ライフサイエンスコーパス: lymphocytic choriomeningitis virus

1 T cells during acute infection of mice with lymphocytic choriomeningitis virus.

2 g an immune response to acute infection with lymphocytic choriomeningitis virus.

3 ibody production during acute infection with lymphocytic choriomeningitis virus.

4 onted with a highly disseminating variant of lymphocytic choriomeningitis virus.

5 loped encephalitis from organ donor-acquired lymphocytic choriomeningitis virus.

6 ls) to antiviral immunity after infection by lymphocytic choriomeningitis virus.

7 or the ability to mount a recall response to lymphocytic choriomeningitis virus.

8 hitecture in mice persistently infected with lymphocytic choriomeningitis virus.

9 nfluenza, H1N1; measles; dengue; rabies; and lymphocytic choriomeningitis virus.

10 pment of T-cell immunity by using the murine lymphocytic choriomeningitis virus.

11 t RNA viruses, including West Nile virus and lymphocytic choriomeningitis virus.

12 cell populations after acute infection with lymphocytic choriomeningitis virus.

13 infection with the prototypic mouse pathogen lymphocytic choriomeningitis virus.

14 ronic infection of mice with viruses such as lymphocytic choriomeningitis virus.

15 as did wild-type mice to four epitopes from lymphocytic choriomeningitis virus.

16 s that also express the gp33-41 epitope from lymphocytic choriomeningitis virus.

17 T cell responses during acute infection with lymphocytic choriomeningitis virus.

18 conditionally ablated in T cells, with acute lymphocytic choriomeningitis virus.

19 ell generation in response to infection with lymphocytic choriomeningitis virus.

20 determinant derived from the glycoprotein of lymphocytic choriomeningitis virus.

21 The clone 13 (Cl13) variant of lymphocytic choriomeningitis virus--a prototype of Old W

22 RNA was also detected in cells infected with lymphocytic choriomeningitis virus, an ambisense RNA vir

23 maintained on CD8(+) T cells during chronic lymphocytic choriomeningitis virus and hepatitis C virus

24 complex I was also required for infection of lymphocytic choriomeningitis virus and human parainfluen

25 4 failed to expand and to protect from acute lymphocytic choriomeningitis virus and Leishmania major

26 s and we describe its use in the contexts of lymphocytic choriomeningitis virus and Mycobacterium tub

27 d CD8(+) T cell responses to infections with lymphocytic choriomeningitis virus and the intracellular

28 only two mammarenaviruses, the widely spread lymphocytic choriomeningitis virus and the recently desc

29 cross-reactive CD8+ T cells recognizing both lymphocytic choriomeningitis virus and vaccinia virus an

30 luenza A virus and EBV in humans and between lymphocytic choriomeningitis virus and vaccinia virus in

31 I MHC protein H-2K(b) carrying peptides from lymphocytic choriomeningitis virus and vaccinia virus, a

32 infection of a naive C57BL/6 recipient with lymphocytic choriomeningitis virus and vaccinia virus, f

33 t in Atf3 showed enhanced viral clearance in lymphocytic choriomeningitis virus and vesicular stomati

34 vity of A3 against representative Old World (lymphocytic choriomeningitis virus) and New World (Junin

35 for JUNV and for vesicular stomatitis virus, lymphocytic choriomeningitis virus, and dengue virus but

36 uthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis virus, and West Nile virus.

37 Mice persistently infected from birth with lymphocytic choriomeningitis virus are tolerant to the p

38 t3, and importantly, anti-CD137 treatment of lymphocytic choriomeningitis virus Armstrong infected St

39 When administered at the beginning of lymphocytic choriomeningitis virus Armstrong infection a

40 yzed viperin expression in vivo during acute lymphocytic choriomeningitis virus Armstrong infection,

41 Using lymphocytic choriomeningitis virus Armstrong to probe th

42 e was followed by acute viral infection with lymphocytic choriomeningitis virus Armstrong.

