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1 o directly affect GC T cell migration within lymphoid follicles.
2 ty and functional activity of GCs in ectopic lymphoid follicles.
3 e thymocytes, and formation of thymic B cell lymphoid follicles.
4 to apoptosis that was localized to tonsillar lymphoid follicles.
5 inversely related to acute inflammation and lymphoid follicles.
6 of gastric inflammation, or the presence of lymphoid follicles.
7 n follicular structures resembling secondary lymphoid follicles.
8 capitulate many features of normal secondary lymphoid follicles.
9 und to be required for B-cell migration into lymphoid follicles.
10 around both immune and stromal cells within lymphoid follicles.
11 assive numbers of apoptotic cells within the lymphoid follicles.
12 idermis, hair follicles, and subconjunctival lymphoid follicles.
13 ng cells, strategically located over mucosal lymphoid follicles.
14 associated with an accumulation of pulmonary lymphoid follicles.
15 is and lymphoplasmacytic infiltrates without lymphoid follicles.
16 CR6 deficiency and remained clustered within lymphoid follicles.
17 f CXCL13 was observed within B-cell areas of lymphoid follicles.
18 progressive luminal occlusion, and tertiary lymphoid follicles.
19 are a subset of CD4(+) T cells in secondary lymphoid follicles.
20 ls, which accumulated in T-cell areas of the lymphoid follicles.
21 st immune response organized, remarkably, as lymphoid follicles.
22 lymphoid tissue, cryptopatches, and isolated lymphoid follicles.
23 followed by destruction of the integrity of lymphoid follicles.
24 of lymph node and were largely absent within lymphoid follicles.
25 ral replication is concentrated in secondary lymphoid follicles.
26 is (present in 42% of sections), presence of lymphoid follicles (32%), a plasma cell-enriched inflamm
27 ugh potentially Ag-reactive cells within the lymphoid follicle accumulate following antigenic challen
28 -1, which correlated with greater degrees of lymphoid follicle adherence and lesser degrees of ileal
29 ns demonstrated some degree of Peyer's patch lymphoid follicle adherence, ranging from diffuse covera
30 ion, we examined the changes in the appendix lymphoid follicle (ALF) and Peyer's patches (PP) in thes
32 crease in gastric B cells, plasma cells, and lymphoid follicles, along with enhanced H. pylori-specif
33 ic architecture in A/WySnJ, with few primary lymphoid follicles and a second abnormal phenotype, mast
34 on at the boundary of T cell:B cell areas in lymphoid follicles and ability to express IL-4 and CD40L
35 dysfunction as measured by the formation of lymphoid follicles and alteration of the thymocyte subse
36 as characterized by morphometric analysis of lymphoid follicles and by differentiating lymphocyte pop
37 atoid arthritis (RA) synovium often resemble lymphoid follicles and contain clonally related Ig trans
38 , we aimed at interrogating the link between lymphoid follicles and development of pulmonary inflamma
39 l disorganization, with reduction of defined lymphoid follicles and expansion of red pulp, a greater
40 rely ill mice was characterized by disrupted lymphoid follicles and fragmented nuclei, while the sple
41 mph nodes and had a defect in forming normal lymphoid follicles and germinal centers in spleen and ly
43 umbers and prevented CS-induced formation of lymphoid follicles and increases in immunoglobulin level
44 y which the TSE agent initially localizes to lymphoid follicles and interacts with FDCs are unknown.
