ライフサイエンスコーパス: lumbar spinal cord

1 ha and -beta mRNA on the injured side of the lumbar spinal cord.

2 ically connected to the motor neurons in the lumbar spinal cord.

3 structures in the septum, hypothalamus, and lumbar spinal cord.

4 ent the degeneration of motor neurons in the lumbar spinal cord.

5 ells of origin of the two projections in the lumbar spinal cord.

6 cle leads to gene transfer in motoneurons of lumbar spinal cord.

7 to suppress FOS-immunoreactivity (FOS-IR) in lumbar spinal cord.

8 f KCC2 and NKCC1 toward normal levels in the lumbar spinal cord.

9 together with Fos protein expression in the lumbar spinal cord.

10 of motor neurons in the ventral horn of the lumbar spinal cord.

11 heat-evoked fos-like immunoreactivity in the lumbar spinal cord.

12 horn neurones in laminae II-VI of the lower lumbar spinal cord.

13 r autonomic nuclei of the thoracic and upper lumbar spinal cord.

14 systems entirely contained within the local lumbar spinal cord.

15 ence in the density of DA innervation in the lumbar spinal cord.

16 serves a sexually dimorphic function in the lumbar spinal cord.

17 beled motoneurons in the ventral horn of the lumbar spinal cord.

18 on neurons in the rat lumbar DRG and in the lumbar spinal cord.

19 t produce NK1R or MOR internalization in the lumbar spinal cord.

20 atin, captopril and phosphoramidon) onto the lumbar spinal cord.

21 gulation of neuronal/astrocytic GluR1 in the lumbar spinal cord.

22 rocytes and motoneurons (MNs) from the chick lumbar spinal cord.

23 fluorescein dextran amine was applied to the lumbar spinal cord.

24 bnormal isoform of the prion protein, in the lumbar spinal cord.

25 mmunoreactivity as well as their mRNA in the lumbar spinal cord.

26 rathecal catheter with the tip placed in the lumbar spinal cord.

27 r the potential of prion interference in the lumbar spinal cord.

28 on in the abundance of HY TME PrP(Sc) in the lumbar spinal cord.

29 eptor function and subunit expression in the lumbar spinal cord.

30 PTP expression levels in the embryonic chick lumbar spinal cord.

31 hypothalamus project to motor columns in the lumbar spinal cord.

32 d Renshaw cell-motoneuron pairs in the mouse lumbar spinal cord.

33 locomotor leg movements resides in the upper lumbar spinal cord.

34 d the release of inhibitory molecules in the lumbar spinal cord.

35 ical analyses were performed in cervical and lumbar spinal cords.

36 -disulfide isomerase (PDI) family members in lumbar spinal cords.

37 hyperalgesia and Fos protein staining in the lumbar spinal cord 1, 7, 14 and 28 days after unilateral

38 ificantly increased microvascular density in lumbar spinal cord, (6) IgG microvascular leakage, (7) r

39 g directly in 12 sporadic ALS and 10 control lumbar spinal cords acquired by a rapid autopsy system t

40 F-alpha below the level of the lesion in the lumbar spinal cord after T13 lateral hemisection that co

41 was detected at 3 weeks postinfection in the lumbar spinal cord and ascended to the brain at a rate o

42 mRNAs and proteins for NT-3 and TrkC in the lumbar spinal cord and associated soleus muscle followin

43 The lumbar spinal cord and brainstem were evaluated for immu

44 en examined in the L4-5 dorsal root ganglia, lumbar spinal cord and gracile nucleus at 2, 6 and 14 we

45 c changes in motor neuron cell bodies in the lumbar spinal cord and in motor neuron axons in ventral

46 investigate the response of microglia in the lumbar spinal cord and in the brainstem to a tonic noxio

47 blots and immunohistochemistry, ipsilateral lumbar spinal cord and L5 and L6 DRG were analyzed for t

48 rived neurotrophic factor (BDNF) in both the lumbar spinal cord and soleus muscle.

