ライフサイエンスコーパス: lumbar

1 etabolomics from the sciatic nerve (SN), the lumbar 4/5 dorsal root ganglia (DRG), and the trigeminal

2 lexors, and enhances the motor output during lumbar afferent-induced locomotor rhythms.

3 g from venous-derived lymph sacs, vessels of lumbar and dorsal midline skin form via assembly of non-

4 We evaluated the accuracy of the best single lumbar and midthigh MRI slice to assess whole-body SM, v

5 itional lesions compared with limited axial (lumbar and pelvic) studies, especially in the thoracic s

6 4-year-old woman presented with a history of lumbar and perineal pain and painful defecation.

7 ory bulb, brainstem, and cervical, thoracic, lumbar and sacral spinal cord).

8 with misshapen, fused and reduced number of lumbar and sacral vertebrae, under-developed hind limb b

9 , chimpanzees walking bipedally rotate their lumbar and thoracic regions in a manner similar to human

10 B-12 were not significantly associated with lumbar and total-body BMD.

11 easibility of stem cell transplantation into lumbar and/or cervical spinal cord regions in amyotrophi

12 ple inferior mesenteric artery (IMA), simple lumbar artery (LA), complex LA, and complex IMA-LA type

13 tients with an external ventricular drain or lumbar catheter in place for more than 24 hours.

14 erebrospinal fluid collection via indwelling lumbar catheter over 36 to 48 hours before, during, and

15 ventricular drain, 17 patients (6.3%) had a lumbar catheter, and four patients (1.5%) had both exter

16 .5%) had both external ventricular drain and lumbar catheter.

17 Use of external ventricular drains and lumbar catheters is associated with a risk of ventriculi

18 the locomotor-related signal produced by the lumbar central pattern generator for locomotion selectiv

19 l, we randomly assigned 400 patients who had lumbar central spinal stenosis and moderate-to-severe le

20 ks can activate and modulate the limb-moving lumbar circuitry, it is important to clarify the functio

21 However, if the lumbar circuits are rhythmically active, these latter ci

22 sses an intrinsic rhythmogenic capacity, the lumbar circuits, if they are rhythmically active, will e

23 Illumination of the lumbar cord in mice expressing eNpHR or Arch in ChAT(+)

24 with the locomotor activity expressed by the lumbar cord.

25 NAGLU activity was detected in lumbar CSF and was 15-20% of that in unaffected children

26 ly, the HMW tau species was also detected in lumbar CSF from AD patients, and its levels were signifi

27 el and postmortem ventricular and antemortem lumbar CSF from AD patients.

28 imates were highest (0.3-0.8 mGy/MBq) in the lumbar CSF space.

29 oid beta lowering was observed in plasma and lumbar CSF when single and multiple doses of LY2886721 w

30 wins from TwinsUK database assessed for LBP, lumbar disc degeneration (LDD) as its possible cause, an

31 The pathogenesis of pain in lumbar disc herniation (LDH) remains poorly understood.

32 ssociated with increased risk of surgery for lumbar disc herniation (OR = 1.89, 95% CI: 1.25, 2.86; n

33 y, brain metastases, lumbar spinal stenosis, lumbar disc herniation, childhood hydrocephalus, trauma

34 tients with chronic back pain diagnosed with lumbar disk degeneration and unresponsive to conservativ

35 with acute radiculopathy due to a herniated lumbar disk, a short course of oral steroids, compared w

36 treatment increased the number of T cells in lumbar dorsal root ganglia (DRG), where CD8(+) T cells w

37 n needed through placement of a percutaneous lumbar drain or ventriculostomy.

38 or a combined treatment approach of IVF plus lumbar drains (LDs).

39 el with a reduction in all components in the lumbar DRGs.

40 on adrenoceptor-dependent sacral control of lumbar flexor motoneuron firing in newborn rats.

41 nking adrenoceptor-activated sacral CPGs and lumbar flexor motoneurons, thereby providing novel insig

42 e required for direct rhythmic activation of lumbar flexor motoneurons.

