コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ephalitis, and defective immune responses to lymphocytic choriomeningitis virus infection.
2 virus infection to those in acute or chronic lymphocytic choriomeningitis virus infection.
3 cked gp33-specific CD8(+) T cells during RRV-lymphocytic choriomeningitis virus infection.
4 ere also found in mouse liver within 24 h of lymphocytic choriomeningitis virus infection.
5 i-CD20 before or different times after acute lymphocytic choriomeningitis virus infection.
6 d day-8 effector CD8 T cells following acute lymphocytic choriomeningitis virus infection.
7 e a crucial function in viral clearance upon lymphocytic choriomeningitis virus infection.
8 ptional profiles and chromatin states during lymphocytic choriomeningitis virus infection.
9 ry CD8 T-cell generation and responses after lymphocytic choriomeningitis virus infection.
10 exhaustion by Tim-3 and PD-1 during chronic lymphocytic choriomeningitis virus infection.
11 xhaustion and the ability to contain chronic lymphocytic choriomeningitis virus infection.
12 ively normal numbers of memory T cells after lymphocytic choriomeningitis virus infection.
13 sufficient wild-type CD8 T cells after acute lymphocytic choriomeningitis virus infection.
14 massively accelerated type 1 diabetes after lymphocytic choriomeningitis virus infection.
15 onses against vesicular stomatitis virus and lymphocytic choriomeningitis virus infection.
16 posttranscriptional level during persistent lymphocytic choriomeningitis virus infection.
17 selectively to CD8(+) T cells responding to lymphocytic choriomeningitis virus infection.
18 risk of developing HLH immunopathology after lymphocytic choriomeningitis virus infection.
19 bility of CD40L-/- mice to control a chronic lymphocytic choriomeningitis virus infection.
20 G (IgG), and were unable to resolve chronic lymphocytic choriomeningitis virus infections.
21 CD8 T cells responding to acute and chronic lymphocytic choriomeningitis virus infections.
22 tal data on the cytotoxic T cell response to lymphocytic choriomeningitis virus infections.
23 equently challenged with acute or persistent lymphocytic choriomeningitis virus infections.
24 erial (Listeria monocytogenes) and/or viral (lymphocytic choriomeningitis virus) infections.
25 Here, we show that during chronic murine lymphocytic choriomeningitis virus infection, activation
26 on of the T cell repertoire during secondary lymphocytic choriomeningitis virus infection alters the
27 investigated the CD8(+) T cell responses to lymphocytic choriomeningitis virus infection and DNA imm
28 to CD8+ T-cell contraction following viral (lymphocytic choriomeningitis virus) infection and bacter
29 ults in reduced viral clearance in models of lymphocytic choriomeningitis virus infection, and also p
30 y respond during the early stages of chronic lymphocytic choriomeningitis virus infection, and that t
32 after protein immunization; however, during lymphocytic choriomeningitis virus infection, B cells in
34 exhibited a normal acute response (day 8) to lymphocytic choriomeningitis virus infection but generat
35 Consistent with this finding, we show that lymphocytic choriomeningitis virus infection can directl
37 ry CD8 T cells that developed after an acute lymphocytic choriomeningitis virus infection could persi
38 peak of the CTL response following an acute lymphocytic choriomeningitis virus infection, effector C
39 tment of mice with rapamycin following acute lymphocytic choriomeningitis virus infection enhanced no
42 isingly, they also show that chronic HIV and lymphocytic choriomeningitis virus infections have a ver
43 ined T cell responses to acute or persistent lymphocytic choriomeningitis virus infection in IFN-lamb
44 erived T cells are comparable in controlling lymphocytic choriomeningitis virus infection in mice and
45 ncoding the TGF-beta receptor during chronic lymphocytic choriomeningitis virus infection in mice, an
46 regulates T cell dysfunction during chronic lymphocytic choriomeningitis virus infection in mice, an
48 each stable levels of memory following acute lymphocytic choriomeningitis virus infection in mice.
49 s in virus-specific CD8 T cells during acute lymphocytic choriomeningitis virus infection in mice.
50 notherapeutic effects of IL-7 during chronic lymphocytic choriomeningitis virus infection in mice.
51 owed enhanced activation and expansion after lymphocytic choriomeningitis virus infection in vivo but
53 odeficiency virus infections in macaques and lymphocytic choriomeningitis virus infections in mice, t
54 sive CD8+ T cell expansion characteristic of lymphocytic choriomeningitis virus infection, leading to
55 rrantly upregulated during memory to chronic lymphocytic choriomeningitis virus infection, limiting f
59 c memory CD4(+) T cell subpopulations in the lymphocytic choriomeningitis virus infection model, we f
61 urthermore, in response to vaccinia virus or lymphocytic choriomeningitis virus infection, more Ag-sp
62 TE/TEM and TRM subsets was overcome by acute lymphocytic choriomeningitis virus infection; neverthele
64 fic CD8 T cells at various time points after lymphocytic choriomeningitis virus infection of mice.
65 We used a murine model of HLH, involving lymphocytic choriomeningitis virus infection of perforin
67 udies evaluating CD8 T cell responses during lymphocytic choriomeningitis virus infections of mice.
70 ve clonal expansion that occurs during acute lymphocytic choriomeningitis virus infection, telomere l
71 ice mounted a more robust immune response to lymphocytic choriomeningitis virus infection than did wi
72 cell-extrinsic manner early following acute lymphocytic choriomeningitis virus infection to suppress
73 n cytochrome c-specific T cell population or lymphocytic choriomeningitis virus infection tracked wit
74 effectiveness of vaccination against lethal lymphocytic choriomeningitis virus infection using plasm
76 rvation of antiviral immune responses, acute lymphocytic choriomeningitis virus infection was used.
80 nd naive CD8(+) T cells to acute and chronic lymphocytic choriomeningitis virus infection, we show th
81 rechallenging mice that had cleared primary lymphocytic choriomeningitis virus infections, we reveal
83 cell contraction after L. monocytogenes and lymphocytic choriomeningitis virus infections were indep
84 or CD8+ T cell responses to acute and memory lymphocytic choriomeningitis virus infections were unaff
85 4hi CD8 and CD4 T cells at days 2 to 3 after lymphocytic choriomeningitis virus infection, when type
86 ses we compared responses that cleared acute lymphocytic choriomeningitis virus infection with those
87 CD8(+) T cells to mount a robust response to lymphocytic choriomeningitis virus infection, with both
88 sion and increased cytokine production after lymphocytic choriomeningitis virus infection, yet DGK-de
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。