ライフサイエンスコーパス: lymph

1 living frogs is produced by a combination of lymph and glandular emission, with pigmentary cell filte

2 Exosomes in postshock mesenteric lymph are key mediators of acute lung injury triggering

3 ons coordinated along lymphangions to propel lymph, but the underlying signalling pathways are unknow

4 downstream mesenteric lymphatic vessels and lymph drainage into mesenteric lymph nodes may be compro

5 The model indicates that lymph flow shapes intranodal CCL21 gradients, and that C

6 Levels of S1P in liver, bone marrow, and lymph fluid were measured using an enzyme-linked immunos

7 cases with features of B cell-rich tertiary lymph follicles, along the cortical meningeal surface.

8 e of intracellular transport in steady-state lymph formation and suggest that LECs use transcellular

9 ling by ROS levels in the PSC/niche controls lymph gland hematopoiesis under parasitism.

10 parasitism non-cell autonomously induces the lymph gland immune response.

11 n with a loss of the majority of the primary lymph gland lobes.

12 In the Drosophila hematopoietic organ, the lymph gland, the posterior signaling center (PSC) acts a

13 blood, secondary lymphoid organs (SLOs), and lymph in the steady state.

14 rectal liver metastasis (CRLM), intrahepatic lymph invasion and lymph node metastasis are poor progno

15 fficking of tissue dendritic cells (DCs) via lymph is critical for the generation of cellular immune

16 nflammatory mediators carried via mesenteric lymph (ML).

17 d localization with, T cells in the draining lymph node (dLN).

18 cluded 17 patients with ipsilateral axillary lymph node (LN) metastases.

19 To identify indicators of lymph node (LN) metastasis in thin melanoma in a large,

20 DR and PD-1 were measured in blood (n = 48), lymph node (LN; n = 9), and rectal tissue (n = 17) from

21 tokine, and Tgfbeta expression in mesenteric lymph node (MLN) CD4(+) T cells and jejunum were monitor

22 er of the primary lesion (T), of the largest lymph node (N), and of the largest metastatic lesion (M)

23 rgical Oncology (SSO) guideline for sentinel lymph node (SLN) biopsy in melanoma.

24 Conclusion Women without sentinel lymph node (SLN) metastases should not receive axillary

25 , retropharyngeal lymph node, nasopharyngeal lymph node and pharyngeal tonsil collected at the peak o

26 ongitudinal intravital imaging of the murine lymph node and surrounding structures for up to 14 d.

27 ed for (99m)Tc activity, which established a lymph node as an SLN.

28 the results clearly identified the sentinel lymph node basin and delineated the lymphatic drainage.

29 precisely BCL2-IGH translocations present in lymph node biopsies of follicular lymphoma patents.

30 Observations: Sentinel lymph node biopsy (SLNB) after NAC in patients presentin

31 concerting lymphedema rates in both sentinel lymph node biopsy (SLNB) and axillary lymph node dissect

32 he extension of neoplastic cells through the lymph node capsule into the perinodal adipose tissue.

33 was observed in ex vivo culture of cervical lymph node cells and splenocytes, indicating that in all

34 (+) T cells and IL-5 and IL-13 production by lymph node cells but had no effect on IgE production.

35 Lymph node cells from untreated HIV-infected individuals

36 ype of cancer with an inherent potential for lymph node colonization, which is generally preceded by

37 stics were not improved with the addition of lymph node CT morphology criteria.

38 Mesenteric lymph node cultures from VDR KO and B-VDR KO mice secret

39 roarray in Node-Negative and 1 to 3 Positive Lymph Node Disease May Avoid Chemotherapy) study to eval

40 ode dissection (SLND) alone without axillary lymph node dissection (ALND) is noninferior to that of w

41 ntinel lymph node biopsy (SLNB) and axillary lymph node dissection (ALND) may be because of unrecogni

42 (SLN) metastases should not receive axillary lymph node dissection (ALND).

43 entinel node biopsy (SNB) to extended pelvic lymph node dissection (ePLND) remains controversial.

44 Minimally invasive inguinal lymph node dissection (MILND) is a novel approach to ing

45 with breast-conserving therapy and sentinel lymph node dissection (SLND) alone without axillary lymp

46 survival for patients treated with sentinel lymph node dissection alone was noninferior to overall s

47 front SLNB and reduces the need for axillary lymph node dissection compared with SLNB prior to NAC.

