ライフサイエンスコーパス: lumbar spine

1 ervertebral disc and other structures of the lumbar spine.

2 nations showing no relevant pathology of the lumbar spine.

3 led also focal tenderness in the area of the lumbar spine.

4 tative computed tomography of the trabecular lumbar spine.

5 women with SLE had lower BMD at the hip and lumbar spine.

6 as the change in bone mineral density at the lumbar spine.

7 ined as a Z score -1.0 or less at the hip or lumbar spine.

8 thy subjects) had osteoporosis at the hip or lumbar spine.

9 ctive against loss of trabecular bone at the lumbar spine.

10 lumbar spine, and 11.8% (6.8) at the lateral lumbar spine.

11 ional radiography or rapid MR imaging of the lumbar spine.

12 Similar results were seen at the lumbar spine.

13 RP) on BMD at the forearm, femoral neck, and lumbar spine.

14 ugh much less frequently, in the thoracic or lumbar spine.

15 cium group (P for time-by-group interaction: lumbar spine, 0.002; total hip, 0.03; whole body, 0.03).

16 ter first scan) was average for age and sex (lumbar spine, +0.7 +/- 1.6; femoral neck, -0.1 +/- 1.1;

17 , mean bone density Z scores have increased (lumbar spine, -0.2 +/- 1.6; femoral neck, -0.6 +/- 1; to

18 elow average for age and sex (mean Z scores: lumbar spine, -0.4 +/- 1.6; femoral neck, -0.7 +/- 1.1;

19 n hip BMD: 0.83 (women), 0.95 (men) g/cm(2); lumbar spine: 0.86 (women), 0.93 (men) g/cm(2)].

20 e interval [95% CI] -0.70, -0.13) and at the lumbar spine (-1.03 versus 0.10; group difference -1.13;

21 change in BMD between the two groups at the lumbar spine (-1.11+/-0.42%, high bone-turnover, vs. 0.6

22 ensity decreased by 3.3+/-0.7 percent in the lumbar spine, 2.1+/-0.6 percent in the trochanter, and 1

23 (-7.24%) compared with the tamoxifen group (lumbar spine, +2.77%; total hip, +0.74%).

24 r Zic1 was the most up-regulated gene in the lumbar spine (202-fold; P<10(-7)) in comparison with the

25 At the lumbar spine, 3 year mean BMD change for the 77 women re

26 omen who received placebo (342 women) at the lumbar spine (-4.0% [-4.5 to -3.4] vs -1.2% [-1.7 to -0.

27 se in median BMD from baseline to 5 years in lumbar spine (-6.08%) and total hip (-7.24%) compared wi

28 /- 1.0%; whole body, -3.6 +/- 0.5%) and F52 (lumbar spine, -6.2 +/- 0.9%; total hip, -10.3 +/- 1.4%;

29 ased from extension baseline by 16.5% at the lumbar spine, 7.4% at total hip, 7.1% at femoral neck, a

30 rsisted at NPNL and F52 (P </= 0.001): NPNL (lumbar spine, -7.5 +/- 0.7%; total hip, -10.5 +/- 1.0%;

31 reased from FREEDOM baseline by 21.7% at the lumbar spine, 9.2% at total hip, 9.0% at femoral neck, a

32 bone mineral density of 13.7 percent at the lumbar spine (95 percent confidence interval, 12.0 to 15

33 It was unaccompanied by substantial lumbar spine abbreviation, an adaptation restricted to v

34 was strongly associated with low BMD at the lumbar spine (adjusted odds ratio 4.42; 95% CI 2.19, 8.9

35 ted with loss of BMD at the femoral neck and lumbar spine after 3 years of treatment.

36 ip, femoral neck, and trabecular bone of the lumbar spine also differed significantly between groups

37 one mineral density T scores are -2.6 at the lumbar spine and -1.9 at the total hip, and spine imagin

38 m(3) (2.7%; 95% CI 2.0-3.4; p<0.0001) at the lumbar spine and 0.025 g/cm(3) (1.4%; 0.8-1.9; p<0.0001)

39 Of the 374 SN, 153 (41%) were in the lumbar spine and 221 (59%) were in the thoracic spine.

40 ensive bone marrow metastases throughout the lumbar spine and a soft tissue mass in the lower sacral

41 rabecular bone mineral density (vBMD) of the lumbar spine and coronary artery calcium (CAC) and abdom

42 sing dual-energy X-ray absorptiometry at the lumbar spine and femoral neck (FN).