43 alternating rhythmic bursting (0.15-1 Hz) in lumbar flexor motoneurons.

44 echanisms by which sacral circuitry recruits lumbar flexors, and enhances the motor output during lum

45 or (SEG) has been identified in the rat with lumbar galaninergic interneurons playing a pivotal role

46 his receptor causes sex-common glutamatergic lumbar ganglion interneurons (LUA) to potentiate downstr

47 ive laminectomy in patients with symptomatic lumbar grade I degenerative spondylolisthesis with spina

48 uing) both under control conditions and with lumbar intrathecal fentanyl impairing feedback from mu-o

49 als under control conditions (CTRL) and with lumbar intrathecal fentanyl impairing lower limb muscle

50 Lumbar intrathecal fentanyl was used to attenuate the ce

51 udy was to assess the safety and efficacy of lumbar intrathecal HPbetaCD.

52 delivered a lentiviral vector using a single lumbar intrathecal injection and a myelin-specific promo

53 of the notochord remnants with aging in the lumbar IVDs of BALB/c mice.

54 pulposus and annulus fibrosus regions of all lumbar IVDs were assessed by means of principal frequenc

55 Finally, nerve injury upregulated CXCL12 in lumbar L4-L6 DRGs, and this upregulation caused migratio

56 in Control neurons and neurons from the 4th lumbar (L4) and 5th lumbar (L5) dorsal root ganglia afte

57 and neurons from the 4th lumbar (L4) and 5th lumbar (L5) dorsal root ganglia after L5 spinal nerve li

58 Injection of NMDA into the fifth lumbar (L5)-DRG induced hyperalgesia in the rat hind paw

59 f serotonergic reticulospinal innervation at lumbar levels, the propriospinal projection network, neu

60 seal facets that shift from thoracic-like to lumbar-like at the penultimate rib-bearing level, rather

61 ce of an ascending excitatory influence from lumbar locomotor CPG circuitry to the medullary respirat

62 ion in some thoracic areas revealed that the lumbar locomotor network could trigger locomotor burstin

63 We conclude that the lumbar locomotor network plays a central role in the gen

64 osynaptic component coming directly from the lumbar locomotor network.

65 tigated some of the interactions between the lumbar locomotor networks that control limb movements an

66 ological process that preferentially affects lumbar lower motor neurons, with or without additional u

67 cerebroventricular (i.c.v.) leptin increases lumbar (LSNA) and renal (RSNA) SNA and baroreflex contro

68 A large proportion of these upper lumbar LVST-responsive dCINs project to contralateral L5

69 e cell patch-clamp recordings of fluorescent lumbar MN cell bodies from ChAT-eGFP or superoxide dismu

70 lice recordings from the fluorescent-labeled lumbar MN cell bodies to establish that fast and slow fi

71 KCC2 expression in lumbar MNs is reduced after spinal cord injury (SCI) res

72 rons provides a novel way to recruit rostral lumbar motoneurons and modulate the output required to e

73 wasting, associated with reduced numbers of lumbar motor neurons and is caused by mutations in DYNC1

74 d an increase in axonal reinnervation of the lumbar motor neurons.

75 ir receptor complexes in distinct subsets of lumbar motor neurons: HGF supports hindlimb motor neuron

76 nated fibers and fewer inflammatory cells in lumbar motor roots, as well as in the femoral motor and

77 wo-photon imaging to monitor the activity of lumbar motorneurons.

78 Methods In a cross-sectional study, in vivo lumbar MR elastography was performed once in the morning

79 , chair stand test, sitting and rising test; lumbar multifidus: timed up and go) as well as trunk mus

80 fy the functional organization of sacral and lumbar networks and their linking pathways.

81 ated sacral CPGs to modulate the activity of lumbar networks via sacral VF neurons provides a novel w

82 To investigate correlation between lumbar opening pressure (LOP) and radiological scores ba

83 he patients who declared decreasing pain had lumbar or cervical MRI.

