ライフサイエンスコーパス: lymph node cell

1 ts (median frequency, 2 infected cells/10(7) lymph node cells).

2 d decreased STAT4 induction in submandibular lymph node cells.

3 th full-length and cleaved SIRT1 in draining lymph node cells.

4 -12 secretion, respectively, in restimulated lymph node cells.

5 or without measurable BCG mRNA expression in lymph node cells.

6 vel roughly equivalent to 107 unfractionated lymph node cells.

7 -regulation of IL-10 in cultures of draining lymph node cells.

8 s anti-CII IFNgamma production by CII-primed lymph node cells.

9 d upon ex vivo stimulation of vaccine-primed lymph node cells.

10 0 and suppression of IFN-gamma production by lymph node cells.

11 ficiencies in primary IFN-gamma responses by lymph node cells.

12 Egr-dependent FasL promoter activity in DKO lymph node cells.

13 icantly decreased the encephalitogenicity of lymph node cells.

14 IFN-gamma and IL-2 by Ag-activated draining lymph node cells.

15 iferation and IFN-gamma production by primed lymph node cells.

16 ted immunization of BALB/c mice with porcine lymph node cells.

17 proach or by adoptive transfer of spleen and lymph node cells.

18 plasma and elevated IFN-gamma production by lymph node cells.

19 d tyrosine phosphorylation concentrations in lymph node cells.

20 reased CD28 and IFN-gamma mRNA expression in lymph node cells.

21 roduced by spleen, mediastinal, and cervical lymph node cells.

22 not because of transfer of antigen-activated lymph node cells.

23 of interferon-gamma by stimulated mesenteric lymph node cells.

24 ated macrophages, in the draining (cervical) lymph node cells.

25 by schistosome egg Ag-stimulated mesenteric lymph node cells.

26 d Th2 cytokines produced by OVA-restimulated lymph node cells.

27 ut decreased levels of IFNgamma in serum and lymph node cells.

28 n physiological stimulation in mouse primary lymph node cells.

29 In contrast, unseparated lymph node cells acutely rejected both GRKO and wild-typ

30 Eleven of 16 such peptides elicited lymph node cell and spleen cell T cell proliferation in

31 immune gene expression in antigen-stimulated lymph node cells and a poor antibody response.

32 ranscripts were altered by THC in both total lymph node cells and CD4(+) T cells.

33 regulated by THC in super antigen-activated lymph node cells and CD4(+) T cells.

34 Lymph node cells and CD4(+) T-cell lines raised with CW/

35 okine production of activated tumor-draining lymph node cells and enhanced their therapeutic efficacy

36 ic joint damage, and cytokine secretion from lymph node cells and from bone marrow-derived macrophage

37 timulated IFN-gamma production by mesenteric lymph node cells and hepatic egg granuloma size.

38 Cytokine messenger RNA (mRNA) expression in lymph node cells and in synovial cells from arthritic pa

39 imulated proliferation of megalin-sensitized lymph node cells and induced high-titer anti-megalin aut

40 53-specific cells is observed among cervical lymph node cells and intrathyroidal lymphocytes.

41 , prevents binding of freshly isolated mouse lymph node cells and of in vivo activated lymphocytes to

42 t correlation between the frequency of vRNA+ lymph node cells and plasma vRNA.

43 We also show that mouse lymph node cells and purified CD4(+) T cells express tra

44 Their lymph node cells and splenocytes produced a distinct pat

45 Lymph node cells and splenocytes were hypo-responsive to

46 was observed in ex vivo culture of cervical lymph node cells and splenocytes, indicating that in all

47 The T cell receptor Vbeta phenotypes of lymph node cells and T cells infiltrating arthritic join

48 responses (protein and/or mRNA) in draining lymph node cells, and IgG2a vs IgG1 Ab isotypes to IRBP,

49 both cognate and bystander specificities of lymph node cells, and reduced inflammatory lesions and s

