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1 ic background, display a high rate of thymic lymphoblastic lymphoma.
2 transformation to acute myeloid leukemia and lymphoblastic lymphoma.
3 ansformation to acute myeloid leukemia and T-lymphoblastic lymphoma.
4 The patient's diagnosis was T-lymphoblastic lymphoma.
5 lopment of human multiple myeloma and T-cell lymphoblastic lymphoma.
6 -cell acute lymphoblastic leukemia or T-cell lymphoblastic lymphoma.
7 e, were observed due to an aggressive T-cell lymphoblastic lymphoma.
8 ession, and a biopsy showed precursor B cell lymphoblastic lymphoma.
9 eas T/T(F) causes a long-latency, pre-B-cell lymphoblastic lymphoma.
10 (DK/DK) mice suffered from rapid-onset acute lymphoblastic lymphoma.
11 therapy was effective only in patients with lymphoblastic lymphoma.
12 cute lymphoblastic leukemia (ALL) and T-cell lymphoblastic lymphoma.
13 ke cell line established from a patient with lymphoblastic lymphoma.
14 similar to most human and mouse T-cell acute lymphoblastic lymphomas.
15 tical in the inception of human T cell acute lymphoblastic lymphomas.
16 s not detected in either case of precursor B-lymphoblastic lymphomas.
17 two Burkitt's lymphomas, and two precursor B-lymphoblastic lymphomas.
19 ts afflicted with this syndrome present with lymphoblastic lymphoma and a myeloproliferative disorder
21 rom small non-cleaved-cell lymphoma, 16 from lymphoblastic lymphoma, and five from large-cell lymphom
22 %, and 1% of small noncleaved cell lymphoma, lymphoblastic lymphoma, and large-cell cases, respective
23 pe of NHL that includes large-cell lymphoma, lymphoblastic lymphoma, and small noncleaved-cell lympho
27 -cyclin-transgenic mice developed high-grade lymphoblastic lymphoma between 3 and 12 months of age.
28 -C3 mice develop aggressive malignant thymic lymphoblastic lymphomas between 4 and 8 months of age.
31 he oncogenic potential of Stat5, with thymic lymphoblastic lymphomas developing in a significant prop
32 pstein-Barr virus-negative, precursor B cell lymphoblastic lymphoma diagnosed 6 months after transpla
33 omas, mantle cell lymphomas, and precursor B-lymphoblastic lymphomas) expressed significant levels of
35 study, the genetic basis of GD/T-cell acute lymphoblastic lymphoma in 2 affected siblings was deciph
36 nt growth to Ba/F3 cells and caused a T-cell lymphoblastic lymphoma in a murine bone marrow transplan
39 now report the development of thymic T cell lymphoblastic lymphomas in transgenic mice in which Stat
40 ent-free survival (pEFS) in pediatric T-cell lymphoblastic lymphoma is about 80%, whereas survival in
42 about the outcome of pediatric patients with lymphoblastic lymphoma (LBL) who suffer from progressive
43 lymphoblastic leukemia [ALL], 13 of 21 T-ALL/lymphoblastic lymphomas [LBL], 9 of 10 B-non-Hodgkin's l
45 en during embryonic development and in acute lymphoblastic lymphoma-like biphenotypic acute leukemias
48 rose after therapy for one case of T-lineage lymphoblastic lymphoma (LyL) and two cases of acute lymp
51 was not seen in small-cell T-cell lymphomas, lymphoblastic lymphomas, or other tumor types, including
53 ce demonstrate complete penetrance of thymic lymphoblastic lymphomas, strongly suggesting that Prkdc
54 ctivation in adult mice induces T-cell acute lymphoblastic lymphoma (T-ALL), a tumor type known to ca
55 ntribute to the pathogenesis of T cell acute lymphoblastic lymphoma (T-ALL), but how activated Notch1
56 eviously, precursor T lymphoblastic leukemia/lymphoblastic lymphoma (T-ALL/LyL) was found to express
59 e lymphoblastic leukemia (T-ALL) or advanced lymphoblastic lymphoma (T-NHL) were randomized at diagno
62 reases of lymphoblasts to overt monoclonal T-lymphoblastic lymphomas that involved multiple organs.
63 cases of large cell lymphoma, five cases of lymphoblastic lymphoma, two cases of small, noncleaved-c
64 survival among the patients with early-stage lymphoblastic lymphoma was inferior to that among the pa
65 Tp53-deficient mouse model for T-cell acute lymphoblastic lymphoma, we reported that loss of heteroz
68 adult patients with newly diagnosed ALL and lymphoblastic lymphoma were treated with a dose-intense
69 n with acute lymphoblastic leukemia (ALL) or lymphoblastic lymphoma who received one of three CNS-dir
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