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1  (DTT) showed that the majority of remaining lymphocytotoxicity against GalT-KO swine was mediated by
2 ed by sensitive antihuman globulin-augmented lymphocytotoxicity (AHG-CDC).
3   Screening was done by complement-dependent lymphocytotoxicity and antihuman globulin augmentation t
4              Preoperative crossmatches using lymphocytotoxicity and flow cytometry were negative.
5 onsiveness was demonstrated by cell-mediated lymphocytotoxicity and mixed lymphocyte response, and su
6 xed lymphocyte reaction (MLR), cell-mediated lymphocytotoxicity, and cytokine profile of unresponsive
7 fractoriness in longitudinal panels from 170 lymphocytotoxicity assay (LCA)(-) and 20 LCA(+) TRAP par
8 sms to sensitize appropriate target cells in lymphocytotoxicity assays and found that the MTF is deri
9 g 29 patients, FCXM and complement-dependent lymphocytotoxicity assays were performed 10+/-2 and 28+/
10 ytotoxic cross-matches (complement-dependent lymphocytotoxicity assays) were stratified on the basis
11 on of the four methods, complement-dependent lymphocytotoxicity (CDC) and C1q-, C4d-, and IgG-Luminex
12  than was obtainable by complement-dependent lymphocytotoxicity (CDC) assays has resulted in a new pa
13 phocyte reaction (MLR)-induced cell-mediated lymphocytotoxicity (CML) assay.
14  lymphocyte cultures (MLC) and cell-mediated lymphocytotoxicity (CML) culture systems analogous to th
15  lymphocyte reaction (MLR) and cell-mediated lymphocytotoxicity (CML).
16 nts had a positive T-cell or positive B-cell lymphocytotoxicity crossmatch against their donors.
17  or donors selected by leukoagglutination or lymphocytotoxicity crossmatching.
18                  In the complement-dependent lymphocytotoxicity (n=49; PRA=84.1+/-12.1%) and antihuma
19 lity was determined at study entry only by a lymphocytotoxicity screening assay (s-LCA) against a pan
20 lysis is a more useful clinical parameter of lymphocytotoxicity testing than simple reporting of % PR

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