43 PD patients had lower hip, lumbar spine and femoral neck BMD levels compared with h

44 ith the use of random-effects models for the lumbar spine and femoral neck for all studies providing

45 avone therapies for treating BMD loss at the lumbar spine and femoral neck in estrogen-deficient wome

46 Bone mineral density (BMD) of lumbar spine and femoral neck was measured, and tryptase

47 ry bone resorption markers (n = 2929) at the lumbar spine and femoral neck were performed in perimeno

48 BMD was assessed at the lumbar spine and femur and vertebral fracture by morphom

49 mulant use had lower DXA measurements of the lumbar spine and femur compared with nonusers.

50 roved bone mass and microarchitecture in the lumbar spine and femur in F508del mice.

51 Mean bone mineral density z scores (lumbar spine and femur) remained stable and were maintai

52 puted tomography, as well as with BMD of the lumbar spine and hip at dual x-ray absorptiometry.

53 S-986001 groups showed a smaller decrease in lumbar spine and hip bone mineral density but greater ac

54 Substantial loss of BMD in the lumbar spine and hip was seen in patients who discontinu

55 Bone mineral density was measured at the lumbar spine and hip, and hip geometry was extracted fro

56 (CAL) and bone mineral density (BMD) at the lumbar spine and hip, lifestyle, smoking, sociodemograph

57 ome measure was 9-month change in BMD of the lumbar spine and hip, measured by dual-energy x-ray abso

58 and 0.8% (0.3-1.4; p=0.003) in year 2 at the lumbar spine and hip, respectively.

59 All women underwent bone densitometry of the lumbar spine and hip.

60 was associated with greater BMD loss at both lumbar spine and hip.

61 sion in human bone biopsy samples taken from lumbar spine and iliac crest, sites that experience high

62 Royal College of Radiologists' guidelines on lumbar spine and knee radiographs.

63 utative somatosensory representations of the lumbar spine and leg.

64 of 64 y underwent (18)F-fluoride PET of the lumbar spine and measurements of biochemical markers of

65 ineral density (rs3736228, p=6.3x10(-12) for lumbar spine and p=1.9x10(-4) for femoral neck) and an i

66 nsity (top SNP, rs4355801: p=7.6x10(-10) for lumbar spine and p=3.3x10(-8) for femoral neck) and incr

67 al examination, and general knowledge of the lumbar spine and pelvic anatomy relevant to the child in

68 Bone mineral density (BMD) of the lumbar spine and proximal femur (by DXA), liver function

69 Lumbar spine and proximal femur bone mineral densities (

70 BMD of the lumbar spine and proximal femur were measured at entry a

71 BMD of the posteroanterior lumbar spine and proximal femur were measured by dual-en

72 chment level and bone mineral density of the lumbar spine and proximal femur, whether examined on a c

73 ified disease starting simultaneously in the lumbar spine and sacroiliac joints in a proportion of pa

74 ages obtained by magnetic resonance scans of lumbar spine and the clinical symptoms of the disease in

75 to increase the bone mineral density at the lumbar spine and the femoral neck in men.

76 Bone mineral density was measured at lumbar spine and the hip.

77 Bone mineral density of the lumbar spine and the proximal femur was measured by dual

78 changes in bone mineral density (BMD) in the lumbar spine and total hip between patients treated with

79 A + R resulted in a significant increase in lumbar spine and total hip BMD compared with A + P treat

80 In the H stratum, lumbar spine and total hip BMD increased significantly (

81 Lumbar spine and total hip BMD were assessed at baseline

82 The primary endpoints were changes in lumbar spine and total hip bone mineral densities (BMDs)

83 Lumbar spine and total hip bone mineral density (BMD) we

84 ndpoints were the mean percentage changes in lumbar spine and total hip bone mineral density at week

85 Bone mineral density was measured at the lumbar spine and total hip by dual-energy X-ray absorpti

86 nts were percent change of BMD at 2 years in lumbar spine and total hip for both groups.