50 agen-induced interferon-gamma secretion from lymph node cells, and reduced the levels of tumor necros

51 re rapidly and to greater levels than CD8(+) lymph node cells, and they acquire the phenotype of full

52 Examination of lymph node cell antigen recall responses identified elev

53 study we show that SHIP(-/-) splenocytes and lymph node cells are poor stimulators of allogeneic T ce

54 Data from fractionated lymph node cells as well as rmIL-12-treated B cell-defic

55 Proliferation of CII-stimulated spleen and lymph node cells as well as the change in serum levels o

56 mphoid tissues (Peyer's patch and mesenteric lymph node cells) as a source of soluble factor(s) that

57 d 40-kDa dextran was mostly contained within lymph node cells at both 4 and 24 hours after injection.

58 Prions arrived in draining lymph nodes cell autonomously within two hours of intrap

59 (+) T cells and IL-5 and IL-13 production by lymph node cells but had no effect on IgE production.

60 ts of CD25+-depleted Tgf-beta1-/- spleen and lymph node cells, but suppression was incomplete when co

61 cement of polyclonal immune responses of rat lymph node cells by a Fab fragment from a CD5 mAb shown

62 Adoptive transfer experiments and lymph node cell cocultures were used to investigate the

63 halomyelitis, that autoantigen-sensitized XX lymph node cells, compared with XY, are more encephalito

64 covery that a small subpopulation of CD4high lymph node cells contained all of the alloantigen-specif

65 cell-depleted mice had greatly reduced total lymph node cell counts; 2) the T cells were derived equa

66 L. mexicana Ag-stimulated lymph node cell culture from the immunized/challenged mi

67 Lymph node cells cultured from animals with peak disease

68 e revealed that IL-12 is rapidly produced in lymph node cell cultures from egg-injected mice.

69 sed by the production of interferon-gamma in lymph node cell cultures.

70 ted with periodontal disease-induced IL-6 in lymph node cell cultures.

71 Both MVA-029 and MVA-043 peptide-stimulated lymph node cells degranulated similarly as assessed by A

72 Flow cytometric analysis of popliteal lymph node cells demonstrated similar profiles of T cell

73 f spleen mononuclear cells and peribronchial lymph node cells demonstrated that the response to SRW i

74 production, cytokine elaboration from local lymph node cells, development of airway hyperresponsiven

75 Freshly isolated ovalbumin-immune lymph node cells did not prevent retinitis, indicating t

76 entry mediator, because LIGHT Tg mesenteric lymph node cells do not cause intestinal inflammation wh

77 Splenocytes and lymph node cells draining both the site of inflammation

78 in the face of decreased parasite survival, lymph node cells draining cutaneous lesions of Deltaarg

79 , but not all, of the epitopes recognized by lymph node cells elicited by 6B- and 19F-CRM(197) as wel

80 nses to synthetic peptides demonstrated that lymph node cells elicited by the poorly immunogenic conj

81 n cells of naive animals, that in spleen and lymph node cells exposed to various stimuli was enhanced

82 s of IL-4, IL-5, IL-10 and IL-13 produced by lymph node cells fell significantly in FTY720-treated an

83 Lymph node cells from (SWRxSJL)F1 mice immunized with th

84 detected by flow cytometry in disaggregated lymph node cells from 11 subjects and constituted a sign

85 suppressed in vitro IFN-gamma production by lymph node cells from AChR-immunized, DR3-bearing transg

86 of myeloid suppressor cells, splenocytes and lymph node cells from adult mice with induced SHIP defic

87 ed equally to a mitogenic stimulus, but only lymph node cells from alopecia areata affected mice disp

88 stantiated by the observations that draining lymph node cells from anti-4-1BB-treated mice failed to

89 At this time, lymph node cells from both IL-18+/+ and IL-18-/- mice pr

90 Lymph node cells from burn-injured mice also produced hi

91 Lymph node cells from C57BL/6 (H-2b) recipients of C3H (

92 Spleen and lymph node cells from CII-immunized mice cultured with C

93 ejection was induced by adoptive transfer of lymph node cells from col(V)-immunized rats.