84 T], magnetic resonance imaging [MRI]) of the lumbar or thoracic spine within 6 weeks of the index vis

85 .14, 4.15) were related to increased risk of lumbar osteoporosis.

86 a 3-month history of left inguinocrural and lumbar pain and anorexia with weight loss.

87 Adverse events consisted of five cases of lumbar pain and one case of thoracic pain which occurred

88 fected the same muscle groups (proximal leg, lumbar paraspinal and medial gastrocnemius muscles).

89 roup was more likely to receive an indicated lumbar puncture (86% vs 32%, p<0.001), and more likely t

90 CrAg-positive patients were offered lumbar puncture (LP) and treated with antifungals.

91 nfected adult patients undergoing diagnostic lumbar puncture (LP) at a single center between 2011 and

92 erized tomography (CT) is recommended before lumbar puncture (LP) if mental impairment.

93 uted tomography (CT) scan of the head before lumbar puncture (LP) in adults with community-acquired m

94 bone marrow stromal cells (BMSCs) following lumbar puncture alleviates early- and late-phase neuropa

95 erior chest wall mass was nondiagnostic, and lumbar puncture and bone marrow biopsies were negative.

96 Patients with CIS underwent a lumbar puncture and magnetic resonance imaging scan with

97 en appropriate, the time between imaging and lumbar puncture assessments.

98 We performed lumbar puncture at 3-5 time points in human immunodefici

99 itted with CNS symptoms or signs requiring a lumbar puncture at Mahosot Hospital, Vientiane, Laos.

100 They underwent lumbar puncture for collection of CSF samples, from whic

101 ent, including blood pressure assessment and lumbar puncture for determination of cerebral spinal flu

102 ey are similar in age to patients undergoing lumbar puncture for evaluation of neonatal fever and are

103 f CSF) may be associated with transient post-lumbar puncture headache, without increasing rates of pe

104 ospinal fluid (CSF) obtained through routine lumbar puncture in 53 patients with suspected or known C

105 tic needles and conventional needles for any lumbar puncture indication.

106 Neonatal and infant lumbar puncture is a commonly performed procedure in eme

107 Success of lumbar puncture on first attempt, failure rate, mean num

108 Incidence of IIH, lumbar puncture opening pressures, and body mass index.

109 We directly evaluated associations of 3 post-lumbar puncture outcomes (immediate postprocedural heada

110 Newborns with cCMV and a lumbar puncture performed were included and classified a

111 A lumbar puncture sample did not contain lymphoma cells.

112 eviously known as T807) who also underwent a lumbar puncture to assess cerebrospinal fluid levels of

113 A lumbar puncture was performed in all participants to mea

114 Polysomnography and lumbar puncture were performed in OSA and control groups

115 sion for patients having elective diagnostic lumbar puncture with a platelet count less than 50 x 109

116 a-analysis to compare patient outcomes after lumbar puncture with atraumatic needles and conventional

117 am or continuous video electroencephalogram, lumbar puncture, and genetic testing may be considered i

118 ated with simultaneous ICP, assessed through lumbar puncture, and IOP measurements when supine, sitti

119 INTERPRETATION: Among patients who had lumbar puncture, atraumatic needles were associated with

120 edle gauge, patient position, indication for lumbar puncture, bed rest after puncture, or clinician s

121 atory tests, magnetic resonance imaging, and lumbar puncture, were nondiagnostic.

122 al alternation test (MAT), venipuncture, and lumbar puncture.

123 ts to undergo magnetic resonance imaging and lumbar puncture.

124 nts thought to be CNS negative by diagnostic lumbar puncture.

125 use of intravenous (IV) fluid bolus prior to lumbar puncture.

126 with the most common adverse event following lumbar puncture.

127 CSF was tested after lumbar puncture.

128 ural 3T magnetic resonance imaging (MRI) and lumbar puncture.

129 s a superior option for patients who require lumbar puncture.

130 n proposed to lower complication rates after lumbar puncture.

131 mortality, and guidelines recommend frequent lumbar punctures (LPs) to control ICP.

