ライフサイエンスコーパス: lymphoid cell

1 per development of adaptive, but not innate, lymphoid cells.

2 3aR(-), except some LP-derived type 3 innate lymphoid cells.

3 T cells, NK cells, and other group 1 innate lymphoid cells.

4 d OPN (sOPN) increase the population size of lymphoid cells.

5 s, basophils, eosinophils, and type 2 innate lymphoid cells.

6 romoting interleukin-13 production by innate lymphoid cells.

7 us expansion of IL-5-producing type 2 innate lymphoid cells.

8 ays recombinase activity in both myeloid and lymphoid cells.

9 ases the frequency of IL-17-producing innate lymphoid cells.

10 evels by gamma-delta T cells, NKT and innate lymphoid cells.

11 flammatory cytokines from myeloid and innate lymphoid cells.

12 O mice, which lack T cells but retain innate lymphoid cells.

13 ar receptors and is expressed in myeloid and lymphoid cells.

14 stable engraftment of functionally corrected lymphoid cells.

15 hemokine axis and by IL-13 expressing innate lymphoid cells.

16 eosinophils, mast cells, and group 3 innate lymphoid cells.

17 ), lymphoid tissue inducer (LTi), and innate lymphoid cell 1 (ILC1) cells, but not ILC2 or ILC3 cells

18 th pathways contribute to death of malignant lymphoid cells after treatment with dual mTORC1/mTORC2 i

19 Type 2 innate lymphoid cells and basophils were scarce in BAL fluid.

20 tion of chemokine responses in primary human lymphoid cells and cell lines that express CXCR4.

21 colon tissues, which activated type 2 innate lymphoid cells and dendritic cells to promote differenti

22 in the TH17 subset of helper T cells, innate lymphoid cells and gammadelta T cells resulted in the pr

23 the studies that formally identified innate lymphoid cells and highlight their emerging roles in con

24 mbers of eosinophils, IL-13(+) type 2 innate lymphoid cells and IL-13(+)CD4(+) T cells and IL-5 and I

25 ding eosinophils, mast cells, group 2 innate lymphoid cells and lymphocytes, which participate in the

26 light-chain-enhancer of activated B cells in lymphoid cells and monocytes.

27 cells and antigen-presenting cells to innate lymphoid cells and regulatory T cells.

28 derived from virions produced by infected T lymphoid cells and show that a single site is exclusivel

29 ulted in loss of AHR-dependent type 3 innate lymphoid cells and T helper 17 cells and increased susce

30 at engages unconventional type-1-like innate lymphoid cells and type 1 innate-like T cells.

31 ant role for vitamin A in controlling innate lymphoid cells and, consequently, postnatal formed lymph

32 CD300a is broadly expressed on myeloid and lymphoid cells, and its expression is differentially reg

33 ress, required extrinsic signals from innate lymphoid cells, and limited bacterial dissemination.

34 ely, eosinophils, Th2 T cells, type 2 innate lymphoid cells, and possibly Foxp3+ Tregs protect agains

35 d IL-13 coming from Th2 cells, type 2 innate lymphoid cells, and probably mast cells.

36 epithelial cells, macrophages, type 2 innate lymphoid cells, and TH2 cells along with increased Il33

37 l developmental intermediates, non-NK innate lymphoid cells, and the capacity for NK cells to adapt a

38 lly, we provide evidence that group 2 innate lymphoid cells are a source of IL-13, which promotes lun

39 ant IL-22 production, whereas group 1 innate lymphoid cells are accumulated in chronic inflammation o

40 Innate lymphoid cells are functionally diverse subsets of immun

41 Group 3 innate lymphoid cells are implicated in intestinal homeostasis

42 s, including Th17, gamma/delta T, and innate lymphoid cells, are differentially distributed along the

43 of eosinophils, Th2 cells, and type 2 innate lymphoid cells, associated with an increase in type 2 cy

44 Adipose type 1 innate lymphoid cells (AT1-ILCs) promote pro-inflammatory macro

45 d mortality after blunt trauma and suggest a lymphoid cell-based switch from self-resolving to self-s

46 ich previously has only been associated with lymphoid cell biology.

