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1 ion in Jijoye cells (an EBV-positive Burkitt lymphoma cell line).
2 ith western blotting in the HDLM-2 Hodgkin's lymphoma cell line.
3  well as in a latently gamma HV68-infected B-lymphoma cell line.
4 egative control IgG2 in a CD20(+) human Raji lymphoma cell line.
5 F-1alpha), as was binding to the 3201 feline lymphoma cell line.
6 n during malignant progression of a murine B-lymphoma cell line.
7 GH in a three-way translocation in a Burkitt lymphoma cell line.
8 irus induced from a body cavity-based B-cell lymphoma cell line.
9  5 to 10 minutes of contact with the RAMOS B-lymphoma cell line.
10  cell line SW-2 than for an antigen-negative lymphoma cell line.
11 cent CpG sites are methylated in a Burkitt's lymphoma cell line.
12  germinal center, memory) and from a Burkitt lymphoma cell line.
13 the highly IFN-sensitive Daudi human Burkitt lymphoma cell line.
14 vels were observed in HL and part of Burkitt lymphoma cell lines.
15 5 correlate with resistance to vorinostat in lymphoma cell lines.
16 n the growth of tumor xenografts produced by lymphoma cell lines.
17 anidin-3-rutinoside, in several leukemia and lymphoma cell lines.
18 ptosis including Bcl-xl and Mcl-1 in several lymphoma cell lines.
19 riable antigenic expression on the different lymphoma cell lines.
20 erum starvation-induced apoptosis in human T lymphoma cell lines.
21 ssion is similar to that of primary effusion lymphoma cell lines.
22 he monoclonal antibody rituximab in vitro in lymphoma cell lines.
23 eceptor (BCR)-induced apoptosis in Burkitt's lymphoma cell lines.
24 rved in PC3 prostate cancer and Ramos B-cell lymphoma cell lines.
25 ied in over half of the PMBLs and in Hodgkin lymphoma cell lines.
26 fectively induce apoptosis in several B-cell lymphoma cell lines.
27 -cell lymphoid cell lines and in non-Hodgkin lymphoma cell lines.
28 entified in a fusion transcript with bcl6 in lymphoma cell lines.
29 ibody resulted in apoptosis of human Burkitt lymphoma cell lines.
30 or lines and 0.025-0.22 micro M for leukemia/lymphoma cell lines.
31 entially identical to EBV-positive Burkitt's lymphoma cell lines.
32 ative, patient-derived anaplastic large cell lymphoma cell lines.
33 lymphomas, we overexpressed NR4A1 in several lymphoma cell lines.
34 metalloproteinase (TIMP)-1 in various B cell lymphoma cell lines.
35 expressed in HHV-8-positive pleural effusion lymphoma cell lines.
36 on carcinoma cell lines but not in Burkitt's lymphoma cell lines.
37 TFO was also effective in other leukemia and lymphoma cell lines.
38 hosphorylation in human diffuse large B-cell lymphoma cell lines.
39 roliferation and induce apoptosis in Burkitt lymphoma cell lines.
40 or genes that increase the invasiveness of T lymphoma cell lines.
41 l death in aggressive histology human B-cell lymphoma cell lines.
42  when transfected into M12.4.5 and M12.4.1 B lymphoma cell lines.
43  or Wp in LCLs, as well as in some Burkitt's lymphoma cell lines.
44 n in wild type B cells and human mantle cell lymphoma cell lines.
45 rated very little activity against two human lymphoma cell lines.
46  translation in both non-Hodgkin and Hodgkin lymphoma cell lines.
47 tive activities against multiple myeloma and lymphoma cell lines.
48 platin and were up to 10-fold more active in lymphoma cell lines.
49 oma cell lines, primary MCL and other B cell lymphoma cell lines.
50 osphorylated TAK1, p38, and IkappaB-alpha in lymphoma cell lines.
51 rcoma-associated herpesvirus (KSHV)-infected lymphoma cell lines.
52 tle cell, anaplastic large cell, and Hodgkin lymphoma cell lines.
53 f NF-kappaB and p38 and induced apoptosis in lymphoma cell lines.