132 tudy of consecutive patients who underwent 2 lumbar punctures between the beginning of 1995 and the e

133 We performed lumbar punctures in 3 patients with this presentation an

134 univariate and multivariable analyses of 338 lumbar punctures in the Dominantly Inherited Alzheimer N

135 departments, yet traumatic and unsuccessful lumbar punctures occur 30% to 50% of the time.

136 ult patients undergoing clinically indicated lumbar punctures or other CSF-related procedures.

137 The mean (SE) interval between lumbar punctures was 2.0 (0.1) years, and the mean (SE)

138 Lumbar punctures were performed and assayed for cerebros

139 Lumbar punctures were performed in GWI, CFS and control

140 )-infected Ugandan adults with CM had serial lumbar punctures with measurement of CSF opening pressur

141 All participants underwent 3 lumbar punctures, blood draw, clinical assessment of str

142 ration may be a risk factor for unsuccessful lumbar punctures, but to our knowledge, no studies have

143 cy by actigraphy in the six nights preceding lumbar punctures, was associated with higher tau (r = 0.

144 overweight and obesity are risk factors for lumbar radicular pain and sciatica in men and women, wit

145 associations of overweight and obesity with lumbar radicular pain and sciatica using a meta-analysis

146 1.27, 1.55; n = 19,165) were associated with lumbar radicular pain.

147 (glial cells markers) were quantified in the lumbar region of the spinal cord, prefrontal cortex (PFC

148 AP images of the lumbar region of the spine were performed in each patien

149 ound examination because of the pain in left lumbar region.

150 ate caudally to the lower thoracic and upper lumbar regions.

151 This lumbar rhythm depended on continuity of the ventral funi

152 ole in somitogenesis and the pathogenesis of lumbar/sacral vertebral anomalies.

153 ll as Western blot analyses, on cervical and lumbar sections of the spinal cord in patients diagnosed

154 orn of the gray matter, in both cervical and lumbar sections.

155 ements in response to ES and/or IS of spinal lumbar segments L1-L7 in decerebrate cats.

156 Here, we investigated whether lumbar segments of cats with a chronic hemisection were

157 gments, we show that the motor output of the lumbar segments produced by SCA stimulation is enhanced

158 which most dCINs are LVST responsive, upper lumbar segments stand out because they contain a much sm

159 In contrast to thoracic and lower lumbar segments, in which most dCINs are LVST responsive

160 the gray rami to the spinal nerves near the lumbar sensory ganglia, we avoided widespread sympatheti

161 eparation and trabecular number of femur and lumbar, serum osteocalcin, total calcium, intact parathy

162 e analysis of CT scans was used to calculate Lumbar skeletal muscle index (LSMI), and mean muscle att

163 pothalamus (PVN) dose-dependently suppressed lumbar SNA (LSNA) and its baroreflex regulation, and the

164 n-dependent increase in OVLT cell discharge, lumbar SNA and ABP.

165 n (20 nl) of 1.0 m NaCl significantly raised lumbar SNA, adrenal SNA and ABP.

166 Interactions between cervical and lumbar spinal circuits are mediated by long propriospina

167 taneously record from hundreds of neurons in lumbar spinal circuits of turtles and establish the neur

168 scular Blockade (NMB) and another undergoing lumbar spinal cord (SC) transection, both serving as con

169 Inosine did not affect CST sprouting in the lumbar spinal cord but did restore levels of the growth-

170 that epidural electrical stimulation of the lumbar spinal cord can reproduce the natural activation

171 Neural circuitry in the lumbar spinal cord governs two principal features of loc

172 bers and initially entered the simian CNS at lumbar spinal cord levels.

173 ALS lumbar spinal cord lysates similarly show increased cyto

174 ha and monocyte chemoattractant protein 1 in lumbar spinal cord of paclitaxel-treated WT mice.

175 Lumbar spinal cord PET signal was significantly higher i

176 In lumbar spinal cord, inflammatory signaling is reduced in

177 d Renshaw cell-motoneuron pairs in the mouse lumbar spinal cord.