47 ng not only to the altered properties of RCS lymphoid cells but also to developmental defects.

48 eletion led to an overall increase in innate lymphoid cells (CD45(+)lin(-)CD25(+) cells) and IL-13(+)

49 d myeloid dendritic cells and group 2 innate lymphoid cells compared with mature TSLP.

50 , NK T cells, macrophages, and type 2 innate lymphoid cells compared with wild-type control mice.

51 (HSCs) generate more myeloid cells and fewer lymphoid cells compared with young HSCs, contributing to

52 al killer (NK) cells, a subset of the innate lymphoid cell compartment, are protective in viral myoca

53 lly resolved and organ-specific detection of lymphoid cell death during polymicrobial sepsis.

54 Here, we tested in vivo imaging of lymphoid cell death using a near-infrared annexin V (AV-

55 Sepsis-induced lymphoid cell death was detected by fluorescent AV-750 a

56 whether AHR antagonism could promote innate lymphoid cell differentiation from hESCs.

57 iciency reduced the number of group 2 innate lymphoid cells during allergen challenge but was not req

58 goblet cells, eosinophils, and type 2 innate lymphoid cells during parasite colonization.

59 damage-associated molecular patterns, innate lymphoid cells, epithelial-derived cytokines and chemoki

60 In adaptive lymphoid cells EZH2 prevented the premature expression o

61 d protein axis specifies innate and adaptive lymphoid cell fate.

62 itecture by an interfollicular infiltrate of lymphoid cells (Fig 1).

63 fficiently delivers tenofovir diphosphate to lymphoid cells following oral administration.

64 ficient mice, generating myeloerythoid and B-lymphoid cells for up to 4 months in vivo.

65 Type 2 innate lymphoid cells from polyps produce IL-5 and IL-13 in res

66 , we detect ERbeta protein in testis, ovary, lymphoid cells, granulosa cell tumours, and a subset of

67 lly divergent lineages, type 1 helper innate lymphoid cells (hILC1s) and conventional NK cells (cNKs)

68 blood DCs (ie, C1Q and CD141) or shared with lymphoid cells (ie, FcgammaRIIIA, GATA3, and RIPK4) refl

69 nd in ex vivo CD27(-)CD4(+) cells and innate lymphoid cell (ILC) 2 from patients with grass pollen al

70 The current model of murine innate lymphoid cell (ILC) development holds that mouse ILCs ar

71 ate early T cell progenitor (ETP) and innate lymphoid cell (ILC) development remains unclear.

72 Commitment to the innate lymphoid cell (ILC) lineage is determined by Id2, a tran

73 rogenitor population specified toward innate lymphoid cell (ILC) lineages, but their relationship wit

74 Little is known about innate lymphoid cell (ILC) populations in the human gut, and th

75 ed the development of tissue-resident innate lymphoid cell (ILC) subsets.

76 Innate lymphoid cells (ILC) are a heterogeneous group of cellul

77 signals that maintain tissue-resident innate lymphoid cells (ILC) in different microenvironments are

78 Innate lymphoid cells (ILC) play an important role in many immu

79 al infection regulates the balance of innate lymphoid cells (ILC), a diverse class of lymphocytes tha

80 r lymphocytes, specifically Th cells, innate lymphoid cells (ILC), and gammadelta T cells.

81 Recently, several groups of innate lymphoid cells (ILC), distinct from NK cells in developm

82 Group 2 innate lymphoid cells (ILC-2s) regulate immune responses to pat

83 ritical negative regulator of group 2 innate lymphoid cells (ILC-2s).

84 , we show that tissue-resident type 1 innate lymphoid cells (ILC1) serve an essential early role in h

85 the features that distinguish type 1 innate lymphoid cells (ILC1s) from natural killer (NK) cells is

86 binant HpARI abrogated IL-33, group 2 innate lymphoid cell (ILC2) and eosinophilic responses to Alter

87 l11b has a role in specifying type II innate lymphoid cell (ILC2) identity and blocks their conversio

88 y gene expression, and TH and group 2 innate lymphoid cell (ILC2) responses.