94 Furthermore, adoptive transfer of lymph node cells from diseased chimeras to RAG2-/- recip

95 In addition, draining lymph node cells from FHL2-KO mice show reduced levels o

96 Mediastinal lymph node cells from Flt3L-treated mice secreted higher

97 Within sorted lymph node cells from four HIV-infected individuals, CXC

98 The tAChR-primed lymph node cells from IL-4(-/-) mice vigorously prolifer

99 Lymph node cells from immunized MCP-1(-/-) mice synthesi

100 videnced by the reduced response of isolated lymph node cells from immunized mice to in vitro restimu

101 essed B7.1 expression on APCs in cultures of lymph node cells from immunized mice.

102 When lymph node cells from infected BALB/c mice were stimulat

103 ed Leishmania Ag-specific IL-4 production by lymph node cells from infected BALB/c mice.

104 ation and IFN-gamma production by spleen and lymph node cells from infected mice.

105 Stimulation of mesenteric lymph node cells from infected transgenic mice with para

106 CD4(+) lymph node cells from IRBP-immunized WT mice transferred

107 In this study, a comparison of draining lymph node cells from L. amazonensis- and L. major-infec

108 Restimulated lymph node cells from L. major-infected BALB/c-CXCR3(Tg)

109 2 wk after infection, although Ag-stimulated lymph node cells from L. major-infected IL-18+/+ and IL-

110 ents with lupus nephritis, and IL-23-treated lymph node cells from lupus-prone mice may transfer dise

111 Lymph node cells from lupus-prone mice, but not control

112 Lymph node cells from mice sensitized in the absence of

113 ed proliferative responses were exhibited by lymph node cells from mice treated with Crry-Ig.

114 Lymph node cells from mice treated with p28/p40 blocked

115 Interestingly, antigen-stimulated lymph node cells from MIF(-/-) mice produced more interl

116 Following ex vivo restimulation, draining lymph node cells from misoprostol-treated mice secreted

117 ion and interleukin-17 (IL-17) production by lymph node cells from MKK-6(-/-) mice in response to typ

118 dependently suppress EAE, we purged draining lymph node cells from MOG-immunized, iTreg treated mice

119 Lymph node cells from MOG-primed D6(-/-) mice showed wea

120 Spleen or lymph node cells from naive and BCG-vaccinated guinea pi

121 Polyclonal activation of mesenteric lymph node cells from naive WSX-1 KO or wild-type mice d

122 preceded by adoptive transfer of spleen and lymph node cells from normal untreated BALB/c mice, then

123 onstitute a significant fraction of cervical lymph node cells from older mice deficient in both E- an

124 FN-gamma and IL-17 were detected in draining lymph node cells from P2rx7(-/-) mice.

125 wild type (WT) controls and that spleen and lymph node cells from P2X7R-/- mice proliferated more vi

126 ificant proliferative T cell responses using lymph node cells from peptide-primed mice and production

127 on was significantly enhanced in cultures of lymph node cells from protein-deprived tuberculous anima

128 CD4+ mesenteric lymph node cells from SAMP-1/Yit donor mice were adoptiv

129 Splenocytes and lung lymph node cells from sensitized EP2(-/-) mice produced

130 Finally, the number of parabronchial lymph node cells from sensitized LTC(4)S(null) mice and

131 bition in T-cell activation, because CCR6-/- lymph node cells from sensitized mice produced threefold

132 However, intravenous injection of CCR6-/- lymph node cells from sensitized mice were unable to pri

133 (but not IL-10) were produced by spleen and lymph node cells from several different strains of mice,

134 Mesenteric lymph node cells from STAT6(-/-) mice with colitis exhib

135 2 wk postinfection, Leishmania Ag-stimulated lymph node cells from STAT6-/- mice produced significant

136 Lymph node cells from susceptible C3H and AKR mice also

137 In lymph node cells from the arthritic mice treated with BB

138 d in the in vitro restimulated skin-draining lymph node cells from the EGF-treated mice.