54 KSHV+ but not in Epstein-Barr virus (EBV)+ B-lymphoma cell lines.
55 nsive apoptosis in a variety of leukemia and lymphoma cell lines.
56 nscription factor PU.1 and CD10 in several B-lymphoma cell lines.
57 ion is also observed in other CD9-positive B lymphoma cell lines.
58 CT-1 oncogene was originally identified from lymphoma cell lines.
59 lected RNA isolated from a human body cavity lymphoma cell line 48 hr after butyrate induction of KSH
60 -induced apoptosis in the GC-sensitive pre-B lymphoma cell line, 697.
61                Here, using the murine B-cell lymphoma cell line A20, we show that the TNF-alpha gene
62 ectable protein 12-24 h later in the mouse B lymphoma cell line A20.
63                           Stimulation of a B lymphoma cell line, A20, with intact anti-IgG antibody i
64 ivation with HHV-8-positive primary effusion lymphoma cell lines, activated the lacZ gene of T1H6 in
65 , and HS-Sultan) and in diffuse large B cell lymphoma cell lines, although yin yang 1 protein (YY1) b
66 tivation of CD5-associated CK2 in a murine B-lymphoma cell line and a human T-leukemia cell line and
67                                In a murine B lymphoma cell line and an I-E(d)-transfected fibroblast
68 l progression, we have used an E2A-deficient lymphoma cell line and DNA microarray analysis to identi
69  of exosomes derived from the DG75 Burkitt's lymphoma cell line and its sublines (LMP1 transfected an
70 stitutive CCL17 secretion of a human Hodgkin lymphoma cell line and prevent upregulation of costimula
71 spond to HA22, we isolated an HA22-resistant lymphoma cell line and showed that resistance was due to
72 de in vitro cytotoxicity data for additional lymphoma cell lines and also chronic lymphocytic leukemi
73 hore A23187 to induce apoptosis in Burkitt's lymphoma cell lines and by measuring classical markers o
74 ssed in three different types of non-Hodgkin lymphoma cell lines and clinical samples as well as in m
75 titutively activated in primary tumors and B lymphoma cell lines and curcumin down-modulates Syk acti
76  of the multivalent aptamer was confirmed on lymphoma cell lines and fresh clinical leukemia samples.
77 f NRP-1 in representative human leukemia and lymphoma cell lines and in a panel of bone marrow specim
78                  Per2 levels were reduced in lymphoma cell lines and in acute myeloid leukemia (AML)
79                  Further validation in human lymphoma cell lines and in primary B-cell tumors demonst
80 hibitors bortezomib or carfilzomib in select lymphoma cell lines and induced potent mitochondrial mem
81 onstrate that PRMT5 knockdown in non-Hodgkin lymphoma cell lines and mouse primary lymphoma cells lea
82 on of global cap-dependent synthesis in both lymphoma cell lines and normal donor lymphocytes.
83 uce reactive oxygen species (ROS) in human B-lymphoma cell lines and primary B-cell chronic lymphocyt
84 hat trigger nonapoptotic PCD in a range of B-lymphoma cell lines and primary B-cell malignancies.
85 ellular cytotoxicity against B-cell leukemia/lymphoma cell lines and primary chronic lymphocytic leuk
86 diated adhesion to fibronectin and VCAM-1 of lymphoma cell lines and primary CLL cells.
87 a with hypocalcemic drugs sensitized human B lymphoma cell lines and primary human lymphoma cells to
88 FDC-mediated protection against apoptosis in lymphoma cell lines and primary lymphoma cells.
89                                       B-cell lymphoma cell lines and primary malignant B cells from p
90 ositive and -negative, anaplastic large cell lymphoma cell lines and primary patient tumours using th
91 we report the use of transformed non-Hodgkin lymphoma cell lines and primary samples to identify the
92            Using gene expression profiles of lymphoma cell lines and primary thymocytes treated with
93 tion of EZH2 A677 to a glycine (A677G) among lymphoma cell lines and primary tumor specimens.