178 ically connected to the motor neurons in the lumbar spinal cord.

179 ells of origin of the two projections in the lumbar spinal cord.

180 f KCC2 and NKCC1 toward normal levels in the lumbar spinal cord.

181 of motor neurons in the ventral horn of the lumbar spinal cord.

182 rough a density cushion were inoculated into lumbar spinal cords of 100-day-old mice carrying a human

183 ical analyses were performed in cervical and lumbar spinal cords.

184 comparing lumbar spinal decompression versus lumbar spinal decompression plus instrumented fusion for

185 5-center randomized clinical trial comparing lumbar spinal decompression versus lumbar spinal decompr

186 dicle screws affixed to titanium alloy rods) lumbar spinal fusion in addition to decompressive lamine

187 e grade I spondylolisthesis, the addition of lumbar spinal fusion to laminectomy was associated with

188 s improved by MRI imaging which demonstrated lumbar spinal nerve root enhancement and clumping or les

189 32 in up to 50% of Schwann cells in multiple lumbar spinal roots and peripheral nerves.

190 cellularity of the CC lining in reference to lumbar spinal segment L4 during the postnatal developmen

191 physiological recordings from the sacral and lumbar spinal segments, we show that the motor output of

192 ment decisions for patients with symptomatic lumbar spinal stenosis (LSS) are challenging, and nonsur

193 ients between 50 and 80 years of age who had lumbar spinal stenosis at one or two adjacent vertebral

194 ondylolisthesis, 3 to 14 mm) and symptomatic lumbar spinal stenosis to undergo either decompressive l

195 ections are widely used to treat symptoms of lumbar spinal stenosis, a common cause of pain and disab

196 me prediction in epilepsy, brain metastases, lumbar spinal stenosis, lumbar disc herniation, childhoo

197 Among patients with lumbar spinal stenosis, with or without degenerative spo

198 o decompression surgery in patients who have lumbar spinal stenosis, with or without degenerative spo

199 omen who received placebo (342 women) at the lumbar spine (-4.0% [-4.5 to -3.4] vs -1.2% [-1.7 to -0.

200 otein intake may have a protective effect on lumbar spine (LS) bone mineral density (BMD) compared wi

201 in areal bone mineral density (aBMD) of the lumbar spine (LS), as determined by dual-energy X-ray ab

202 -miRTS)-centric multistage meta-analysis for lumbar spine (LS)-, total hip (HIP)- and femoral neck (F

203 t total femur (TFBMD), femoral neck (FNBMD), lumbar spine (LSBMD), and physician-diagnosed osteoporos

204 ary statistics from the GEFOS consortium for lumbar spine (n = 31,800) and femoral neck (n = 32,961)

205 one mineral density T scores are -2.6 at the lumbar spine and -1.9 at the total hip, and spine imagin

206 ensive bone marrow metastases throughout the lumbar spine and a soft tissue mass in the lower sacral

207 PD patients had lower hip, lumbar spine and femoral neck BMD levels compared with h

208 ith the use of random-effects models for the lumbar spine and femoral neck for all studies providing

209 avone therapies for treating BMD loss at the lumbar spine and femoral neck in estrogen-deficient wome

210 mulant use had lower DXA measurements of the lumbar spine and femur compared with nonusers.

211 roved bone mass and microarchitecture in the lumbar spine and femur in F508del mice.

212 Mean bone mineral density z scores (lumbar spine and femur) remained stable and were maintai

213 S-986001 groups showed a smaller decrease in lumbar spine and hip bone mineral density but greater ac

214 Bone mineral density was measured at the lumbar spine and hip, and hip geometry was extracted fro

215 was associated with greater BMD loss at both lumbar spine and hip.

216 utative somatosensory representations of the lumbar spine and leg.

217 al examination, and general knowledge of the lumbar spine and pelvic anatomy relevant to the child in

218 Bone mineral density was measured at lumbar spine and the hip.

219 changes in bone mineral density (BMD) in the lumbar spine and total hip between patients treated with

220 ndpoints were the mean percentage changes in lumbar spine and total hip bone mineral density at week

221 ooked for if abnormalities in the MRI of the lumbar spine are not found.

