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1 xic T lymphocytes, natural killer cells, and lymphokine-activated killer cells.
2 cytotoxic T-lymphocytes, natural killer, or lymphokine-activated killer cells.
3 IL-4 was required to limit the induction of lymphokine-activated killer cells.
4 d in perforin- and granzyme-resistant CTL or lymphokine-activated killer cells.
6 cytotoxic T lymphocyte, natural killer, and lymphokine-activated killer cell activities compared wit
7 hocyte activity, and both natural killer and lymphokine-activated killer cell activities of graft non
8 oxic assay of PBL indicated the induction of lymphokine-activated killer cell activity, but no eviden
12 ions in T cell differentiation, induction of lymphokine-activated killer cells, and regulation of imm
14 otoxic lymphocytes, natural killer cells and lymphokine-activated killer cells depend primarily on th
15 actions of different human effector cells, (lymphokine-activated killer cells, gammadelta T cells, c
16 ytotoxic T lymphocytes and natural killer or lymphokine-activated killer cells is not blocked by BCR-
18 or T cell fraction derived from heterologous lymphokine activated killer cells kills those tumor cell
20 tion of murine and human T cell-mediated and lymphokine-activated killer cell-mediated cytotoxicity,
21 uces production of IFN-gamma and augments NK/lymphokine-activated killer cell proliferation and funct
22 mC and F genes in their MLR-derived CTLs and lymphokine-activated killer cells; removal of the PGK-ne
23 ndothelium and cytotoxicity against tumor by lymphokine-activated killer cells were not affected by P
25 cid mice previously reconstituted with human lymphokine-activated killer cells, whereas treatment wit
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