ライフサイエンスコーパス: lymphoblastoid cell line

1 od-related cell types (CD3 and CD4+ T cells, lymphoblastoid cell lines).

2 m log-phase GM06990, a karyotypically normal lymphoblastoid cell line.

3 tibility complex class I-deficient 721.221 B-lymphoblastoid cell line.

4 P and EBNA2 to be associated with HDAC4 in a lymphoblastoid cell line.

5 astoid cell lines, and EBV genome number per lymphoblastoid cell line.

6 RRP12 in LNCaP, USP14 in DU-145 and SMIN3 in lymphoblastoid cell line.

7 Whole-genome sequencing reads were from a lymphoblastoid cell-line.

8 ession in human aortic endothelial cells and lymphoblastoid cell lines.

9 rase reporter, and EBV genome maintenance in lymphoblastoid cell lines.

10 n human adipose tissue, skeletal muscle, and lymphoblastoid cell lines.

11 f virus gene expression and the outgrowth of lymphoblastoid cell lines.

12 the TwinsUK microarray and RNA-Seq cohort in lymphoblastoid cell lines.

13 available RNA-seq expression data sets from lymphoblastoid cell lines.

14 ed IRF7 is detected in latently infected EBV lymphoblastoid cell lines.

15 ype and reduced BAK1 expression was shown in lymphoblastoid cell lines.

16 phase arrest and apoptosis in leukemic and B-lymphoblastoid cell lines.

17 normal tissue pairs and 17 matched SCLC and lymphoblastoid cell lines.

18 sing whole-genome expression array data from lymphoblastoid cell lines.

19 lded aberrant results in the majority of SLE lymphoblastoid cell lines.

20 esses that prevent LTIII BHRF1 expression in lymphoblastoid cell lines.

21 hosphorylation was also noted in 2 of 16 SLE lymphoblastoid cell lines.

22 er of studies have explored this area, using lymphoblastoid cell lines.

23 Gene expression of SLC1A1 is heritable in lymphoblastoid cell lines.

24 ion 2 (hPMS2)-deficient and proficient human lymphoblastoid cell lines.

25 mary B lymphocytes into continuously growing lymphoblastoid cell lines.

26 for a subset of genes in a separate group of lymphoblastoid cell lines.

27 and vaccinia virus (expressing gB)-infected lymphoblastoid cell lines.

28 197 ethnically defined Human Variation Panel lymphoblastoid cell lines.

29 ivation of EBV-positive Burkitt lymphoma and lymphoblastoid cell lines.

30 ,599 genes in Epstein-Barr virus-transformed lymphoblastoid cell lines.

31 with daunorubicin IC50 values in a panel of lymphoblastoid cell lines.

32 DV-infected CEF, MDV-induced tumors, and MDV lymphoblastoid cell lines.

33 y that inhibits B-cell receptor signaling in lymphoblastoid cell lines.

34 nic B220(+) splenocytes than in EBV-infected lymphoblastoid cell lines.

35 ed in HL, diffuse large B-cell lymphoma, and lymphoblastoid cell lines.

36 d in proband versus control EBV-immortalized lymphoblastoid cell lines.

37 (ChIP-exo) genome-wide analysis of 27 HapMap lymphoblastoid cell lines.

38 and RP1-10D13.2 expression levels in the CAP lymphoblastoid cell lines.

39 oning patterns are observed in two different lymphoblastoid cell lines.

40 the expression level of both transcripts in lymphoblastoid cell lines.

41 es less stable or leakier in EBV-transformed lymphoblastoid cell lines.

42 or each of the four genes in the region in B lymphoblastoid cell lines.

43 ys were performed on patient and control EBV lymphoblastoids cell lines.

