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1 g, and to refine the RAR beta effects in the lung field.
2 th factor Fgf10 and bud morphogenesis in the lung field.
3 en continues to deposit in the radio-exposed lung field.
4 icant for the lower lobes but not the middle lung fields.
5 axial helical CT through the upper and lower lung fields.
6  <=20 degrees , and an adequate view of both lung fields.
7    It was most common in bilateral posterior lung fields.
8 glass opacities in the right upper and lower lung fields.
9 led diffuse granular shadow in the bilateral lung fields and "Tree-in-bud appearance" in the peripher
10  tended to underestimate the activity in the lung fields and in chest tumors (slope = 0.79 and 0.94,
11 ocal opacities or opacities located in upper lung fields and predominant airway involvement, in addit
12 uating the extent of involvement over 6 or 8 lung fields and the finding of alveolar opacities in the
13 suitable for scanner calibration assessment, lung fields, and 6 hot spheric lesions with diameters ra
14  silicotic and control animals for the total lung field, but there were no statistically significant
15 sts that the buccal epithelium is altered in lung field carcinogenesis.
16 ing systems taking into consideration 6 or 8 lung fields (designated 6A, 6B, and 8) to quantify lung
17                       The number of affected lung fields for the two CXRs was assessed using the AI t
18 ate the amount of contrast that entered each lung field from the start of contrast injection and unti
19  range, 2 to 20 Gy) to a large volume (whole-lung field) had a high risk of breast cancer (standardiz
20 ccurately predicted alveolitis in the middle lung fields, HRCT did not detect all sites of inflammati
21            Chest radiographs revealed normal lung fields in 24 patients and streaky bibasilar densiti
22 ntrilobular nodules throughout the bilateral lung fields in a chest CT scan, and mixed ventilatory im
23 omputational model started by segmenting the lung field into six subregions.
24 wed bud formation and gene expression in the lung field of both Raldh2-null and BMS493-treated foregu
25     Progression of pneumonic extension >=0.5 lung fields per day compared to pneumonia onset, detecte
26  sufficient to rescue the absence of primary lung field specification in embryos overexpressing Dkk1,
27 controls Tgfbeta activity in the prospective lung field to allow local expression of Fgf10 and induct
28 we show that RA activity is essential in the lung field to maintain lung cell identity in the endoder
29 ted, but the liver was not enlarged, and the lung fields were clear.
30            ROIs were defined by dividing the lung field with equally spaced coronal, sagittal, and tr