89 osinophils and both type 2 and type 3 innate lymphoid cells (ILC2 and ILC3), specifically in the port

90 to deregulated activation of group 2 innate lymphoid cells (ILC2 cells) and infection-associated typ

91 Group 2 innate lymphoid cells (ILC2 cells) are important for type 2 imm

92 Type-2 innate lymphoid cells (ILC2) are a prominent source of type II

93 Group 2 innate lymphoid cells (ILC2) are important in effector function

94 The recent discovery of group 2 innate lymphoid cells (ILC2) has increased our understanding of

95 Group 2 innate lymphoid cells (ILC2) have been implicated in the pathog

96 creased the proportion of ST2-bearing innate lymphoid cells (ILC2) in blood and kidneys, and adoptive

97 oduction by type 2 cytokine-producing innate lymphoid cells (ILC2) in the FALC.

98 Group 2 innate lymphoid cells (ILC2) include IL-5- and IL-13-producing

99 Type 2 innate lymphoid cells (ILC2) mediate inflammatory immune respon

100 Type 2 innate lymphoid cells (ILC2) share cytokine and transcription f

101 )-Rorasg/sg mice deficient in group 2 innate lymphoid cells (ILC2), and C57BL/6 wild-type mice treate

102 3, secreted by CD4(+) Th2 and group 2 innate lymphoid cells (ILC2), but whether certain metabolic enz

103 Here we show that type 2 innate lymphoid cells (ILC2), important mediators of barrier im

104 elper (NH) cells, a member of group 2 innate lymphoid cells (ILC2), to secrete Th2 type-cytokines.

105 ppressor cells (M-MDSCs), and group 2 innate lymphoid cells (ILC2).

106 It also caused a reduction in innate lymphoid cell, ILC2, and IL-9(+) and IL-13(+) ILC2 numbe

107 ted with the expansion of lung type 2 innate lymphoid cells (ILC2s) and are dependent on IL-13 and th

108 Group 2 innate lymphoid cells (ILC2s) and CD4(+) type 2 helper T cells

109 production by tissue-resident group 2 innate lymphoid cells (ILC2s) and recruited type 2 helper T cel

110 Type-2 innate lymphoid cells (ILC2s) and the acquired CD4(+) Th2 and T

111 of RSV infection to stimulate group 2 innate lymphoid cells (ILC2s) and the associated mechanism in a

112 Group 2 innate lymphoid cells (ILC2s) and type 2 helper T cells (Th2 ce

113 Type 2 innate lymphoid cells (ILC2s) and type 3 innate lymphoid cells

114 Group 2 innate lymphoid cells (ILC2s) are a potential innate source of

115 Group 2 innate lymphoid cells (ILC2s) are a recently identified group o

116 Group 2 innate lymphoid cells (ILC2s) are a subset of ILCs that play a

117 Group 2 innate lymphoid cells (ILC2s) are effector cells within the muc

118 Interleukin (IL)-33-dependent group 2 innate lymphoid cells (ILC2s) are enriched at barrier surfaces

119 Group 2 innate lymphoid cells (ILC2s) are important effector cells driv

120 Group 2 innate lymphoid cells (ILC2s) are involved in human diseases, s

121 Group 2 innate lymphoid cells (ILC2s) are involved in the initial phase

122 Group 2 innate lymphoid cells (ILC2s) are key regulators of type 2 infl

123 Type 2 innate lymphoid cells (ILC2s) are tissue sentinel mediators of

124 d interleukin (IL)-9-producing type 2 innate lymphoid cells (ILC2s) as the mediators of a molecular a

125 Type 2 innate lymphoid cells (ILC2s) both contribute to mucosal homeos

126 xpressed during development of type 2 innate lymphoid cells (ILC2s) but is not present at the mature

127 ablished, the newly identified type 2 innate lymphoid cells (ILC2s) can also contribute to orchestrat