43 of type I IFNs, and in persistently infected lymphocytic choriomeningitis virus carrier mice, which c

44 pecific CD8 T cells during acute and chronic lymphocytic choriomeningitis virus challenges, but did n

45 Pathogenic HIV or lymphocytic choriomeningitis virus chronic infections di

46 matopoietic or nonhematopoietic cells during lymphocytic choriomeningitis virus clone 13 (LCMV CL-13)

47 We find that the Old World arenaviruses lymphocytic choriomeningitis virus clone 13 (LCMV Cl13)

48 n PTPN22 resist chronic viral infection with lymphocytic choriomeningitis virus clone 13 (LCMV cl13).

49 s control and prevented chronic infection in lymphocytic choriomeningitis virus clone 13- and reduced

50 We found that in mice infected with lymphocytic choriomeningitis virus, colocalization of vi

51 signaling does occur upon stimulation with a lymphocytic choriomeningitis virus-derived escape mutant

52 The G protein from lymphocytic choriomeningitis virus endowed VSV with the

53 virus viral vectors expressing a polytope of lymphocytic choriomeningitis virus epitopes with differe

54 isella strains expressing well-characterized lymphocytic choriomeningitis virus epitopes, we found th

55 recombinant adenovirus vector expressing the lymphocytic choriomeningitis virus glycoprotein (LCMVgp)

56 After infection of mice with lymphocytic choriomeningitis virus, IL-2Ralpha-deficient

57 ely transferring splenocytes from individual lymphocytic choriomeningitis virus-immune donors into pa

58 in the form of panniculitis are observed in lymphocytic choriomeningitis virus-immune mice after vac

59 ections with murine CMV (MCMV), but not with lymphocytic choriomeningitis virus, induce CD25 on NK ce

60 mmune diabetes development in the CD8-driven lymphocytic choriomeningitis virus-induced model of type

61 at Bim has a dual role in the development of lymphocytic choriomeningitis virus-induced, T cell-media

62 Infection with lymphocytic choriomeningitis virus induces monopoiesis i

63 exhibited a normal acute response (day 8) to lymphocytic choriomeningitis virus infection but generat

64 Consistent with this finding, we show that lymphocytic choriomeningitis virus infection can directl

65 tment of mice with rapamycin following acute lymphocytic choriomeningitis virus infection enhanced no

66 ined T cell responses to acute or persistent lymphocytic choriomeningitis virus infection in IFN-lamb

67 erived T cells are comparable in controlling lymphocytic choriomeningitis virus infection in mice and

68 regulates T cell dysfunction during chronic lymphocytic choriomeningitis virus infection in mice, an

69 ncoding the TGF-beta receptor during chronic lymphocytic choriomeningitis virus infection in mice, an

70 In a model of chronic lymphocytic choriomeningitis virus infection in mice, we

71 each stable levels of memory following acute lymphocytic choriomeningitis virus infection in mice.

72 s in virus-specific CD8 T cells during acute lymphocytic choriomeningitis virus infection in mice.

73 notherapeutic effects of IL-7 during chronic lymphocytic choriomeningitis virus infection in mice.

74 During lymphocytic choriomeningitis virus infection in vivo, PY

75 c memory CD4(+) T cell subpopulations in the lymphocytic choriomeningitis virus infection model, we f

76 We used a murine model of HLH, involving lymphocytic choriomeningitis virus infection of perforin

77 cell-extrinsic manner early following acute lymphocytic choriomeningitis virus infection to suppress

78 effectiveness of vaccination against lethal lymphocytic choriomeningitis virus infection using plasm

79 rvation of antiviral immune responses, acute lymphocytic choriomeningitis virus infection was used.

80 Here, we show that during chronic murine lymphocytic choriomeningitis virus infection, activation

81 ults in reduced viral clearance in models of lymphocytic choriomeningitis virus infection, and also p

82 y respond during the early stages of chronic lymphocytic choriomeningitis virus infection, and that t

83 after protein immunization; however, during lymphocytic choriomeningitis virus infection, B cells in

84 In chronic lymphocytic choriomeningitis virus infection, blockade o

85 Upon lymphocytic choriomeningitis virus infection, Cmah(-/-)

86 rrantly upregulated during memory to chronic lymphocytic choriomeningitis virus infection, limiting f

