ライフサイエンスコーパス: lymphocytic choriomeningitis

1 NA viruses that cause human diseases such as lymphocytic choriomeningitis, Bolivian hemorrhagic fever

2 clearance of murine gamma-herpesvirus 68 and lymphocytic choriomeningitis clone 13 and reversed T cel

3 IgG-antigen complexes formed during chronic lymphocytic choriomeningitis infection can interfere wit

4 f HIV infection in humans and during chronic lymphocytic choriomeningitis infection in mice.

5 lls were demonstrated in vivo during chronic lymphocytic choriomeningitis infection.

6 and therapy, we focused on two RNA viruses: lymphocytic choriomeningitis (LCMV) and influenza (Flu).

7 s of coinfection, including the well-studied lymphocytic choriomeningitis (LCMV) and Pichinde (PICV)

8 for diabetes antigen tetramers and to LCMV (lymphocytic choriomeningitis)-reactive CD8+ T cells.

9 his concept, we have developed a recombinant lymphocytic choriomeningitis virus (LCMV) (rLCMVDeltaGP/

10 oteins of all known pathogenic arenaviruses, lymphocytic choriomeningitis virus (LCMV) and Lassa, Jun

11 essing of GPC from the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) and LASV, whic

12 infection with two natural mouse pathogens, lymphocytic choriomeningitis virus (LCMV) and murine cyt

13 -deficient (Prf1(--)) mice are infected with lymphocytic choriomeningitis virus (LCMV) and secondary

14 T cells were generated after infection with lymphocytic choriomeningitis virus (LCMV) and that these

15 Here we show that the arenaviruses lymphocytic choriomeningitis virus (LCMV) and the clinic

16 eoproteins (NPs) of the Old World arenavirus lymphocytic choriomeningitis virus (LCMV) and the New Wo

17 ce with Leishmania major and 2 wk later with lymphocytic choriomeningitis virus (LCMV) and then monit

18 Mice were infected with lymphocytic choriomeningitis virus (LCMV) and treated wi

19 te viral infections, such as infections with lymphocytic choriomeningitis virus (LCMV) and vaccinia v

20 topes from unrelated and pathogenic viruses, lymphocytic choriomeningitis virus (LCMV) and vaccinia v

21 In this study, using lymphocytic choriomeningitis virus (LCMV) and Zika virus

22 yelin oligodendrocyte glycoprotein (MOG) and lymphocytic choriomeningitis virus (LCMV) antigens, resp

23 Using lymphocytic choriomeningitis virus (LCMV) as a model of

24 owing infection with a persistent variant of lymphocytic choriomeningitis virus (LCMV) but have no im

25 We show that lymphocytic choriomeningitis virus (LCMV) can also inhib

26 Lymphocytic choriomeningitis virus (LCMV) can cause acut

27 Lymphocytic choriomeningitis virus (LCMV) can establish

28 ablishment of life-long chronic infection by lymphocytic choriomeningitis virus (LCMV) Cl 13 but does

29 nic phase of infection with HIV in humans or lymphocytic choriomeningitis virus (LCMV) clone 13 in mi

30 ) is maximally induced on APCs at day 2 post-lymphocytic choriomeningitis virus (LCMV) clone 13 infec

31 effector CD8 T cells from mice infected with lymphocytic choriomeningitis virus (LCMV) clone 13 into

32 During chronic infection with lymphocytic choriomeningitis virus (LCMV) clone 13, miR-

33 aride (LPS) with that of a persistent virus, lymphocytic choriomeningitis virus (LCMV) clone 13, on e

34 Lymphocytic choriomeningitis virus (LCMV) clone 13, whic

35 rst 12-24 hours after mice are infected with lymphocytic choriomeningitis virus (LCMV) clone 13, whic

36 ses during persistent infection of mice with lymphocytic choriomeningitis virus (LCMV) clone 13.