222 from baseline in bone mineral density at the lumbar spine at 12 months.

223 Mean lumbar spine BMC was significantly lower in stimulant us

224 r 3 months or longer had significantly lower lumbar spine BMD (0.89 g/cm2; 95% CI, 0.85-0.93 g/cm2 vs

225 95% CI, 13.26-13.51 g; P = .02), as was mean lumbar spine BMD (0.90 g/cm2; 95% CI, 0.87-0.94 g/cm2 vs

226 combined OA phenotype (hip and/or knee) and lumbar spine BMD (rg=0.18, P = 2.23 x 10-2), which may b

227 n of previously reported common variants for lumbar spine BMD (rs11692564(T), MAF = 1.6%, replication

228 lts were seen for change in femoral neck and lumbar spine BMD and across a range of subgroup analyses

229 The ZOL arm had an 8% higher lumbar spine BMD at 12 weeks relative to the placebo arm

230 Lumbar spine BMD decline was also less with MVC (median

231 CD4(+)CD38(+)HLA-DR(+)) were associated with lumbar spine BMD loss.

232 t 5 years from diagnosis, with whole-body or lumbar spine BMD z scores of -1.0 or lower.

233 ulant use and total femur, femoral neck, and lumbar spine bone mineral content (BMC) and bone mineral

234 Lumbar spine bone mineral density showed a mean increase

235 decline in HIV-uninfected individuals at the lumbar spine but not at the hip.

236 ssociated with less bone loss at the hip and lumbar spine compared with TDF.

237 enal stone protocols (26.2%) and thoracic or lumbar spine examinations (6.6%).

238 he dual-energy x-ray absorptiometry scans of lumbar spine in 39 KTR and 77 controls.

239 be used to assess MAT content and BMD of the lumbar spine in a single examination and provides data t

240 phic (CT) trabecular texture analysis of the lumbar spine in patients with anorexia nervosa and norma

241 The finding of a VT on MRI imaging of the lumbar spine is often incidental but may be found in pat

242 T and MR imaging procedures were head CT and lumbar spine MR imaging.

243 improvement initiatives include head CT and lumbar spine MR imaging.

244 examinations within 2 months, comprising the lumbar spine of 40 patients, were included.

245 vertebral body fractures in the thoracic and lumbar spine on CT images with a high sensitivity and a

246 A were independently associated with risk of lumbar spine osteoporosis.

247 ng a mean follow-up of 6.5 years, additional lumbar spine surgery was performed in 22% of the patient

248 bone mineral density (BMD) loss at the L2-L4 lumbar spine vertebra (P < 0.05), femoral neck (P < 0.01

249 rcentage loss in bone mineral density in the lumbar spine was greater in the standard group than in t

250 The patient initially had a CT scan of the lumbar spine which only revealed a protrusion of the L5-

251 ter first scan) was average for age and sex (lumbar spine, +0.7 +/- 1.6; femoral neck, -0.1 +/- 1.1;

252 , mean bone density Z scores have increased (lumbar spine, -0.2 +/- 1.6; femoral neck, -0.6 +/- 1; to

253 elow average for age and sex (mean Z scores: lumbar spine, -0.4 +/- 1.6; femoral neck, -0.7 +/- 1.1;

254 At the lumbar spine, 3 year mean BMD change for the 77 women re

255 ased from extension baseline by 16.5% at the lumbar spine, 7.4% at total hip, 7.1% at femoral neck, a

256 reased from FREEDOM baseline by 21.7% at the lumbar spine, 9.2% at total hip, 9.0% at femoral neck, a

257 orptiometry (DXA) bone outcomes (whole body, lumbar spine, and hip), controlling for known determinan

258 BMC, or bone area for the total-body radius, lumbar spine, and total hip were observed between subjec

259 spondylodiscitis include: involvement of the lumbar spine, ill-defined paraspinal abnormal contrast e

260 ant increases in bone mineral density at the lumbar spine, including an increase of 11.3% with the 21

261 At the lumbar spine, isoflavone treatment was associated with a

262 ual-energy X-ray absorptiometry, we compared lumbar spine, total hip, and femoral neck bone mineral d

263 as well as bone mineral density (BMD) at the lumbar spine, total hip, femoral neck, and one-third rad

264 al density (a T score of -2.0 or less at the lumbar spine, total hip, or femoral neck and -3.5 or mor

265 Similar results were seen at the lumbar spine.

266 ugh much less frequently, in the thoracic or lumbar spine.

