ライフサイエンスコーパス: lymphotoxin beta receptor

1 ands, including those that bind CD30 and the lymphotoxin beta receptor.

2 that CD30 signals predated those through the lymphotoxin beta receptor.

3 IGHT receptors herpesvirus entry mediator or lymphotoxin beta receptor.

4 75 neurotrophin receptor and weakly with the lymphotoxin-beta receptor.

5 to the cytosolic domains of CD30, CD40, and lymphotoxin-beta receptors.

6 Induction of PNAd in mutant PPs requires lymphotoxin beta receptor activity, and its upregulation

7 f lymphotoxin was confirmed, as the use of a lymphotoxin beta receptor agonist results in partial res

8 both of its identified signaling receptors, lymphotoxin beta receptor and herpes virus entry mediato

9 rmation was dependent on lymphotoxin and the lymphotoxin beta receptor and independent of lymphocytes

10 LIGHT protected mice from colitis via the lymphotoxin beta receptor and was expressed mainly by my

11 SF-14) is a ligand of stromal cell-expressed lymphotoxin-beta receptor and T cell-expressed herpes vi

12 of six single chain-Fv fragments of the anti-lymphotoxin-beta receptor antibody, statistically signif

13 ts receptors, herpesvirus entry mediator and lymphotoxin beta receptor, are found in T cells and stro

14 Conversely, stimulation of lymphotoxin-beta receptor by agonistic antibody leads to

15 Signaling through the lymphotoxin-beta receptor controlled the fate of adipocy

16 ion of LT and LIGHT signaling with a soluble lymphotoxin beta receptor decoy protein attenuated the d

17 tinal inflammation when transferred into the lymphotoxin beta receptor-deficient mice, and herpes vir

18 on in the thymi of lymphotoxin-deficient and lymphotoxin-beta receptor-deficient mice contributes to

19 de development during embryogenesis involves lymphotoxin-beta receptor engagement and subsequent diff

20 LIGHT, a ligand for the lymphotoxin-beta receptor, establishes lymphoid-like tis

21 activated, LTalphabeta(+), lymphocytes with lymphotoxin beta-receptor-expressing fibrosarcoma tumor

22 al-associated lymphoid tissue by LTbetaR-Ig (lymphotoxin-beta receptor human Ig fusion protein) treat

23 tion, BALB/c mice were treated in utero with lymphotoxin beta-receptor Ig fusion protein to generate

24 belonging to the TNF superfamily, by soluble lymphotoxin beta receptor-Ig (LTbetaR-Ig) inhibits the c

25 Blockade of LIGHT by its soluble receptors, lymphotoxin beta receptor-Ig or HVEM-Ig, inhibits the in

26 In lymphotoxin-beta receptor-Ig-treated mice, which lack LN

27 Coexpression of a soluble lymphotoxin beta-receptor:Ig fusion protein, an inhibito

28 nobese diabetic (NOD) mice were treated with lymphotoxin-beta receptor immunoglobulin fusion protein

29 d state, blockade of this stimulation with a lymphotoxin beta receptor-immunoglobulin fusion protein

30 ions including BO, and that treatment with a lymphotoxin-beta receptor-immunoglobulin fusion protein

31 LIGHT signals through the lymphotoxin beta receptor in the colon to regulate the i

32 CICD was strongly activated by both TNF and lymphotoxin-beta receptor ligation in IKKbeta-/- MEFs an

33 eatment with interferon-gamma, which induced lymphotoxin beta receptor (LT beta R) expression, was re

34 implex virus entry mediator (HVEM) and LIGHT/lymphotoxin beta receptor (LT beta R) interactions decre

35 The lymphotoxin beta receptor (LT beta R) was originally des

36 is study demonstrates enhanced expression of lymphotoxin beta receptor (Lt betaR) during the demyelin

37 Recent studies revealed that the lymphotoxin/lymphotoxin beta receptor (LT)/LTbetaR system activates

38 We found that a soluble decoy lymphotoxin beta receptor (LT-betaR)-Fc, which can block

39 4 interacts with the cytosolic domain of the lymphotoxin-beta receptor (LT beta R) and weakly with th

40 Here we report that blocking LT alpha beta/lymphotoxin-beta receptor (LT beta R) interaction in viv

41 Lymphotoxin-beta receptor (LT beta R) is a member of tum

42 hese clones encode the cytoplasmic domain of lymphotoxin-beta receptor (LT betaR), which is a member

43 osis factor-alpha (TNF-alpha), whereas decoy lymphotoxin-beta receptor (LT-betaR) fusion protein had

44 nsduction of the TNF receptor 2 (TNFR2), the lymphotoxin-beta receptor (LT-betaR), CD40, CD30, and LM

45 ult mice was reduced by systemic blockade of lymphotoxin-beta receptor (LTbeta R) signaling with a so