49 rmalities included vacuoles initially in the lumbar spinal cord and subsequently in more cervical are

50 corticospinal tracts from the medulla to the lumbar spinal cord and the sensory tracts from the lumba

51 We examined gene expression in the lumbar spinal cord and the specific response of motoneur

52 Lumbar spinal cords and LA muscles from gonadally intact

53 arasagittal and transverse slices of the rat lumbar spinal cord, and examine how these inputs differe

54 Levels of TNFalpha in the thoraco-lumbar spinal cord are also increased at day 8 post-liga

55 is model, UPR activation was detected in the lumbar spinal cord at 6 months then declined at 12 month

56 oduces (a) a decrease in mu receptors in the lumbar spinal cord; (b) a decrease in the affinity of li

57 the levels of both neurotransmitters in the lumbar spinal cord beyond normal.

58 ability to TNF was increased not only in the lumbar spinal cord but also in brain and distal spinal c

59 Inosine did not affect CST sprouting in the lumbar spinal cord but did restore levels of the growth-

60 hroughout the gray matter in control and ALS lumbar spinal cord, but was significantly and selectivel

61 ssociated with enhanced reinnervation of the lumbar spinal cord by 5-HT axons.

62 30-40% more motoneurons in the brachial and lumbar spinal cord by the end of the normal period of ce

63 that epidural electrical stimulation of the lumbar spinal cord can reproduce the natural activation

64 (i.e., periaqueductal gray and subarachnoid lumbar spinal cord) can reduce pain sensitivity of rats

65 In embryonic lumbar spinal cord, cell-cell coupling is widespread in

66 ce results in DA concentrations in the adult lumbar spinal cord comparable to those of males.

67 a population of spinothalamic neurons in the lumbar spinal cord containing several neuropeptides incl

68 Regions of the thoraco-lumbar spinal cord containing sympathetic preganglionic

69 f lumbar-projecting A(11) DA neurons and the lumbar spinal cord DA concentrations, organized by the p

70 mals without manifestation of disease had no lumbar spinal cord demyelination.

71 early genes have been shown to occur in the lumbar spinal cord dorsal horn after peripheral inflamma

72 ary tract (NTS), and caudal thoracic/rostral lumbar spinal cord dorsal horn.

73 ns of spinocerebellar neurons in the thoraco-lumbar spinal cord: dorsal spinocerebellar tract neurons

74 phosphatase, were severely diminished in the lumbar spinal cord, DRG, and hindlimb skin.

75 nd p75, appeared qualitatively normal in the lumbar spinal cord, DRG, and hindlimb skin.

76 tion and impulse conduction in the Lewis rat lumbar spinal cord during the clinical course of acute E

77 In rodent lumbar spinal cord, extensive gap junctional coupling am

78 Moreover, in caudal lumbar spinal cord extracts from a set of paraplegic rab

79 HY TME spread from the sciatic nerve to the lumbar spinal cord followed by transsynaptic spread and

80 we found no obvious staining differences in lumbar spinal cord following the chronic constriction in

81 ffects of estrogen on gene expression in the lumbar spinal cord, following sciatic nerve crush injury

82 eparate extraction of cervical, thoracic and lumbar spinal cord for immunochemical assay.

83 (heart rate and mean arterial pressure), and lumbar spinal cord Fos expression in control rats and in

84 Neural circuitry in the lumbar spinal cord governs two principal features of loc

85 hen administered intrathecally (i.t.) to the lumbar spinal cord, GSK189254 produced robust effects in

86 cells in the superficial dorsal horn of the lumbar spinal cord have silent synapses: they do not res

87 e denervated gray matter of the cervical and lumbar spinal cord; however, EPO treatment further signi

88 or neuron density in the ventral horn of the lumbar spinal cord in G93A mice.

89 We examined whether the lumbar spinal cord in postnatal day 5 neonatal spinally

90 al analysis of cell extracts from the caudal lumbar spinal cord indicated that there was increased pr

91 f SB 203580 blocked the increased GFAP-IR in lumbar spinal cord induced by pSNL.