48 dings do not support routine use of axillary lymph node dissection in this patient population based o

49 We have previously found that axillary lymph node dissection, both clinically and in a mouse mo

50 y followed by radical cystectomy with pelvic lymph node dissection, which disclosed residual high-gra

51 ed by breast-conserving surgery and axillary lymph node dissection, which revealed residual disease i

52 all survival for those treated with axillary lymph node dissection.

53 may require chemotherapy, radiotherapy, and lymph node dissection.

54 the primary producer of IL-4 in the reactive lymph node during type 2 immune responses.

55 G uptake by day 4 postinfection with minimal lymph node enlargement, indicating that elevated cell me

56 endothelial venules and exhibited defects in lymph node entrance and egress.

57 Lobectomy with systematic lymph node evaluation remains the recommended treatment,

58 antigen-induced T cell expansion in draining lymph node experiments.

59 uently CXCR5(+) and entered and persisted in lymph node follicles throughout the follow-up (240 d pos

60 livering antigens and soluble factors to the lymph node for immune surveillance.

61 vere loss of dendritic cells in the draining lymph node had no impact on viral replication in this or

62 However, lymph node hyperplasia was clearly visible postviremia b

63 endritic cells and T cells into the draining lymph node immediately following infection and for the r

64 We developed a quantitative PCR-based lymph node infiltration assay to address the slowness of

65 Lymph node inflammation was higher in HIV-infected indiv

66 0 days or less (HR, 2.6; P = 0.01) and hilar lymph node invasion (HR = 2.2; P = 0.03), but not pre-LT

67 This may explain our findings of increased lymph node invasion and new metastatic sites in 30% of s

68 positive surgical margins, and/or pathologic lymph node invasion.

69 the pathological tumor stage and presence of lymph node invasion.

70 l disease, T stage, resection margin status, lymph node involvement, and postoperative complications.

71 lude intestinal lymphangiectasia, mesenteric lymph node lymphadenopathy, and lymphangiogenesis in bot

72 n from the iliac crests and in vivo sentinel lymph node mapping were performed during open standard o

73 situ hybridization, fluorescence imaging for lymph node mapping, nonmalignant lesions, nonsurgical pu

74 ed UNO (FAM-UNO) homed to tumor and sentinel lymph node MEMs in different cancer models: 4T1 and MCF-

75 ignificantly associated with the presence of lymph node metastases and invasive tumor stages.

76 platin-based chemotherapy, and it eliminated lymph node metastases by targeting CSCs and the tumor bu

77 r overall survival of patients with sentinel lymph node metastases treated with breast-conserving the

78 -DCFPyL and (18)F-PSMA-1007 for local tumor, lymph node metastases, and bone metastases.

79 tudy population was limited to patients with lymph node metastases, tumors 4 cm or larger, or local e

80 h disease progression and a higher number of lymph node metastases.

81 ive, early-stage breast cancer without overt lymph node metastases.

82 with stage III cutaneous melanoma (excluding lymph node metastasis </=1 mm or in-transit metastasis)

83 e positively correlated with invasion depth, lymph node metastasis and negatively correlated with the

84 encoding cIAP1 and cIAP2, is associated with lymph node metastasis and poor clinical outcome in OSCC.

85 To ascertain the roles of cIAP1/cIAP2 in lymph node metastasis and radioresistance, we use an in

86 asis (CRLM), intrahepatic lymph invasion and lymph node metastasis are poor prognostic factors.

87 difference between pulmonary metastasis and lymph node metastasis showing that the androgen receptor

88 h aggressive features that include increased lymph node metastasis, reduced responsiveness to neoadju

89 relation of 11q22.1-q22.2 amplification with lymph node metastasis, reduced survival, and increased c

90 ntly associated with histological grades and lymph node metastasis.

91 entral tumour, the tumour invasive front and lymph node metastasis.

92 tively correlated with tumor stage, size and lymph node metastasis.

93 The lymph node periphery is an important site for many immun

94 squamous cell carcinoma of the anus (SCCA), lymph node positivity (LNP) indicates poor prognosis for

95 ent age is an important factor in estimating lymph node positivity in thin melanoma independent of tr

96 tions, more positive lymph nodes, and higher lymph node ratios (P < 0.05).

97 Lymph node recurrence following CRLM resection was assoc

98 es was distinct and could be used for guided lymph node resection, such as by using Cerenkov luminesc

99 Absolute lymph node retrieval was not related to survival (P = 0.

100 fied PCV3 antigen in skin, kidney, lung, and lymph node samples localized in typical PDNS lesions, in

101 wed amelioration of lymphoproliferation with lymph node sizes and spleen volumes reduced by 39% (mean

102 A better EFS was associated with negative lymph node status ( P < .01) and absence of LOH 1p or 16

103 only 74% in patients with combined positive lymph node status and LOH 1p or 16q.