94 usokine treatment led to direct apoptosis of lymphoma cell lines and primary tumors that otherwise we
95                In in vivo analyses of B-cell lymphoma cell lines and primary tumors, Syk inhibition i
96  and during cisplatin-induced apoptosis in B-lymphoma cell lines and spontaneous apoptosis of primary
97 phoma kinase-positive, anaplastic large cell lymphoma cell lines and that ectopically expressed JunB
98  in lysis of BL-3 cells (cells from a bovine lymphoma cell line) and sheep and human erythrocytes.
99 ckdowns of IRF8 expression in a mouse B cell lymphoma cell line, and examined the effects of a null m
100 yc promoter by 46-fold in the Raji Burkitt's lymphoma cell line, and it was the most active enhancer
101  of target genes in stable STAT6 transfected lymphoma cell lines, and elevated baseline expression le
102 etal and adult human CNS, in human leukemia, lymphoma cell lines, and in a variety of human cancers d
103 BV-negative lines, in EBV-infected Burkitt's lymphoma cell lines, and in EBV-transformed lymphoblasto
104 ession of miR-34a was not toxic in several B lymphoma cell lines, and inhibition of miR-34a impaired
105 herpesvirus (KSHV)-infected primary effusion lymphoma cell lines, and its expression correlates close
106 hronic lymphocytic leukaemia and mantle cell lymphoma cell lines, and patients treated with idelalisi
107  cells mediate the apoptotic death of murine lymphoma cell lines, and whether dendritic cell effector
108 n a subset of KSHV-infected primary effusion lymphoma cell lines as a consequence of altered processi
109  down-regulated in a variety of leukemia and lymphoma cell lines as well as in CD34+ cells from the b
110 toxicity in retargeting human PBMC against B-lymphoma cell lines as well as in mediating autologous B
111 nes, whereas it was expressed in non-Hodgkin lymphoma cell lines at levels comparable with normal B c
112 y shown to inhibit pro-liferation of several lymphoma cell lines at nanomolar concentrations in the a
113 hat anti-IgM-induced cell death in a human B lymphoma cell line, B104, is associated with early intra
114                                  The human B lymphoma cell line, B104, possesses characteristics that
115 cation in two KSHV-infected primary effusion lymphoma cell lines (BC-3 and BC-1) and in MTX-resistant
116 pression of siRNAs into the primary effusion lymphoma cell line BCBL-1 caused a substantial reduction
117 omes of a latently infected pleural effusion lymphoma cell line BCBL1.
118 f RNA extracted from frozen DLCL samples and lymphoma cell lines, BCL8 expression was detected in all
119            Mfn2-knockdown clones of a B-cell lymphoma cell line BJAB exhibited an increased rate of c
120 could be induced to kill the TRAIL-sensitive lymphoma cell line BJAB through a TRAIL-dependent mechan
121 mmortalized cell line IB4, and the Burkitt's lymphoma cell line BJAB.
122  the B cell receptor (BCR) on the immature B lymphoma cell line BKS-2 induces growth inhibition and a
123  WiDr) but promoted cell growth of Burkitt's lymphoma cell lines (BL30 and BL41) in a dose-dependent
124                                 In a Burkitt lymphoma cell line, both types of agents likewise stimul
125 as expressed in the poorly tumorigenic DAC B-lymphoma cell line but was significantly down-regulated
126              Infection of a primary effusion lymphoma cell line by vesicular stomatitis virus type G-
127 NA expression in a panel of primary effusion lymphoma cell lines by real-time RT-PCR recapitulated so
128 vities against Y3-Ag1.2.3 and the rat T cell lymphoma cell line C58 (NT) D.1.G.OVAR.1.
