ライフサイエンスコーパス: lymphoreticular

1 tissue to be tested anonymously for abnormal lymphoreticular accumulation of prion protein (PrP) was

2 tection of protease-resistant PrP(Sc) within lymphoreticular and central nervous system tissues.

3 Brucellae display strong tissue tropism for lymphoreticular and reproductive systems with an intrace

4 issues before neuroinvasion, suggesting that lymphoreticular biopsy samples may allow early diagnosis

5 is immune privileged due to lack of resident lymphoreticular cells or due to antigenic sequestration

6 dritic cells, BP3(+) stromal cells, ERTR7(+) lymphoreticular cells, and peripheral node addressin(+)

7 ic cells, BP3(+) stromal cells, and ERTR7(+) lymphoreticular cells.

8 essive fibrosis and infiltration by abnormal lymphoreticular cells.

9 n incubation time, brain neuropathology, and lymphoreticular involvement.

10 the pathway of initiation and progression of lymphoreticular malignancies in the absence of ATM.

11 Lymphoreticular malignancies, collectively called posttr

12 unodeficiency with increased risk to develop lymphoreticular malignancies.

13 rtunistic organisms, autoimmune disease, and lymphoreticular malignancies.

14 om patients with an uncommon complication of lymphoreticular malignancy, chronic disseminated candidi

15 Sc, also accumulates in the spleen and other lymphoreticular organs.

16 While prion infection of the lymphoreticular system (LRS) is necessary for neuroinvas

17 al tissues involves agent replication in the lymphoreticular system (LRS) prior to entry into the ner

18 e central nervous system (CNS) and often the lymphoreticular system (LRS).

19 replication is thought to occur first in the lymphoreticular system and then spread into the brain.

20 f PrP(Sc) from the spleen suggested that the lymphoreticular system does not play a role in neuroinva

21 Q and ARQ/VRQ) and low (ARR/VRQ and AHQ/VRQ) lymphoreticular system involvement in scrapie pathogenes

22 The results show that involvement of the lymphoreticular system is a defining feature of variant

23 s molecules that accumulate in the brain and lymphoreticular system of the host consist of three diff

24 rP(d) accumulated in elements of the cranial lymphoreticular system prior to neuroinvasion.

25 trafficking, and retention of prions in the lymphoreticular system, hallmark sites of early prion pr

26 fficking by B and dendritic cells within the lymphoreticular system, intranodal prion replication by

27 o malignancies are those of the skin and the lymphoreticular system.

28 lly striatum) and the human exon h1D mRNA in lymphoreticular system.

29 lar strategy for invading and colonizing the lymphoreticular system.

30 certain other cells, including those of the lymphoreticular system.

31 arily restricted to cells of the nervous and lymphoreticular systems.

32 whether targeting nasopharyngeal-associated lymphoreticular tissue (NALT) DCs by a different deliver

33 By contrast, nasopharyngeal-associated lymphoreticular tissue function remains intact during ag

34 lignant lymphocyte migration into and within lymphoreticular tissue is an important aspect of chronic

35 glands (SMGs), and nasopharyngeal-associated lymphoreticular tissue of mice given nCT.

36 nostic tests and prevalence studies based on lymphoreticular tissue or blood.

37 nses were noted in nasopharyngeal-associated lymphoreticular tissue, cervical lymph nodes, and spleen

38 To test whether BHV-1 persisted in lymphoreticular tissue, we analyzed tonsils of latently

39 ry of vaccines for nasopharyngeal-associated lymphoreticular tissue-based mucosal immunity offers an

40 eractions, as is the case for gut-associated lymphoreticular tissue.

41 sease-associated prion protein in peripheral lymphoreticular tissue.

42 Lymphoreticular tissues (68 tonsils, 64 spleens, and 40

43 of immune cells in organized gut-associated lymphoreticular tissues (GALT) and diffuse lamina propri

44 Prions replicate in lymphoreticular tissues before neuroinvasion, suggesting

45 her molecule was present in nasal-associated lymphoreticular tissues beyond 24 h.

46 We studied lymphoreticular tissues from a necropsy series and asses

47 ant prion protein (PrP(res)) accumulation in lymphoreticular tissues indicates prion infection.

48 All lymphoreticular tissues obtained at necropsy from patien

49 ter numbers in the nasopharyngeal-associated lymphoreticular tissues of 1-year-old mice than of young

50 and tonsils of the nasopharyngeal-associated lymphoreticular tissues represent a distinct component o

51 and there was limited PrP immunostaining in lymphoreticular tissues such as spleen and appendix.

52 distinct component of the mucosal-associated lymphoreticular tissues with features of both systemic a

53 lium-processes important for the invasion of lymphoreticular tissues, a major determinant of disease

54 ne alterations occur first in gut-associated lymphoreticular tissues, and thus nasal delivery of vacc

55 A reduction in gut-associated lymphoreticular tissues, intestinal antigen-specific IgA

56 ould target, in addition to nasal-associated lymphoreticular tissues, the olfactory nerves/epithelium

57 ed high-level accumulation of PrP(Sc) within lymphoreticular tissues.

58 n protein (PrP(Sc)) is readily detectable in lymphoreticular tissues.

59 in and abundant blood vessels in infiltrated lymphoreticular tissues.

60 ignant mesotheliomas, gliomas, sarcomas, and lymphoreticular tumors were also stained.