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1 he need for an alternative mechanism for the lymphocytopenia.
2 n 7 in a patient with idiopathic CD4+ T-cell lymphocytopenia.
3 icient patients develop a severe progressive lymphocytopenia.
4 ected patients, which are characterized by T lymphocytopenia.
5 ns or seizures even in the absence of severe lymphocytopenia.
6 lase (PNP) deficiency in humans results in T lymphocytopenia.
7 of splenic CD4(+)CD8(-) cells and a systemic lymphocytopenia.
8  was not predicted by the severity of CD4+ T lymphocytopenia.
9 ents included seizure, pleural effusion, and lymphocytopenia.
10 d are absent in patients with idiopathic CD4 lymphocytopenia.
11 21); and grade 3 or 4 thrombocytopenia (29), lymphocytopenia (18), and neutropenia (18).
12 e frequent in the cladribine groups included lymphocytopenia (21.6% in the 3.5-mg group and 31.5% in
13 d alopecia, a condition we named enteropathy-lymphocytopenia-alopecia.
14                                       Severe lymphocytopenia and an extent of GGO >50% on chest CT we
15 ia-telangiectasia (A-T) syndrome with severe lymphocytopenia and greatly increased risk for T-cell ly
16       In the second family, 2 brothers had B lymphocytopenia and immunoglobulin deficiencies.
17 Rdelta locus as the molecular origin of both lymphocytopenia and the signature t(12;14) translocation
18                Monocytopenia, B, NK, and CD4 lymphocytopenia correlated with the presence of disease
19  and 4 treatment-related adverse events were lymphocytopenia (eight [20%] of 40 patients), fatigue (e
20                            Idiopathic CD4+ T lymphocytopenia (ICL) is a rare heterogeneous disorder d
21                            Idiopathic CD4(+) lymphocytopenia (ICL) is a rare non-HIV-related syndrome
22 tion of microbial products in idiopathic CD4 lymphocytopenia (ICL), a rare disorder characterized by
23 this syndrome, a subset of idiopathic CD4+ T lymphocytopenia (ICL), is uncertain.
24 on of lymphocytes in white pulp and a marked lymphocytopenia in 15 of 19 patients.
25       Mutations in XLF/Cernunnos (XLF) cause lymphocytopenia in humans, and various studies suggest a
26           The repeated induction of profound lymphocytopenia in patients undergoing multiple cycles o
27  RAG chimeric mice exhibited profound T cell lymphocytopenia in the peripheral circulation.
28 sponse, underlies the progressive and severe lymphocytopenia in XLF-deficient patients in the absence
29                                       Severe lymphocytopenia (less than 300/microL) at the time of fi
30                                        CD8 T lymphocytopenia (< 180/mm(3)) and the degree of CD4 T ce
31 a consideration in patients with significant lymphocytopenia (<0.5 x 10(9) cells/L).
32 , who had developed severe and long-standing lymphocytopenia (<500/mm(3) ).
33 ells/microL; median, 4 cells/microL), and NK lymphocytopenia (mean, 16 cells/microL; median, 5.5 cell
34  cells/microL; median, 14.5 cells/microL), B lymphocytopenia (mean, 9.4 cells/microL; median, 4 cells
35                   ATM(-/-) mice recapitulate lymphocytopenia of A-T patients and routinely succumb to
36 eficient mice recapitulate the age-dependent lymphocytopenia of patients.
37 d of having miliary tuberculosis with severe lymphocytopenia or an extent of ground glass opacity (GG
38 herapy is warranted for patients with severe lymphocytopenia or positive blood PCR, and in those in w
39 examined using the key words: burn patients, lymphocytopenia, skin oxidative stress, antioxidant, mel
40 leoside phosphorylase (PNP) deficiency and T lymphocytopenia suggested that inhibition of this enzyme
41 cy (SCID), XLF mutations cause a progressive lymphocytopenia that is generally less severe than SCID.
42 ukoencephalopathy and idiopathic CD4+ T-cell lymphocytopenia were established, a 61-year-old man was
43 l patients displayed a profound, generalized lymphocytopenia, with few lymphocytes present in the lym

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