87 Following acute lymphocytic choriomeningitis virus infection, memory CD8

88 urthermore, in response to vaccinia virus or lymphocytic choriomeningitis virus infection, more Ag-sp

89 Using the experimental mouse model of lymphocytic choriomeningitis virus infection, we demonst

90 Following lymphocytic choriomeningitis virus infection, we found m

91 nd naive CD8(+) T cells to acute and chronic lymphocytic choriomeningitis virus infection, we show th

92 4hi CD8 and CD4 T cells at days 2 to 3 after lymphocytic choriomeningitis virus infection, when type

93 CD8(+) T cells to mount a robust response to lymphocytic choriomeningitis virus infection, with both

94 sion and increased cytokine production after lymphocytic choriomeningitis virus infection, yet DGK-de

95 ere also found in mouse liver within 24 h of lymphocytic choriomeningitis virus infection.

96 ephalitis, and defective immune responses to lymphocytic choriomeningitis virus infection.

97 i-CD20 before or different times after acute lymphocytic choriomeningitis virus infection.

98 d day-8 effector CD8 T cells following acute lymphocytic choriomeningitis virus infection.

99 e a crucial function in viral clearance upon lymphocytic choriomeningitis virus infection.

100 ptional profiles and chromatin states during lymphocytic choriomeningitis virus infection.

101 ry CD8 T-cell generation and responses after lymphocytic choriomeningitis virus infection.

102 exhaustion by Tim-3 and PD-1 during chronic lymphocytic choriomeningitis virus infection.

103 risk of developing HLH immunopathology after lymphocytic choriomeningitis virus infection.

104 xhaustion and the ability to contain chronic lymphocytic choriomeningitis virus infection.

105 virus infection to those in acute or chronic lymphocytic choriomeningitis virus infection.

106 cked gp33-specific CD8(+) T cells during RRV-lymphocytic choriomeningitis virus infection.

107 TE/TEM and TRM subsets was overcome by acute lymphocytic choriomeningitis virus infection; neverthele

108 isingly, they also show that chronic HIV and lymphocytic choriomeningitis virus infections have a ver

109 odeficiency virus infections in macaques and lymphocytic choriomeningitis virus infections in mice, t

110 CD8 T cells responding to acute and chronic lymphocytic choriomeningitis virus infections.

111 G (IgG), and were unable to resolve chronic lymphocytic choriomeningitis virus infections.

112 CD8(+) T cells per mouse) and 1 in 2,958 for lymphocytic choriomeningitis virus (LCMV) ( approximatel

113 his concept, we have developed a recombinant lymphocytic choriomeningitis virus (LCMV) (rLCMVDeltaGP/

114 oteins of all known pathogenic arenaviruses, lymphocytic choriomeningitis virus (LCMV) and Lassa, Jun

115 essing of GPC from the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) and LASV, whic

116 infection with two natural mouse pathogens, lymphocytic choriomeningitis virus (LCMV) and murine cyt

117 -deficient (Prf1(--)) mice are infected with lymphocytic choriomeningitis virus (LCMV) and secondary

118 T cells were generated after infection with lymphocytic choriomeningitis virus (LCMV) and that these

119 Here we show that the arenaviruses lymphocytic choriomeningitis virus (LCMV) and the clinic

120 eoproteins (NPs) of the Old World arenavirus lymphocytic choriomeningitis virus (LCMV) and the New Wo

121 ce with Leishmania major and 2 wk later with lymphocytic choriomeningitis virus (LCMV) and then monit

122 Mice were infected with lymphocytic choriomeningitis virus (LCMV) and treated wi

123 te viral infections, such as infections with lymphocytic choriomeningitis virus (LCMV) and vaccinia v

124 topes from unrelated and pathogenic viruses, lymphocytic choriomeningitis virus (LCMV) and vaccinia v

125 In this study, using lymphocytic choriomeningitis virus (LCMV) and Zika virus

126 yelin oligodendrocyte glycoprotein (MOG) and lymphocytic choriomeningitis virus (LCMV) antigens, resp

127 Using lymphocytic choriomeningitis virus (LCMV) as a model of

128 owing infection with a persistent variant of lymphocytic choriomeningitis virus (LCMV) but have no im