104 leason grade, pathological T score, positive lymph node status and primary therapy failure.

105 that WNV may migrate from the skin into the lymph node through another mechanism.

106 To evaluate the impact of lymph node yield (LNY) on survival in patients treated w

107 Lymph node yield has been used as a surrogate for extent

108 Outcomes according to lymph node yield were determined.

109 or positive resection margins, and >/=23 for lymph node yield.

110 103(+) dendritic cells, in the lung-draining lymph node, as well as increased expression of the costi

111 issues bronchial lymph node, retropharyngeal lymph node, nasopharyngeal lymph node and pharyngeal ton

112 K) cells are located at the periphery of the lymph node, predominantly in the medulla, and we found t

113 esions and in the lymphoid tissues bronchial lymph node, retropharyngeal lymph node, nasopharyngeal l

114 dent changes in RNA profiles of the draining lymph node, suggesting a change in cell profile followin

115 duction in peripheral CD4 memory T cells and lymph node-derived follicular helper T cells of patients

116 excessive IFN-gamma production by blood and lymph node-derived T cells of patients with CVID with im

117 In solid tumors, the presence of lymph node-like structures called tertiary lymphoid stru

118 phoid-tissue inducer (LTi) cells residing in lymph node-like structures in the gut called solitary in

119 erall survival of lymph node-positive versus lymph node-negative patients decreased significantly fro

120 %, the hazard ratios for overall survival of lymph node-positive versus lymph node-negative patients

121 n of any age with hormone receptor-positive, lymph node-positive, and operable breast cancer for whic

122 disease correctly identified in the sentinel lymph node.

123 endritic cells of the metastasis-infiltrated lymph node.

124 piration of a palpable, ipsilateral axillary lymph node.

125 36 of whom had at least one mapped sentinel lymph node.

126 ent lymph nodes and 1 pig exhibited a single lymph node.

127 d to calculate the amount of dye within each lymph node.

128 enal antigens to CD8 T cells in the draining lymph node.

129 inical trials will evaluate whether sentinel-lymph-node biopsy can be avoided altogether in selected

130 Refinement of the sentinel-lymph-node biopsy technique might overcome the slightly

131 prove this concept, trans-/intramucosal and lymph-node delivery of PLGA-PEG nanoparticles was demons

132 rker molecular assay to immediate completion lymph-node dissection (dissection group) or nodal observ

133 -lymph-node involvement, completion axillary-lymph-node dissection can be omitted or replaced by axil

134 Immediate completion lymph-node dissection was not associated with increased

135 elper T cells, thymic T cell development and lymph-node genesis.

136 For selected patients with limited sentinel-lymph-node involvement, completion axillary-lymph-node d

137 injection of indocyanine green and sentinel-lymph-node mapping followed by pelvic lymphadenectomy wi

138 Sentinel-lymph-node mapping has been advocated as an alternative

139 de-positive disease with successful sentinel-lymph-node mapping who had metastatic disease correctly

140 Sentinel-lymph-node mapping with complete pelvic lymphadenectomy

141 colorectal cancers that was associated with lymph-node metastases (INHBB, AXL, FGFR1, and PDFGRB) an

142 ration but no apoptosis, presenting frequent lymph-node metastasis.

143 rimary endpoint, sensitivity of the sentinel-lymph-node-based detection of metastatic disease, was de

144 s CTVs were bone (23/52, 44%) and perirectal lymph nodes (16/52, 31%).

145 on therapy to the primary tumor and regional lymph nodes (45 to 54 Gy) plus eight once-weekly doses o

146 rine melanoma cell line highly metastatic to lymph nodes (B16F10) was implanted subcutaneously on the

147 of peripheral activation events in cervical lymph nodes (CLN) to driving humoral immune responses in

148 intestinal lamina propria (LPL) and cervical lymph nodes (CLN).

149 T cells, were enriched in SIV DNA in blood, lymph nodes (LN), spleen, and gut, and contained replica

150 erns, highest in blood, bone marrow (BM), or lymph nodes (LN), with the frequency and function in blo

151 ells underwent several divisions in draining lymph nodes (LN; DLNs) while maintaining expression of T

152 pathogenic, B cells rapidly increase in both lymph nodes (LNs) and intestine.