44 Twenty lymphoblastoid cell lines (15 SMA and 5 control lines) w

45 324 Centre d' Etude du Polymorphisme Humain lymphoblastoid cell lines (24 pedigrees) was evaluated f

46 ttempt to isolate human chromosome 15 from a lymphoblastoid cell line, a chromosome 15 centromere-spe

47 appear to regulate gene expression in human lymphoblastoid cell lines, a tightly controlled, largely

48 as well as in the highly IFN-sensitive Daudi lymphoblastoid cell line, abrogates NF-kappaB activation

49 e method was first applied to RNA-Seq from a lymphoblastoid cell-line, achieving 99.7% precision and

50 eQTLs each affect hundreds of transcripts in lymphoblastoid cell lines across three African populatio

51 ong chromosomes from primary lymphocytes and lymphoblastoid cell lines adapted to long-term growth in

52 the analysis of DNA adducts in the human TK6 lymphoblastoid cell line after exposure to N-hydroxy-4-a

53 ells, HeLa cells, HEK293 cells, and 16 human lymphoblastoid cell lines, all genotyped for the 9p21.3

54 edish patients, and to KMO expression in 717 lymphoblastoid cell lines and 138 hippocampal biopsies.

55 d the structure of HLA-F on the surface of B lymphoblastoid cell lines and activated lymphocytes by d

56 s from human, chimpanzee, and rhesus macaque lymphoblastoid cell lines and compared them to transcrip

57 stimulation with autologous EBV-transformed lymphoblastoid cell lines and correlated with EBV load i

58 rm that CYFIP1 is upregulated in transformed lymphoblastoid cell lines and demonstrate its upregulati

59 Here we measure both f and mu by culturing B-lymphoblastoid cell lines and first eliminating preexist

60 R-181c, were significantly down-regulated in lymphoblastoid cell lines and fresh peripheral blood cel

61 990620, that differentially recruits CTCF in lymphoblastoid cell lines and human brain to influence C

62 and association with TMEM106B expression in lymphoblastoid cell lines and human brain.

63 hromatin signature to infer MAE for genes in lymphoblastoid cell lines and human fetal brain tissue.

64 ructure of BMPR1A, we performed 5' RACE from lymphoblastoid cell lines and normal colon tissue, which

65 sociated with lower CTSH expression in human lymphoblastoid cell lines and pancreatic tissue.

66 with a decline in CLDN14 expression in both lymphoblastoid cell lines and T cells (Padj = 0.003 and

67 med histone acetylation ChIP-seq on 57 human lymphoblastoid cell lines and used the resulting reads t

68 e associated with lower FBXO33 expression in lymphoblastoid cell lines and with reduced frontal gray

69 etal muscle, subcutaneous adipose tissue and lymphoblastoid cell lines) and observed a tissue-specifi

70 from GM06990, a near-normal EBV-transformed lymphoblastoid cell line, and have compared origin distr

71 clear factor kappaB (p65), were mapped in 10 lymphoblastoid cell lines, and 25 and 7.5% of the respec

72 iP luciferase reporter, EBNA1 DNA binding in lymphoblastoid cell lines, and EBV genome number per lym

73 large-effect eQTLs identified in the HapMap lymphoblastoid cell lines, and examined the association

74 DLX6 in mouse x human somatic cell hybrids, lymphoblastoid cell lines, and frontal cortex from contr

75 lymphoma and Epstein-Barr virus-transformed lymphoblastoid cell lines, and in T cells activated via

76 te in man can now be measured routinely in B-lymphoblastoid cell lines, and it is elevated in cancer

77 ed BZLF1 expression in latently EBV-infected lymphoblastoid cell lines, and knockdown of BGLF2 reduce

78 cted lymphoblastoid JY cells or autologous B lymphoblastoid cell lines, and the cytolytic activity wa

79 n both peripheral blood leukocytes (PBL) and lymphoblastoid cell lines; and a study of postoperative

80 als on whom both haplotypes and DH status in lymphoblastoid cell lines are publicly available.

81 d this effect is substantially reversed when lymphoblastoid cell lines are stably infected with a ret

82 -state levels of the adduct in the human TK6 lymphoblastoid cell line as a function of dose (0.5, 1.0

83 ng Salmonella typhimurium infection of human lymphoblastoid cell lines as a means of dissecting the g

84 n transcriptional response in fibroblast and lymphoblastoid cell lines, as well as circulating monocy

85 ce the death of established, EBV-transformed lymphoblastoid cell lines at doses nontoxic to normal ce

86 Patient-derived lymphoblastoid cell lines bearing a range of expanded al

87 We then evaluated [ATP/ADP] in 40 human lymphoblastoid cell lines, bearing non-HD CAG lengths (9

88 ized endogenously presented targets on EBV B lymphoblastoid cell lines (BLCLs), but not peripheral bl