129 We show that lymphocytic choriomeningitis virus (LCMV) can also inhib

130 Lymphocytic choriomeningitis virus (LCMV) can cause acut

131 Lymphocytic choriomeningitis virus (LCMV) can establish

132 ablishment of life-long chronic infection by lymphocytic choriomeningitis virus (LCMV) Cl 13 but does

133 nic phase of infection with HIV in humans or lymphocytic choriomeningitis virus (LCMV) clone 13 in mi

134 ) is maximally induced on APCs at day 2 post-lymphocytic choriomeningitis virus (LCMV) clone 13 infec

135 effector CD8 T cells from mice infected with lymphocytic choriomeningitis virus (LCMV) clone 13 into

136 During chronic infection with lymphocytic choriomeningitis virus (LCMV) clone 13, miR-

137 aride (LPS) with that of a persistent virus, lymphocytic choriomeningitis virus (LCMV) clone 13, on e

138 Lymphocytic choriomeningitis virus (LCMV) clone 13, whic

139 rst 12-24 hours after mice are infected with lymphocytic choriomeningitis virus (LCMV) clone 13, whic

140 ses during persistent infection of mice with lymphocytic choriomeningitis virus (LCMV) clone 13.

141 from a septic event are more susceptible to lymphocytic choriomeningitis virus (LCMV) clone-13 infec

142 diabetic NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus (LCMV) delayed diabet

143 Lymphocytic choriomeningitis virus (LCMV) establishes li

144 report that mice persistently infected with lymphocytic choriomeningitis virus (LCMV) exhibit a seve

145 this study we found that mice infected with lymphocytic choriomeningitis virus (LCMV) exhibit global

146 rmore, pDC-deficient animals failed to clear lymphocytic choriomeningitis virus (LCMV) from hematopoi

147 Mice immunized with H(2)O(2)-inactivated lymphocytic choriomeningitis virus (LCMV) generated cyto

148 -defective adenovirus vectors expressing the lymphocytic choriomeningitis virus (LCMV) glycoprotein (

149 Ad5, Ad26, Ad35, and Ad48 vectors expressing lymphocytic choriomeningitis virus (LCMV) glycoprotein (

150 we immunized mice with Ad5 vectors encoding lymphocytic choriomeningitis virus (LCMV) glycoprotein (

151 However, the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) has proven to

152 enic CD4(+) and CD8(+) T cells responding to lymphocytic choriomeningitis virus (LCMV) identified mul

153 onse during acute primary infection with the lymphocytic choriomeningitis virus (LCMV) in mice is sig

154 n with hepatitis B and C virus in humans and lymphocytic choriomeningitis virus (LCMV) in mice.

155 viruses pseudotyped with the glycoprotein of lymphocytic choriomeningitis virus (LCMV) in vitro.

156 in mice changes the outcome to a subsequent lymphocytic choriomeningitis virus (LCMV) infection and

157 Cs) is amplified during chronic versus acute lymphocytic choriomeningitis virus (LCMV) infection and

158 Although much is known about lymphocytic choriomeningitis virus (LCMV) infection and

159 ulated on virus-specific CTLs during chronic lymphocytic choriomeningitis virus (LCMV) infection and

160 d the role of IAPs in T-cell immunity during lymphocytic choriomeningitis virus (LCMV) infection by p

161 Although cellular immunity to acute lymphocytic choriomeningitis virus (LCMV) infection has

162 IL-10 production over the course of chronic lymphocytic choriomeningitis virus (LCMV) infection in a

163 ls contribute to establishment of persistent lymphocytic choriomeningitis virus (LCMV) infection in m

164 ector function of CD8 T cells during chronic lymphocytic choriomeningitis virus (LCMV) infection in m

165 t CD70 blockade during an acute or a chronic lymphocytic choriomeningitis virus (LCMV) infection in m

166 In contrast to MCMV, lymphocytic choriomeningitis virus (LCMV) infection in m

167 Lymphocytic choriomeningitis virus (LCMV) infection indu

168 Using acute and chronic lymphocytic choriomeningitis virus (LCMV) infection mode

169 Using the lymphocytic choriomeningitis virus (LCMV) infection mode

170 Recently, several cases of fatal lymphocytic choriomeningitis virus (LCMV) infection occu

171 ll activation kinetics and viral loads after lymphocytic choriomeningitis virus (LCMV) infection of C