139 Following Ag stimulation in vitro, lymph node cells from the immunized Gal-3-/- mice produc

140 Lymph node cells from the malnourished, infected mice pr

141 ximum activation was present in the draining lymph node cells from the progesterone-treated group, an

142 Lymph node cells from the same recipients displayed redu

143 mma-/- mice resolve L. mexicana lesions, and lymph node cells from these mice produced less IL-10 and

144 control groups was not observed in draining lymph node cells from TLR2(-/-) mice.

145 Isolated lung draining lymph node cells from TLR3-/- mice produced significant

146 could be adoptively transferred by removing lymph node cells from tolerance-induced donors and givin

147 analyzed by flow cytometry in disaggregated lymph node cells from untreated HIV-1-infected women (n

148 Lymph node cells from untreated HIV-infected individuals

149 orreliacidal antibody production by cultured lymph node cells from vaccinated mice peaked soon after

150 Lymph node cells from wild-type and IL-4Ralpha-/- DO11.1

151 We performed adoptive transfer of lymph node cells from wild-type mice or FoxP3-deficient

152 ed in levels of IL-10 and GM-CSF produced by lymph nodes cells from estradiol-treated mice.

153 Transferring lymph nodes cells from UV-irradiated, FITC-sensitized mi

154 een identified among spleen and particularly lymph node cells, further characterization of these cell

155 sage was random, but the CDR3 regions of the lymph node cells had a conserved Gly-Gly motif.

156 In contrast i.v. injection of cervical lymph node cells harvested 8 days after OVA injection in

157 Unlike CD8(+) lymph node cells, IELs express high levels of the FasL g

158 oduction and similar increases in mesenteric lymph node cell IL-4 production were observed in B7-1/B7

159 roliferative responses from tracheobronchial lymph node cells, immunoglobulin M (IgM) and IgG antibod

160 proliferative immune responses of mesenteric lymph node cells in C57BL/6J mice infected with differen

161 ed with controls and suppressed their primed lymph node cells in culture.

162 analysis revealed that the proliferation of lymph node cells in response to antigenic stimulation wa

163 as well as the cytokines produced by primed lymph node cells in response to IRBP showed a distinct p

164 sitized ICE-deficient mice, proliferation of lymph node cells in response to the specific antigen was

165 taining of peripheral lymphocytes as well as lymph node cells in situ.

166 gene expression or cytokine response by host lymph node cells in the developing bubo.

167 iferation of anti-CD3-activated SHP-1 mutant lymph node cells in the presence or absence of IL-12 wer

168 f mice in proliferation indices of spleen or lymph node cells in vitro after stimulation with type II

169 Restimulation of spleen and lymph node cells in vitro yielded T-cell lines that spec

170 kines in vivo as that observed in stimulated lymph node cells in vitro.

171 tion of Th-1-type cytokines in peribronchial lymph node cells in vitro.

172 d spectrum of cytokine responses by cervical lymph node cells in vitro.

173 These changes were not observed on lymph node cells, including those isolated from lymph no

174 Moreover, analysis of pancreatic lymph node cells indicated that lack of mast cells has n

175 ined free of inflammation, even though their lymph node cells induced SMG inflammation in Rag1(-/-) r

176 A and by adoptive transfer of peptide-primed lymph node cells into naive recipient hosts, but neither

177 ated with FTY720, which blocks the export of lymph node cells into peripheral tissue.

178 for CD by adoptive transfer of Tg mesenteric lymph node cells into RAG(-/-) mice.