89 nomic and transcriptomic data from 445 human lymphoblastoid cell lines by combining an RNA editing QT

90 ne expression levels generated for 373 human lymphoblastoid cell lines by the Geuvadis consortium and

91 virus (EBV) to transform human B cells into lymphoblastoid cell lines compared to that of type 2 EBV

92 us (EBV)-encoded RNAs (EBERs) were tested in lymphoblastoid cell lines containing EBER mutants of EBV

93 In this study, lymphoblastoid cell line cultures (LCLs) from women with

94 Application of the methods to the human lymphoblastoid cell line data on chromosomes 14 and 22 f

95 study the response to 23 treatments in three lymphoblastoid cell lines demonstrating that it should a

96 ical cross-correction in an enzyme-deficient lymphoblastoid cell line derived from patients with muco

97 associations with gene expression levels in lymphoblastoid cell lines derived from 480 participants

98 onstructed by transferring mitochondria from lymphoblastoid cell lines derived from a Chinese family

99 generated by transferring mitochondria from lymphoblastoid cell lines derived from a Chinese family

100 structed by transferring mitochondria from 9 lymphoblastoid cell lines derived from a Chinese family

101 quantitative immunoblotting of lysates from lymphoblastoid cell lines derived from affected individu

102 Lymphoblastoid cell lines derived from carriers of misse

103 iated (P = .01) with FPGS gene expression in lymphoblastoid cell lines derived from combined HapMap A

104 , cytosine modification levels in 133 HapMap lymphoblastoid cell lines derived from individuals of Eu

105 ed gene expression in the full set of HapMap lymphoblastoid cell lines derived from individuals of Eu

106 In this study, utilizing lymphoblastoid cell lines derived from International Hap

107 We performed functional assays by using lymphoblastoid cell lines derived from members of Chines

108 ofiling by CpG island microarray analysis of lymphoblastoid cell lines derived from monozygotic twins

109 ition, signaling, and repair mechanisms in B lymphoblastoid cell lines derived from patients with ped

110 used an unbiased whole-genome approach using lymphoblastoid cell lines derived from persons of Europe

111 d expression information for EBV-transformed lymphoblastoid cell lines derived from populations acros

112 expression by affecting the binding to GR in lymphoblastoid cell lines derived from the same patients

113 We sequenced RNA from 69 lymphoblastoid cell lines derived from unrelated Nigeria

114 ar susceptibility to cisplatin in 176 HapMap lymphoblastoid cell lines derived from Yoruba individual

115 Nonleukemic EBV-transformed lymphoblastoid cell lines displayed highly stable replic

116 in retroviral infection using the chicken B lymphoblastoid cell line DT40.

117 cific quantification assays to a panel of HD lymphoblastoid cell lines, each carrying the major Europ

118 Surprisingly, in lymphoblastoid cell lines, EBNA3C is extremely stable, a

119 a demonstration, Epstein-Barr virus-infected lymphoblastoid cell lines (EBV-LCL) were isolated based

120 We show for lymphoblastoid cell lines established from individuals o

121 Total RNA was isolated from lymphoblastoid cell lines established from patients, par

122 The B-lymphoblastoid cell lines exhibited a unimodal distribut

123 otein from V362I and R381Q variants in human lymphoblastoid cell lines exhibited lower expression lev

124 these mouse T cells efficiently killed human lymphoblastoid cell lines expressing endogenous HA-1 or

125 C1 ligand group are activated in vitro by B lymphoblastoid cell lines expressing the C2 group.

126 ines, four adult stem cell populations, four lymphoblastoid cell lines, five normal human tissues, an

127 udies, we collected RNA-sequencing data from lymphoblastoid cell lines for 431 Hutterite individuals.

128 red the cytotoxicity of 156 compounds in 884 lymphoblastoid cell lines for which genotype and transcr

129 measure chromatin accessibility in 70 Yoruba lymphoblastoid cell lines, for which genome-wide genotyp

130 A lymphoblastoid cell line from one affected individual sh

131 ed the genomes of a malignant melanoma and a lymphoblastoid cell line from the same person, providing

132 s to evaluate transcriptional profiles using lymphoblastoid cell lines from 62 prostate cancer patien

133 ne relative protein levels of 5,953 genes in lymphoblastoid cell lines from 95 diverse individuals ge

134 ormed a genome-wide gene expression study in lymphoblastoid cell lines from 96 Hutterites.