92 In lumbar spinal cord, inflammatory signaling is reduced in

93 o the same populations of neurons within the lumbar spinal cord, interference between HY TME and DY T

94 ective antagonist) in the dorsal quadrant of lumbar spinal cord ipsilateral to nerve injury.

95 bers and initially entered the simian CNS at lumbar spinal cord levels.

96 ly invasive, intrathecal delivery of NPCs at lumbar spinal cord (lumbar puncture) represents an impor

97 ALS lumbar spinal cord lysates similarly show increased cyto

98 s and recordings of motor output, in the rat lumbar spinal cord maintained in vitro, to demonstrate t

99 Treatment with CLC increased the number of lumbar spinal cord motoneurons that survived the cell de

100 There was no significant loss of lumbar spinal cord motor neurons in the MOG peptide-EAE

101 pinal IGF-1 expression and partial rescue of lumbar spinal cord motor neurons, as well as sex-specifi

102 These results suggest that lumbar spinal cord neurons are sensitized during the dev

103 (NR1) subunit was selectively deleted in the lumbar spinal cord of adult mice by the localized inject

104 s and retrodorsolateral nucleus of the lower lumbar spinal cord of adult rats.

105 significant elevation of C(16:0)-Cer in the lumbar spinal cord of EAE mice.

106 id; DOPAC) of A11 DA neurons innervating the lumbar spinal cord of male and female C57/BL6 mice, and

107 tau, MAP2, synaptophysin and drebrin in the lumbar spinal cord of non-diabetic and streptozotocin-di

108 ha and monocyte chemoattractant protein 1 in lumbar spinal cord of paclitaxel-treated WT mice.

109 The lumbar spinal cord of rats contains the sexually dimorph

110 The lumbar spinal cord of rats contains the sexually dimorph

111 of Fos protein-like immunoreactivity in the lumbar spinal cord of rats with complete spinal transect

112 s injected into the intrathecal space of the lumbar spinal cord of rats.

113 d white matter of the medulla, cervical, and lumbar spinal cord of SALS patients are novel findings.

114 al spinal neural stem cells (hNSCs) into the lumbar spinal cord of SOD1(G93A) rats leads to a moderat

115 Grafting of hNSCs into the lumbar spinal cord of SOD1(G93A) rats protected alpha-mo

116 d the levels of glutamate and glycine in the lumbar spinal cord of spinally transected rats.

117 oduct of membrane lipid peroxidation, in the lumbar spinal cord of sporadic amyotrophic lateral scler

118 sed TNF-alpha and FasL immunostaining in the lumbar spinal cord of the G93A SOD1 transgenic mice occu

119 ized sexually dimorphic motor nucleus in the lumbar spinal cord of the male rat.

120 n-dependent population of motoneurons in the lumbar spinal cord of the rat, to partial motoneuron dep

121 both sexually dimorphic motor nuclei in the lumbar spinal cord of the rat.

122 rough a density cushion were inoculated into lumbar spinal cords of 100-day-old mice carrying a human

123 Tissue concentrations of hIGF-1 in lumbar spinal cords of mice infused with IGF-1:TTC were

124 l hyperalgesia and Fos expression within the lumbar spinal cord on days 14, 35 and 55 after either CC

125 , chosen to include (hindbrain, cervical and lumbar spinal cord), or exclude (dorsal telencephalon) p

126 vector encoding human IGF-1 (CERE-130) into lumbar spinal cord parenchyma of SOD1(G93A) mice.

127 disruption of the BBB by compression of the lumbar spinal cord, permeability to TNF was increased no

128 Lumbar spinal cord PET signal was significantly higher i

129 n, paw skin, lumbar dorsal root ganglia, and lumbar spinal cord, postinfection (p.i.).

