ライフサイエンスコーパス: lymphoblastoid

1 atency III proliferating B cells, on various lymphoblastoid and Burkitt lymphoma cell lines, some of

2 me and xeroderma pigmentosum patient-derived lymphoblastoid and fibroblast cells.

3 ased levels of CSF KYNA (P=0.03) and reduced lymphoblastoid and hippocampal KMO expression (P</=0.05)

4 of which were profiled in both the GM12878 (lymphoblastoid) and K562 (erythroleukemic) human hematop

5 covered that in human cancer cells (myeloma, lymphoblastoid, and breast cancer), when expression of h

6 of transcription start sites (TSSs) in human lymphoblastoid B cell (GM12878) and chronic myelogenous

7 aluated by site-directed mutagenesis using a lymphoblastoid B cell line (B-LCL) and U3A cells.

8 merase (PARP) on global gene expression in a lymphoblastoid B cell line.

9 EBV-transformed lymphoblastoid B cell lines (LCLs) derived from subjects

10 resent study, we report that EBV-transformed lymphoblastoid B-cell lines (LCLs) and primary PTLDs ove

11 Epstein-Barr virus immortalized lymphoblastoid B-cell lines (LCLs) can be generated from

12 ow that Epstein-Barr virus (EBV) transformed lymphoblastoid B-cell lines (LCLs) not only express DEC-

13 *01:01 and HLA-G*01:01 were transfected into lymphoblastoid C1R cells expressing low endogenous HLA.

14 BV) super-enhancers (ESEs) are essential for lymphoblastoid cell (LCL) growth and survival.

15 uses NCL and RPL4 to establish persistent B-lymphoblastoid cell infection.

16 rom the exosome fractions derived from human lymphoblastoid cell line (LCL) culture media.

17 o compare skin eQTLs to a published panel of lymphoblastoid cell line (LCL) eQTLs.

18 p16(INK4A) is essential for immortal human B-lymphoblastoid cell line (LCL) growth.

19 Lymphoblastoid cell line (LCL) is a common tool to study

20 n and subsequently almost completely ablated lymphoblastoid cell line (LCL) outgrowth.

21 (WES) are whole blood (WB) and immortalized lymphoblastoid cell line (LCL).

22 In this study, lymphoblastoid cell line cultures (LCLs) from women with

23 Application of the methods to the human lymphoblastoid cell line data on chromosomes 14 and 22 f

24 in retroviral infection using the chicken B lymphoblastoid cell line DT40.

25 A lymphoblastoid cell line from one affected individual sh

26 Integration with ENCODE data from lymphoblastoid cell line GM12878, demonstrates that IRF4

27 cell RNA-seq protocol to study the reference lymphoblastoid cell line GM12878.

28 lic occupancy of 24 TFs and EP300 in a human lymphoblastoid cell line GM12878.

29 depletion and gene activation necessary for lymphoblastoid cell line growth and survival.

30 BNA-2 confers a type 1 growth phenotype in a lymphoblastoid cell line growth maintenance assay.

31 Moreover, NCL silencing impaired lymphoblastoid cell line growth.

32 Here, we show that growth of a human B lymphoblastoid cell line infected with Epstein-Barr viru

33 Infection of the JY lymphoblastoid cell line limited the accumulation of a m

34 -infected B lymphocytes and are critical for lymphoblastoid cell line outgrowth.

35 C-seq by sequencing the methylome of a human lymphoblastoid cell line to approximately 8.6x high-qual

36 from GM06990, a near-normal EBV-transformed lymphoblastoid cell line, and have compared origin distr

37 Applied to high-resolution Hi-C data in a lymphoblastoid cell line, HiC-DC detects significant int

38 Furthermore, in the GM12878 lymphoblastoid cell line, these three genes are in a con

39 kata-EBV and Raji), and cells from an EBV(+) lymphoblastoid cell line, thus suggesting a specific ass

40 for PBL-to-atrium; and from 0.81 to 0.98 for lymphoblastoid cell line-to-PBL based on cross-validatio

41 m log-phase GM06990, a karyotypically normal lymphoblastoid cell line.

42 astoid cell lines, and EBV genome number per lymphoblastoid cell line.