129 r describe a unique feature of the Burkitt's lymphoma cell line CA46 that allowed us to clearly demon
130 rated, using a cellular assay in a Burkitt's lymphoma cell line (CA46-specific), that these effects w
131                                           In lymphoma cell lines carrying BCL6 translocations, small
132 JAB cell line or v-Rel in the chicken DT40 B-lymphoma cell line causes reduced expression of PU.1.
133                                   In certain lymphoma cell lines, cellular proliferation is stimulate
134                        Twenty-five Burkitt's lymphoma cell lines (CL) and seven normal lymphoblastoid
135 s isotype class switching in a murine B-cell lymphoma cell line; conversely, B cells from mice where
136 have performed in vitro studies with a human lymphoma cell line (Daudi) because its response to anti-
137                  Here we describe two B-cell lymphoma cell lines (DB and RL) that differ in their sen
138                            We show that in a lymphoma cell line derived from an ATM(-/-) mouse, two t
139 the status of NF-kappaB in two non-Hodgkin's lymphoma cell lines derived from mantle cell lymphoma (M
140 ng of a conditionally BCL6-deficient Burkitt lymphoma cell line, DG75-AB7, with a library of small mo
141      Expression of the Fas receptor on the B lymphoma cell lines did not correlate with their capacit
142                          Using the chicken B lymphoma cell line DT40 as a model system, we investigat
143 oculation of both melanoma cell line B16 and lymphoma cell line EL-4.
144        When inoculated with a transplantable lymphoma cell line, EL4, the treated old mice showed sta
145                               The murine EL4 lymphoma cell line exists in variants that are either se
146   Analysis of p27kip1 derived from Burkitt's lymphoma cell lines expressing high levels of p27kip1, B
147                        In a range of Burkitt lymphoma cell lines, expression of DeltaCD79b was relati
148                                Human Burkitt lymphoma cell lines give rise to progressively growing s
149 examined by FACS analysis, only Raji (B cell lymphoma cell line), GM847 (fibroblast cell line), 293 (
150  lines, including two EBV-positive Burkitt's lymphoma cell lines, grew normally in the absence of miR
151 ation of endogenous CCR7 in the human T cell lymphoma cell line H9.
152                          The t(14;18) B-cell lymphoma cell line had low GSH levels and sensitivity to
153                     The diffuse large B cell lymphoma cell lines had a distinctive slow-migrating com
154 , on transfection of Raji cells, a Burkitt's lymphoma cell line harboring a transcriptionally inactiv
155 ivo, the stable lentiviral-transduced SuDHL4 lymphoma cell line harboring an inducible NR4A1 construc
156                                          Six lymphoma cell lines have 4- to 9-fold more binding sites
157  SHP-1 mRNA observed in various leukemia and lymphoma cell lines have been attributed to either the m
158 us, these results indicated that mantle cell lymphoma cell lines have distinct mechanisms sustaining
159 BV vIL-10 locus exclusively in the Hodgkin's lymphoma cell line, Hs 611.T, the expression of which we
160 rin on the viability of the cutaneous T-cell lymphoma cell lines HuT-78 and HH by using 3-(4,5-dimeth
161 ibitors on T-cell lymphoma, the human T-cell lymphoma cell line HUT78 was tested for sensitivity and
162 expression differences between two related T lymphoma cell lines, HuT78 and H9, were identified.
163 TAT6 phosphorylation in immune and Hodgkin's lymphoma cell lines, IL-13 binding, and cytotoxicity of
164                                           In lymphoma cell lines, IMGN529 induced G2/M cell cycle arr
165              Here we show that incubation of lymphoma cell lines in acidic conditions (pH 6.5) blocks
166 o analysis of KSHV-infected primary effusion lymphoma cell lines in the presence of 12-O-tetradecanoy
167 t the proliferation of myeloma and Burkitt's lymphoma cell lines in vitro without showing toxicity to
168 le of target-dependent killing of nonHodgkin lymphoma cell lines in vitro.
169 to inhibit the growth of several different B-lymphoma cell lines in vitro.
170 ved successful latent infection are cultured lymphoma cell lines.) In most latently infected lines, s
171 endent cytotoxicity against a broad panel of lymphoma cell lines including mantle cell lymphoma (MCL)
172 ncrease over HA22 were observed in various B lymphoma cell lines including WSU-CLL that contains only
173 xamination of a number of human leukemia and lymphoma cell lines, including Jurkat, Molt-4, and Sup-T
174 f TAK1 is abundantly expressed in a panel of lymphoma cell lines, including mantle cell, anaplastic l
175 d murine T cell acute lymphoblastic leukemia/lymphoma cell lines indicating that NOTCH1 signals are r
176 a library prepared from the BC-1 body cavity lymphoma cell line induced into KSHV lytic gene expressi
177 al Gag polyprotein and is shared by leukemia/lymphoma cell lines induced by Friend, Moloney, and Raus
178 atinase B by the high grade B-cell Burkitt's lymphoma cell lines is consistent with previous findings
179  initiation within newly established Burkitt lymphoma cell lines is consistent with the transcription
180  domain of MDC-L supported adhesion of the T-lymphoma cell line, Jurkat, in a concentration- and diva
181 ts ability to support cell adhesion of the T-lymphoma cell line, Jurkat.