87 subregions, as measured by QCT, but only the lumbar spine and total hip, as measured by DXA, were sig

88 neral density (BMD) and content (BMC) at the lumbar spine, and (2) focal lesions in x-rays of long bo

89 otal hip, 10.4% (5.4) at the posteroanterior lumbar spine, and 11.8% (6.8) at the lateral lumbar spin

90 ft tibia determined by pQCT, and whole-body, lumbar spine, and femoral neck measurements by DXA.

91 one mineral density (BMD) at the total body, lumbar spine, and hip (total and femoral neck) were eval

92 BMC of the total body, lumbar spine, and hip and dietary phylloquinone intake w

93 orptiometry (DXA) bone outcomes (whole body, lumbar spine, and hip), controlling for known determinan

94 DXA was used to determine BMD of the radius, lumbar spine, and hip.

95 BMC, or bone area for the total-body radius, lumbar spine, and total hip were observed between subjec

96 moral neck; -0.09, 95% CI -0.15 to -0.03 for lumbar spine; and -0.05, 95% CI -0.07 to -0.03 for total

97 or lower at the total hip, femoral neck, or lumbar spine; and a history of fracture.

98 with dual-energy x-ray absorbtiometry at the lumbar spine (anterior-posterior and lateral) and proxim

99 ooked for if abnormalities in the MRI of the lumbar spine are not found.

100 The mean increase in lumbar spine areal BMD after 1 year was 16.3% in the ris

101 y efficacy endpoint was percentage change in lumbar spine areal bone mineral density (BMD) at 1 year.

102 tment who are also receiving HRT, BMD of the lumbar spine as measured by QCT, but not DXA, is an inde

103 from baseline in bone mineral density at the lumbar spine at 12 months.

104 from baseline in bone mineral density at the lumbar spine at 12 months.

105 ercent change in bone mineral density at the lumbar spine at 24 months.

106 neral density (BMD) at the femur, tibia, and lumbar spine at 3 months and at the lumbar spine at 4 mo

107 bia, and lumbar spine at 3 months and at the lumbar spine at 4 months, with full normalization of the

108 ident, with higher lumbar spine BMC (13.9%), lumbar spine BA (6.2%), and lumbar spine BMD (10.6%) in

109 Supplemented mothers had higher lumbar spine BA (6.7%; P = 0.002) and lumbar spine BMC (

110 etween groups were more evident, with higher lumbar spine BMC (13.9%), lumbar spine BA (6.2%), and lu

111 higher lumbar spine BA (6.7%; P = 0.002) and lumbar spine BMC (7.9%, P = 0.08) than did mothers who c

112 Mean lumbar spine BMC was significantly lower in stimulant us

113 ients (n = 98) had lower rates of decline in lumbar spine BMD (-0.004 +/- 0.003 vs. -0.015 +/- 0.003

114 e L stratum showed a significant decrease in lumbar spine BMD (-2.1%; P = .0109) and a numerical decr

115 r 3 months or longer had significantly lower lumbar spine BMD (0.89 g/cm2; 95% CI, 0.85-0.93 g/cm2 vs

116 95% CI, 13.26-13.51 g; P = .02), as was mean lumbar spine BMD (0.90 g/cm2; 95% CI, 0.87-0.94 g/cm2 vs

117 ine BMC (13.9%), lumbar spine BA (6.2%), and lumbar spine BMD (10.6%) in the supplemented group (P </

118 ctors of incident fracture were the baseline lumbar spine BMD (for each 1-point decrease in T score,

119 tly greater femoral neck BMD (P = 0.008) and lumbar spine BMD (P = 0.007) than did those who never co

120 between SNPs in the Osterix region and adult lumbar spine BMD (P = 9.9 x 10(-11)).

121 al hip BMD (r=-0.33, P<0.0001), but not with lumbar spine BMD (r=-0.09, P=0.27).