43 RRP12 in LNCaP, USP14 in DU-145 and SMIN3 in lymphoblastoid cell line.

44 ized endogenously presented targets on EBV B lymphoblastoid cell lines (BLCLs), but not peripheral bl

45 a demonstration, Epstein-Barr virus-infected lymphoblastoid cell lines (EBV-LCL) were isolated based

46 addition, human LRRK2 G2019S patient-derived lymphoblastoid cell lines (LCL) demonstrated increased m

47 l transcriptomics data from a panel of human lymphoblastoid cell lines (LCL) to infer drug response n

48 tore iNKT recognition in EBV-infected cells, lymphoblastoid cell lines (LCL) were treated with AM580,

49 inhibitor olaparib (AZD2281) of 5 ATM mutant lymphoblastoid cell lines (LCL), an ATM mutant MCL cell

50 allowed the establishment of MDV-transformed lymphoblastoid cell lines (LCL).

51 latency II NPC C666-1 cells, and latency III lymphoblastoid cell lines (LCL).

52 ntly reported gene expression changes in 480 lymphoblastoid cell lines (LCLs) after in vitro simvasta

53 lysis of RNA stability in seven human HapMap lymphoblastoid cell lines (LCLs) and analyzed the effect

54 lomere repeat elements (SREs) in transformed lymphoblastoid cell lines (LCLs) and human embryonic ste

55 otein isoforms across 68 Yoruba (YRI) HapMap lymphoblastoid cell lines (LCLs) and identified 12 cis a

56 on measured via RNA-seq analysis in adipose, lymphoblastoid cell lines (LCLs) and skin.

57 Using EBV-immortalized lymphoblastoid cell lines (LCLs) as a model, we found th

58 umented the DNA methylation pattern in human lymphoblastoid cell lines (LCLs) as well as identified s

59 Patient-derived cell lines such as lymphoblastoid cell lines (LCLs) could represent the ide

60 Using a data set of gene expression in lymphoblastoid cell lines (LCLs) derived from 210 HapMap

61 tosine modifications at 283,540 CpG sites in lymphoblastoid cell lines (LCLs) derived from independen

62 Here we report that RP-mutated lymphoblastoid cell lines (LCLs) established from DBA pa

63 Using lymphoblastoid cell lines (LCLs) established with EBV re

64 ene expression profiles using microarrays on lymphoblastoid cell lines (LCLs) from 413 cases and 446

65 romatin profiling for three histone marks in lymphoblastoid cell lines (LCLs) from 75 sequenced indiv

66 Previously we hypothesized that a subset of lymphoblastoid cell lines (LCLs) from children with auti

67 Here we analyze m(6)A mRNA modifications in lymphoblastoid cell lines (LCLs) from human, chimpanzee

68 tified H3K4me3-associated genomic regions in lymphoblastoid cell lines (LCLs) from humans, chimpanzee

69 enome-wide association studies involving 523 lymphoblastoid cell lines (LCLs) from individuals of Eur

70 A21 to assess trisomic protein expression in lymphoblastoid cell lines (LCLs) from patients with DS a

71 Lymphoblastoid cell lines (LCLs) from some children with

72 Using RNA-sequence data from lymphoblastoid cell lines (LCLs) from the TwinsUK cohort

73 EBV to convert human B cells into long-lived lymphoblastoid cell lines (LCLs) in vitro requires the c

74 lymphocytes into continuously proliferating lymphoblastoid cell lines (LCLs) in vitro through manipu

75 rther demonstrated that knockdown of H2AX in lymphoblastoid cell lines (LCLs) led to the upregulation

76 ion, elicited lytic EBV in latently infected lymphoblastoid cell lines (LCLs) partially via Toll-like

77 Epstein-Barr virus (EBV) transformed lymphoblastoid cell lines (LCLs) provide a conveniently

78 r Virus (EBV) conversion of B-lymphocytes to Lymphoblastoid Cell Lines (LCLs) requires four EBV nucle

79 transformed into continuously proliferating lymphoblastoid cell lines (LCLs) that carry EBV DNA as e

