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1 DNA from the cell lines and tissues from the lymphomatous animal demonstrated the presence of a 536-b
2 h node and the other from the blood of the 2 lymphomatous animals.
3                       A 94-year-old man with lymphomatous ascites.
4 etional mapping of chromosome 6q in 22 acute/lymphomatous ATL samples using 24 highly informative mic
5 ery important role in the evolution of acute/lymphomatous ATL.
6                           Three patients had lymphomatous bone marrow infiltrates, and four had perip
7                                 Furthermore, lymphomatous bone marrow involvement and low CD4 count w
8 imens revealed a thickened gastric wall with lymphomatous cell infiltration.
9 oninvasive way of differentiating immune and lymphomatous cell populations.
10            Phenotypic analysis revealed that lymphomatous cells were cluster of differentiation (CD)3
11 AIDS-related lymphoma usually have extensive lymphomatous disease, with relatively frequent involveme
12 iled follow-ups, 3 had a prior or concurrent lymphomatous disease; 8 had stage I disease (limited to
13 adicate both untreated and Rituxan-resistant lymphomatous EBV-LPD, with failures ascribable to impair
14 isk factor (13/13), presented initially as a lymphomatous effusion (14/15), remained localized to the
15        This cell line was established from a lymphomatous effusion obtained from an HIV- patient, and
16 between KSHV-positive PELs and KSHV-negative lymphomatous effusions and also between KSHV-positive, E
17            In contrast, all 15 KSHV-positive lymphomatous effusions exhibited a distinctive morpholog
18                       All four KSHV-negative lymphomatous effusions exhibited Burkitt or Burkitt-like
19 rpesvirus-8) infection, present as malignant lymphomatous effusions in body cavities.
20 ficiency virus (HIV)-positive individuals as lymphomatous effusions in the serous cavities without a
21 his hypothesis by investigating 19 malignant lymphomatous effusions occurring in the absence of a con
22 gly suggest that the KSHV-positive malignant lymphomatous effusions represent a distinct clinicopatho
23                                              Lymphomatous effusions that lack KSHV also occur; these
24 mas that grow mainly in the body cavities as lymphomatous effusions without an identifiable contiguou
25 c and molecular characteristics, and grow as lymphomatous effusions without an identifiable contiguou
26 ene expression and the pathogenesis of these lymphomatous effusions.
27 ty-based lymphomas (BCBLs), which present as lymphomatous effusions.
28 and extends overall survival of mice bearing lymphomatous effusions.
29                                   Those with lymphomatous features were excluded.
30 or age > or = 10 years, excluding those with lymphomatous features) who achieved an RER (< or = 25% m
31 failure (ARF) is an unusual manifestation of lymphomatous infiltration of the kidneys.
32 eless, because it often responds to therapy, lymphomatous infiltration should be suspected in any pat
33 ining 15 cases showed histologic evidence of lymphomatous infiltration.
34 homa (PCNSL) is caused mostly by intraocular lymphomatous involvement (vitritis and retinal infiltrat
35  those used in clinical trials, such as <25% lymphomatous involvement of the bone marrow.
36 enia lower than 100 000 mm(3), high LDH, and lymphomatous involvement of the CSF.
37                       Five of 7 patients had lymphomatous involvement of the spleen.
38 ukemia that can present with leukemic and/or lymphomatous manifestations.
39 provement in skin lesions, and regression of lymphomatous masses and splenomegaly.
40 tion of cerebrospinal fluid samples detected lymphomatous meningitis in 2 patients.
41                                      Because lymphomatous meningitis is a frequent complication of no
42 athecal rituximab in patients with recurrent lymphomatous meningitis will be implemented based on the
43 luded acute (n = 7), smoldering (n = 2), and lymphomatous (n = 1).
44  by slowly progressive visual deterioration, lymphomatous ONI is rare and characterized by rapidly pr
45 -like RNA expression was demonstrated in the lymphomatous organs and in 2 transformed T-cell lines, o
46 evealed no other sites of involvement by the lymphomatous process.
47 leted form may be preferentially selected in lymphomatous processes.
48 istology and immunophenotype and studied the lymphomatous tissue from the parotid, cervical lymph nod
49 -depth analysis due to its extreme rarity in lymphomatous tissue.
50 eas aneuploidy was frequently evident in pre-lymphomatous tissues, end-stage E micro -K cyclin/p53(-/
51 onses, autoantibody formation, and potential lymphomatous transformation.

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