128 Group 2 innate lymphoid cells (ILC2s) can regulate adaptive immunity an

129 RATIONALE: Newly characterized type 2 innate lymphoid cells (ILC2s) display potent type 2 effector fu

130 Group 2 innate lymphoid cells (ILC2s) expand in the lungs of mice durin

131 associated with the number of type 2 innate lymphoid cells (ILC2s) expressing IL-33Ralpha and IL-13

132 Group 2 innate lymphoid cells (ILC2s) have recently been shown to play

133 ced eosinophilia and expanded group 2 innate lymphoid cells (ILC2s) in aspirin-exacerbated respirator

134 tology, and real-time PCR; and type 2 innate lymphoid cells (ILC2s) in lung single-cell preparations

135 models of allogeneic BMT, that type 2 innate lymphoid cells (ILC2s) in the lower GI tract are sensiti

136 Finally, group 2 innate lymphoid cells (ILC2s) in the lungs showed robust expres

137 servations in prostate cancer.Group 2 innate lymphoid cells (ILC2s) modulate inflammatory and allergi

138 The recently identified type 2 innate lymphoid cells (ILC2s) play significant roles in the pat

139 Type 2 innate lymphoid cells (ILC2s) produce large amounts of Th2 cyto

140 Group 2 innate lymphoid cells (ILC2s) produce type 2 cytokines, and alt

141 Group 2 innate lymphoid cells (ILC2s) promote allergic inflammation.

142 Type 2 innate lymphoid cells (ILC2s) promote anti-helminth responses a

143 In innate immunity, IL-33 and group 2 innate lymphoid cells (ILC2s) provide an essential axis for rap

144 Group 2 innate lymphoid cells (ILC2s) regulate tissue inflammation and

145 Type 2 innate lymphoid cells (ILC2s) represent an important type 2 imm

146 Type 2 innate lymphoid cells (ILC2s) resemble TH2 cells and produce th

147 Type 2 innate lymphoid cells (ILC2s) resemble type 2 helper (Th2) cell

148 Group 2 innate lymphoid cells (ILC2s) reside in multiple organs in the

149 Group 2 innate lymphoid cells (ILC2s) robustly produce IL-5 and IL-13,

150 Resident intestinal type 2 innate lymphoid cells (ILC2s) were identified as the major prod

151 luding mast cells, basophils, group 2 innate lymphoid cells (ILC2s), and a subset of tissue-resident

152 initiates the accumulation of group 2 innate lymphoid cells (ILC2s), eosinophils, and alternatively a

153 3 are prominently secreted by group 2 innate lymphoid cells (ILC2s), which are stimulated by the proa

154 Group 2 innate lymphoid cells (ILC2s), which promote tissue eosinophili

155 cells and innate responses by group 2 innate lymphoid cells (ILC2s), with these latter being well cha

156 lated with reduced numbers of group 2 innate lymphoid cells (ILC2s).

157 le of the recently discovered group 2 innate lymphoid cells (ILC2s).

158 (Treg) cells and the emerging type 2 innate lymphoid cells (ILC2s).

159 activity of islet-associated group 2 innate lymphoid cells (ILC2s).

160 inflammation, eosinophils and group 2 innate lymphoid cells (ILC2s).

161 ne (IL-5, IL-13) production by type 2 innate lymphoid cells (ILC2s).

162 cNK development and supported group 3 innate lymphoid cell (ILC3) differentiation.

163 ing IL-22 production from the group 3 innate lymphoid cell (ILC3) in an aryl hydrocarbon receptor dep

164 ht vagus decreased peritoneal group 3 innate lymphoid cell (ILC3) numbers and altered peritoneal macr

165 dritic-cell-derived IL-10 and group 3 innate lymphoid cell (ILC3)-derived IL-22.