122 combined OA phenotype (hip and/or knee) and lumbar spine BMD (rg=0.18, P = 2.23 x 10-2), which may b

123 n of previously reported common variants for lumbar spine BMD (rs11692564(T), MAF = 1.6%, replication

124 ly beer had a positive significant effect on lumbar spine BMD after adjustment for lifestyle (P = 0.0

125 lts were seen for change in femoral neck and lumbar spine BMD and across a range of subgroup analyses

126 r ALN trial for followup measurements of the lumbar spine BMD and hip BMD, and retrospective informat

127 However, only lumbar spine BMD as measured by QCT was a significant pr

128 The ZOL arm had an 8% higher lumbar spine BMD at 12 weeks relative to the placebo arm

129 We measured femoral neck and lumbar spine BMD at baseline and after 1 and 3 years, an

130 influence of all protein supplementation on lumbar spine BMD but showed no association with relative

131 Lumbar spine BMD decline was also less with MVC (median

132 ponse parameter was the percentage change in lumbar spine BMD from the end of year 1 to the followup

133 n evidence of association with adult hip and lumbar spine BMD in an Icelandic population, as well as

134 ositive effect of protein supplementation on lumbar spine BMD in randomized placebo-controlled trials

135 nosis could reduce the likelihood of reduced lumbar spine BMD in these patients by prompting interven

136 At 12 and 24 months, lumbar spine BMD increased by 5.5% and 7.6%, respectivel

137 In the combined group, the lumbar spine BMD increased by 7.2%, and total hip BMD in

138 Lumbar spine BMD increased in the placebo group by 0.98%

139 At 12 months, posterior-anterior lumbar spine BMD increased more in the combination group

140 CD4(+)CD38(+)HLA-DR(+)) were associated with lumbar spine BMD loss.

141 etween the coronary artery calcium score and lumbar spine BMD was -0.57 (P = 0.04), and between the c

142 ucocorticoid dose, neither total hip BMD nor lumbar spine BMD was significantly associated with focal

143 Hip and lumbar spine BMD were measured by dual-energy x-ray abso

144 ns between lycopene intake and 4-y change in lumbar spine BMD were significant for women (P for trend

145 ficantly higher than that in patients with a lumbar spine BMD Z score higher than -1.5 (P = 0.038).

146 In patients with a lumbar spine BMD Z score of -1.5 or lower, the RANKL:OPG

147 accretion and a subsequent reduction in the lumbar spine BMD Z score.

148 Lumbar spine BMD Z scores (mean +/- SD -0.13 +/- 1.19 [r

149 t 5 years from diagnosis, with whole-body or lumbar spine BMD z scores of -1.0 or lower.

150 ration of untreated juvenile DM have reduced lumbar spine BMD Z scores.

151 ing variables (including menstrual history), lumbar spine BMD, bone mineral content, and BMD z score

152 ied for soy protein or milk basic protein on lumbar spine BMD.

153 of DNA pools prepared from individuals with lumbar spine-BMD (LS-BMD) values falling into the top an

154 ge correlation (r) of -0.24 with femoral and lumbar spine BMDs.

155 amounts of alcohol was associated with less lumbar spine bone loss (P < 0.01 for quartile of alcohol

156 d that soy protein with isoflavones lessened lumbar spine bone loss in midlife women.

157 ts with low calcium intake results in higher lumbar spine bone mass and a reduced rate of femoral nec

158 sts that the observed deficits in height and lumbar spine bone mass may not be related to suboptimal

159 The lumbar spine bone mass was measured in 45 consecutive pa

160 ulant use and total femur, femoral neck, and lumbar spine bone mineral content (BMC) and bone mineral

161 graph 4 years later, and for whom a baseline lumbar spine bone mineral density (BMD) measurement was

162 rcentage change from baseline at month 12 in lumbar spine bone mineral density (BMD).

163 and superior to risedronate for increase of lumbar spine bone mineral density in both the treatment

164 Lumbar spine bone mineral density showed a mean increase

165 Total-body and lumbar spine bone mineral density were measured in adole

166 point was percentage change from baseline in lumbar spine bone mineral density.

167 cts were significantly shorter and had lower lumbar spine bone mineral density; the deficits were gre

168 decline in HIV-uninfected individuals at the lumbar spine but not at the hip.

169 pression fractures of the lower thoracic and lumbar spine by using the Genant visual semiquantitative

170 obtained for other reasons that include the lumbar spine can be used to identify patients with osteo

171 ssociated with less bone loss at the hip and lumbar spine compared with TDF.