182 sed the protein tyrosine kinase Src in the B lymphoma cell line K46-17 mu m lambda.
183 cing of PTPN1 by RNA interference in Hodgkin lymphoma cell line KM-H2 resulted in hyperphosphorylatio
184 mples with data from RCOR1 knockdowns in the lymphoma cell lines KM-H2 and Raji yielded an RCOR1 loss
185 nfection of murine B cell blasts, a murine B lymphoma cell line (L10A), and immortalized human B cell
186                             The human B-cell lymphoma cell line, L3055, formed solid tumors only when
187 erimental model using an FDC line, HK, and a lymphoma cell line, L3055, that resembles centroblasts.
188 r at the mRNA and protein levels relative to lymphoma cell lines lacking a 1q21 rearrangement.
189 ytic replication of EBV induced in a Burkitt lymphoma cell line latently infected with EBV.
190 rast to previous reports using CD20-positive lymphoma cell lines, lenalidomide down-regulated CD20 su
191 apoptosis has been investigated in two mouse lymphoma cell lines: line LY-as is radiation sensitive,
192 o an Epstein-Barr virus-negative Burkitt's B-lymphoma cell line, Louckes, and compared to virus induc
193 proliferation results were observed in two B lymphoma cell lines (LP-1 and U266) but interestingly no
194  the NF kappa B status for two murine B-cell lymphoma cell lines, LY-as (apoptosis-sensitive) and LY-
195 the expression of Bcl-2 in two murine B-cell lymphoma cell lines: LY-as, an apoptosis-proficient line
196  and Fas activation on three CD30+ cutaneous lymphoma cell lines (Mac-1, Mac-2 A, JK) derived from no
197 r transfectants generated in a murine B cell lymphoma cell line migrated in transwell chemotaxis assa
198 rmation of cytosolic Myddosome aggregates in lymphoma cell lines, mimicking the effect of dimerized T
199   We discovered a novel oncogene in a T-cell lymphoma cell line, multiple copies in T-cell lymphoma-1
200           Here we used 2 Myc-induced mouse B lymphoma cell lines, Myc5-M5 and Myc5-M12, which spontan
201 (P-gp) pump in a multidrug-resistant (MDR) B-lymphoma cell line, Namalwa/MDR1, and that this effect i
202                                   L3055, a B lymphoma cell line of germinal center (GC) origin, is de
203 y expressed the highest TIMP1 level among 29 lymphoma cell lines of various subtypes.
204 thylated, DAP-Kinase nonexpressing Burkitt's lymphoma cell line, only did so when treated with 5-aza-
205 erfering RNA treatment of the Jurkat human T lymphoma cell line or human peripheral blood T cells dis
206 The results were confirmed using a Burkitt's lymphoma cell line overexpressing transfected bcl-2.
207 bited growth of a MYC-dependent human B cell lymphoma cell line (P493) by blocking DNA replication, l
208                                      Burkitt lymphoma cell lines phenocopy the primary tumors with re
209 ith ibrutinib induced synergistic killing of lymphoma cell lines, primary human lymphoma specimens ex
210 ed cell survival and lymphoma progression in lymphoma cell lines, primary MCL and other B cell lympho
211 -transformed lymphoblasts, and many standard lymphoma cell lines produce high levels of granzyme B in
212 BC-1, a KSHV-DNA-positive, body cavity-based lymphoma cell line, produces infective herpesviral parti
213                        In a cutaneous T-cell lymphoma cell line, promoter hypermethylation was shown
214   Reduction of SLIP-GC levels in the Burkitt lymphoma cell line Raji and in non-Hodgkin lymphoma cell
215      TIMP-2 promoter hypermethylation in the lymphoma cell line Raji and the leukemia cell line KG1a
216                Here we show that the Burkitt lymphoma cell line Raji represents an experimental syste
217 phorylation of Akt (serine 473) in Burkitt's lymphoma cell line Ramos and in gastric carcinoma cell l
218 gions for isotype switching, an IgM+ Burkitt lymphoma cell line (Ramos 2G6) was assayed for the up-re
219  rituximab against a variety of human B-cell lymphoma cell lines (Ramos, DHL-4, and FL-18) in vivo.