135 rometry to perform an integrated analysis in lymphoblastoid cell lines from a diverse group of indivi

136 We established lymphoblastoid cell lines from a G319S homozygote and co

137 Research design and methods: We established lymphoblastoid cell lines from a G319S homozygote and co

138 Using immortalized lymphoblastoid cell lines from a healthy study populatio

139 Protein blot analysis using lymphoblastoid cell lines from affected individuals show

140 OGT protein levels was observed in isolated lymphoblastoid cell lines from affected individuals, con

141 - to 3-fold overexpression of DIAPH3 mRNA in lymphoblastoid cell lines from affected individuals.

142 This miniATM variant was also highlighted in lymphoblastoid cell lines from AT patients and was shown

143 f NK cells to kill freshly established human lymphoblastoid cell lines from autologous or allogeneic

144 enes in Epstein-Barr virus (EBV)-transformed lymphoblastoid cell lines from children in the asthma fa

145 and extended in three human EBV-transformed lymphoblastoid cell lines from individuals with MSS, lea

146 rigin effect, except for differentiating the lymphoblastoid cell lines from other cell types.

147 Using simvastatin and sham incubated lymphoblastoid cell lines from participants of the Chole

148 tein (FMRP), its upregulation in transformed lymphoblastoid cell lines from patients with duplication

149 to interrogate DSB repair and recognition in lymphoblastoid cell lines from patients with pediatric S

150 suggest that DSB repair is defective in some lymphoblastoid cell lines from pediatric patients with S

151 In addition, we established lymphoblastoid cell lines from subjects homozygous for e

152 Lymphoblastoid cell lines from subjects with the p.V228F

153 We used 1,086 lymphoblastoid cell lines from the 1000 Genomes Project,

154 , we present a genome-wide model using human lymphoblastoid cell lines from the International HapMap

155 In lymphoblastoid cell lines from two translocation subject

156 we utilized gene expression association from lymphoblastoid cells lines from 754 p.Phe508del CF-affec

157 r virus-transformed B-cell cultures (human B-lymphoblastoid cell lines) from 19 healthy donors.

158 Lymphoblastoid cell lines generated from affected childr

159 Integration with ENCODE data from lymphoblastoid cell line GM12878, demonstrates that IRF4

160 cell RNA-seq protocol to study the reference lymphoblastoid cell line GM12878.

161 lic occupancy of 24 TFs and EP300 in a human lymphoblastoid cell line GM12878.

162 depletion and gene activation necessary for lymphoblastoid cell line growth and survival.

163 BNA-2 confers a type 1 growth phenotype in a lymphoblastoid cell line growth maintenance assay.

164 Moreover, NCL silencing impaired lymphoblastoid cell line growth.

165 DNA methylation (DNAm) measured in lymphoblastoid cell lines has been repeatedly demonstrat

166 icular, studies using FRDA patient blood and lymphoblastoid cell lines have detected increased DNA me

167 Applied to high-resolution Hi-C data in a lymphoblastoid cell line, HiC-DC detects significant int

168 k haplotype show no binding of STAT1, and in lymphoblastoid cell lines homozygous for the CAD non-ris

169 Lymphoblastoid cell lines homozygous for the CAD risk ha

170 in nonsynonymous substitutions in all three lymphoblastoid cell lines in our study, unlike RNA editi

171 nd transform B lymphocytes into immortalized lymphoblastoid cell lines in vitro.

172 using >300 expression microarrays (from 117 lymphoblastoid cell lines) in corticosteroid (dexamethas

173 h resulted in the generation of EBV-positive lymphoblastoid cell lines, indicating that the virus in

174 Here, we show that growth of a human B lymphoblastoid cell line infected with Epstein-Barr viru

175 rom the exosome fractions derived from human lymphoblastoid cell line (LCL) culture media.

176 o compare skin eQTLs to a published panel of lymphoblastoid cell line (LCL) eQTLs.

177 p16(INK4A) is essential for immortal human B-lymphoblastoid cell line (LCL) growth.

