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3 crosis and vascular damage from EBV-positive lymphomatoid granulomatosis and nasal or nasal-type T/na
4 mally elevated in patients with EBV-positive lymphomatoid granulomatosis and nasal or nasal-type T/NK
6 , and he had a biopsy-confirmed diagnosis of lymphomatoid granulomatosis that evolved into fatal B-ce
7 y-one patients with pathologically confirmed lymphomatoid granulomatosis were enrolled in a natural h
8 monary T- or NK-cell angiocentric lymphomas (lymphomatoid granulomatosis), 12/19 T-cell anaplastic la
10 ma closely mimics the full spectrum of human lymphomatoid granulomatosis, an EBV-associated malignanc
12 t this translocation at the genomic level in lymphomatoid papulosis (14 cases), primary cutaneous CD3
14 ression of skin lesions is characteristic of lymphomatoid papulosis (LyP), a clonal cutaneous lymphop
15 phoproliferative disorders (LPDs), including lymphomatoid papulosis (LyP), anaplastic and nonanaplast
16 was undertaken to determine the clonality of lymphomatoid papulosis (LyP), its clonal relationship to
17 t for the CD30+ lymphoproliferative disorder lymphomatoid papulosis (LyP), which currently has no app
19 0 ligand expression in regressing lesions of lymphomatoid papulosis and cutaneous CD30+ anaplastic la
20 he large-cell lymphoma cases, and six of the lymphomatoid papulosis cases were then subjected to dot
23 mors of two patients who had progressed from lymphomatoid papulosis to systemic anaplastic large cell
25 btained from 16 patients with MF, seven with lymphomatoid papulosis, seven with primary cutaneous CD3
27 l tolerated in cutaneous T-cell lymphoma and lymphomatoid papulosis, with an overall response rate of
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