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1 t them to the site of infection and to local lymphatic tissue.
2  tumor infiltration into the bone marrow and lymphatic tissue.
3  with Ag-specific activation as it occurs in lymphatic tissue.
4 l immunological status comparable to that of lymphatic tissue.
5  within the allograft as well as in draining lymphatic tissue.
6 hat will fully suppress viral replication in lymphatic tissues.
7 vated (CD86(+)) dendritic cells in secondary lymphatic tissues.
8 e inoculum traffic from the vaginal lumen to lymphatic tissues.
9 es at the site of inoculation, in liver, and lymphatic tissues.
10 meostasis by inducing collagen deposition in lymphatic tissues.
11 s also highly expressed in hematopoietic and lymphatic tissues.
12 trointestinal tissues, peripheral blood, and lymphatic tissues.
13 mphoma/leukemia 10 (Bcl10)-mucosa-associated lymphatic tissue 1(MALT1) (CBM) complex, which appears t
14 at, even if it is largely free of neural and lymphatic tissue, a comprehensive effort to map the dist
15  the upstream Bcl10-MALT1 (mucosa-associated lymphatic tissue) adapter complex.
16 nograft and persistence of microchimerism in lymphatic tissue after graft removal.
17 munodeficiency virus type 1 (HIV-1)-infected lymphatic tissues after treatment, and we undertook mapp
18 on based on its association with substantial lymphatic tissue, although the specific nature of that p
19 ficiency virus type 1 (HIV-1) replication in lymphatic tissues and partially reverses the pathologica
20            This is also effective in sealing lymphatic tissues and thereby facilitating dissection.
21 o BALB/c mice, but the mutants colonized the lymphatic tissues and were sufficiently immunogenic to p
22 of IFN-alpha/beta at peak acute infection in lymphatic tissues, and (iii) natural SIV hosts downregul
23 in collagen deposition and disruption of the lymphatic tissue architecture, and this damage contribut
24  fibrosis within the T cell zone disrupt the lymphatic tissue architecture, contributing to depletion
25 ith preservation of CD4(+) T cell counts and lymphatic tissue architecture.
26  center T follicular helper cells (GCTfh) in lymphatic tissue are critical for B cell differentiation
27  promotes CLL cell survival and expansion in lymphatic tissue areas designated proliferation centers.
28                      Virus was isolated from lymphatic tissue as early as 2 days post-IN inoculation;
29 nt in smooth muscle and heart, but scarce in lymphatic tissues, brain, and liver.
30 f encephalitogenic T cells in the peripheral lymphatic tissue, but Nlrx1(-/-) mice are more susceptib
31                         In vivo infection of lymphatic tissues by the human immunodeficiency virus ty
32                                           In lymphatic tissues, chronic lymphocytic leukemia (CLL) ce
33 ociated with progressive CD4(+) T-cell loss, lymphatic tissue destruction and premature death.
34 , viral replication and immune activation in lymphatic tissue drive a premature immunosuppressive res
35 nent of the inflammatory response induced by lymphatic tissue-dwelling filariae.
36 cells first settle in secondary (2 degrees ) lymphatic tissues (e.g., the spleen) where, in the absen
37 inducible T(reg) cell stimulation of primary lymphatic tissue fibroblasts to produce collagen type I
38 ALT or CLN to address the necessity of these lymphatic tissues for the development of a local protect
39                                  Analysis of lymphatic tissues from 12/15-LO-deficient mice confirmed
40 y virus-1 (HIV-1) infections, gut-associated lymphatic tissue (GALT), the largest component of the ly
41 tion of CD4+ T lymphocytes in gut-associated lymphatic tissue (GALT).
42 d incomplete, particularly in gut-associated lymphatic tissues (GALT).
43 ized that systemically targeting antigens to lymphatic tissue in vivo might modulate immunity.
44 e follicular dendritic cell (FDC) network in lymphatic tissues in HIV-1 and simian immunodeficiency v
45 aging and immunohistochemical examination of lymphatic tissues in mice heterozygous (+/-) for a targe
46 eripheral blood mononuclear cells (PBMC) and lymphatic tissues in the acutely infected baboons were i
47 al blood mononuclear cells (PBMCs) and other lymphatic tissues in vivo.
48 sive early replication in the gut-associated lymphatic tissue, including intestinal Peyer's patches,
49 d the transcriptional profiles of intestinal lymphatic tissue infected with Y. enterocolitica.
50  in microarray studies of HIV-1 infection in lymphatic tissue (LT) and show in this study that increa
51 ection establishes a persistent infection in lymphatic tissues (LT).
52                                              Lymphatic tissues (LTs) are structurally organized to pr
53 emination, and establishment of infection in lymphatic tissues (LTs) during the acute stage of infect
54 trol but do not eradicate infection from the lymphatic tissues (LTs) or prevent the particularly mass
55 tic ulcer disease, gastric mucosa-associated lymphatic tissue lymphoma, or gastric adenocarcinoma.
56                     In this study, we report lymphatic tissue microarray analyses, revealing for the
57 lication of M. avium and SIV was analyzed in lymphatic tissues obtained from rhesus macaques experime
58                                       In the lymphatic tissue of simian immunodeficiency virus (SIV)-
59                         The cells within the lymphatic tissue of the gut are likely to be central for
60  cells and mouse macrophages in the skin and lymphatic tissues of humanized mice.
61  microbes and restricted bacterial access to lymphatic tissues of the mice, thereby reducing microbio
62 adily colonize and induce disease within the lymphatic tissues of the small intestine.
63 of CD4 memory T cells in blood and secondary lymphatic tissues of these patients.
64                         We hypothesized that lymphatic tissues play a critical role in HO formation.
65  AIDS-related disease progression within the lymphatic tissues, such as vascular proliferation and ly
66          To identify genetic determinants in lymphatic tissue that critically affect HIV-1 replicatio
67 ors in vivo that impact viral replication in lymphatic tissue, the primary anatomical site of virus-h
68 ain "specified offals," including neural and lymphatic tissues, thought to contain high titers of pri
69 s study was to determine the contribution of lymphatic tissue to heterotopic ossification (HO).
70 rformance of an extended PLND, including all lymphatic tissue to the level of the aortic bifurcation.
71                         Enrichment of mature lymphatic tissues was noted 2 weeks after injury at the
72 ool for Yersinia pseudotuberculosis-infected lymphatic tissues, we revealed numerous alterations of h
73  from peripheral blood to the gut-associated lymphatic tissue, where they may contribute to immune ac
74 ls proliferate in pseudofollicles within the lymphatic tissues, where signals from the microenvironme

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