153 reticular cells (FRCs) in the T cell zone of lymph nodes (LNs) are pivotal for T cell survival, mobil

154 ent co-delivery of CpG and antigens (Ags) to lymph nodes (LNs) by albumin/AlbiVax than benchmark inco

155 or required for homing to GCs) and expand in lymph nodes (LNs) following pathogenic SIV infection in

156 f primary tumour and locoregional metastatic lymph nodes (LNs) in breast cancer and to look for poten

157 zation of MHCII-bound peptides isolated from lymph nodes (LNs) of C57BL/6 mice.

158 .25-294 ng/mL), 362 (68)Ga-PSMA PET-positive lymph nodes (LNs) were identified.

159 The NQF endorses evaluating >/=12 lymph nodes (LNs), adjuvant chemotherapy (AC) for stage

160 osal tissues (lungs, intestines), associated lymph nodes (LNs), and other lymphoid sites from 78 indi

161 activation are asymmetrically distributed in lymph nodes (LNs), but how this affects adaptive respons

162 n HIV, inflammation is also increased within lymph nodes (LNs), tissues known to harbor the virus eve

163 7 drives leukocyte migration into and within lymph nodes (LNs).

164 ion of cellular immune responses in draining lymph nodes (LNs).

165 psy sites were bone (n = 7), pleura (n = 3), lymph nodes (n = 2), and liver (n = 2).

166 eral blood (PB) and from pancreatic draining lymph nodes (PLN) of T1D patients and non-diabetic subje

167 rivesical fat and involving two of 20 pelvic lymph nodes (pT3N2).

168 ral DNA levels in PBMCs after 2 weeks and in lymph nodes after 10 weeks.

169 odes were detected; 2 pigs had 2 fluorescent lymph nodes and 1 pig exhibited a single lymph node.

170 exhibited reduced lymphocyte trafficking to lymph nodes and a corresponding increase in T cell popul

171 but were barely detectable within noninvaded lymph nodes and absent in peripheral blood.

172 ncies of ZIKV-specific Ab-secreting cells in lymph nodes and bone marrow, correlating with low Ab tit

173 re present in para-aortic adipose tissue and lymph nodes and display an inflammatory-like phenotype a

174 homing of tumor-infiltrating DC to draining lymph nodes and increased infiltration of T cells into t

175 it the entry of circulating lymphocytes into lymph nodes and long-term parabiosis experiments, we hav

176 ositively correlated with enlargement of the lymph nodes and peaked on day 10 postinfection.

177 ilar to DCs, they homeostatically migrate to lymph nodes and present antigen to antigen-specific T ce

178 4 production by CD4(+) T cells isolated from lymph nodes and prevented IgE-dependent oral allergen-in

179 sustained on allergen recall response in the lymph nodes and spleen.

180 uripotency and the ability to migrate to the lymph nodes and spleen.

181 +) DCs, which enhances migration to draining lymph nodes and Th2 priming capacity.

182 In particular, they could shuttle between lymph nodes and the CNS and produced encephalitogenic cy

183 These cells traffic between lymph nodes and the skin, and are identified by their co

184 are trapped for long periods of time within lymph nodes and the spleen in the steady state.

185 uNP that targeted myeloid dendritic cells in lymph nodes as a peptide antigen carrier, substantially

186 astatic lesions in the liver, bone, lung, or lymph nodes before and after Sandostatin LAR administrat

187 68% of lungs and 36% of spleens and cervical lymph nodes but fewer than 20% of axillary lymph nodes,

188 None of the three sentinel lymph nodes contained metastatic carcinoma.

189 Two of the 11 axillary lymph nodes contained metastatic carcinoma.

190 ble axillary adenopathy, and 1 or 2 sentinel lymph nodes containing metastases, 10-year overall survi

191 ble axillary adenopathy, and 1 or 2 sentinel lymph nodes containing metastases.

192 f the type 2 immune response in the reactive lymph nodes during parasitic helminth infection.

193 w in mice that local delivery of Rapa MPs to lymph nodes during vaccination either suppresses or enha

194 of NKT cells at the interfollicular areas of lymph nodes facilitates both their direct priming by res

195 port a role of macrophage efflux to draining lymph nodes following treatment with infliximab.

