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1 nal activity by competing with T-cell factor/lymphoid enhancer factor.
7 ate the transcriptional activities of STAT1, lymphoid enhancer factor 1 (LEF-1), and the androgen rec
8 A FLS by transfection with dominant-negative lymphoid enhancer factor 1 (LEF-1)/T cell factor 4 (TCF-
9 ssociation with transcription factors of the lymphoid enhancer factor 1 (LEF-1)/T-cell factor (TCF) f
10 Contrasting examples are provided by SRY and lymphoid enhancer factor 1 (LEF-1): whereas the SRY tail
11 unique function for the transcription factor lymphoid enhancer factor 1 (LEF1) in the postselection e
12 that expression of the transcription factor lymphoid enhancer factor 1 (Lef1) is Notch dependent in
13 that the movo1 promoter is activated by the lymphoid enhancer factor 1 (LEF1)/beta-catenin complex,
14 beta-catenin and transcription activation of lymphoid enhancer factor 1 (LEF1)/T-cell factor (TCF) ta
15 ocket protein inhibitory complex, as well as lymphoid enhancer factor 1 and Pitx2, by dismissal of hi
16 omplex, which interacts with T-cell factor 4/lymphoid enhancer factor 1 transcription factors and act
20 lity group (HMG) DNA binding domain of human lymphoid enhancer factor-1 (hLEF-1) as bait in a yeast t
22 on of beta-catenin, which interacts with the lymphoid enhancer factor-1 (LEF)/T-cell factor (TCF) fam
24 d the ability of each Axin mutant to inhibit lymphoid enhancer factor-1 (Lef-1) reporter activity in
25 tion of beta-catenin and T cell factor (TCF)/lymphoid enhancer factor-1 (LEF-1), a transcriptional fa
26 s (-199 C to A) within a potential motif for lymphoid enhancer factor-1 (LEF-1), a transcriptional me
29 of the beta-catenin-dependent T-cell factor.lymphoid enhancer factor-1 transactivation and invasiven
30 yposis coli, GSK3beta, axin 1, beta-catenin, lymphoid enhancer factor-1, cyclin D1, and c-myc, reveal
31 dent on the cyclin D1 promoter T-cell factor/lymphoid enhancer factor-1-binding site between -81 to -
34 gher cytosolic beta-catenin and beta-catenin/lymphoid enhancer factor-1/T cell factor (beta-catenin/L
35 cleus, where it cooperates with LEF/TCF (for lymphoid enhancer factor and T-cell factor) transcriptio
36 ggest that HRG regulate Tiam1 activation and lymphoid enhancer factor/beta-catenin nuclear signaling
37 ain a single, highly conserved T-cell factor/lymphoid enhancer factor binding site that is required f
38 ts Wnt signaling, beta-catenin/T-cell factor/lymphoid-enhancer factor-dependent transcription and cel
39 y of Dvl3 to activate TCF/LEF (T-cell factor/lymphoid enhancer factor)-driven transcription and induc
40 ed to the nucleus, binds to T cell factor or lymphoid enhancer factor family members, and transactiva
42 ioned, chromatin-bound T-cell factor (Tcf) 1/lymphoid enhancer factor (Lef) 1 to facilitate recruitme
43 ted nuclear beta-catenin/T-cell factor (Tcf)-lymphoid enhancer factor (LEF) complex formation have be
45 Sox17 and Sox4 physically interact with TCF/lymphoid enhancer factor (LEF) family members via their
46 by beta-catenin and the T-cell factor (TCF)-lymphoid enhancer factor (LEF) family transcription fact
47 sient transfections with T cell factor (TCF)/lymphoid enhancer factor (LEF) optimized promoter report
49 scription factors of the T-cell Factor (Tcf)/Lymphoid Enhancer Factor (LEF) subfamily and mediates di
50 pathway, which activates T-cell factor (TCF)/lymphoid enhancer factor (LEF) transcription and is ofte
51 ation of beta-catenin, and activation of the lymphoid enhancer factor (LEF) transcription factor.
52 hrough the activation of T-cell factor (TCF)/lymphoid enhancer factor (LEF) transcription factors.
53 its interaction with T cell factor (TCF) or lymphoid enhancer factor (Lef) transcription factors.
54 expression causing formation of beta-catenin-lymphoid enhancer factor (LEF)-1 complexes that initiate
55 to Galpha(q), activates T-cell factor (Tcf)/lymphoid enhancer factor (Lef)-mediated transcriptional
56 r Ras, Neu, and Src, and by the beta-catenin/lymphoid enhancer factor (LEF)/T cell factor (TCF) pathw
58 criptional activation of T cell factor (Tcf)/lymphoid-enhancer factor (Lef), and expression of target
59 Wnt/Frizzled-1 signaling to the beta-catenin-lymphoid-enhancer factor (LEF)-T cell factor (Tcf) pathw
60 dence in the distribution of K17 and that of lymphoid-enhancer factor (lef-1), a DNA-bending protein
61 us and form complexes with any member of the lymphoid enhancer factor (LEF1) and T-cell factor (TCF1,
62 nscription factors of multiple T-cell factor/lymphoid enhancer factor regulatory elements, conserved
63 drives beta-catenin to bind to T-cell factor/lymphoid-enhancer factor response elements identified in
65 osphorylation and beta-catenin/T-cell factor/lymphoid enhancer factor signaling leading to MIG-7 upre
66 d, as it induced CD4 mRNA, and T cell factor/lymphoid enhancer factor sites were identified within th
67 ar receptor, androgen receptor (AR), and the Lymphoid Enhancer Factor/T Cell Factor family member, Le
70 e pathway to target genes are members of the lymphoid enhancer factor/T-cell factor (LEF/TCF) family.
71 an evolutionarily conserved binding site for lymphoid enhancer factor/T-cell factor family proteins,
73 vation, as judged by increased activity of a lymphoid enhancer factor/T-cell factor promoter reporter
75 cks beta-catenin accumulation, activation of lymphoid enhancer factor/T-cell factor protein-sensitive
77 presence of exogenous Wnt3a, as revealed by lymphoid enhancer factor/T-cell factor/beta-catenin repo
78 criptional coactivator for the T-cell factor/lymphoid enhancer factor (TCF/LEF) family of transcripti
80 ased the beta-catenin-mediated T cell factor/lymphoid enhancer factor (TCF/LEF) reporter activity, an
81 slocation and interaction with T-cell factor/lymphoid enhancer factor (TCF/LEF) transcription factors
83 ssor complex to a Wnt-mediated T-cell factor/lymphoid enhancer factor (TCF/LEF) transcriptional activ
84 er, Wnt3a-induced beta-catenin/T-cell factor/lymphoid enhancer factor (TCF/LEF) transcriptional activ
85 growth factor (EGF) stimulates T-cell factor/lymphoid enhancer factor (Tcf/Lef) transcriptional activ
86 uring liver regeneration using T cell factor/lymphoid enhancer factor (TCF/LEF)-reporter mice (TOPGal
88 ear stress-induced increase in T-cell factor/lymphoid enhancer factor transcriptional activity was ab
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