172 It is dramatic at 2 to 4 days following lymphocytic choriomeningitis virus (LCMV) infection of m

173 We show here that persistent lymphocytic choriomeningitis virus (LCMV) infection of m

174 this study, we used a mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection to a

175 se ranging from aseptic meningitis following lymphocytic choriomeningitis virus (LCMV) infection to h

176 The primary CD8(+) T-cell response to acute lymphocytic choriomeningitis virus (LCMV) infection was

177 s and Ag-Ab complexes in T cell responses to lymphocytic choriomeningitis virus (LCMV) infection were

178 minally differentiated CD8(+) T cells during lymphocytic choriomeningitis virus (LCMV) infection, and

179 found that during the first week of chronic lymphocytic choriomeningitis virus (LCMV) infection, bef

180 (+) T cells are essential for clearance of a lymphocytic choriomeningitis virus (LCMV) infection, but

181 ty, Hegazy et al. report that in response to lymphocytic choriomeningitis virus (LCMV) infection, ful

182 During persistent lymphocytic choriomeningitis virus (LCMV) infection, IFN

183 (+) T-cell formation following immunization, lymphocytic choriomeningitis virus (LCMV) infection, or

184 evelop fatal pathology during early systemic lymphocytic choriomeningitis virus (LCMV) infection, sug

185 In response to lymphocytic choriomeningitis virus (LCMV) infection, the

186 viral replication in the context of chronic lymphocytic choriomeningitis virus (LCMV) infection.

187 d IL-21 production at late stages of chronic lymphocytic choriomeningitis virus (LCMV) infection.

188 D-L1) blockade in the mouse model of chronic lymphocytic choriomeningitis virus (LCMV) infection.

189 y reproduced the manifestations of HLH after lymphocytic choriomeningitis virus (LCMV) infection.

190 of CD8 T cells are virus specific following lymphocytic choriomeningitis virus (LCMV) infection.

191 differentiation in vitro and in vivo during lymphocytic choriomeningitis virus (LCMV) infection.

192 e subsets to HLH-like disease severity after lymphocytic choriomeningitis virus (LCMV) infection.

193 induction of exhaustion in mice with chronic lymphocytic choriomeningitis virus (LCMV) infection.

194 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

195 e globally distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

196 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is a neglected

197 worldwide-distributed prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is an importan

198 The prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) is widely used

199 g (ARM) or chronic Clone 13 (C13) strains of lymphocytic choriomeningitis virus (LCMV) led to two dis

200 Ins2-GP(Tg) mice injected with lymphocytic choriomeningitis virus (LCMV) lost islet sym

201 lowing infection with the clone 13 strain of lymphocytic choriomeningitis virus (LCMV) or influenza v

202 t Z protein of the Old World (OW) arenavirus lymphocytic choriomeningitis virus (LCMV) or Lassa virus

203 In mice infected with lymphocytic choriomeningitis virus (LCMV) or Listeria mo

204 ow-derived DC (BMDC) that were infected with lymphocytic choriomeningitis virus (LCMV) or loaded with

205 evidence that infection of mice with either lymphocytic choriomeningitis virus (LCMV) or pneumonia v

206 anasal infection with the systemic pathogens lymphocytic choriomeningitis virus (LCMV) or vaccinia vi

207 t isografts using RIP-LCMV mice expressing a lymphocytic choriomeningitis virus (LCMV) protein in the

208 Intracranial (i.c.) infection of mice with lymphocytic choriomeningitis virus (LCMV) results in ano

209 Systemic low-dose infection of mice with lymphocytic choriomeningitis virus (LCMV) results in mas

210 l differentiation after acute infection with lymphocytic choriomeningitis virus (LCMV) strain Armstro

211 combinant antigen variant-expressing chronic lymphocytic choriomeningitis virus (LCMV) strains, we un

212 generated in mice persistently infected with lymphocytic choriomeningitis virus (LCMV) suppressed mul