196 eally injected eosinophils into paratracheal lymph nodes from distal alveolar lung was diminished in

197 d immune activation in vivo, was enhanced in lymph nodes from pigtailed macaques infected with simian

198 patients with adenocarcinoma, 41 (93.2%) had lymph nodes harvested, with nodal metastases in only 14

199 rostate bed in 27% of patients, locoregional lymph nodes in 39%, and distant metastatic disease in 16

200 nto the lung lumen, parenchyma, and draining lymph nodes in HDM-sensitized mice.

201 FDG followed by Cerenkov-guided resection of lymph nodes in healthy mice has previously been introduc

202 he airways and on DC subsets in the lung and lymph nodes in murine model of allergic airway inflammat

203 ary dendritic cells in the lung and draining lymph nodes in wild-type BALB/c mice after RSV infection

204 Nineteen of 26 lymph nodes involved by NLPHL demonstrated a population

205 c vessels and lymph drainage into mesenteric lymph nodes may be compromised.

206 but (68)Ga-PSMA-11 PET/CT showed additional lymph nodes metastasis.

207 l metastases were identified in the sentinel lymph nodes of 35 (97%) of these 36 patients, yielding a

208 ed in CD4+ T cells isolated from spleens and lymph nodes of arthritic mice treated with CM-MSC or MSC

209 resulted in detectable virus in brain and/or lymph nodes of fetuses and/or pups.

210 maintain the Th2 response in the mesenteric lymph nodes of infected mice.

211 ation of viral Gag and CD169 was observed in lymph nodes of infected pigtailed macaques, suggesting p

212 ioned the in vivo mobilization to mesenteric lymph nodes of intestinal migratory CD103(+) DCs carryin

213 In contrast, the bone marrow and lymph nodes of nonsurvivors showed increased [(18)F]-FDG

214 G35-55-specific T cells in the skin draining lymph nodes of primed mice, but it is not required for t

215 In lymph nodes of surviving monkeys, changes in [(18)F]-FDG

216 involvement (M1a or M1b due to extrathoracic lymph nodes only) or disseminated metastatic disease.

217 cell migration from the skin to the draining lymph nodes plays a prominent role in activating systemi

218 on therapy to the primary tumor and regional lymph nodes plus eight once-weekly doses of concurrent c

219 .01), but a higher proportion of cases with lymph nodes positive for cancer (40.9% [442 of 1080] vs

220 The number of positive lymph nodes seems to be an appropriate selection factor

221 e out of the skin and mucosa to the draining lymph nodes to present antigens to T and B cells.

222 In selected mice, popliteal lymph nodes underwent Cerenkov luminescence imaging.

223 es from the oral cavity, larynx-pharynx, and lymph nodes using 16S rRNA sequencing.

224 n detection and identification of metastatic lymph nodes was distinct and could be used for guided ly

225 inhibition, positing that efflux to draining lymph nodes was involved.

226 T cells subsets in blood, spleen and lymph nodes were detected dynamically by flow cytometry.

227 A total of 5 fluorescent lymph nodes were detected; 2 pigs had 2 fluorescent lymp

228 the intestinal lamina propria and mesenteric lymph nodes were GFP(+) However, in vitro infection of t

229 The six dissected lymph nodes were negative for malignancy.

230 Several peripancreatic lymph nodes were observed that measured up to 11 mm x 5

231 Sentinel lymph nodes were successfully detected in all patients.

232 x transcription factor (T-bet)(+) B cells in lymph nodes, and an accumulation of T-bet(+)CD21(low) B

233 e tumor cells can escape into the blood, the lymph nodes, and at times the visceral organs.

234 oE(-/-)Irf5(-/-) mice in the aorta, draining lymph nodes, and bone marrow cell cultures, indicating t

235 hearts, pericardial AT, spleen, mediastinal lymph nodes, and bone marrow were quantified by flow cyt

236 eural invasion, R1 resections, more positive lymph nodes, and higher lymph node ratios (P < 0.05).

237 , with occasional viremia; tonsil, mesentery lymph nodes, and intestinal mucosa served as major targe

238 neck disease, increasing number of positive lymph nodes, and lower neck disease.

239 -day mortality, the total number of resected lymph nodes, and R0 resection rates was evaluated with m

240 e 30-day mortality, total number of resected lymph nodes, and R0 resection rates.