46 inal fluid, and presence of EBV in meningeal lymphoid follicles and perivenular infiltrates in the wh
47 ymphocytes is known to block cell entry into lymphoid follicles and promote accumulation in T cell zo
48 he more inducible lymphoid tissues, isolated lymphoid follicles and tertiary lymphoid tissues, is unc
49 igration of Ag-transporting cells (ATC) into lymphoid follicles and the phenotypic similarity between
51 ophy of intestinal villus and colon-resident lymphoid follicle, and degeneration and atrophy of brain
53 g intestinal Peyer's patches (PPs), isolated lymphoid follicles, and nasal-associated lymphoid tissue
54 cells; the major stromal cell components in lymphoid follicle are the follicular dendritic cells (FD
57 and IL-8 and marked hyperplasia of secondary lymphoid follicles are early consequences of H. pylori i
59 hanisms underlying the exclusion of CTL from lymphoid follicles as well as the role of lymphoid folli
60 he rectal mucosa, specialized M cells of the lymphoid follicle-associated epithelium conduct vesicula
61 y not correlate with the formation of thymic lymphoid follicles but may correlate with the expansion
63 node may have resulted from colonization of lymphoid follicles by MALT lymphoma cells, following whi
64 A class switching in systemic and intestinal lymphoid follicles by shuttling Nef from infected macrop
65 ated with emphysema severity; (4) there were lymphoid follicles (CD20(+)IgM(+)) with active B cells (
66 rved ectopic lymphoid structures, defined as lymphoid follicles comprising clusters of B lymphocytes
67 P in the small intestine; in both instances, lymphoid follicles covered by surface epithelium (dome-f
69 occurs only in the epithelium over organized lymphoid follicles) deliver samples of foreign material
70 shown that advanced COPD is characterized by lymphoid follicles, drawing attention to immunological m
71 ssociated osteopontin covary with changes in lymphoid follicles during acute and late stages of infec
73 on recruits plasma cells and induces ectopic lymphoid follicle formation beneath the mucosal epitheli
77 of DCs that migrate rapidly into the primary lymphoid follicles from marginal zone after immunization
78 is expressed in the alveolar epithelium and lymphoid follicles from patients with IPF, and AKAP13 mR
79 lthy intestine, Peyer's patches and isolated lymphoid follicles generate protective and homeostatic i
80 hronic obstructive pulmonary disease (COPD), lymphoid follicles have been associated with disease sev
83 le to initiate development of CP or isolated lymphoid follicles (ILF) after transfer to CD132-null mi
84 mal cells in cryptopatches (CP) and isolated lymphoid follicles (ILF) in the small intestine of C57BL
88 tally 'imprinted' cryptopatches and isolated lymphoid follicles (ILFs), but not embryonically 'imprin
89 uce cryptopatches to transform into isolated lymphoid follicles (ILFs), which subsequently act as sit
92 tion response of Ag delivery by B cells into lymphoid follicles in an autoimmune condition has not be
94 ive Th1 response, the formation of secondary lymphoid follicles in granulomas and the role of Th1 res
98 tes within the germinal centers of secondary lymphoid follicles in normal and reactive nodes were TRA
99 om lymphoid follicles as well as the role of lymphoid follicles in perpetuating other chronic pathoge
101 ) DCs, expressing CXCR5, localize to primary lymphoid follicles in response to CXC ligand 13 (B lymph
102 2 or GPR183) directs B-cell migration in the lymphoid follicles in response to its endogenous ligands
104 MS, Th17 cells specifically induced ectopic lymphoid follicles in the central nervous system (CNS).
108 cells in the submucosa and ectopic tertiary lymphoid follicles in the ectocervix and vagina; and 3)
110 , for the first time, we illustrate isolated lymphoid follicles in the large intestine, consisting of
111 ties such as pleuritis and the effacement of lymphoid follicles in the regional lymph nodes and splee
115 last of these exhibited elements of classic lymphoid follicles, including networks of follicular den
116 eceptor needed for lymphocyte migration into lymphoid follicles indicates that multiple chemoattracti
119 13, a critical chemokine for B-cell entry to lymphoid follicles, is one of the most highly up-regulat
121 is known about what drives the appearance of lymphoid follicles (LFs), which may function as lymphoid
123 anced generation of IgA(+) cells in isolated lymphoid follicles of intestines offset defective intest
126 zed epithelia known as M cells overlying the lymphoid follicles of Peyer's patches are important in t
128 s, and they are located predominantly in the lymphoid follicles of the spleen and the lymph nodes.
130 issue and lead to the formation of inducible lymphoid follicles or aggregates that can mediate local
131 b2 as a nonredundant regulator that controls lymphoid follicle organization and germinal center react
132 ecursors was identified, suggesting that the lymphoid follicles represent newly formed, ectopic lymph
134 e the formation and persistence of meningeal lymphoid follicles suggest persistence of antigens to dr
135 issue-specific antiviral immune responses in lymphoid follicles that limit SIV replication in this pa
136 sustains the production of germinal centers, lymphoid follicles that ordinarily are anatomical signat
137 ity maturation occurs in germinal centers in lymphoid follicles through a critical interaction betwee
139 ntly, another subset of T cells in secondary lymphoid follicles was described, follicular regulatory
145 obule leukocytes and numerous active mucosal lymphoid follicles were present in infected animals.
146 eaths (PALS) and subsequently migrate to the lymphoid follicle where they enter nascent germinal cent
147 phoid tissues, but fail to accumulate within lymphoid follicles where HIV-1 replication is concentrat
149 these is a unique lymphoid tissue, isolated lymphoid follicles, which shares properties of both Peye
151 ated and typically leads to the formation of lymphoid follicles with germinal center (GC) reactions.
153 rs examined and clear evidence of extranodal lymphoid follicles with germinal center-like architectur
154 In a series of 64 synovial tissue biopsies, lymphoid follicles with germinal centers (GCs) were foun
156 es in infection and in the reconstitution of lymphoid follicles with treatment and that mapping genes
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