220                       The human large B-cell lymphoma cell line RC-K8 has a rearranged REL locus that
221 ed at a chromosomal breakpoint in the B-cell lymphoma cell line, RC-K8; Drosophila ME31B, encoded by
222 t lymphoma cell line Raji and in non-Hodgkin lymphoma cell lines resulted in an increase in DNA break
223 on of CARD11 coiled-coil domain mutants into lymphoma cell lines resulted in constitutive NF-kappaB a
224 ic expression of TSC-22 in mouse leukemia or lymphoma cell lines resulted in delayed in vivo tumor fo
225 d following anti-Ig stimulation of a human B-lymphoma cell line, resulting in activation of phosphati
226 MP-1 expression in an EBV-negative Burkitt's lymphoma cell line results in a biphasic, in vivo tumor
227 s when challenged with the established mouse lymphoma cell line RMA-S-RAE-1beta, which overexpresses
228 is of the gammaHV68 latently infected B-cell lymphoma cell line S11E, but were also detected during l
229 glucocorticoid-induced apoptosis, the murine lymphoma cell lines S49.A2 and WEHI7.2.
230                         Remarkably, a murine lymphoma cell line, several human B cell cancer lines, a
231 cells, on various lymphoblastoid and Burkitt lymphoma cell lines, some of them being inducible or not
232 toxin uptake and toxin-mediated killing of B lymphoma cell lines, suggesting an alternative approach
233 udies of the t(2;5) translocation-containing lymphoma cell line SUP-M2 showed NPM-ALK to be localized
234 xpression of this tRNA-derived microRNA in a lymphoma cell line suppresses proliferation and modulate
235 SUPT-11 T-cell leukemia and P3HR-1 Burkitt's lymphoma cell lines, TCL1 interacts with endogenous Akt1
236 s significantly reduced off-rates in 3 human lymphoma cell lines tested, as well as increased complem
237 o PEL in vitro and in vivo, but not to other lymphoma cell lines tested.
238                   In RL and CA46, two B-cell lymphoma cell lines, TGF-beta1 treatment caused down-reg
239 itors are far more cytotoxic for myeloma and lymphoma cell lines than for hepatocarcinoma or non-acti
240 identified as being overexpressed in a mouse lymphoma cell line that had gained resistance to cell de
241                  In BC-1, a primary effusion lymphoma cell line that is dually infected with EBV and
242                                     A T-cell lymphoma cell line that is SHP-1 deficient (Karpas 299)
243                          In pleural effusion lymphoma cell lines that express variable levels of KSHV
244 ct processes, we isolated stably transfected lymphoma cell lines that expressed either murine wild-ty
245 nt a mechanism of resistance to apoptosis in lymphoma cell lines that might be relevant for the devel
246 ngle agent efficacy against human follicular lymphoma cell lines that overexpress Bcl-2, and efficacy
247 4 hours varied widely in the seven different lymphoma cell lines that were tested, ranging from 35 to
248        We further show, in the BL2 Burkitt's lymphoma cell line, that KLHL6 interacts with Cullin3, b
249 ransformed B lymphoblastoid and many Burkitt lymphoma cell lines, the EBV EBNA-1 protein is one of si
250                   As in all primary effusion lymphoma cell lines, there is a low level of spontaneous
251 sublines were isolated by exposing a Burkitt lymphoma cell line to increasing concentrations of vincr
252                              Using a Burkitt lymphoma cell line to model human B-cell apoptosis in vi
253            Exposure of splenic marginal zone lymphoma cell lines to a demethylating agent caused part
254  Bcl-2 and susceptibility of human Burkitt's lymphoma cell lines to H2O2-induced killing.
255 2 and BCR ligation triggered several Burkitt lymphoma cell lines to undergo apoptosis, and the 2 stim
256 rther, transient Flag-LMP1 expression in a B-lymphoma cell line transduces signals that upregulate TR
257 osensitivity assays, using the L5178 mouse T lymphoma cell line transfected with the human MDR1 gene.