178 ytes with the autologous virus-transformed B-lymphoblastoid cell line (LCL) in vitro, can be used to

179 nt for virus infection in vivo, we studied a lymphoblastoid cell line (LCL) infected with an unusual

180 Lymphoblastoid cell line (LCL) is a common tool to study

181 n and subsequently almost completely ablated lymphoblastoid cell line (LCL) outgrowth.

182 (WES) are whole blood (WB) and immortalized lymphoblastoid cell line (LCL).

183 addition, human LRRK2 G2019S patient-derived lymphoblastoid cell lines (LCL) demonstrated increased m

184 l transcriptomics data from a panel of human lymphoblastoid cell lines (LCL) to infer drug response n

185 tore iNKT recognition in EBV-infected cells, lymphoblastoid cell lines (LCL) were treated with AM580,

186 inhibitor olaparib (AZD2281) of 5 ATM mutant lymphoblastoid cell lines (LCL), an ATM mutant MCL cell

187 latency II NPC C666-1 cells, and latency III lymphoblastoid cell lines (LCL).

188 allowed the establishment of MDV-transformed lymphoblastoid cell lines (LCL).

189 ntly reported gene expression changes in 480 lymphoblastoid cell lines (LCLs) after in vitro simvasta

190 lysis of RNA stability in seven human HapMap lymphoblastoid cell lines (LCLs) and analyzed the effect

191 lomere repeat elements (SREs) in transformed lymphoblastoid cell lines (LCLs) and human embryonic ste

192 otein isoforms across 68 Yoruba (YRI) HapMap lymphoblastoid cell lines (LCLs) and identified 12 cis a

193 ntinuous proliferation of primary B cells as lymphoblastoid cell lines (LCLs) and if EBV-negative BL-

194 on measured via RNA-seq analysis in adipose, lymphoblastoid cell lines (LCLs) and skin.

195 Using EBV-immortalized lymphoblastoid cell lines (LCLs) as a model, we found th

196 umented the DNA methylation pattern in human lymphoblastoid cell lines (LCLs) as well as identified s

197 B lymphocytes converted into lymphoblastoid cell lines (LCLs) by an Epstein-Barr viru

198 Bortezomib induced apoptosis of EBV lymphoblastoid cell lines (LCLs) by inducing cleavage of

199 pitopes were assayed against EBV-transformed lymphoblastoid cell lines (LCLs) containing lytically in

200 e lytically infected subset of early-passage lymphoblastoid cell lines (LCLs) could potentially contr

201 Patient-derived cell lines such as lymphoblastoid cell lines (LCLs) could represent the ide

202 lymphocytes into indefinitely proliferating lymphoblastoid cell lines (LCLs) depends on the concerte

203 Using a data set of gene expression in lymphoblastoid cell lines (LCLs) derived from 210 HapMap

204 Early-passage lymphoblastoid cell lines (LCLs) derived from EBV mutant

205 tosine modifications at 283,540 CpG sites in lymphoblastoid cell lines (LCLs) derived from independen

206 Here we report that RP-mutated lymphoblastoid cell lines (LCLs) established from DBA pa

207 Using lymphoblastoid cell lines (LCLs) established with EBV re

208 ene expression profiles using microarrays on lymphoblastoid cell lines (LCLs) from 413 cases and 446

209 romatin profiling for three histone marks in lymphoblastoid cell lines (LCLs) from 75 sequenced indiv

210 Previously we hypothesized that a subset of lymphoblastoid cell lines (LCLs) from children with auti

211 Here we analyze m(6)A mRNA modifications in lymphoblastoid cell lines (LCLs) from human, chimpanzee

212 tified H3K4me3-associated genomic regions in lymphoblastoid cell lines (LCLs) from humans, chimpanzee

213 enome-wide association studies involving 523 lymphoblastoid cell lines (LCLs) from individuals of Eur

214 A21 to assess trisomic protein expression in lymphoblastoid cell lines (LCLs) from patients with DS a

215 ng events in immortalized peripheral blood B lymphoblastoid cell lines (LCLs) from patients with RA a

