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1 wer endometrial expression of FST during the luteal phase.
2 id-follicular phase to the symptomatic, late luteal phase.
3 ncreased from the mid-follicular to the late luteal phase.
4 r for half of the menstrual cycle during the luteal phase.
5 teins (P = 5.62E-4) were elevated during the luteal phase.
6 e of 18 proteins that best distinguished the luteal phase.
7 n </= 5 ng/mL and no LH peak in the mid/late luteal phase.
8 rgery during the follicular phase versus the luteal phase.
9 its precursors) were associated with shorter luteal phase.
10 eft fronto-polar cortex more than during the luteal phase.
11 ating hormone and luteinizing hormone in the luteal phase.
12 d a 0.06-log(PdG) decrease (p = 0.03) in the luteal phase.
13 ficantly associated with length of the prior luteal phase.
14 icular phase and then decreased again in the luteal phase.
15 ls the entire night in the luteal and pseudo luteal phase.
16 se, and 0.76 +/- 0.07 kJ/min during the late luteal phase.
17 ity (P = 0.09) during menses than during the luteal phase.
18 -cycle compared with both the follicular and luteal phases.
19 E1 were determined during the follicular and luteal phases.
20 follicular phase to the mid luteal and late luteal phases.
21 levels during the follicular, ovulatory, and luteal phases.
22 easured in the early and late follicular and luteal phases.
23 sion showed that decreases in follicular and luteal phase 17beta-estradiol levels were positively ass
24 hthalate (MCOP) were associated with shorter luteal phase [2nd tertile vs. 1st tertile: -0.5 days (95
26 men comprised the study cohort: 230 (28%) in luteal phase; 363 (44%) in follicular phase; and 241 gro
27 ase (59 [17]) compared with women during the luteal phase (53 [14]) and compared with men (46 [16]; P
28 tal cortex and amygdala more than during the luteal phase (6-10 days after luteinizing hormone surge)
30 ition of uILCs in the endometrium during the luteal phase and in the decidua during early pregnancy.
31 traception could mimic the high-progesterone luteal phase and predispose women to human immunodeficie
32 cular phase, 0.70 +/- 0.10 kJ/min during the luteal phase, and 0.76 +/- 0.07 kJ/min during the late l
33 e onset of melatonin levels for women in the luteal phase, but it had little effect on melatonin leve
36 poradic anovulation, irregular cycle length, luteal phase deficiency, long menses, and heavy blood lo
37 mones measured either midcycle or during the luteal phase, despite good statistical power to detect m
39 e anxiety and dysphoria associated with late luteal phase dysphoria disorder and major unipolar depre
40 lection of gene expression profiles from mid-luteal phase endometrial biopsies (n = 115) from women e
41 o [OR] 1.11, 95% CI 1.03-1.20; p=0.0063) and luteal phase endometrial thickness lower (0.90, 0.83-0.9
43 5-1988) of a prospective study, midcycle and luteal-phase estrogens and progestins were measured in 1
48 articular susceptibility observed during the luteal phase in nonhuman primate models and ex vivo huma
49 th, 16.0 (standard deviation, 4.4) days; and luteal phase length, 12.9 (standard deviation, 1.7) days
51 and increased follicular phase and decreased luteal phase lengths; Hispanic ethnicity with anovulatio
52 sing linear mixed models for follicular- and luteal-phase lengths, discrete-time fecundability models
53 ese chemicals in relation to follicular- and luteal-phase lengths, time to pregnancy, and early pregn
55 any of the following cycle endpoints: short luteal phase (< or = 10 days), long follicular phase (>
56 to an increased risk for anovulation, short luteal phase (< or =10 days), long follicular phase (> o
59 ons comparable to levels observed during the luteal phase of premenopausal women and were significant
61 ats mimics the progesterone component of the luteal phase of the human menstrual cycle, these finding
62 ature levels in women were tested during the luteal phase of the menstrual cycle (n=30) or the pseudo
63 omen using no long-term contraceptive in the luteal phase of the menstrual cycle also had a 3.25 time
64 h levels comparable to those observed in the luteal phase of the menstrual cycle and modestly increas
66 egnanolone levels from the follicular to the luteal phase of the menstrual cycle by blocking the conv
67 s were significantly (P=0.0078) lower in the luteal phase of the menstrual cycle compared to the foll
68 Changes in neurosteroid levels during the luteal phase of the menstrual cycle may precipitate affe
69 onadotropin secretion was blocked during the luteal phase of the menstrual cycle with a gonadotropin-
70 istered to female rhesus macaques during the luteal phase of the menstrual cycle, 40 min before admin
71 T cell populations were detected during the luteal phase of the menstrual cycle, and longitudinal an
73 often occur during pregnancy and during the luteal phase of the menstrual cycle, when levels of prog
80 rovided) were measured in the follicular and luteal phases of 2 menstrual cycles before a single inje
81 of the first menstrual cycle and during the luteal phases of both the first and third menstrual cycl
83 l females, and females in the follicular and luteal phases of the menstrual cycle (FDR-adjusted p-val
85 e glucuronide (E1G) in the periovulatory and luteal phases of the menstrual cycle, and to assess the
88 ding with the follicular, periovulatory, and luteal phases of their menstrual cycle were studied.
93 s increased sixfold to eightfold in the late luteal phase (P < 0.001) and those of swelling or bloati
97 nsive pregnancies were tested during the mid-luteal phase (PRE) and early pregnancy (EARLY; 6.2 +/- 1
98 previously, we showed more inhibition in the luteal phase relative to the midfollicular menstrual pha
102 inary sodium loss, not retention, during the luteal phase; severity of menstrual symptoms was unchang
104 ed in women with PMDD from follicular to the luteal phases, suggesting the absence of effect of the l
106 t is greater during the early follicular and luteal phases than in the late follicular (periovulatory
109 re collected, characterized as follicular or luteal phase using days since last menstrual period, and
110 ng postovulation (average of luteal and late luteal phases), when it was 0.73 +/- 0.07 kJ/min, compar
111 antly higher than those measured in the late luteal phase, whereas aging and cigarette smoking reduce
112 mmunocytochemically detectable GAL-R1 in the luteal phase, whereas only a twentieth expressed GAL-R1
113 offer candidate mechanisms through which the luteal phase, wherein progesterone is dominant relative
114 modeling and leukocyte infiltration with the luteal phase, which may represent potential hormone-asso
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