37 from a septic event are more susceptible to lymphocytic choriomeningitis virus (LCMV) clone-13 infec

38 diabetic NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus (LCMV) delayed diabet

39 report that mice persistently infected with lymphocytic choriomeningitis virus (LCMV) exhibit a seve

40 this study we found that mice infected with lymphocytic choriomeningitis virus (LCMV) exhibit global

41 rmore, pDC-deficient animals failed to clear lymphocytic choriomeningitis virus (LCMV) from hematopoi

42 Mice immunized with H(2)O(2)-inactivated lymphocytic choriomeningitis virus (LCMV) generated cyto

43 -defective adenovirus vectors expressing the lymphocytic choriomeningitis virus (LCMV) glycoprotein (

44 Ad5, Ad26, Ad35, and Ad48 vectors expressing lymphocytic choriomeningitis virus (LCMV) glycoprotein (

45 we immunized mice with Ad5 vectors encoding lymphocytic choriomeningitis virus (LCMV) glycoprotein (

46 However, the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) has proven to

47 enic CD4(+) and CD8(+) T cells responding to lymphocytic choriomeningitis virus (LCMV) identified mul

48 onse during acute primary infection with the lymphocytic choriomeningitis virus (LCMV) in mice is sig

49 n with hepatitis B and C virus in humans and lymphocytic choriomeningitis virus (LCMV) in mice.

50 viruses pseudotyped with the glycoprotein of lymphocytic choriomeningitis virus (LCMV) in vitro.

51 in mice changes the outcome to a subsequent lymphocytic choriomeningitis virus (LCMV) infection and

52 Cs) is amplified during chronic versus acute lymphocytic choriomeningitis virus (LCMV) infection and

53 Although much is known about lymphocytic choriomeningitis virus (LCMV) infection and

54 ulated on virus-specific CTLs during chronic lymphocytic choriomeningitis virus (LCMV) infection and

55 d the role of IAPs in T-cell immunity during lymphocytic choriomeningitis virus (LCMV) infection by p

56 Although cellular immunity to acute lymphocytic choriomeningitis virus (LCMV) infection has

57 IL-10 production over the course of chronic lymphocytic choriomeningitis virus (LCMV) infection in a

58 ls contribute to establishment of persistent lymphocytic choriomeningitis virus (LCMV) infection in m

59 ector function of CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection in m

60 t CD70 blockade during an acute or a chronic lymphocytic choriomeningitis virus (LCMV) infection in m

61 In contrast to MCMV, lymphocytic choriomeningitis virus (LCMV) infection in m

62 Lymphocytic choriomeningitis virus (LCMV) infection indu

63 Using acute and chronic lymphocytic choriomeningitis virus (LCMV) infection mode

64 Using the lymphocytic choriomeningitis virus (LCMV) infection mode

65 Recently, several cases of fatal lymphocytic choriomeningitis virus (LCMV) infection occu

66 ll activation kinetics and viral loads after lymphocytic choriomeningitis virus (LCMV) infection of C

67 It is dramatic at 2 to 4 days following lymphocytic choriomeningitis virus (LCMV) infection of m

68 We show here that persistent lymphocytic choriomeningitis virus (LCMV) infection of m

69 this study, we used a mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection to a

70 se ranging from aseptic meningitis following lymphocytic choriomeningitis virus (LCMV) infection to h

71 The primary CD8(+) T-cell response to acute lymphocytic choriomeningitis virus (LCMV) infection was

72 minally differentiated CD8(+) T cells during lymphocytic choriomeningitis virus (LCMV) infection, and

73 found that during the first week of chronic lymphocytic choriomeningitis virus (LCMV) infection, bef

74 (+) T cells are essential for clearance of a lymphocytic choriomeningitis virus (LCMV) infection, but

75 ty, Hegazy et al. report that in response to lymphocytic choriomeningitis virus (LCMV) infection, ful

76 During persistent lymphocytic choriomeningitis virus (LCMV) infection, IFN

77 (+) T-cell formation following immunization, lymphocytic choriomeningitis virus (LCMV) infection, or

78 evelop fatal pathology during early systemic lymphocytic choriomeningitis virus (LCMV) infection, sug

79 In response to lymphocytic choriomeningitis virus (LCMV) infection, the

80 induction of exhaustion in mice with chronic lymphocytic choriomeningitis virus (LCMV) infection.