258              In Jurkat cells, a human T-cell lymphoma cell line, tumor necrosis factor-alpha (TNF-alp
259 The assay was evaluated with the histiocytic lymphoma cell line U937 and the topoisomerase inhibitors
260 O was used to screen the proteome of a human lymphoma cell line (U937) sensitive to CDDO-induced apop
261 d with full-length BAFF, or by a histiocytic lymphoma cell line (U937) that expresses BAFF endogenous
262 er pretargeted RIT varied depending upon the lymphoma cell line used, with 1F5 Ab-SA and Lym-1 Ab-SA
263 rom initially sensitive OCI-Ly1 and SU-DHL-4 lymphoma cell lines via long-term exposure.
264 n siRNA-mediated IRF8 knockdown mouse B cell lymphoma cell line, we showed that IRF8 represses Bcor a
265 liferation of a number of human leukemia and lymphoma cell lines, we examined the role of p38 mitogen
266   In the current study with four mantle cell lymphoma cell lines, we report a new major metabolic pat
267  leads to apoptosis of EBV-infected LCLs and lymphoma cell lines, we sought to determine whether LMP1
268 phate receptor (IP(3)R) levels in two murine lymphoma cell lines, WEHI7.2 and S49.A2.
269  Subclones of the NK-92 human natural killer lymphoma cell line were derived by treatment with 5-aza-
270              Growth and survival of multiple lymphoma cell lines were studied with either drug alone
271 ell) or epithelial origin and five Burkitt's lymphoma cell lines were studied.
272                        Ramos cells, a B-cell lymphoma cell line, were used as target cells for the ge
273  DNA from the KS-1 cells (a primary effusion lymphoma cell line which is estimated to have 16 copies
274 G methylation of the EBV genome in Burkitt's lymphoma cell lines which maintain type I latency, and l
275                             A mutant YAC-1 T lymphoma cell line, which was selected for resistance to
276 in expression is also increased in Burkitt's lymphoma cell lines, which are known to have increased c
277                          A panel of murine B lymphoma cell lines, which express different levels of F
278 eath (apoptosis) response in three different lymphoma cell lines, which plays a key role in the devel
279 , of the ICOS gene, whereas cutaneous T-cell lymphoma cell lines, which strongly express ICOS, show n
280 oligonucleotides had no effect on two B-cell lymphoma cell lines, which were not infected with KSHV.
281 n t(8;14;12)(q24.1;q32.3;q24.1) in a Burkitt lymphoma cell line (Wien 133) was performed; all four tr
282                  E710.2.3 is a murine thymic lymphoma cell line with an immature phenotype (CD4-CD8-)
283 An Epstein-Barr virus (EBV)-negative Burkitt lymphoma cell line with ectopic TIMP-1 expression (TIMP-
284                                   In Burkitt lymphoma cell lines with chromosome 1q21 abnormalities,
285                        In the current study, lymphoma cell lines with constitutively active NF-kappaB
286  reports the effects of bortezomib in B-cell lymphoma cell lines with differing sensitivity to bortez
287                          We labeled 2 murine lymphoma cell lines with dual function reporter genes an
288 h of human prostate cancer, lung cancer, and lymphoma cell lines with EC(50) values of 0.13, 0.56, an
289 th of human lung cancer and BP3 human B-cell lymphoma cell lines with EC(50) values of 0.33 and 0.66
290 ependent reduction in cell viability in five lymphoma cell lines, with EC(50) values ranging from 6 n
291 ntitumor efficacy against a syngeneic B cell lymphoma cell line within a background of normal CD19(+)

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