80 The ZIIRmt-infected lymphoblastoid cell lines (LCLs) that did grow out exhib

81 Lymphoblastoid cell lines (LCLs) were cocultured with au

82 Populations of human-derived HapMap lymphoblastoid cell lines (LCLs) were infected with RSV.

83 between vitamin D receptor (VDR) binding in lymphoblastoid cell lines (LCLs), chromatin states in LC

84 In contrast to wild-type lymphoblastoid cell lines (LCLs), dividing LCLs establis

85 ChIP-seq performed on lymphoblastoid cell lines (LCLs), expressing epitope-tag

86 Human lymphoblastoid cell lines (LCLs), generated through Epst

87 In lytic-permissive lymphoblastoid cell lines (LCLs), pulse exposure to the

88 In GS-derived lymphoblastoid cell lines (LCLs), the proportion of ITPR

89 amining Epstein-Barr virus (EBV)-transformed lymphoblastoid cell lines (LCLs), we identified four EBV

90 sforms B cells to continuously proliferating lymphoblastoid cell lines (LCLs), which represent an exp

91 ll proliferation leading to the outgrowth of lymphoblastoid cell lines (LCLs).

92 oth in newly infected primary B cells and in lymphoblastoid cell lines (LCLs).

93 ISPR/Cas9 loss-of-function screens in BL and lymphoblastoid cell lines (LCLs).

94 A polymerase II (Pol II) occupancy in Yoruba lymphoblastoid cell lines (LCLs).

95 s drives their indefinite proliferation into lymphoblastoid cell lines (LCLs).

96 roliferation and through transformation into lymphoblastoid cell lines (LCLs).

97 critical for B-lymphocyte transformation to lymphoblastoid cell lines (LCLs).

98 ansformation into indefinitely proliferating lymphoblastoid cell lines (LCLs).

99 n vitro, EBV transforms primary B cells into lymphoblastoid cell lines (LCLs).

100 conducted a pharmacogenomic study using 266 lymphoblastoid cell lines (LCLs).

101 lation from chromatin to proteins, in Yoruba lymphoblastoid cell lines (LCLs).

102 s, nasopharyngeal carcinoma (NPC) cells, and lymphoblastoid cell lines (LCLs).

103 a panel of iPSCs from 58 well-studied Yoruba lymphoblastoid cell lines (LCLs); 14 of these lines were

104 vitro results in their immortalization into lymphoblastoid cell lines (LCLs); this latency program i

105 loci in genome-wide expression data sets of lymphoblastoid cell lines (n = 1,830) and were related t

106 common CNVs in a cohort of 50 radiosensitive lymphoblastoid cell lines (RS-LCLs) derived from patient

107 CAMKK2 in human brains (P=1.1 x 10(-6)) and lymphoblastoid cell lines (the lowest P=8.4 x 10(-6)).

108 eQTLs each affect hundreds of transcripts in lymphoblastoid cell lines across three African populatio

109 ong chromosomes from primary lymphocytes and lymphoblastoid cell lines adapted to long-term growth in

110 edish patients, and to KMO expression in 717 lymphoblastoid cell lines and 138 hippocampal biopsies.

111 d the structure of HLA-F on the surface of B lymphoblastoid cell lines and activated lymphocytes by d

112 s from human, chimpanzee, and rhesus macaque lymphoblastoid cell lines and compared them to transcrip

113 stimulation with autologous EBV-transformed lymphoblastoid cell lines and correlated with EBV load i

114 rm that CYFIP1 is upregulated in transformed lymphoblastoid cell lines and demonstrate its upregulati

115 R-181c, were significantly down-regulated in lymphoblastoid cell lines and fresh peripheral blood cel

116 990620, that differentially recruits CTCF in lymphoblastoid cell lines and human brain to influence C

117 and association with TMEM106B expression in lymphoblastoid cell lines and human brain.

118 hromatin signature to infer MAE for genes in lymphoblastoid cell lines and human fetal brain tissue.

119 ructure of BMPR1A, we performed 5' RACE from lymphoblastoid cell lines and normal colon tissue, which

120 sociated with lower CTSH expression in human lymphoblastoid cell lines and pancreatic tissue.

121 with a decline in CLDN14 expression in both lymphoblastoid cell lines and T cells (Padj = 0.003 and

122 med histone acetylation ChIP-seq on 57 human lymphoblastoid cell lines and used the resulting reads t

123 e associated with lower FBXO33 expression in lymphoblastoid cell lines and with reduced frontal gray

124 als on whom both haplotypes and DH status in lymphoblastoid cell lines are publicly available.