81 viral replication in the context of chronic lymphocytic choriomeningitis virus (LCMV) infection.

82 d IL-21 production at late stages of chronic lymphocytic choriomeningitis virus (LCMV) infection.

83 D-L1) blockade in the mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection.

84 y reproduced the manifestations of HLH after lymphocytic choriomeningitis virus (LCMV) infection.

85 of CD8 T cells are virus specific following lymphocytic choriomeningitis virus (LCMV) infection.

86 differentiation in vitro and in vivo during lymphocytic choriomeningitis virus (LCMV) infection.

87 e subsets to HLH-like disease severity after lymphocytic choriomeningitis virus (LCMV) infection.

88 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

89 e globally distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

90 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

91 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is an importan

92 The prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is widely used

93 g (ARM) or chronic Clone 13 (C13) strains of lymphocytic choriomeningitis virus (LCMV) led to two dis

94 Ins2-GP(Tg) mice injected with lymphocytic choriomeningitis virus (LCMV) lost islet sym

95 lowing infection with the clone 13 strain of lymphocytic choriomeningitis virus (LCMV) or influenza v

96 t Z protein of the Old World (OW) arenavirus lymphocytic choriomeningitis virus (LCMV) or Lassa virus

97 In mice infected with lymphocytic choriomeningitis virus (LCMV) or Listeria mo

98 ow-derived DC (BMDC) that were infected with lymphocytic choriomeningitis virus (LCMV) or loaded with

99 evidence that infection of mice with either lymphocytic choriomeningitis virus (LCMV) or pneumonia v

100 anasal infection with the systemic pathogens lymphocytic choriomeningitis virus (LCMV) or vaccinia vi

101 t isografts using RIP-LCMV mice expressing a lymphocytic choriomeningitis virus (LCMV) protein in the

102 Systemic low-dose infection of mice with lymphocytic choriomeningitis virus (LCMV) results in mas

103 l differentiation after acute infection with lymphocytic choriomeningitis virus (LCMV) strain Armstro

104 combinant antigen variant-expressing chronic lymphocytic choriomeningitis virus (LCMV) strains, we un

105 generated in mice persistently infected with lymphocytic choriomeningitis virus (LCMV) suppressed mul

106 The lymphocytic choriomeningitis virus (LCMV) system constit

107 d mice persistently infected from birth with lymphocytic choriomeningitis virus (LCMV) to demonstrate

108 We used the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to develop a g

109 The recombinant engineering of trisegmented lymphocytic choriomeningitis virus (LCMV) to express two

110 ted the ability of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

111 ility of the prototypic Old World arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

112 l where the viral nucleoprotein (NP) gene of lymphocytic choriomeningitis virus (LCMV) was expressed

113 leoprotein (NP) of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) with the least

114 tion) of the NP of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV), as well as th

115 costimulatory molecule expression induced by lymphocytic choriomeningitis virus (LCMV), CD28/B7-media

116 per that the prototype member in the family, lymphocytic choriomeningitis virus (LCMV), disables the

117 deficient (Prf1(-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV), disease is dr

118 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a model fo

119 worldwide-distributed prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a neglecte

120 d by coinfecting mice with L. guyanensis and lymphocytic choriomeningitis virus (LCMV), or the sand f

121 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), provides inve

122 ruses, including Ebola virus, Marburg virus, lymphocytic choriomeningitis virus (LCMV), rabies virus,

123 Lymphocytic choriomeningitis virus (LCMV), the prototype

124 us (IAV) and in memory phase challenged with lymphocytic choriomeningitis virus (LCMV), which we show

125 T(M) from lymphocytic choriomeningitis virus (LCMV)-immune and H60

126 replication is also partially controlled in lymphocytic choriomeningitis virus (LCMV)-immune C57BL/6

127 ate protein (AP)-fed controls, LP feeding in lymphocytic choriomeningitis virus (LCMV)-immune mice re

128 In this environment, lymphocytic choriomeningitis virus (LCMV)-infected iFRCs

129 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8(+

130 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8+

131 duced expression of CCL21 in murine spleens, lymphocytic choriomeningitis virus (LCMV)-specific T cel

132 itro and in vivo during acute infection with lymphocytic choriomeningitis virus (LCMV).