172 ethnicity was associated with low BMD at the lumbar spine controlling for relevant clinical covariate

173 eprogrammed for full cervical, thoracic, and lumbar spine coverage (combined 70-cm FOV, seven section

174 Reviewing the full-FOV images from lumbar spine CT examinations will result in the detectio

175 were present on images in 162 (40.5%) of 400 lumbar spine CT examinations; 59 (14.8%) patients had in

176 212 male and 188 female patients) undergoing lumbar spine CT for low back pain and/or radiculopathy.

177 ne mineral density of the posterior-anterior lumbar spine decreased by 2.5% +/- 0.5% in the leuprolid

178 Mean (+/- SE) BMD of the posteroanterior lumbar spine decreased by 3.1% +/- 1.0% in men assigned

179 group, the mean bone mineral density at the lumbar spine decreased by 3.2 percent (P=0.03 for the co

180 mean trabecular bone mineral density of the lumbar spine decreased by 8.5+/-1.8 percent (P<0.001 for

181 data exist concerning the natural history of lumbar spine disc degeneration and associated risk facto

182 y to examine the radiographic progression of lumbar spine disc degeneration over the course of 9 year

183 idual radiographic features of AO and DSN in lumbar spine disc degeneration.

184 MATERIALS AND Lumbar spine diskograms and prediskogram MR images of 73

185 ompared in 782 participants with symptomatic lumbar spine disorders who were referred to orthopedists

186 Lumbar spine dual X-ray absorptiometry does not consiste

187 Patients were randomly assigned to receive lumbar spine evaluation by rapid MRI or by radiograph.

188 enal stone protocols (26.2%) and thoracic or lumbar spine examinations (6.6%).

189 gy x-ray absorptiometry, was assessed at the lumbar spine, femoral neck, and total femur (grams per s

190 numerically greater decreases in BMD at the lumbar spine, femoral neck, and total hip from the end o

191 lower rib fractures, 7.6% (eight of 105) for lumbar spine fractures, and 5.2% (nine of 174) for pelvi

192 lted in a significant increase in BMD at the lumbar spine (from 0.875 +/- 0.025 to 0.913 +/- 0.026 g/

193 Thirty-four samples of three cadaveric lumbar spines (from subjects who died at ages 51, 57, an

194 For lumbar spine fusion, rhBMP-2 and iliac crest bone graft

195 At one year, the bone mineral density at the lumbar spine had decreased by a mean of 0.7 percent in t

196 At 24 months, bone mineral density of the lumbar spine had increased by 5.6% in the denosumab grou

197 the mean (+/-SE) bone mineral density at the lumbar spine had increased more in the teriparatide grou

198 Measurements of BMD by DXA of the lumbar spine, hip (and subregions), and forearm (and sub

199 Bone mineral density of the lumbar spine, hip, and total body was measured yearly fo

200 The changes in BMD at the lumbar spine, hip, and wrist over the period of the stud

201 spondylodiscitis include: involvement of the lumbar spine, ill-defined paraspinal abnormal contrast e

202 To improve the accuracy of lumbar spine imaging-based marrow dosimetry, one can adj

203 he dual-energy x-ray absorptiometry scans of lumbar spine in 39 KTR and 77 controls.

204 be used to assess MAT content and BMD of the lumbar spine in a single examination and provides data t

205 amidronate prevents bone loss in the hip and lumbar spine in men receiving treatment for prostate can

206 phic (CT) trabecular texture analysis of the lumbar spine in patients with anorexia nervosa and norma

207 limitations regarding load-relaxation of the lumbar spine in response to flexion exposures and the in

208 nst 4-y loss in trochanter BMD in men and in lumbar spine in women.

209 ant increases in bone mineral density at the lumbar spine, including an increase of 11.3% with the 21

210 The bone mineral density at the lumbar spine increased significantly more in men treated

211 The bone mineral density of the lumbar spine increased significantly more in the combina

212 h as physical therapy, oral medications, and lumbar spine injections.

213 The finding of a VT on MRI imaging of the lumbar spine is often incidental but may be found in pat

214 At the lumbar spine, isoflavone treatment was associated with a

215 Up to five BMD measurements of the lumbar spine (L(2-4)) and the femoral neck were obtained

216 rmed a cross-sectional audit of MRI scans of lumbar spine (L-spine) and sacroiliac (SI) joints.