267 RP) on BMD at the forearm, femoral neck, and lumbar spine.

268 n hip BMD: 0.83 (women), 0.95 (men) g/cm(2); lumbar spine: 0.86 (women), 0.93 (men) g/cm(2)].

269 moral neck; -0.09, 95% CI -0.15 to -0.03 for lumbar spine; and -0.05, 95% CI -0.07 to -0.03 for total

270 or lower at the total hip, femoral neck, or lumbar spine; and a history of fracture.

271 Thirty-four samples of three cadaveric lumbar spines (from subjects who died at ages 51, 57, an

272 tients with symptomatic grade I degenerative lumbar spondylolisthesis with spinal stenosis.

273 produced a significantly greater increase in lumbar sympathetic nerve activity (SNA), adrenal SNA and

274 on of PF4800567 increased blood pressure and lumbar sympathetic nerve activity in WKY rats, and this

275 OVLT injection of hypertonic NaCl increases lumbar sympathetic nerve activity, adrenal sympathetic n

276 ficantly reduced arterial blood pressure and lumbar sympathetic nerve discharges in SHRs.

277 d to route of administration (ie, cisternal, lumbar, ventricular).

278 r and humerus) and non-weight bearing (2(nd) lumbar vertebra and calvarium) bones in the context of o

279 nimals compared to ground control animals in lumbar vertebra and distal femur metaphysis and epiphysi

280 using computed tomography scans of the third lumbar vertebra before and during palliative chemotherap

281 letal muscle index (muscle area at the third lumbar vertebra divided by squared height).

282 (aorta, liver, spleen, kidney, small bowel, lumbar vertebra, psoas muscle, urinary bladder) as well

283 re calculated from manual delineation of the lumbar vertebrae and blood samples, assuming a fixed RM-

284 ivo BMD assessment of the trabecular bone of lumbar vertebrae and enables freely rotatable color-code

285 region with the largest effect for number of lumbar vertebrae and thoracolumbar vertebrae were locate

286 l-energy CT software, the trabecular bone of lumbar vertebrae L1-L4 were analyzed and segmented.

287 Single slices were taken at lumbar vertebrae L1-L5 plus intervertebral discs and the

288 r the posterior cortex were performed in the lumbar vertebrae of 10 pigs by a single operator.

289 In lumbar vertebrae reduced vertebral body area and wall th

290 ography (microCT) analyses of the femurs and lumbar vertebrae revealed delayed or incomplete endochon

291 A total of 210 thoracic and lumbar vertebrae showed compression fractures and were e

292 Although the total biomechanical strength of lumbar vertebrae was reduced by 35%, the strength of the

293 One hundred sixty lumbar vertebrae were analyzed in 40 patients (mean age,

294 nsity and bone mineral content in femurs and lumbar vertebrae when compared with the wild-type (WT) l

295 tment reduced intervertebral disc defects of lumbar vertebrae, loss of synchondroses, and foramen-mag

296 higher turnover rate in the humerus than in lumbar vertebrae, suggesting enhanced bone formation and

297 r of thoracic vertebrae as well as number of lumbar vertebrae.

298 res and measure bone density of thoracic and lumbar vertebral bodies on computed tomographic (CT) ima

299 There were 141 thoracic and lumbar vertebral body fractures in the case set.

300 Although several complete lumbar vertebral columns are known for early hominins, t

301 The lumbar vertebral level 4/5 IVDs harvested from 15-day-,