125 d this effect is substantially reversed when lymphoblastoid cell lines are stably infected with a ret

126 ng Salmonella typhimurium infection of human lymphoblastoid cell lines as a means of dissecting the g

127 ce the death of established, EBV-transformed lymphoblastoid cell lines at doses nontoxic to normal ce

128 Patient-derived lymphoblastoid cell lines bearing a range of expanded al

129 nomic and transcriptomic data from 445 human lymphoblastoid cell lines by combining an RNA editing QT

130 ne expression levels generated for 373 human lymphoblastoid cell lines by the Geuvadis consortium and

131 virus (EBV) to transform human B cells into lymphoblastoid cell lines compared to that of type 2 EBV

132 us (EBV)-encoded RNAs (EBERs) were tested in lymphoblastoid cell lines containing EBER mutants of EBV

133 study the response to 23 treatments in three lymphoblastoid cell lines demonstrating that it should a

134 associations with gene expression levels in lymphoblastoid cell lines derived from 480 participants

135 onstructed by transferring mitochondria from lymphoblastoid cell lines derived from a Chinese family

136 generated by transferring mitochondria from lymphoblastoid cell lines derived from a Chinese family

137 Lymphoblastoid cell lines derived from carriers of misse

138 iated (P = .01) with FPGS gene expression in lymphoblastoid cell lines derived from combined HapMap A

139 , cytosine modification levels in 133 HapMap lymphoblastoid cell lines derived from individuals of Eu

140 We performed functional assays by using lymphoblastoid cell lines derived from members of Chines

141 ofiling by CpG island microarray analysis of lymphoblastoid cell lines derived from monozygotic twins

142 ition, signaling, and repair mechanisms in B lymphoblastoid cell lines derived from patients with ped

143 d expression information for EBV-transformed lymphoblastoid cell lines derived from populations acros

144 expression by affecting the binding to GR in lymphoblastoid cell lines derived from the same patients

145 We sequenced RNA from 69 lymphoblastoid cell lines derived from unrelated Nigeria

146 ar susceptibility to cisplatin in 176 HapMap lymphoblastoid cell lines derived from Yoruba individual

147 Nonleukemic EBV-transformed lymphoblastoid cell lines displayed highly stable replic

148 We show for lymphoblastoid cell lines established from individuals o

149 The B-lymphoblastoid cell lines exhibited a unimodal distribut

150 otein from V362I and R381Q variants in human lymphoblastoid cell lines exhibited lower expression lev

151 udies, we collected RNA-sequencing data from lymphoblastoid cell lines for 431 Hutterite individuals.

152 red the cytotoxicity of 156 compounds in 884 lymphoblastoid cell lines for which genotype and transcr

153 ne relative protein levels of 5,953 genes in lymphoblastoid cell lines from 95 diverse individuals ge

154 ormed a genome-wide gene expression study in lymphoblastoid cell lines from 96 Hutterites.

155 rometry to perform an integrated analysis in lymphoblastoid cell lines from a diverse group of indivi

156 Using immortalized lymphoblastoid cell lines from a healthy study populatio

157 OGT protein levels was observed in isolated lymphoblastoid cell lines from affected individuals, con

158 - to 3-fold overexpression of DIAPH3 mRNA in lymphoblastoid cell lines from affected individuals.

159 This miniATM variant was also highlighted in lymphoblastoid cell lines from AT patients and was shown

160 f NK cells to kill freshly established human lymphoblastoid cell lines from autologous or allogeneic

161 and extended in three human EBV-transformed lymphoblastoid cell lines from individuals with MSS, lea

162 Using simvastatin and sham incubated lymphoblastoid cell lines from participants of the Chole

163 tein (FMRP), its upregulation in transformed lymphoblastoid cell lines from patients with duplication

164 to interrogate DSB repair and recognition in lymphoblastoid cell lines from patients with pediatric S

165 suggest that DSB repair is defective in some lymphoblastoid cell lines from pediatric patients with S

