

257 (AIH) in remission, presented with bilateral lower limb ulcers 4 years after the diagnosis of AIH.

258 Many people with diabetes experience lower-limb ulcers.

259 c troponin I, brain natriuretic peptide, and lower limb ultrasound testing.

260 sitive pulmonary angiogram and four positive lower-limb US scans.

261 ction and timing of the change of non-active lower limb vascular conductance at the onset of contrala

262 than in young healthy sedentary men due to a lower limb vascular conductance.

263 mptomatic pulmonary embolism associated with lower-limb vein thrombosis and at least 1 criterion for

264 mittent pneumatic calf compression increased lower limb venous return, causing acute but transient de

265 Angiogenesis in the lower limb was also increased and was associated with in

266 Atherosclerosis in the arteries of the lower limb was assessed in 186 people by the ankle-brach

267 educed motor function in the right upper and lower limb was associated with lower gamma-aminobutyric

268 d blood flow in the non-active contralateral lower limb was recorded at 15 s intervals.

269 ermittent pneumatic compression (IPC) of the lower limbs was effective in reducing venous thromboembo

270 d trunk with more-modest amelioration in the lower limbs was observed in all patients.

271 complete compression ultrasonography of the lower limbs was performed after 8 +/- 2 days of antithro

272 a motor-neurological disorder manifested by lower limb weakness and spasticity and length-dependent

273 Distal lower limb weakness began between the ages of 8 and 16 y

274 ch less common but a milder degree of distal lower limb weakness is often observed.

275 lograft recipients experience expiratory and lower limb weakness that may contribute to exercise into

276 Patients show adult onset proximal lower limb weakness with highly raised serum creatine ki

277 wly growing VT in a patient with progressing lower limb weakness without any history or imaging findi

278 adult-onset myopathy characterized by distal lower limb weakness, calf hypertrophy and progressive de

279 ic distribution of deep venous reflux in the lower limb were determined across the clinical classes o

280 and vessel maturation (angiogenesis) in the lower limb were enhanced in TNFR1-KO but were reduced in

281 er incidence of inattention than those whose lower limbs were most affected.

282 ested in eight patients in a setup where the lower limbs were simulated using immersive virtual reali

283 Mild atrophy in the left upper and lower limbs without pain, swelling, or skin lesions was