179 12-induced IFN-gamma secretion from cultured lymph node cells is accessory cell dependent and can be

180 Analysis of lymph node cells isolated from donor and recipient mice

181 motility of syncytia and adhesion to CD4(+) lymph node cells led to the formation of long membrane t

182 hoproliferation, flow cytometric analysis of lymph node cells (LN), or histologic analysis of the kid

183 ignificantly enhance IFN-gamma production by lymph node cells (LNC) and LNC-derived CD4(+) T-cell lin

184 ollowing Ag-specific stimulation of draining lymph node cells (LNC) from males as compared with femal

185 SRW extract or individual peptides s.c. and lymph node cells (LNC) were challenged in vitro.

186 Spleen, but not lymph node, cells lost proliferative response against do

187 suppression was transferable with mesenteric lymph node cells (MLNC) from infected animals to uninfec

188 CD44 on CD4(+) T cells; increased mesenteric lymph node cell numbers; and increased production of imm

189 Its blood and lymph node cells obtained at 49 weeks after intrarectal

190 ce protein A (OspA), was readily produced by lymph node cells obtained from C3H/HeJ mice vaccinated w

191 was detected in supernatants of cultures of lymph node cells obtained on day 7 after vaccination, pe

192 rary was constructed from RNA extracted from lymph node cells of a simian immunodeficiency virus (SIV

193 The draining lymph node cells of anti-CTLA4-treated BALB/c mice produ

194 Lymph node cells of BALB/c mice with progressive leishma

195 the amount of antibody produced by cultured lymph node cells of C3H/HeJ mice vaccinated with outer s

196 mu, were found in the first intron of Myc in lymph node cells of IL-6 transgenic BALB/c mice.

197 w that during the innate immune response the lymph node cells of L. major-infected C3H mice upregulat

198 icant suppression of IL-12 production by the lymph node cells of MBP1-11-injected mice.

199 u) complexes), was increased in the draining lymph node cells of me(v+/-) mice compared with wild-typ

200 fect of THC on global histone methylation in lymph node cells of mice immunized with a superantigen,

201 ndritic cells obtained ex vivo from cervical lymph node cells of NaI-fed or control mice, suggesting

202 in bronchoalveolar lavage fluid and draining lymph node cells of Nur77-KO mice, as well as increased

203 Similarly, lung-draining lymph node cells of pretreated mice secreted significant

204 interferon (IFN)-gamma production ex vivo by lymph node cells of protected mice were not reduced.

205 in supernatants from cultures of mesenteric lymph node cells of SAMP1/YitFc mice ( P < .05 vs vehicl

206 In vitro, lymph node cells of susceptible and resistant mice respo

207 Freshly explanted lymph node cells of treated mice showed inhibition of IF

208 SEA-stimulated bulk mesenteric lymph node cells or CD4 T cells from 7-week-infected WSX

209 with little or no reduction in lung-draining lymph node cells or their cytokine production and with n

210 fic transfusions of splenocytes, thymocytes, lymph node cells, or buffy coat cells can prolong skin a

211 Transfusion of splenocytes, thymocytes, lymph node cells, or buffy coat cells led to prolonged s

212 predicted viral load or HIV-1 RNA-producing lymph node cells (P >/= .24), after adjusting for CD4(+)

213 Donor pancreatic lymph node cells (PLNC) protect islet transplants in Non

214 owed by coadministration of donor pancreatic lymph node cells (PLNCs).

215 mulatory ability resided within the adherent lymph node cell population.

216 c stem cells, present in the donor spleen or lymph node cell populations, in the thymus of irradiated

217 ngle-cell suspensions of spleen and cervical lymph node cells prepared from normal C57BL/6 and SjS-su

218 ive response to mTg, mTg1677, and mTg2342 of lymph node cells primed to each Ag.