166 Lymphoblastoid cell lines from subjects with the p.V228F

167 We used 1,086 lymphoblastoid cell lines from the 1000 Genomes Project,

168 In lymphoblastoid cell lines from two translocation subject

169 Lymphoblastoid cell lines generated from affected childr

170 DNA methylation (DNAm) measured in lymphoblastoid cell lines has been repeatedly demonstrat

171 k haplotype show no binding of STAT1, and in lymphoblastoid cell lines homozygous for the CAD non-ris

172 Lymphoblastoid cell lines homozygous for the CAD risk ha

173 in nonsynonymous substitutions in all three lymphoblastoid cell lines in our study, unlike RNA editi

174 nd transform B lymphocytes into immortalized lymphoblastoid cell lines in vitro.

175 gh-throughput sequencing data sets for human lymphoblastoid cell lines mapped to the EBV genome.

176 Lymphoblastoid cell lines obtained from a patient and fr

177 elationship between COMETs and haplotypes in lymphoblastoid cell lines of African and European origin

178 In seven lymphoblastoid cell lines of Asian, European and African

179 lls or NK-cell clones with HLA-C2(+) CCR7(+) lymphoblastoid cell lines resulted in increased CCR7 upt

180 exhibits higher expression in BLM-sensitive lymphoblastoid cell lines than insensitive cell lines up

181 s, which leads to continuously proliferating lymphoblastoid cell lines through examination of the exp

182 logous dendritic cells and EBV-transformed B-lymphoblastoid cell lines transduced with an adenoviral

183 y members were enrolled, blood was obtained, lymphoblastoid cell lines were immortalized, deoxyribonu

184 r virus-transformed B-cell cultures (human B-lymphoblastoid cell lines) from 19 healthy donors.

185 using >300 expression microarrays (from 117 lymphoblastoid cell lines) in corticosteroid (dexamethas

186 od-related cell types (CD3 and CD4+ T cells, lymphoblastoid cell lines).

187 appear to regulate gene expression in human lymphoblastoid cell lines, a tightly controlled, largely

188 ells, HeLa cells, HEK293 cells, and 16 human lymphoblastoid cell lines, all genotyped for the 9p21.3

189 iP luciferase reporter, EBNA1 DNA binding in lymphoblastoid cell lines, and EBV genome number per lym

190 ed BZLF1 expression in latently EBV-infected lymphoblastoid cell lines, and knockdown of BGLF2 reduce

191 n transcriptional response in fibroblast and lymphoblastoid cell lines, as well as circulating monocy

192 cific quantification assays to a panel of HD lymphoblastoid cell lines, each carrying the major Europ

193 measure chromatin accessibility in 70 Yoruba lymphoblastoid cell lines, for which genome-wide genotyp

194 h resulted in the generation of EBV-positive lymphoblastoid cell lines, indicating that the virus in

195 entified as eQTL in monocytes, liver tissue, lymphoblastoid cell lines, T cells, and fibroblasts are

196 Here we report that, in lymphoblastoid cell lines, the translocation additionall

197 nd drug sensitivity measurements in 24 human lymphoblastoid cell lines, was applied to a panel of 12

198 Using the HapMap lymphoblastoid cell lines, we combine 1000 Genomes genot

199 Applying riboHMM to human lymphoblastoid cell lines, we identified 7273 novel codi

200 Using metaphase spreads from human lymphoblastoid cell lines, we previously showed how immu

201 to the establishment of permanently growing lymphoblastoid cell lines, whereas CD40L/IL-4 blasts hav

202 transcription factor binding sites in human lymphoblastoid cell lines, which is comprised of sites c

203 iation in genome-wide gene expression in 107 lymphoblastoid cell lines, with alleles ranging from 15

204 ession in human aortic endothelial cells and lymphoblastoid cell lines.

205 or each of the four genes in the region in B lymphoblastoid cell lines.

206 rase reporter, and EBV genome maintenance in lymphoblastoid cell lines.

207 n human adipose tissue, skeletal muscle, and lymphoblastoid cell lines.

208 f virus gene expression and the outgrowth of lymphoblastoid cell lines.

209 the TwinsUK microarray and RNA-Seq cohort in lymphoblastoid cell lines.

210 available RNA-seq expression data sets from lymphoblastoid cell lines.