213 The lymphocytic choriomeningitis virus (LCMV) system constit

214 d mice persistently infected from birth with lymphocytic choriomeningitis virus (LCMV) to demonstrate

215 We used the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to develop a g

216 The recombinant engineering of trisegmented lymphocytic choriomeningitis virus (LCMV) to express two

217 ted the ability of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

218 ility of the prototypic Old World arenavirus lymphocytic choriomeningitis virus (LCMV) to interfere w

219 l where the viral nucleoprotein (NP) gene of lymphocytic choriomeningitis virus (LCMV) was expressed

220 leoprotein (NP) of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV) with the least

221 tion) of the NP of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV), as well as th

222 costimulatory molecule expression induced by lymphocytic choriomeningitis virus (LCMV), CD28/B7-media

223 per that the prototype member in the family, lymphocytic choriomeningitis virus (LCMV), disables the

224 deficient (Prf1(-/-)) mice are infected with lymphocytic choriomeningitis virus (LCMV), disease is dr

225 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a model fo

226 worldwide-distributed prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), is a neglecte

227 d by coinfecting mice with L. guyanensis and lymphocytic choriomeningitis virus (LCMV), or the sand f

228 The prototypic arenavirus, lymphocytic choriomeningitis virus (LCMV), provides inve

229 ruses, including Ebola virus, Marburg virus, lymphocytic choriomeningitis virus (LCMV), rabies virus,

230 Lymphocytic choriomeningitis virus (LCMV), the prototype

231 us (IAV) and in memory phase challenged with lymphocytic choriomeningitis virus (LCMV), which we show

232 T(M) from lymphocytic choriomeningitis virus (LCMV)-immune and H60

233 replication is also partially controlled in lymphocytic choriomeningitis virus (LCMV)-immune C57BL/6

234 ate protein (AP)-fed controls, LP feeding in lymphocytic choriomeningitis virus (LCMV)-immune mice re

235 In this environment, lymphocytic choriomeningitis virus (LCMV)-infected iFRCs

236 ed the fate of virus-specific CD8 T cells in lymphocytic choriomeningitis virus (LCMV)-infected mice

237 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8(+

238 Here, we show that lymphocytic choriomeningitis virus (LCMV)-specific CD8+

239 duced expression of CCL21 in murine spleens, lymphocytic choriomeningitis virus (LCMV)-specific T cel

240 itro and in vivo during acute infection with lymphocytic choriomeningitis virus (LCMV).

241 oma cells that replicate the negative-strand lymphocytic choriomeningitis virus (LCMV).

242 were challenged with the Armstrong strain of lymphocytic choriomeningitis virus (LCMV).

243 protection against viral infection, such as lymphocytic choriomeningitis virus (LCMV).

244 eted mice infected with the mouse arenavirus lymphocytic choriomeningitis virus (LCMV).

245 T) donor mice and infected the chimeras with lymphocytic choriomeningitis virus (LCMV).

246 mphohistiocytosis (HLH) after infection with lymphocytic choriomeningitis virus (LCMV).

247 fection of WASP-deficient (WAS KO) mice with lymphocytic choriomeningitis virus (LCMV).

248 pe I IFN response to many viruses, including lymphocytic choriomeningitis virus (LCMV).

249 e in perforin-deficient mice is triggered by lymphocytic choriomeningitis virus (LCMV).

250 iral loads in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

251 -Z interaction for the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

252 Ia and class II molecules were infected with lymphocytic choriomeningitis virus (LCMV).

253 ells generated after systemic infection with lymphocytic choriomeningitis virus (LCMV).

254 ctivating TCR signals after clearance of the lymphocytic choriomeningitis virus (LCMV).

255 nfection with vaccinia virus (VV) but not to lymphocytic choriomeningitis virus (LCMV).

256 ed in the murine model of chronic infection, lymphocytic choriomeningitis virus (LCMV).

257 ry pathway in mice chronically infected with lymphocytic choriomeningitis virus (LCMV).

258 D8(+) T cells following acute infection with lymphocytic choriomeningitis virus (LCMV).

259 on with vesicular stomatitis virus (VSV) and lymphocytic choriomeningitis virus (LCMV).

