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1 n increases both meningeal and parenchymal T lymphocyte number.
2 etrial angiogenesis and resident endometrial lymphocyte number.
3 hanced by combining this result with the CD4 lymphocyte number.
4 zygous mice show reduced CD4(+) and CD8(+) T lymphocyte numbers.
5 nockout) leads to a reduction in cytotoxic T lymphocyte numbers.
6 s that showed a significant increase in CD4+ lymphocyte numbers.
7 s in AIDS patients and with a decline in CD4 lymphocyte numbers.
8 .59 x (5) [0.39], p<0.001) and by suppressed lymphocyte numbers (0.092 x 10(5) [0.044] vs 0.45 x 10(5
12 hocyte deletion is required for reduction of lymphocyte numbers after expansion in response to antige
14 autoimmunity, with relatively normal T and B lymphocyte numbers and a diverse TCR repertoire expands
15 nd are important for regulating peripheral T-lymphocyte numbers and can mediate apoptosis of mature T
17 l, multisystem correction, and nearly normal lymphocyte numbers and mitogenic proliferative responses
19 ng-associated lymph node CD4(+)- and CD8(+)- lymphocyte numbers and rates of proliferation, and CD4(+
20 MLNs, which was caused by a 85% reduction in lymphocyte numbers and reduced short-term B cell homing.
21 IGF-I stimulates thymic growth and increases lymphocyte numbers and that IGF-binding protein (IGFBP)-
22 osome 7 locus was identified to be linked to lymphocyte numbers, and genetic evaluation of the interv
23 able, had normal peripheral blood and spleen lymphocyte numbers, and had normal specific Ab responses
24 nonuclear cell (PBMC) ADA activity, improved lymphocyte numbers, and normal proliferative responses t
25 correlates with viral load and peripheral T lymphocyte numbers, and suggest that HIV-induced suscept
27 this syndrome, we studied viral load, CD4+ T-lymphocyte numbers as well as cellular and humoral immun
28 ls correlated significantly with circulating lymphocyte numbers but not with the IGHV mutation status
29 iciency led to small reduction of peripheral lymphocyte number by compromising both synapse and end-j
30 tress-induced changes in CD95 expression and lymphocyte number could be blocked by naltrexone or nalo
33 nd found that although these mice had normal lymphocyte numbers, gamma(c)/TSLPR double KO mice had a
35 did not observe significant differences in B lymphocyte numbers in any of the genotypes or treatment
36 2 mice did not have elevated erythrocyte and lymphocyte numbers in blood and marrow and did not have
37 ation significantly increased eosinophil and lymphocyte numbers in bronchoalveolar lavage fluid 24 h
38 cal restraint failed to induce a decrease in lymphocyte numbers in CD95-deficient (Fas(lpr/lpr)) mice
41 ded a dose-dependent decrease in circulating lymphocyte numbers in multiple species and showed a ther
43 e increases in RL, as well as eosinophil and lymphocyte numbers in the bronchoalveolar lavage fluid (
45 ion in the lung and increased macrophage and lymphocyte numbers in the lavage, indicating a dysregula
49 X40 not only increased CD4(+)CD44(+)CD62L(-) lymphocyte number, it upregulated IL-7Ralpha expression
50 bitor, significantly reduced the decrease in lymphocyte numbers observed during stress and significan
53 P < 0.05) and an increase in bronchoalveolar lymphocyte numbers (P < 0.05), but not a generalized inf
54 try showed increased neutrophil and CD3(+) T-lymphocyte numbers post-UVR in both groups (P < 0.01) wi
58 ing was highly reflective of the decrease in lymphocyte numbers, suggesting iatrogenic gaps in the VB
59 monia and reduced the bronchoalveolar lavage lymphocyte number to one-quarter of the total measured i
64 ver, it was surprising that lamina propria T lymphocyte numbers were diminished, as E-cadherin is not
67 ecause circulating neutrophil, monocyte, and lymphocyte numbers were increased markedly in L-selectin
69 intrapulmonary, Peyer's patch, and splenic T lymphocyte numbers were not reduced in alpha E-deficient
70 formation and increases cytokine-containing lymphocyte number while maintaining endometrial connecti
71 des airway hyperresponsiveness and increased lymphocyte numbers, with the latter trait being influenc
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