133 oma cells that replicate the negative-strand lymphocytic choriomeningitis virus (LCMV).

134 were challenged with the Armstrong strain of lymphocytic choriomeningitis virus (LCMV).

135 protection against viral infection, such as lymphocytic choriomeningitis virus (LCMV).

136 eted mice infected with the mouse arenavirus lymphocytic choriomeningitis virus (LCMV).

137 T) donor mice and infected the chimeras with lymphocytic choriomeningitis virus (LCMV).

138 mphohistiocytosis (HLH) after infection with lymphocytic choriomeningitis virus (LCMV).

139 fection of WASP-deficient (WAS KO) mice with lymphocytic choriomeningitis virus (LCMV).

140 pe I IFN response to many viruses, including lymphocytic choriomeningitis virus (LCMV).

141 iral loads in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

142 e in perforin-deficient mice is triggered by lymphocytic choriomeningitis virus (LCMV).

143 -Z interaction for the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

144 Ia and class II molecules were infected with lymphocytic choriomeningitis virus (LCMV).

145 ells generated after systemic infection with lymphocytic choriomeningitis virus (LCMV).

146 ctivating TCR signals after clearance of the lymphocytic choriomeningitis virus (LCMV).

147 nfection with vaccinia virus (VV) but not to lymphocytic choriomeningitis virus (LCMV).

148 ed in the murine model of chronic infection, lymphocytic choriomeningitis virus (LCMV).

149 ry pathway in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

150 D8(+) T cells following acute infection with lymphocytic choriomeningitis virus (LCMV).

151 on with vesicular stomatitis virus (VSV) and lymphocytic choriomeningitis virus (LCMV).

152 protect mice against infection with chronic lymphocytic choriomeningitis virus (LCMV).

153 f cells) during acute infection of mice with lymphocytic choriomeningitis virus (LCMV).

154 n the S segment of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

155 ntiviral effector T cells postinfection with lymphocytic choriomeningitis virus (LCMV).

156 replaced with a variant glycoprotein of the lymphocytic choriomeningitis virus (LCMV-GP), creating a

157 t viruses, murine cytomegalovirus (MCMV) and lymphocytic choriomeningitis virus (LCMV; Cl13), in thei

158 Lymphocytic choriomeningitis virus (LCVM) nucleoprotein

159 Ad5) prime followed by replication-defective lymphocytic choriomeningitis virus (rLCMV) boost elicite

160 We constructed recombinant lymphocytic choriomeningitis virus (rLCMV) featuring eit

161 at recombinants of the prototypic arenavirus lymphocytic choriomeningitis virus (rLCMVs), whose S-IGR

162 maintained on CD8(+) T cells during chronic lymphocytic choriomeningitis virus and hepatitis C virus

163 complex I was also required for infection of lymphocytic choriomeningitis virus and human parainfluen

164 4 failed to expand and to protect from acute lymphocytic choriomeningitis virus and Leishmania major

165 s and we describe its use in the contexts of lymphocytic choriomeningitis virus and Mycobacterium tub

166 d CD8(+) T cell responses to infections with lymphocytic choriomeningitis virus and the intracellular

167 only two mammarenaviruses, the widely spread lymphocytic choriomeningitis virus and the recently desc

168 cross-reactive CD8+ T cells recognizing both lymphocytic choriomeningitis virus and vaccinia virus an

169 luenza A virus and EBV in humans and between lymphocytic choriomeningitis virus and vaccinia virus in

170 I MHC protein H-2K(b) carrying peptides from lymphocytic choriomeningitis virus and vaccinia virus, a

171 infection of a naive C57BL/6 recipient with lymphocytic choriomeningitis virus and vaccinia virus, f

172 t in Atf3 showed enhanced viral clearance in lymphocytic choriomeningitis virus and vesicular stomati

173 t3, and importantly, anti-CD137 treatment of lymphocytic choriomeningitis virus Armstrong infected St

174 When administered at the beginning of lymphocytic choriomeningitis virus Armstrong infection a

175 yzed viperin expression in vivo during acute lymphocytic choriomeningitis virus Armstrong infection,

176 Using lymphocytic choriomeningitis virus Armstrong to probe th

177 e was followed by acute viral infection with lymphocytic choriomeningitis virus Armstrong.