217 ormed before treatment, and Z scores for the lumbar spine (L1-L4) were determined.

218 Single-slice CT measurements of VAT area at lumbar spine L2-3 and L4-5 levels were taken in 110 Afri

219 the femoral neck, trochanter, total hip, and lumbar spine (L2-L4) was associated with a 0.005-0.008-g

220 the femoral neck, trochanter, total hip, and lumbar spine (L2-L4) was measured by using dual-energy X

221 osteopenia at the trochanter, total hip, and lumbar spine (L2-L4) were lower by 14% (OR: 0.86; 95% CI

222 e femoral neck, trochanter, total femur, and lumbar spine (L2-L4) were measured by using dual-energy

223 The primary outcome was percent change in lumbar spine (LS) BMD at 6 months.

224 The primary end point was the change in lumbar spine (LS) BMD from baseline to 1 year.

225 otein intake may have a protective effect on lumbar spine (LS) bone mineral density (BMD) compared wi

226 delayed group received zoledronic acid when lumbar spine (LS) or total hip (TH) T score decreased to

227 in areal bone mineral density (aBMD) of the lumbar spine (LS), as determined by dual-energy X-ray ab

228 Lumbar spine (LS), femoral neck (FN), and distal radius

229 -miRTS)-centric multistage meta-analysis for lumbar spine (LS)-, total hip (HIP)- and femoral neck (F

230 elected because of reduced BMD values at the lumbar spine (LS-BMD) or femoral neck (FN-BMD) in proban

231 t total femur (TFBMD), femoral neck (FNBMD), lumbar spine (LSBMD), and physician-diagnosed osteoporos

232 bdominal computed tomography (CT), brain and lumbar spine magnetic resonance (MR) imaging, and body p

233 ntensity confined to the anterior horns on a lumbar spine magnetic resonance imaging.

234 Five cadaveric lumbar spines (mean age, 61 years +/- 11) were prepared

235 bone histology; the first carrier had normal lumbar spine measurements (L1-L4), as determined by dual

236 The interpretation of general lumbar spine MR characteristics has sufficient reliabili

237 ncomplicated degenerative changes on initial lumbar spine MR images were identified, 71 (30%) of whic

238 ar non-SLIP patients undergoing conventional lumbar spine MR imaging as usual care in calendar year 2

239 demiologic information was included in their lumbar spine MR imaging reports.

240 nt was routinely but arbitrarily included in lumbar spine MR imaging reports.

241 T and MR imaging procedures were head CT and lumbar spine MR imaging.

242 improvement initiatives include head CT and lumbar spine MR imaging.

243 re of the sixth through 12th ribs (n = 216), lumbar spine (n = 105), or pelvis (n = 174).

244 ary statistics from the GEFOS consortium for lumbar spine (n = 31,800) and femoral neck (n = 32,961)

245 ers, consistent with the mean T-score of the lumbar spine of -1.9 by dual energy x-ray absorptiometry

246 n absolute change of -0.07 (-1.3 to 0.9) for lumbar spine of 0.04 (-0.95 to 1.03) for knee radiograph

247 n an increase in bone mineral density at the lumbar spine of 3.0 to 6.7 percent (as compared with an

248 examinations within 2 months, comprising the lumbar spine of 40 patients, were included.

249 and Z scores) of the hip, femoral neck, and lumbar spine of IgE-CMA patients were significantly lowe

250 vertebral body fractures in the thoracic and lumbar spine on CT images with a high sensitivity and a

251 d MRI group vs 4 in the radiograph group had lumbar spine operations (risk difference, 0.34; 95% CI,

252 eral density (T score of -1.8 to -4.0 at the lumbar spine or -1.8 to -3.5 at the proximal femur).

253 All had osteopenia of the lumbar spine or hip, as demonstrated by dual x-ray absor

254 z score of >/= 2.0 SDs below the mean at the lumbar spine or hip, was highly prevalent in all 3 group

255 less than -2.5 but not less than -4.0 at the lumbar spine or total hip.