216 Lymphoblastoid cell lines (LCLs) from some children with

217 Using RNA-sequence data from lymphoblastoid cell lines (LCLs) from the TwinsUK cohort

218 EBV to convert human B cells into long-lived lymphoblastoid cell lines (LCLs) in vitro requires the c

219 lymphocytes into continuously proliferating lymphoblastoid cell lines (LCLs) in vitro through manipu

220 for in vitro transformation of B cells into lymphoblastoid cell lines (LCLs) is activation of the NF

221 rther demonstrated that knockdown of H2AX in lymphoblastoid cell lines (LCLs) led to the upregulation

222 ion, elicited lytic EBV in latently infected lymphoblastoid cell lines (LCLs) partially via Toll-like

223 Epstein-Barr virus (EBV) transformed lymphoblastoid cell lines (LCLs) provide a conveniently

224 r Virus (EBV) conversion of B-lymphocytes to Lymphoblastoid Cell Lines (LCLs) requires four EBV nucle

225 and bisulfite sequencing of EBV-transformed lymphoblastoid cell lines (LCLs) shows extensive Wp meth

226 transformed into continuously proliferating lymphoblastoid cell lines (LCLs) that carry EBV DNA as e

227 The ZIIRmt-infected lymphoblastoid cell lines (LCLs) that did grow out exhib

228 Lymphoblastoid cell lines (LCLs) were cocultured with au

229 Populations of human-derived HapMap lymphoblastoid cell lines (LCLs) were infected with RSV.

230 between vitamin D receptor (VDR) binding in lymphoblastoid cell lines (LCLs), chromatin states in LC

231 In contrast to wild-type lymphoblastoid cell lines (LCLs), dividing LCLs establis

232 ChIP-seq performed on lymphoblastoid cell lines (LCLs), expressing epitope-tag

233 Human lymphoblastoid cell lines (LCLs), generated through Epst

234 In lytic-permissive lymphoblastoid cell lines (LCLs), pulse exposure to the

235 In GS-derived lymphoblastoid cell lines (LCLs), the proportion of ITPR

236 amining Epstein-Barr virus (EBV)-transformed lymphoblastoid cell lines (LCLs), we identified four EBV

237 sforms B cells to continuously proliferating lymphoblastoid cell lines (LCLs), which represent an exp

238 oth in newly infected primary B cells and in lymphoblastoid cell lines (LCLs).

239 ISPR/Cas9 loss-of-function screens in BL and lymphoblastoid cell lines (LCLs).

240 A polymerase II (Pol II) occupancy in Yoruba lymphoblastoid cell lines (LCLs).

241 s drives their indefinite proliferation into lymphoblastoid cell lines (LCLs).

242 roliferation and through transformation into lymphoblastoid cell lines (LCLs).

243 critical for B-lymphocyte transformation to lymphoblastoid cell lines (LCLs).

244 ansformation into indefinitely proliferating lymphoblastoid cell lines (LCLs).

245 response using 277 ethnically defined human lymphoblastoid cell lines (LCLs).

246 ls in response to autologous EBV-transformed lymphoblastoid cell lines (LCLs).

247 ific killing of autologous EBV-transformed B lymphoblastoid cell lines (LCLs).

248 n EBV-infected cells in vitro, as well as in lymphoblastoid cell lines (LCLs).

249 n vitro, EBV transforms primary B cells into lymphoblastoid cell lines (LCLs).

250 conducted a pharmacogenomic study using 266 lymphoblastoid cell lines (LCLs).

251 lation from chromatin to proteins, in Yoruba lymphoblastoid cell lines (LCLs).

252 s, nasopharyngeal carcinoma (NPC) cells, and lymphoblastoid cell lines (LCLs).

253 ll proliferation leading to the outgrowth of lymphoblastoid cell lines (LCLs).

254 a panel of iPSCs from 58 well-studied Yoruba lymphoblastoid cell lines (LCLs); 14 of these lines were

255 vitro results in their immortalization into lymphoblastoid cell lines (LCLs); this latency program i

256 s (CTLs) reactivated using EBV-transformed B-lymphoblastoid cells lines (LCLs) contained minor popula

257 Infection of the JY lymphoblastoid cell line limited the accumulation of a m

258 gh-throughput sequencing data sets for human lymphoblastoid cell lines mapped to the EBV genome.