166 Intestinal T cells and group 3 innate lymphoid cells (ILC3 cells) control the composition of t

167 eased numbers of RORgammat(+) group 3 innate lymphoid cells (ILC3) correlates with an increased likel

168 However, ROR-gammat-dependent group 3 innate lymphoid cells ILC3s provide essential immunity and tiss

169 Type 3 innate lymphoid cells (ILC3s) and enteric glia, an essential st

170 Group 3 innate lymphoid cells (ILC3s) are important for intestinal heal

171 Group 3 innate lymphoid cells (ILC3s) are important regulators of the i

172 Type 3 innate lymphoid cells (ILC3s) are involved in maintenance of mu

173 Group 3 innate lymphoid cells (ILC3s) expressing the transcription fact

174 ate lymphoid cells (ILC2s) and type 3 innate lymphoid cells (ILC3s) have been implicated, respectivel

175 Group 3 innate lymphoid cells (ILC3s) have demonstrated roles in promot

176 s in depletion of intrathymic group 3 innate lymphoid cells (ILC3s) necessary for thymic regeneration

177 IL-23-driven tissue-resident group 3 innate lymphoid cells (ILC3s) play essential roles in intestina

178 :MHCII by RORgamma-expressing group 3 innate lymphoid cells (ILC3s), which are enriched within gut ti

179 were the recently identified group 3 innate lymphoid cells (ILC3s).

180 Innate lymphoid cell (ILCs) subsets differentially populate var

181 Innate lymphoid cells (ILCs) 'preferentially' localize into bar

182 d2 is constitutively expressed in all innate lymphoid cells (ILCs) and is required for their developm

183 precise lineage relationship between innate lymphoid cells (ILCs) and lymphoid tissue-inducer (LTi)

184 Innate lymphoid cells (ILCs) are a family of immune effector ce

185 Innate lymphoid cells (ILCs) are a family of innate immune cell

186 Innate lymphoid cells (ILCs) are a growing family of immune cel

187 Innate lymphoid cells (ILCs) are a new family of immune cells t

188 Innate lymphoid cells (ILCs) are a recently identified populati

189 Innate lymphoid cells (ILCs) are critical for maintaining epith

190 Innate lymphoid cells (ILCs) are critical mediators of mucosal

191 Innate lymphoid cells (ILCs) are effectors and regulators of in

192 Innate lymphoid cells (ILCs) are emerging as important regulato

193 Innate lymphoid cells (ILCs) are important regulators in variou

194 Innate lymphoid cells (ILCs) are increasingly acknowledged as i

195 Innate lymphoid cells (ILCs) are innate immune cells that are u

196 Innate lymphoid cells (ILCs) are known as first responders to i

197 Innate lymphoid cells (ILCs) are part of a heterogeneous family

198 Innate lymphoid cells (ILCs) are rapidly-responding cells that

199 Innate lymphoid cells (ILCs) are the most recently discovered g

200 Innate lymphoid cells (ILCs) are tissue-resident "first respond

201 Innate lymphoid cells (ILCs) are tuned to quickly respond to an

202 s study, we show that IL-7R-dependent innate lymphoid cells (ILCs) block LIP of CD8(+) T cells in neo

203 Innate lymphoid cells (ILCs) communicate with other haematopoie

204 Innate lymphoid cells (ILCs) contribute to barrier immunity, ti

205 Innate lymphoid cells (ILCs) contribute to host defence and tis

206 lls (DCs), but its role in regulating innate lymphoid cells (ILCs) during fetal and adult life is not

207 Innate lymphoid cells (ILCs) function to protect epithelial bar

208 Innate lymphoid cells (ILCs) functionally resemble T lymphocyte

209 In particular, the discovery of innate lymphoid cells (ILCs) has opened entirely new avenues fo

210 Innate lymphoid cells (ILCs) have an important role in the immu

211 Innate lymphoid cells (ILCs) have been classified into "functio

212 In recent years, innate lymphoid cells (ILCs) have emerged as innate correlates

213 etermine the potential involvement of innate lymphoid cells (ILCs) in allergic airway disease exacerb

214 ls have highlighted the importance of innate lymphoid cells (ILCs) in multiple immune responses.