256 Hematuria and fracture of the lower ribs, lumbar spine, or pelvis are objective predictors of miss

257 tly associated with systemic BMD loss at the lumbar spine (osteocalcin, bone-turnover biomarker, p =

258 No association between lumbar spine osteopenia/osteoporosis and radiographic sc

259 A were independently associated with risk of lumbar spine osteoporosis.

260 0(-7)) and lower bone mineral density at the lumbar spine (P = 0.038), but not the femoral neck.

261 f the thoracic spine (P <.05) but not in the lumbar spine (P =.09).

262 subjects had lost a mean of 2.4% BMD at the lumbar spine (P=0.003) but did not experience significan

263 Radiographs of the cervical spine, lumbar spine, pelvis, and hips were scored by using the

264 neral density were measured from total body, lumbar spine, proximal femur, and forearm with dual-ener

265 The bone mineral density of the lumbar spine, proximal femur, radial shaft, and total bo

266 th reduced whole-body (r=0.21, p=0.0088) and lumbar-spine (r=0.17, p=0.03) bone-mineral content in ch

267 tween fracture and BMD in patients with IBD (lumbar spine, r = -0.103, p = 0.17 and femoral neck, r =

268 Seven hundred ninety-six paired lumbar spine radiographs were read by a single reader fo

269 BMD), and BA-adjusted BMC of the whole-body, lumbar spine, radius, and hip were measured by dual-ener

270 dioactivity to the cumulated activity of the lumbar spine region of interest (ROI) from serial gamma-

271 ng a mean follow-up of 6.5 years, additional lumbar spine surgery was performed in 22% of the patient

272 d MRI sequences of the sacroiliac joints and lumbar spine that were scored for active bone marrow ede

273 At the lumbar spine, the rate of BMD change for premenopausal w

274 BMD of the lumbar spine, total hip and hip subregions, as measured

275 ual-energy X-ray absorptiometry, we compared lumbar spine, total hip, and femoral neck bone mineral d

276 as well as bone mineral density (BMD) at the lumbar spine, total hip, femoral neck, and one-third rad

277 al density (a T score of -2.0 or less at the lumbar spine, total hip, or femoral neck and -3.5 or mor

278 Outcomes included lumbar spine, total proximal femur, femoral neck, and wh

279 al density was measured at the total hip and lumbar spine using dual-energy x-ray absorptiometry.

280 seline BMD measurements (femoral neck and/or lumbar spine) using dual x-ray absorptiometry.

281 bone mineral density (BMD) loss at the L2-L4 lumbar spine vertebra (P < 0.05), femoral neck (P < 0.01

282 Subjects lost 1-2% BMD annually at lumbar spine vertebrae 2-4, the forearm, the femoral nec

283 trally and laterally within the thoracic and lumbar spine vertebral bodies.

284 After 18 months, DeltaBMD at the lumbar spine was 0.068 +/- 0.21 and 0.015 +/- 0.034 for

285 rcentage loss in bone mineral density in the lumbar spine was greater in the standard group than in t

286 vidence for age at onset of bone loss at the lumbar spine was inconclusive.

287 Trabecular bone mineral density of the lumbar spine was measured at base line and month 30 by m

288 Diffusion-weighted MR imaging of the lumbar spine was performed in 39 patients (all men; mean

289 BMD of the lumbar spine was significantly (P<or=0.05) and comparabl

290 using a specific exercise that isolated the lumbar spine, was efficacious in preventing steroid-indu

291 c resonance images of the lower thoracic and lumbar spine were analyzed in 516 healthy female twins (

292 disease, after which the cervical spine and lumbar spine were equally involved.

293 ual-energy x-ray absorptiometry scans of the lumbar spine were performed.

294 Vertebral deformities of the thoracic and lumbar spine were radiographically classified by using t

295 The patient initially had a CT scan of the lumbar spine which only revealed a protrusion of the L5-

296 of the pelvis and lateral radiographs of the lumbar spine, which were scored using the Stoke Ankylosi

297 cision of (18)F-fluoride PET measured at the lumbar spine, which will aid in the accurate interpretat

298 Computed tomography, bone scintigraphy, and lumbar spine x-rays were performed at the beginning and

299 Lumbar spine z scores measured 19-44 d after delivery (n

300 A total of 33% (5/15) of adolescents had lumbar spine z scores that met the definition of osteope