178 of type I IFNs, and in persistently infected lymphocytic choriomeningitis virus carrier mice, which c

179 pecific CD8 T cells during acute and chronic lymphocytic choriomeningitis virus challenges, but did n

180 Pathogenic HIV or lymphocytic choriomeningitis virus chronic infections di

181 matopoietic or nonhematopoietic cells during lymphocytic choriomeningitis virus clone 13 (LCMV CL-13)

182 We find that the Old World arenaviruses lymphocytic choriomeningitis virus clone 13 (LCMV Cl13)

183 n PTPN22 resist chronic viral infection with lymphocytic choriomeningitis virus clone 13 (LCMV cl13).

184 s control and prevented chronic infection in lymphocytic choriomeningitis virus clone 13- and reduced

185 The G protein from lymphocytic choriomeningitis virus endowed VSV with the

186 isella strains expressing well-characterized lymphocytic choriomeningitis virus epitopes, we found th

187 recombinant adenovirus vector expressing the lymphocytic choriomeningitis virus glycoprotein (LCMVgp)

188 Infection with lymphocytic choriomeningitis virus induces monopoiesis i

189 exhibited a normal acute response (day 8) to lymphocytic choriomeningitis virus infection but generat

190 Consistent with this finding, we show that lymphocytic choriomeningitis virus infection can directl

191 tment of mice with rapamycin following acute lymphocytic choriomeningitis virus infection enhanced no

192 ined T cell responses to acute or persistent lymphocytic choriomeningitis virus infection in IFN-lamb

193 erived T cells are comparable in controlling lymphocytic choriomeningitis virus infection in mice and

194 regulates T cell dysfunction during chronic lymphocytic choriomeningitis virus infection in mice, an

195 ncoding the TGF-beta receptor during chronic lymphocytic choriomeningitis virus infection in mice, an

196 In a model of chronic lymphocytic choriomeningitis virus infection in mice, we

197 each stable levels of memory following acute lymphocytic choriomeningitis virus infection in mice.

198 s in virus-specific CD8 T cells during acute lymphocytic choriomeningitis virus infection in mice.

199 notherapeutic effects of IL-7 during chronic lymphocytic choriomeningitis virus infection in mice.

200 During lymphocytic choriomeningitis virus infection in vivo, PY

201 c memory CD4(+) T cell subpopulations in the lymphocytic choriomeningitis virus infection model, we f

202 We used a murine model of HLH, involving lymphocytic choriomeningitis virus infection of perforin

203 cell-extrinsic manner early following acute lymphocytic choriomeningitis virus infection to suppress

204 effectiveness of vaccination against lethal lymphocytic choriomeningitis virus infection using plasm

205 rvation of antiviral immune responses, acute lymphocytic choriomeningitis virus infection was used.

206 Here, we show that during chronic murine lymphocytic choriomeningitis virus infection, activation

207 ults in reduced viral clearance in models of lymphocytic choriomeningitis virus infection, and also p

208 y respond during the early stages of chronic lymphocytic choriomeningitis virus infection, and that t

209 after protein immunization; however, during lymphocytic choriomeningitis virus infection, B cells in

210 In chronic lymphocytic choriomeningitis virus infection, blockade o

211 Upon lymphocytic choriomeningitis virus infection, Cmah(-/-)

212 rrantly upregulated during memory to chronic lymphocytic choriomeningitis virus infection, limiting f

213 Following acute lymphocytic choriomeningitis virus infection, memory CD8

214 urthermore, in response to vaccinia virus or lymphocytic choriomeningitis virus infection, more Ag-sp