211 ed IRF7 is detected in latently infected EBV lymphoblastoid cell lines.

212 ype and reduced BAK1 expression was shown in lymphoblastoid cell lines.

213 phase arrest and apoptosis in leukemic and B-lymphoblastoid cell lines.

214 normal tissue pairs and 17 matched SCLC and lymphoblastoid cell lines.

215 sing whole-genome expression array data from lymphoblastoid cell lines.

216 lded aberrant results in the majority of SLE lymphoblastoid cell lines.

217 esses that prevent LTIII BHRF1 expression in lymphoblastoid cell lines.

218 hosphorylation was also noted in 2 of 16 SLE lymphoblastoid cell lines.

219 with daunorubicin IC50 values in a panel of lymphoblastoid cell lines.

220 ed in HL, diffuse large B-cell lymphoma, and lymphoblastoid cell lines.

221 (ChIP-exo) genome-wide analysis of 27 HapMap lymphoblastoid cell lines.

222 d in proband versus control EBV-immortalized lymphoblastoid cell lines.

223 and RP1-10D13.2 expression levels in the CAP lymphoblastoid cell lines.

224 oning patterns are observed in two different lymphoblastoid cell lines.

225 the expression level of both transcripts in lymphoblastoid cell lines.

226 es less stable or leakier in EBV-transformed lymphoblastoid cell lines.

227 uencing in ATAC-seq libraries generated from lymphoblastoid cell lines: targeted cleavage of mitochon

228 n both peripheral blood leukocytes (PBL) and lymphoblastoid cell lines; and a study of postoperative

229 e method was first applied to RNA-Seq from a lymphoblastoid cell-line, achieving 99.7% precision and

230 Whole-genome sequencing reads were from a lymphoblastoid cell-line.

231 ys were performed on patient and control EBV lymphoblastoids cell lines.

232 hese cells do not resemble the proliferating lymphoblastoid cells (LCLs) (latency III) that are gener

233 ls with type I latency and reactivation from lymphoblastoid cells (LCLs) with type III latency.

234 acteristics of MMR-deficient tumor cells) in lymphoblastoid cells (LCs) from 3 patients with CMMRD an

235 strongly associated with CREB1 expression in lymphoblastoid cells (P<0.005) and the prefrontal cortex

236 in transfected murine macrophages and human lymphoblastoid cells affected anthrax toxin binding, int

237 Levels of coenzyme Q10 in lymphoblastoid cells and brain tissue were measured on h

238 Analysis of lymphoblastoid cells and fibroblasts from patients homoz

239 Gene expression profiling was performed on lymphoblastoid cells and levels of CXCL10 were measured

240 F8 mRNA and intracellular FVIII protein in B lymphoblastoid cells and liver biopsies from individuals

241 ntagonizes the growth inhibitory effect in B lymphoblastoid cells and might be used to modulate the f

242 lular fusion transcripts in transduced human lymphoblastoid cells and primary hematopoietic stem/prog

243 We also analyze data from individual lymphoblastoid cells and show that desirable properties

244 l activation of hPer1 was reduced in human B-lymphoblastoid cells carrying the risk genotype of rs302

245 NA from affected individuals' fibroblasts or lymphoblastoid cells confirmed mutant transcripts with p

246 Chromatin immunoprecipitation-sequencing in lymphoblastoid cells confirmed p53 binding to seven poly

247 Lymphoblastoid cells derived from a HapMap Project cohor

248 Similar results were found in lymphoblastoid cells derived from a SMS patient carrying

249 tructure and transcription factor binding in lymphoblastoid cells derived from individuals of geograp

250 Using B-lymphoblastoid cells derived from the HapMap Project coh

251 and associated with PRPF6 mRNA expression in lymphoblastoid cells from 373 Europeans in the 1000 Geno

252 Importantly, lymphoblastoid cells from an individual heterozygous for

253 In lymphoblastoid cells from control individuals, we found

254 Lymphoblastoid cells from individuals with BRCA1 pathoge

255 ce (MECP2-TG), and corrected MECP2 levels in lymphoblastoid cells from MECP2 duplication patients in