241 spite extensive sampling from ileum, rectum, lymph nodes, bone marrow, CSF, circulating CD4+ T cell s

242 seminal vesicles, local lymph nodes, distant lymph nodes, bone, and others) at both time points by vi

243 al vesicle, or extraprostatic, including all lymph nodes, bone, or soft-tissue metastasis), and subje

244 tromal cells regulate leukocyte responses in lymph nodes, but the role of stromal cells in adipose ti

245 sent within tumors and invaded or metastatic lymph nodes, but were barely detectable within noninvade

246 into account the number of involved axillary lymph nodes, clinical tumour stage, oestrogen-receptor s

247 lithiasis, infiltration, biliary dilatation, lymph nodes, complications.

248 C(+) macrophages in the ankles and popliteal lymph nodes, decreased migration of monocytes into the a

249 locations (prostate, seminal vesicles, local lymph nodes, distant lymph nodes, bone, and others) at b

250 to the chest wall and the draining regional lymph nodes, followed by an optional mastectomy scar boo

251 gens to antigen-presenting cells in draining lymph nodes, leading to increased surface presentation w

252 cutaneous tumors were metastatic to regional lymph nodes, liver and lung.

253 l lymph nodes but fewer than 20% of axillary lymph nodes, livers, brown fat samples, kidneys, or bloo

254 t the stomach, with metastases documented in lymph nodes, lung, and liver.

255 haalpha T cells, and CD8alphabeta T cells in lymph nodes, peripheral blood, and bronchoalveolar lavag

256 ) and 54 (67%) metastatic and non-metastatic lymph nodes, respectively.

257 ductions in bacterial burden in the draining lymph nodes, spleen, and liver were observed.

258 c CD8(+) T cells were detected in the blood, lymph nodes, vagina, cervix, uterus, and fallopian tubes

259 essing melanocytes localize to skin-draining lymph nodes, where they induce T-cell proliferation and

260 phatic flow from the donor graft to draining lymph nodes, which may be a factor in promoting cellular

261 ll responses within the local draining iliac lymph nodes, yet robust Th1 and Th17 responses were prom

262 fewer DCs carrying parasite antigens to the lymph nodes.

263 ale for surgical resection of tumor-draining lymph nodes.

264 nd meninges to the peripheral (CNS-draining) lymph nodes.

265 cantly between metastatic and non-metastatic lymph nodes.

266 ongly correlated with metastasis to lung and lymph nodes.

267 cord while wild type DC migrated to cervical lymph nodes.

268 survival of central memory T (TCM) cells in lymph nodes.

269 reduction in IL-17(+) cells in the draining lymph nodes.

270 ation of the immune response in the draining lymph nodes.

271 ultiple tissues in patients, particularly in lymph nodes.

272 spensable for their activation in pancreatic lymph nodes.

273 occurred only in the regional skin-draining lymph nodes.

274 prefer to metastasize to lung rather than to lymph nodes.

275 kine secretion and proliferation in regional lymph nodes.

276 y a major role in viral reservoir control in lymph nodes.

277 before leaving the inflamed skin to draining lymph nodes.

278 ed in duodenum, ileum, colon, and mesenteric lymph nodes.

279 ed role of the receptor in guiding exit from lymph nodes.

280 emory CD4(+) T cells in peripheral blood and lymph nodes.

281 mphangiogenesis in intestinal lesions and in lymph nodes.

282 tumor status in order to identify metastatic lymph nodes.

283 n exam, and multiple palpable right axillary lymph nodes.

284 4(lo)Foxp3(+) central Treg cells in draining lymph nodes.

285 ells were detected in local genital draining lymph nodes.

286 sitively correlates with local metastases in lymph nodes.

287 ) IFN-gamma(+) T cells in the heart-draining lymph nodes.

288 acrophage M1-like polarization within murine lymph nodes.

289 ophil trafficking from lungs to paratracheal lymph nodes.

290 ial AT, which outnumbered DCs and T cells in lymph nodes.

291 lated from tonsils, gut mucosa, and draining lymph nodes.

292 e in donor cells in the mediastinal draining lymph nodes; increased lymphatic vessel area; and graft

293 traction waves along lymphatic muscle during lymph propulsion.

294 ession, which contributed to a reduced liver:lymph S1P gradient and limited HSC egress from the liver

295 g and hemorrhage as well as enlarged jugular lymph sacs and lymphatic vessels.

296 key epigenetic modifier that maintains blood-lymph separation and integrates both extrinsic forces an

297 The brain lacks lymph vessels and must rely on other mechanisms for clea

298 tasis, for instance, by recruiting blood and lymph vessels.

299 n in plasma, while THC concentrations in the lymph were 100-fold higher than in plasma.

300 CBD concentrations in the lymph were 250-fold higher than in plasma, while THC con