259 4 and Epstein-Barr virus-transformed human B-lymphoblastoid cell lines mediated contact-dependent act

260 loci in genome-wide expression data sets of lymphoblastoid cell lines (n = 1,830) and were related t

261 Cellular studies of a lymphoblastoid cell line obtained from an affected patie

262 Lymphoblastoid cell lines obtained from a patient and fr

263 BMPR2 mRNA from lymphoblastoid cell lines of 30 families with PAH and 14

264 elationship between COMETs and haplotypes in lymphoblastoid cell lines of African and European origin

265 profiling of Epstein-Barr virus-transformed lymphoblastoid cell lines of all 270 individuals genotyp

266 In seven lymphoblastoid cell lines of Asian, European and African

267 -infected B lymphocytes and are critical for lymphoblastoid cell line outgrowth.

268 Human lymphoblastoid cell line (Raji), a poor host for HCV pse

269 Expression assays in 62 lymphoblastoid cell lines representing the six genotypes

270 lls or NK-cell clones with HLA-C2(+) CCR7(+) lymphoblastoid cell lines resulted in increased CCR7 upt

271 common CNVs in a cohort of 50 radiosensitive lymphoblastoid cell lines (RS-LCLs) derived from patient

272 perfectly-matched probes were identified in lymphoblastoid cell-line samples through comparison with

273 and in vivo proliferation of EBV-infected B lymphoblastoid cell lines (SLCL), derived from patients

274 entified as eQTL in monocytes, liver tissue, lymphoblastoid cell lines, T cells, and fibroblasts are

275 uencing in ATAC-seq libraries generated from lymphoblastoid cell lines: targeted cleavage of mitochon

276 exhibits higher expression in BLM-sensitive lymphoblastoid cell lines than insensitive cell lines up

277 tive CD23 expression in pt1 B cells, we used lymphoblastoid cell lines that express LMP1 under the co

278 CAMKK2 in human brains (P=1.1 x 10(-6)) and lymphoblastoid cell lines (the lowest P=8.4 x 10(-6)).

279 Excluding common rearrangements in lymphoblastoid cell lines, the fraction of CNVs in offsp

280 Here we report that, in lymphoblastoid cell lines, the translocation additionall

281 Furthermore, in the GM12878 lymphoblastoid cell line, these three genes are in a con

282 s, which leads to continuously proliferating lymphoblastoid cell lines through examination of the exp

283 kata-EBV and Raji), and cells from an EBV(+) lymphoblastoid cell line, thus suggesting a specific ass

284 In this investigation two isogenic human B-lymphoblastoid cell lines, TI-112 and TSCER2, were used

285 The B-lymphoblastoid cell line TK6 (p53 wild-type) and its p53

286 C-seq by sequencing the methylome of a human lymphoblastoid cell line to approximately 8.6x high-qual

287 for PBL-to-atrium; and from 0.81 to 0.98 for lymphoblastoid cell line-to-PBL based on cross-validatio

288 In an expression quantitative trait study in lymphoblastoid cell lines, transcript levels of the MTDH

289 logous dendritic cells and EBV-transformed B-lymphoblastoid cell lines transduced with an adenoviral

290 We show that EBNA3C-expressing lymphoblastoid cell lines treated with the drug nocodazo

291 nd drug sensitivity measurements in 24 human lymphoblastoid cell lines, was applied to a panel of 12

292 Using the HapMap lymphoblastoid cell lines, we combine 1000 Genomes genot

293 Using RNA derived from lymphoblastoid cell lines, we find that of 46 informativ

294 Applying riboHMM to human lymphoblastoid cell lines, we identified 7273 novel codi

295 Using metaphase spreads from human lymphoblastoid cell lines, we previously showed how immu

296 y members were enrolled, blood was obtained, lymphoblastoid cell lines were immortalized, deoxyribonu

297 to the establishment of permanently growing lymphoblastoid cell lines, whereas CD40L/IL-4 blasts hav

298 transcription factor binding sites in human lymphoblastoid cell lines, which is comprised of sites c

299 vant phenotypes in two closely related human lymphoblastoid cell lines with different p53 status, nam

300 iation in genome-wide gene expression in 107 lymphoblastoid cell lines, with alleles ranging from 15