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1 ntigen system was established by analysis of lymphocytotoxic alloantibodies that were made by pregnan
2 ted the hypothesis that replacement of their lymphocytotoxic and nephrotoxic immunosuppression (combi
6 r coagulation, increasing height and weight, lymphocytotoxic antibody positivity, an increasing numbe
7 Platelet refractoriness was associated with lymphocytotoxic antibody positivity, heparin administrat
10 ve historical and pretransplant standard NIH lymphocytotoxic cross-matches and received the same immu
11 r allografts transplanted against a positive lymphocytotoxic crossmatch (CDC+) are susceptible to an
15 activity reported with a single course of a lymphocytotoxic drug in patients with mainly relapsed an
17 data are also relevant to patients receiving lymphocytotoxic mAb therapy for other indications, and t
18 studied patients who had previously received lymphocytotoxic monoclonal antibody (mAb) therapy for rh
19 in 53 RA patients who were treated with the lymphocytotoxic monoclonal antibody alemtuzumab between
20 clinicians and patients who are considering lymphocytotoxic or other immunomodulatory therapy for RA
23 The PRA screen was performed by use of the lymphocytotoxic technique treated with dithiothreitol to
24 correlate with the lymphocyte count prior to lymphocytotoxic therapy; however, after therapy the degr
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