1 th factor Fgf10 and bud morphogenesis in the lung field.

2 g, and to refine the RAR beta effects in the lung field.

3 icant for the lower lobes but not the middle lung fields.

4 axial helical CT through the upper and lower lung fields.

5 glass opacities in the right upper and lower lung fields.

6  tended to underestimate the activity in the lung fields and in chest tumors (slope = 0.79 and 0.94,

7 suitable for scanner calibration assessment, lung fields, and 6 hot spheric lesions with diameters ra

8  silicotic and control animals for the total lung field, but there were no statistically significant

9 sts that the buccal epithelium is altered in lung field carcinogenesis.

10  range, 2 to 20 Gy) to a large volume (whole-lung field) had a high risk of breast cancer (standardiz

11 ccurately predicted alveolitis in the middle lung fields, HRCT did not detect all sites of inflammati

12            Chest radiographs revealed normal lung fields in 24 patients and streaky bibasilar densiti

13 ntrilobular nodules throughout the bilateral lung fields in a chest CT scan, and mixed ventilatory im

14 wed bud formation and gene expression in the lung field of both Raldh2-null and BMS493-treated foregu

15  sufficient to rescue the absence of primary lung field specification in embryos overexpressing Dkk1,

16 controls Tgfbeta activity in the prospective lung field to allow local expression of Fgf10 and induct

17 we show that RA activity is essential in the lung field to maintain lung cell identity in the endoder

18            ROIs were defined by dividing the lung field with equally spaced coronal, sagittal, and tr