219 had high titer IgG1 and no IgG2a, and their lymph node cells produced high levels of IL-4 but no IFN

220 Df-restimulated P2Y(6)-deficient lymph node cells produced higher levels of Th1 and Th2 c

221 NKG2D ligand-negative), and their spleen and lymph node cells produced IFN-gamma in response to RMA b

222 ith each peptide emulsified in CFA, draining lymph node cells produced IFN-gamma on recognition of ce

223 followed by induction of arthritis, inguinal lymph node cells produced IL-4, TGF beta, and IL-10.

224 Mesenteric lymph node cells produced lower levels of cytokines comp

225 h cells from controls, egg Ag-stimulated JHD lymph node cells produced significantly higher amounts o

226 (PLP) sequence 139-151, induced deviated Th2 lymph node cells producing IL-4 instead of IL-2 and aner

227 CTLA-4-deficient (-/-) splenocytes and lymph node cells proliferate without added stimuli in vi

228 This peptide induces lymph node cell proliferation and development of antibod

229 ice was diminished, as were IL-5 production, lymph node cell proliferation, and serum antibody levels

230 at a T cell epitope of alpha3(IV)NC1 induces lymph node cell proliferation, EAG, and intramolecular e

231 SHV-associated diseases, latent infection of lymph node cells provides a mechanism for the persistenc

232 ransforming growth factor beta in mesenteric lymph node cells, purified CD4 T cells, and isolated liv

233 e UV-irradiated donor mice or are induced in lymph node cell recipients and the mechanism of suppress

234 that tolerized proteolipid protein-specific lymph node cells regain the ability to divide, different

235 wk of infection, splenocytes and mesenteric lymph node cells responded to p38/P4 peptides with predo

236 hantavirus, Andes virus, results in a robust lymph node cell response, signatures of T and B cell mat

237 oups exhibited a dominant type 1 response in lymph node cells restimulated ex vivo, the expression of

238 Activation of normal lymph node cells resulted in increased expression of Egr

239 w cytometric analysis of isolated mesenteric lymph node cells revealed evidence of reduced cell activ

240 PCR (peripheral blood mononuclear cells and lymph node cells), RNA PCR (plasma), virus isolation, an

241 Upon stimulation in vitro such PAAIT lymph node cells secrete high amounts of IFN-gamma (129

242 IL-33 augmented mesenteric lymph node cell secretion of IL-5, IL-13, IL-6, and IFN-

243 and CD8(+) neonatal, as compared with adult, lymph node cells showed early cell cycle entry; this was

244 Lymph node cells showed increased expression of the prol

245 Analyses of spleen and mesenteric lymph node cells showed no differences in total cell cou

246 cytokine production by spleen and mesenteric lymph node cells showed production of IL-4, IL-10, IL-13

247 nalysis of CD4(+) NOD I-Ak transgenic primed lymph node cells showed that autoreactive CD4(+) T cells

248 genomic RNA and mRNA obtained from isolated lymph node cells showed the highest mRNA-to-genomic-RNA

249 First, compared with WT, Ccr2(-/-) lymph node cells, splenocytes, BMDCs, and LCs produced l

250 Incubation of lymph node cells/splenocytes from collagen-primed DBA/1

251 Endogenous golli-MBP epitopes within lymph node cells stimulated "classic" MBP 1-44-specific

252 ron (IFN)-gamma and no interleukin (IL)-4 by lymph node cells stimulated with P. gingivalis antigens.

253 In addition, we found that the lymph node cell subsets that are most efficacious in sti

254 -stimulated C57BL/6 granuloma and mesenteric lymph node cells suggested the possibility of apoptosis

255 4), IL-5, and IL-10 production by spleen and lymph node cells, suggesting that both Th1 and Th2 cells

256 as distinct from that seen for DNA from CD4+ lymph node cells, suggesting that TCR alphabeta+ CD8 alp

257 lasma), virus isolation, and the transfer of lymph node cell suspensions (10(8) cells) plus 8 ml of w

258 In the HDM model, lymph node cell T(H)1/T(H)2 signature cytokines were dec

259 sfer of proteolipid protein (PLP)-stimulated lymph node cells to 4MU-fed mice resulted in a delayed E