130 hat neurons in different segments of the rat lumbar spinal cord process information from group II aff

131 ficantly with small fibre axonal loss in the lumbar spinal cord (r = -0.832, P = 0.003) only in HLA-D

132 conjugated to saporin to demyelinate the rat lumbar spinal cord, remove macroglia, and produce parapl

133 haw cells and their dendrites in cat and rat lumbar spinal cord, respectively.

134 However, examination of the lumbar spinal cord revealed the following changes.

135 amyotrophic lateral sclerosis (ALS) than in lumbar spinal cord samples from neurologically normal pa

136 Here, we report higher Par-4 levels in lumbar spinal cord samples from patients with amyotrophi

137 We also compared the levels of Par-4 in lumbar spinal cord samples from wild-type and transgenic

138 amples, higher Par-4 levels were observed in lumbar spinal cord samples prepared from the transgenic

139 xpression arrays were used to compare murine lumbar spinal cord (SC) and dorsal root ganglion (DRG) g

140 scular Blockade (NMB) and another undergoing lumbar spinal cord (SC) transection, both serving as con

141 Silver impregnation of lumbar spinal cord sections and descending motor axon co

142 eased TNF-alpha and FasL immunoreactivity in lumbar spinal cord sections of ALS patients and G93A tra

143 Further analyses of adjacent lumbar spinal cord sections revealed that many, although

144 unohistochemical analyses of human and mouse lumbar spinal cord sections revealed that Par-4 is local

145 Lumbar spinal cord sections showed gamma2 immunostaining

146 ath genes, bcl-2 and bax, in control and ALS lumbar spinal cord sections.

147 rode was placed on the left dorsal column at lumbar spinal cord segments 2-3 in sodium pentobarbital

148 dorsal horn of the caudal thoracic and upper lumbar spinal cord segments.

149 In rat lumbar spinal cord slices, we now demonstrate that condi

150 ade with biocytin-filled electrodes from rat lumbar spinal cord slices.

151 Neurites from rostral (cervical) and caudal (lumbar) spinal cord slices exhibit distinct growth prefe

152 an be activated via lower thoracic and upper lumbar spinal cord stimulation to produce an effective c

153 ignificantly higher DA concentrations in the lumbar spinal cord than either females or males carrying

154 ns, more infectious virus was present in the lumbar spinal cord than in the cervical spinal cord.

155 ), a sexually dimorphic motor nucleus in the lumbar spinal cord that controls penile reflexes involve

156 bitory effects on dorsal horn neurons in the lumbar spinal cord that receive nociceptive input from c

157 evels after tissue injury, in the normal rat lumbar spinal cord, the majority of neurons and radial g

158 ocated group of somatic motor neurons in the lumbar spinal cord, the retrodorsolateral nucleus, showe

159 descending propriospinal projections to the lumbar spinal cord, therefore, produce inhibitory effect

160 Western blot analyses of lumbar spinal cord tissue from spastic animals showed a

161 Lumbar spinal cord tissues were examined 7 days after th

162 ion in parasagittal and transverse slices of lumbar spinal cord to stimulate presynaptic neurons by g

163 brain, and clearance from the brain stem and lumbar spinal cord was delayed, followed by reactivation

164 etic and somatic motor outflows from thoraco-lumbar spinal cord was investigated in a novel arteriall

165 Lumbar spinal cord was removed and immunostained for M4

166 tor-alpha and estrogen receptor-beta mRNA in lumbar spinal cord was shown by traditional RT-PCR.

167 scope examination of brainstem, cervical and lumbar spinal cords was performed in ALS mice at early a

168 DA concentrations in the lumbar spinal cord were higher in males, suggesting a gr

169 inhibition of rat dorsal horn neurons of the lumbar spinal cord were tested.

170 d by real-time PCR, was more abundant in the lumbar spinal cord, while HSV-2 DNA was more abundant in

171 ress increases GABA(B(1a)) expression in the lumbar spinal cord, with no commensurate change in therm

172 ine (by approximately 85% of control) in rat lumbar spinal cord without altering levels of dopamine o