256 RNA and protein abundance in patient-derived lymphoblastoid cells from one NUDT21 deletion and three

257 Pre-rRNA analysis in lymphoblastoid cells from patients bearing mutations in

258 Patient-derived SDH(var+) lymphoblastoid cells had elevated cellular reactive oxyg

259 onal changes in cells, we cultured TK6 human lymphoblastoid cells in a high aspect ratio vessel (bior

260 we utilized gene expression association from lymphoblastoid cells lines from 754 p.Phe508del CF-affec

261 Lymphoblastoid cells obtained from an affected individua

262 Lymphoblastoid cells of a mutant gene carrier had, in ad

263 In addition, the mutant gene carrier lymphoblastoid cells proliferated faster and were less r

264 cleus cytome (CBMN-Cyt) assay with WIL2-NS B lymphoblastoid cells to test the potential genotoxicity,

265 e variant SNP genotypes in estradiol-treated lymphoblastoid cells transfected with estrogen receptor

266 peptide profile of human EC and syngeneic B lymphoblastoid cells was biochemically analyzed and comp

267 Blood and lymphoblastoid cells were collected from patients and co

268 ere readily detected in DNA from the treated lymphoblastoid cells, and both were largely eliminated f

269 cription start site used in brain but not in lymphoblastoid cells, and have detected FMR1 isoforms ge

270 MK) enzyme activity was analyzed in cultured lymphoblastoid cells, and mevalonic acid levels were mea

271 hESC-derived neural precursor cells (NPCs), lymphoblastoid cells, and two human induced pluripotent

272 The densest, in human lymphoblastoid cells, contains 4.9 billion contacts, ach

273 To date, only patient-derived lymphoblastoid cells, fibroblasts and SETX knockdown cel

274 Studies in TK6 human lymphoblastoid cells, in which the analytical data were

275 2-depleted motor neurons, in patient-derived lymphoblastoid cells, induced pluripotent stem cell-deri

276 ction studies in EBV-positive B lymphoma and lymphoblastoid cells, we found that the levels of functi

277 tructures (at the macrodomain resolution) of lymphoblastoid cells, we identify an atlas of stable int

278 In cultured human CEM lymphoblastoid cells, which possess a single hNT type (h

279 NA and transcription factor targets in human lymphoblastoid cells, while being nearly a million times

280 res for mouse embryonic stem cells and human lymphoblastoid cells.

281 on HLA-A2(+) melanoma, breast carcinoma, and lymphoblastoid cells.

282 st and attenuated FBXL2-induced apoptosis of lymphoblastoid cells.

283 related with COMT Val/Met genotypes in human lymphoblastoid cells.

284 ed normal CEP290 splicing in patient-derived lymphoblastoid cells.

285 tact map of chromosome 10 from human GM12878 lymphoblastoid cells.

286 ochondrial and redox abnormalities in autism lymphoblastoid cells: a sibling control study.

287 Global transcriptome analysis of L254F-OGT lymphoblastoids compared with controls revealed a small

288 Surprisingly, lymphoblastoids from affected individuals displayed a ma

289 ised myelogenous leukemia (K562) and healthy lymphoblastoid (GM12878) cell lines to train the learnin

290 Lymphoblastoid haQTLs were highly predictive of autoimmu

291 t fuses to other genes associated with acute lymphoblastoid leukemia (ALL).

292 reast cancer cell line (HCC1395) and matched lymphoblastoid line (HCC1395BL).

293 histone modifications, cohesin, and CTCF in lymphoblastoid lines from 19 individuals of diverse ance

294 ble virion infection, and (iii) immortalized lymphoblastoid lines have partial postentry blocks to ef

295 tagged on the Immunochip, 15 have SNPs in B-lymphoblastoid open chromatin regions in high LD (r2>0.8

296 We apply it to a collection of lymphoblastoid RNA-seq data from the 1000 Genomes Projec

297 virus-driven luciferase expression, or A3R5 lymphoblastoid target cells, in which infectivity was ev

298 cellular resistance to IR or BLM in human B-lymphoblastoid TK6 cells and HCT116 colon tumor cells.

299 obtain such transcriptomes, we sequenced the lymphoblastoid transcriptomes of three family members (G

300 chickens characterized by the development of lymphoblastoid tumors in multiple organs and is transmit