260 sue by transferring neonatal or adult CD4(+) lymph node cells to adoptive adult recombinase-activatin

261 tion, have no increase in the ability of the lymph node cells to bind IL-12 and correspondingly small

262 An increase in the ability of the lymph node cells to bind IL-12 correlates with 9.3- and

263 Adhesion of mesenteric lymph node cells to mucosal addressin cell adhesion mole

264 nsduced MHC-matched APCs stimulated alopecic lymph node cells to release IL-2 and IFN-gamma.

265 found but transient block in the capacity of lymph node cells to secrete IFN-alpha upon stimulation.

266 similar bias in CD4(+) T cells from draining lymph node cells toward IL-17A and away from IFN-gamma.

267 we co-transferred isolated Tregs and Scurfy lymph node cells; Treg repletion significantly attenuate

268 Inflammation of mesenteric and pancreatic lymph node cells was also evaluated.

269 iferative response of OVA-specific popliteal lymph node cells was assessed.

270 e in vitro cytokine secretion of mediastinal lymph node cells was determined using ELISA.

271 mma production by antigen-stimulated, primed lymph node cells was examined by assaying supernatants o

272 ugh antigen-dependent proliferation of their lymph node cells was substantially compromised.

273 Proliferation of recipient lymph node cells was tested against allogeneic, syngenei

274 ulture on antigen-driven responses of primed lymph node cells was tested.

275 CFSE-labeled C57BL/6 (H-2(b)) spleen and lymph node cells were adoptively transferred to C57BL/6x

276 choalveolar lavage fluid (BALF) and draining lymph node cells were analysed for inflammation.

277 istology and IL-12 secretion, and mesenteric lymph node cells were assessed for T-cell activation mar

278 ne secretion and proliferation by spleen and lymph node cells were assessed on days 31 and 65 and cor

279 val, immunohistology, circulating cells, and lymph node cells were assessed.

280 Their lymph node cells were depleted of V beta 6(+)CD4(+) cell

281 Although both spleen and lymph node cells were equally effective, graft-infiltrat

282 Supernatants from superficial cervical lymph node cells were examined by ELISA for IFNgamma, IL

283 ootpads of naive DO11.10 mice whose draining lymph node cells were harvested 4 days later and assayed

284 Draining lymph node cells were harvested from HLA-DR*0401 transge

285 l scoring, and lamina propria and mesenteric lymph node cells were isolated for analysis of activatio

286 Draining lymph node cells were obtained during the innate and ada

287 After 5 or 24 hr, the lymph node cells were removed, and RNA was prepared from

288 The draining lymph node cells were tested for T cell proliferative an

289 fore elicitation or when normally sensitized lymph node cells were transferred to neutrophil-deficien

290 When lymph node cells were treated with interleukin-4 (IL-4),

291 , adoptive transfer of tolerance failed when lymph nodes cells were depleted of CD4(+)CD25(+) T cells

292 nterleukin-2 (IL-2) production in mandibular lymph node cells, while transforming growth factor beta

293 fic IgE, a reduced number of splenocytes and lymph node cells with a decreased number of CD4+ T cells

294 ed by restimulation of spleen and mesenteric lymph node cells with a soluble H. hepaticus antigen (Ag

295 uperinfection of HIV-1-infected individuals' lymph node cells with GFP reporter virus confirmed the p

296 Treatment of lymph node cells with interleukin-6 (IL-6) augmented bor

297 Stimulation of splenic or mesenteric lymph node cells with lactococci resulted in their proli

298 ody was studied in vitro by culturing immune lymph node cells with macrophages and B. burgdorferi.

299 ma secretion following ex vivo activation of lymph node cells with rmIL-12, indicating the presence o

300 eatment of myelin basic protein-primed SJL/J lymph node cells with SB-331750 delayed the onset and re