215 inal role of T-bet-dependent NKp46(+) innate lymphoid cells (ILCs) in the initiation of CD4(+) TH17-m

216 The recognized diversity of innate lymphoid cells (ILCs) is rapidly expanding.

217 Innate lymphoid cells (ILCs) patrol environmental interfaces to

218 Innate lymphoid cells (ILCs) play a central role in the respons

219 Innate lymphoid cells (ILCs) play critical roles in immune home

220 s (Th cells), and recently identified innate lymphoid cells (ILCs) play important roles in host defen

221 Innate lymphoid cells (ILCs) play key roles in host defense, ba

222 by a subset of fetal CD103(+) group 3 innate lymphoid cells (ILCs) producing high levels of IL-17 and

223 Recent findings that innate lymphoid cells (ILCs) regulate adaptive T cell responses

224 s that drive the early development of innate lymphoid cells (ILCs) remain poorly understood.

225 Innate lymphoid cells (ILCs) represent innate versions of T hel

226 Subsets of innate lymphoid cells (ILCs) reside in the mucosa and regulate

227 Innate lymphoid cells (ILCs) serve as sentinels in mucosal tiss

228 Distinct groups of innate lymphoid cells (ILCs) such as ILC1, ILC2, and ILC3 popul

229 ispensable for the development of all innate lymphoid cells (ILCs) that express the interleukin 7 rec

230 ced, envelope (Env)-dependent mucosal innate lymphoid cells (ILCs) that produce interleukin (IL)-17,

231 quired for the expression of IL-22 in innate lymphoid cells (ILCs) upon T. gondii infection.

232 scriptional characteristics of sorted innate lymphoid cells (ILCs) were defined by using quantitative

233 he signals guiding differentiation of innate lymphoid cells (ILCs) within tissues are not well unders

234 Additionally, innate lymphoid cells (ILCs), important producers of cytokines

235 Innate lymphoid cells (ILCs), including NK cells, contribute to

236 s led to the formal identification of innate lymphoid cells (ILCs), increased the understanding of th

237 EP4 signaling acts directly on type 3 innate lymphoid cells (ILCs), promoting their homeostasis and d

238 innate-adaptive continuum and include innate lymphoid cells (ILCs), unconventional T cells (e.g., NKT

239 GR is selectively deleted in NKp46(+) innate lymphoid cells (ILCs), we demonstrated a major role for

240 inal lymph nodes, comprised mainly of innate lymphoid cells (ILCs); approximately 90% of IL-17-produc

241 NK cells are innate lymphoid cells important for immune surveillance, identi

242 al. (2015) demonstrate that resident innate lymphoid cells in subcutaneous fat generate and activate

243 have an increased frequency of type 2 innate lymphoid cells in the skin in comparison with control su

244 ion of IL-33, a stimulator of type II innate lymphoid cells, in lung epithelial cells was associated

245 rived cytokines that activate group 2 innate lymphoid cells, induce migration and activation of dendr

246 other cells of similar phenotype and loss of lymphoid cells infiltrating the islets.

247 pathology, mucus production, group 2 innate lymphoid cell infiltration, IL-5 and IL-13 production, a

248 IL-15 as a growth factor, we have selected a lymphoid cell line derived from rainbow trout head kidne

249 Here we show that exposure of a human lymphoid cell line to hyperthermia causes CDK5 insolubil

250 ne lymphocytic leukemia and mouse pro-B cell lymphoid cell lines, mitotic cells reversibly increase t

251 ocus further interrogation of the drivers of lymphoid cell loss with aging.

252 ges, including natural killer T cell, innate lymphoid cell, mucosal-associated invariant T cell and g

253 population in other organs, including innate lymphoid cells, natural killer cells, natural killer T c

254 The discovery of tissue-resident innate lymphoid cell populations effecting different forms of t

255 roteins were detected in both epithelial and lymphoid cell populations expressing CD155 in the tonsil

256 iscuss the identification of a common innate lymphoid cell precursor characterized by transient expre

257 PLZF expression at the innate lymphoid cell precursor stage has a long-range effect on

258 uced IL-22 expression, which required innate lymphoid cells, prevented microbiota encroachment and pr

259 ents with allergy and asthma, group 2 innate lymphoid cells produce high amounts of IL-5 and IL-13, t

260 e recently described pathway in which innate lymphoid cells produce interleukin-22 (IL-22) in respons

261 Type 3 innate lymphoid cells producing predominantly GM-CSF are expand

262 ly multipotent, yet they display both higher lymphoid cell production and greater capacity to generat

263 helper innate lymphoid progenitor and innate lymphoid cell progenitor compartments.