260 protect mice against infection with chronic lymphocytic choriomeningitis virus (LCMV).

261 f cells) during acute infection of mice with lymphocytic choriomeningitis virus (LCMV).

262 n the S segment of the prototypic arenavirus lymphocytic choriomeningitis virus (LCMV).

263 ntiviral effector T cells postinfection with lymphocytic choriomeningitis virus (LCMV).

264 replaced with a variant glycoprotein of the lymphocytic choriomeningitis virus (LCMV-GP), creating a

265 t viruses, murine cytomegalovirus (MCMV) and lymphocytic choriomeningitis virus (LCMV; Cl13), in thei

266 Lymphocytic choriomeningitis virus (LCVM) nucleoprotein

267 ated stimulation during primary responses to lymphocytic choriomeningitis virus lowered the magnitude

268 Using the highly dynamic model system of lymphocytic choriomeningitis virus-mediated hepatitis an

269 To improve upon this, we used the murine lymphocytic choriomeningitis virus model and adenoviral

270 for CTL activation was also verified in the lymphocytic choriomeningitis virus model, in which IFN-b

271 ction of NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus, neither of which dir

272 did not efficiently promote CD8 immunity to lymphocytic choriomeningitis virus, nor did cav-1(-/-) O

273 r infection by two other NS RNA viruses, the lymphocytic choriomeningitis virus of the Arenaviridae f

274 splantation, the impact of an infection with lymphocytic choriomeningitis virus on prenatal allospeci

275 could be cross-reactive with three different lymphocytic choriomeningitis virus, one Pichinde virus,

276 to acute or protracted viral infection with lymphocytic choriomeningitis virus or during autoimmune

277 ologous rechallenge of mice immune to either lymphocytic choriomeningitis virus or Listeria monocytog

278 und to be in cell cycle after infection with lymphocytic choriomeningitis virus or vesicular stomatit

279 After infection with lymphocytic choriomeningitis virus, pearl mice developed

280 on vesicular stomatitis virus, reovirus, or lymphocytic choriomeningitis virus replication but prote

281 ronic, but not acute, infection of mice with lymphocytic choriomeningitis virus results in a marked e

282 Infection of Spi6 knockout mice with lymphocytic choriomeningitis virus revealed impaired sur

283 Ad5) prime followed by replication-defective lymphocytic choriomeningitis virus (rLCMV) boost elicite

284 We constructed recombinant lymphocytic choriomeningitis virus (rLCMV) featuring eit

285 at recombinants of the prototypic arenavirus lymphocytic choriomeningitis virus (rLCMVs), whose S-IGR

286 namics of this cell turnover, we transferred lymphocytic choriomeningitis virus specific memory CD8 T

287 theless, neither germinal center B cells nor lymphocytic choriomeningitis virus-specific Ab levels we

288 of HIV-specific human CD8 T cells or chronic lymphocytic choriomeningitis virus-specific CD8 T cells

289 The majority (approximately 65% to 80%) of lymphocytic choriomeningitis virus-specific CD8 T cells

290 on fundamental T-cell functions, "exhausted" lymphocytic choriomeningitis virus-specific cells losing

291 ing and expansion of both primary and memory lymphocytic choriomeningitis virus-specific CTL, which c

292 +) memory B cells and the systemic levels of lymphocytic choriomeningitis virus-specific IgG2 Abs wer

293 ed T cells from peripheral T cells using the lymphocytic choriomeningitis virus-specific P14 TCR.

294 is virus infection, Cmah(-/-) mice make more lymphocytic choriomeningitis virus-specific T cells than

295 n T cell responses in mice given variants of lymphocytic choriomeningitis virus that cause acute or p

296 tly reduced in mice lacking B cells, whereas lymphocytic choriomeningitis virus titers were dramatica

297 We infected FtDKO mice with lymphocytic choriomeningitis virus to generate and track

298 and IFN-I signaling blockade on the residual lymphocytic choriomeningitis virus-triggered CTL respons

299 contraction phase of an acute infection with lymphocytic choriomeningitis virus, we found that virus-

300 aring the influenza hemagglutinin or GP from lymphocytic choriomeningitis virus, which enter through