215 Using the experimental mouse model of lymphocytic choriomeningitis virus infection, we demonst

216 Following lymphocytic choriomeningitis virus infection, we found m

217 nd naive CD8(+) T cells to acute and chronic lymphocytic choriomeningitis virus infection, we show th

218 4hi CD8 and CD4 T cells at days 2 to 3 after lymphocytic choriomeningitis virus infection, when type

219 CD8(+) T cells to mount a robust response to lymphocytic choriomeningitis virus infection, with both

220 sion and increased cytokine production after lymphocytic choriomeningitis virus infection, yet DGK-de

221 ere also found in mouse liver within 24 h of lymphocytic choriomeningitis virus infection.

222 ephalitis, and defective immune responses to lymphocytic choriomeningitis virus infection.

223 i-CD20 before or different times after acute lymphocytic choriomeningitis virus infection.

224 d day-8 effector CD8 T cells following acute lymphocytic choriomeningitis virus infection.

225 e a crucial function in viral clearance upon lymphocytic choriomeningitis virus infection.

226 ptional profiles and chromatin states during lymphocytic choriomeningitis virus infection.

227 ry CD8 T-cell generation and responses after lymphocytic choriomeningitis virus infection.

228 exhaustion by Tim-3 and PD-1 during chronic lymphocytic choriomeningitis virus infection.

229 risk of developing HLH immunopathology after lymphocytic choriomeningitis virus infection.

230 xhaustion and the ability to contain chronic lymphocytic choriomeningitis virus infection.

231 virus infection to those in acute or chronic lymphocytic choriomeningitis virus infection.

232 cked gp33-specific CD8(+) T cells during RRV-lymphocytic choriomeningitis virus infection.

233 TE/TEM and TRM subsets was overcome by acute lymphocytic choriomeningitis virus infection; neverthele

234 isingly, they also show that chronic HIV and lymphocytic choriomeningitis virus infections have a ver

235 CD8 T cells responding to acute and chronic lymphocytic choriomeningitis virus infections.

236 G (IgG), and were unable to resolve chronic lymphocytic choriomeningitis virus infections.

237 ated stimulation during primary responses to lymphocytic choriomeningitis virus lowered the magnitude

238 To improve upon this, we used the murine lymphocytic choriomeningitis virus model and adenoviral

239 for CTL activation was also verified in the lymphocytic choriomeningitis virus model, in which IFN-b

240 r infection by two other NS RNA viruses, the lymphocytic choriomeningitis virus of the Arenaviridae f

241 splantation, the impact of an infection with lymphocytic choriomeningitis virus on prenatal allospeci

242 to acute or protracted viral infection with lymphocytic choriomeningitis virus or during autoimmune

243 ologous rechallenge of mice immune to either lymphocytic choriomeningitis virus or Listeria monocytog

244 und to be in cell cycle after infection with lymphocytic choriomeningitis virus or vesicular stomatit

245 on vesicular stomatitis virus, reovirus, or lymphocytic choriomeningitis virus replication but prote

246 ronic, but not acute, infection of mice with lymphocytic choriomeningitis virus results in a marked e

247 Infection of Spi6 knockout mice with lymphocytic choriomeningitis virus revealed impaired sur

248 namics of this cell turnover, we transferred lymphocytic choriomeningitis virus specific memory CD8 T

249 n T cell responses in mice given variants of lymphocytic choriomeningitis virus that cause acute or p

250 tly reduced in mice lacking B cells, whereas lymphocytic choriomeningitis virus titers were dramatica

251 We infected FtDKO mice with lymphocytic choriomeningitis virus to generate and track

252 vity of A3 against representative Old World (lymphocytic choriomeningitis virus) and New World (Junin

253 RNA was also detected in cells infected with lymphocytic choriomeningitis virus, an ambisense RNA vir

254 for JUNV and for vesicular stomatitis virus, lymphocytic choriomeningitis virus, and dengue virus but

255 uthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis virus, and West Nile virus.