264 that was essentially identical to an innate lymphoid cell progenitor.

265 ssed on natural killer (NK) cells and innate lymphoid cells, recognizes PDGF-DD.

266 nantly TC1, TH1, and TH17 cells), and innate lymphoid cells recruited from the circulation.

267 ole in myeloid cell function but its role in lymphoid cells remains poorly defined.

268 TNF-alpha and IFN-gamma by liver myeloid and lymphoid cells, respectively.

269 deletion of the ETS1 transcription factor in lymphoid cells resulted in a loss of ILC2s in the bone m

270 led by the microbiota through group 3 innate lymphoid cells, STAT3 (signal transducer and activator o

271 eased STAT1 and STAT3 signaling responses in lymphoid cell subsets after surgery, consistent with enh

272 PRKCQ gene expression was assessed in innate lymphoid cell subsets purified from human PBMCs and mous

273 mma is known to be predominantly produced by lymphoid cells such as certain subsets of T cells, NK ce

274 rived from T-helper-2 (Th2) cells and innate lymphoid cells, such as interleukins 4, 5, and 13, as un

275 dendritic cell, mast cell, basophil, innate lymphoid cell, T-cell, and B-cell responses to allergens

276 CD, we observed B cells and apparent innate lymphoid cells that had invaded the lymphatic vessel wal

277 atural killer cells constitute potent innate lymphoid cells that play a major role in both tumor immu

278 s) define the origin and generation of early lymphoid cells that play essential roles in establishing

279 (IELs) are a large and diverse population of lymphoid cells that reside between the intestinal epithe

280 orchestrated by TH2 cells and type 2 innate lymphoid cells though release of IL-33 from epithelial c

281 pithelial cells, dendritic cells, and innate lymphoid cells translates to T-cell outcomes, with an em

282 conventional natural killer cells and innate lymphoid cells type 1.

283 ese mice, ozone increased lung IL-13+ innate lymphoid cells type 2 (ILC2) and IL-13+ gammadelta T cel

284 ontributes to differentiation of myeloid and lymphoid cell types, coordinates multicellular immunity,

285 nd that the presence of either donor or host lymphoid cells was required.

286 Unexpectedly, innate lymphoid cells were found to have a potent influence on

287 (vagina and cervix), whereas APCs and innate lymphoid cells were mainly located in the upper tract (u

288 Lymphoid cells were measured in peripheral blood of 55 p

289 inflammation, mast cells, and group 3 innate lymphoid cells were more enriched in adult-onset severe

290 A, and IL-22, all hallmarks of type 3 innate lymphoid cells, were expanded in the blood of patients w

291 Natural killer (NK) cells are innate lymphoid cells which mediate resistance against pathogen

292 ns of specific subsets of T cells and innate lymphoid cells, which are key drivers of inflammatory di

293 PP IgA class switching requires innate lymphoid cells, which promote lymphotoxin-beta receptor

294 ue injury in sepsis, activates type 2 innate lymphoid cells, which promote polarization of M2 macroph

295 increased lung IL-5-producing type 2 innate lymphoid cells, which required protease-activated recept

296 of using an immunomodulatory mAb to regulate lymphoid cells, which then recruit and activate myeloid

297 focus of the 2(nd)EMBO Conference on Innate Lymphoid Cells, which took place from November 30 to Dec

298 LZF, and the lineage relationships of innate lymphoid cells with conventional natural killer cells an

299 es of alloantigens, the cross talk of innate lymphoid cells with damaged epithelia and with the recip

300 Although CD103 marks many lymphoid cells within the gut, its direct functional rol