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1 ibility to infection with roundworm (Ascaris lumbricoides).
2 ate had low efficacy against hookworm and A. lumbricoides.
3 richiura, 443 with hookworm, and 293 with A. lumbricoides.
4 ng early development of the nematode Ascaris lumbricoides.
5 ing up a STH MDA programme targeting Ascaris lumbricoides.
6 otic gene activation in the nematode Ascaris lumbricoides.
7 VD 103-HgR in children infected with Ascaris lumbricoides and investigated the effect of albendazole
9 tamoeba histolytica), and helminths (Ascaris lumbricoides and Trichuris trichiura), as well as two ex
10 n with malaria, Trichuris trichiura, Ascaris lumbricoides, and hookworms were all associated with wor
11 estimate that the prevalence of hookworm, A lumbricoides, and T trichiura among school-aged children
12 space and estimate risk of with hookworm, A lumbricoides, and T trichiura over a grid of roughly 1 m
13 in the parasitic nematode of humans, Ascaris lumbricoides, and the parasitic nematode of dogs, Toxoca
14 oil-transmitted helminths (hookworm, Ascaris lumbricoides, and Trichuris trichiura) are the most wide
15 ommon aeroallergens, and IgG4 and IgE anti-A lumbricoides antibody levels were measured in all childr
16 odeficiency virus type 1 (HIV-1) and Ascaris lumbricoides co-infection has led to significantly impro
18 d IFN-gamma) to CT-B, that infection with A. lumbricoides diminishes the magnitude of this response,
20 eripheral blood leukocytes stimulated with A lumbricoides extract, serum total IgE levels, specific I
21 with the soil-transmitted helminths Ascaris lumbricoides, hookworm (Ancylostoma duodenale and Necato
22 ons with soil-transmitted helminths (Ascaris lumbricoides, hookworm, and Trichuris trichiura) are wid
24 oral cholera vaccine CVD 103-HgR in Ascaris lumbricoides-infected subjects randomized in a double-bl
26 eta-carotene, extra dietary fat, and Ascaris lumbricoides infection on serum retinol concentrations i
29 To define the cytokine response to Ascaris lumbricoides infection, the cellular immune response to
31 ates and treatment group, suggesting that A. lumbricoides infections impair the immune response to or
32 Analysis of these data indicates that A. lumbricoides infections in endemic regions are associate
37 access was associated with lower odds of A. lumbricoides (OR 0.40, 95% CI 0.39-0.41) and T. trichiur
38 richiura (OR 0.61, 95% CI 0.50-0.74), and A. lumbricoides (OR 0.62, 95% CI 0.44-0.88), but not with h
39 tercoralis, T. trichiura, M. perstans and A. lumbricoides prevalence was estimated as 51%, 22%, 12%,
41 kU/L), whereas negative results for Ascaris lumbricoides, T gondii, herpes simplex virus, and EBV we
42 ing susceptibility to infection with Ascaris lumbricoides, the most common soil-transmitted intestina
43 d helminth infections (ie, hookworm, Ascaris lumbricoides, Trichuris trichiura) with the Kato-Katz me
44 improved WASH on infection with STH (Ascaris lumbricoides, Trichuris trichiura, hookworm [Ancylostoma
45 Iodamoeba butschlii, Endolimax nana, Ascaris lumbricoides, Trichuris trichiura, Strongyloides stercor
48 ch-specific IgE [aGMR 2.37 (1.39, 4.06)], A. lumbricoides with wheeze in participants >/=5 years [aOR
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