256 We found that in mice infected with lymphocytic choriomeningitis virus, colocalization of vi

257 After infection of mice with lymphocytic choriomeningitis virus, IL-2Ralpha-deficient

258 ections with murine CMV (MCMV), but not with lymphocytic choriomeningitis virus, induce CD25 on NK ce

259 ction of NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus, neither of which dir

260 did not efficiently promote CD8 immunity to lymphocytic choriomeningitis virus, nor did cav-1(-/-) O

261 could be cross-reactive with three different lymphocytic choriomeningitis virus, one Pichinde virus,

262 After infection with lymphocytic choriomeningitis virus, pearl mice developed

263 contraction phase of an acute infection with lymphocytic choriomeningitis virus, we found that virus-

264 aring the influenza hemagglutinin or GP from lymphocytic choriomeningitis virus, which enter through

265 The clone 13 (Cl13) variant of lymphocytic choriomeningitis virus--a prototype of Old W

266 signaling does occur upon stimulation with a lymphocytic choriomeningitis virus-derived escape mutant

267 ely transferring splenocytes from individual lymphocytic choriomeningitis virus-immune donors into pa

268 in the form of panniculitis are observed in lymphocytic choriomeningitis virus-immune mice after vac

269 mmune diabetes development in the CD8-driven lymphocytic choriomeningitis virus-induced model of type

270 at Bim has a dual role in the development of lymphocytic choriomeningitis virus-induced, T cell-media

271 Using the highly dynamic model system of lymphocytic choriomeningitis virus-mediated hepatitis an

272 theless, neither germinal center B cells nor lymphocytic choriomeningitis virus-specific Ab levels we

273 of HIV-specific human CD8 T cells or chronic lymphocytic choriomeningitis virus-specific CD8 T cells

274 The majority (approximately 65% to 80%) of lymphocytic choriomeningitis virus-specific CD8 T cells

275 on fundamental T-cell functions, "exhausted" lymphocytic choriomeningitis virus-specific cells losing

276 ing and expansion of both primary and memory lymphocytic choriomeningitis virus-specific CTL, which c

277 +) memory B cells and the systemic levels of lymphocytic choriomeningitis virus-specific IgG2 Abs wer

278 ed T cells from peripheral T cells using the lymphocytic choriomeningitis virus-specific P14 TCR.

279 is virus infection, Cmah(-/-) mice make more lymphocytic choriomeningitis virus-specific T cells than

280 and IFN-I signaling blockade on the residual lymphocytic choriomeningitis virus-triggered CTL respons

281 T cells during acute infection of mice with lymphocytic choriomeningitis virus.

282 g an immune response to acute infection with lymphocytic choriomeningitis virus.

283 ibody production during acute infection with lymphocytic choriomeningitis virus.

284 onted with a highly disseminating variant of lymphocytic choriomeningitis virus.

285 loped encephalitis from organ donor-acquired lymphocytic choriomeningitis virus.

286 ls) to antiviral immunity after infection by lymphocytic choriomeningitis virus.

287 or the ability to mount a recall response to lymphocytic choriomeningitis virus.

288 hitecture in mice persistently infected with lymphocytic choriomeningitis virus.

289 nfluenza, H1N1; measles; dengue; rabies; and lymphocytic choriomeningitis virus.

290 pment of T-cell immunity by using the murine lymphocytic choriomeningitis virus.

291 t RNA viruses, including West Nile virus and lymphocytic choriomeningitis virus.

292 cell populations after acute infection with lymphocytic choriomeningitis virus.

293 infection with the prototypic mouse pathogen lymphocytic choriomeningitis virus.

294 ronic infection of mice with viruses such as lymphocytic choriomeningitis virus.

295 as did wild-type mice to four epitopes from lymphocytic choriomeningitis virus.

296 s that also express the gp33-41 epitope from lymphocytic choriomeningitis virus.

297 T cell responses during acute infection with lymphocytic choriomeningitis virus.

298 conditionally ablated in T cells, with acute lymphocytic choriomeningitis virus.

299 ell generation in response to infection with lymphocytic choriomeningitis virus.

300 determinant derived from the glycoprotein of lymphocytic choriomeningitis virus.