46 re NIK for activation of NF-kappaB, only the lymphotoxin-beta receptor (LTbeta-R) is expressed in mel

47 Our previous studies indicated that lymphotoxin beta receptor (LTbetaR) activation controls

48 ediated by at least two receptors, including lymphotoxin beta receptor (LTbetaR) and herpes simplex v

49 cells including HT29 cells that express both lymphotoxin beta receptor (LTbetaR) and HVEM/TR2 recepto

50 tosis of various tumor cells expressing both lymphotoxin beta receptor (LTbetaR) and TR2/HVEM recepto

51 or receptors (TNFRs) as a homotrimer and via lymphotoxin beta receptor (LTbetaR) as a heterotrimeric

52 Lymphotoxin beta receptor (LTbetaR) blockade reduces hom

53 eady-state and after bone marrow transplant, lymphotoxin beta receptor (LTbetaR) controls entry of T

54 Lymphotoxin beta receptor (LTbetaR) deficiency is associ

55 onditional gene-deficient mice, we find that lymphotoxin beta receptor (LTbetaR) directly controls th

56 DC-derived lymphotoxin beta receptor (LTbetaR) ligands were critica

57 Lymphotoxin beta receptor (LTbetaR) ligation-induced Air

58 (LT)alpha(1)beta(2) on inducer cells and the lymphotoxin beta receptor (LTbetaR) on stromal cells ini

59 fusion protein between the IgG Fc domain and lymphotoxin beta receptor (LTbetaR) reduces lung fibrosi

60 bind the cytoplasmic domains of CD40 and the lymphotoxin beta receptor (LTbetaR), both of which are k

61 inflammation mediated by LIGHT, a ligand for lymphotoxin beta receptor (LTbetaR), not only stimulates

62 fic deletion of the thymus medulla regulator lymphotoxin beta receptor (LTbetaR), we show that thymic

63 a substantial reduction in the expression of lymphotoxin beta receptor (LTbetaR)-controlled migration

64 Lymphotoxin beta receptor (LTbetaR)-driven induction of

65 Administration of lymphotoxin beta receptor (LTbetaR)-Ig to wild-type mice

66 Lymphotoxin beta receptor (LTbetaR)-induced activation o

67 these genes in response to engagement of the lymphotoxin beta receptor (LTbetaR).

68 to that of homozygosity for deletion of the lymphotoxin beta receptor (LTbetaR).

69 The lymphotoxin-beta receptor (LTbetaR) activates the NF-kap

70 Lymphotoxin-beta receptor (LTbetaR) and CD40 are members

71 K) and TNF receptor family members including lymphotoxin-beta receptor (LTbetaR) and CD40.

72 ammatory mechanism, including membrane-bound lymphotoxin-beta receptor (LTbetaR) and CXC chemokine re

73 LIGHT engages the lymphotoxin-beta receptor (LTbetaR) and HVEM (TNFRSF14),

74 r, which binds two known cellular receptors, lymphotoxin-beta receptor (LTbetaR) and the herpesvirus

75 this effect through the interaction with the lymphotoxin-beta receptor (LTbetaR) but not herpes virus

76 Ligation of the lymphotoxin-beta receptor (LTbetaR) by LIGHT (lymphotoxi

77 Moreover, we show that signals through the lymphotoxin-beta receptor (LTbetaR) in DC are also requi

78 R4-activated canonical NF-kappaB pathway and lymphotoxin-beta receptor (LTbetaR) induced non-canonica

79 The lymphotoxin-beta receptor (LTbetaR) plays critical roles

80 Ligation of the lymphotoxin-beta receptor (LTbetaR) recruits tumor necro

81 romal cell microenvironments is dependent on lymphotoxin-beta receptor (LTbetaR) signaling.

82 Lymphotoxin-beta receptor (LTbetaR), a member of the tum

83 equires innate lymphoid cells, which promote lymphotoxin-beta receptor (LTbetaR)-dependent maintenanc

84 ors, herpes virus entry mediator (HVEM), and lymphotoxin-beta receptor (LTbetaR).

85 high), is dependent on signaling through the lymphotoxin-beta receptor (LTbetaR).

86 ated by intragraft TLOs and whether blocking lymphotoxin-beta-receptor (LTbetaR) signaling, a pathway

87 nt region of IgG and extracellular domain of lymphotoxin beta receptor (LTbetaRIg) interrupted clonal

88 Components of lymphotoxin beta receptor (LTBR)-associated signaling co

89 as well as pharmacological inhibition of the lymphotoxin-beta receptor markedly delays tumor developm

90 xin axis, and pharmacological stimulation of lymphotoxin beta receptor might represent a novel therap

91 s, herpesvirus entry mediator on T cells and lymphotoxin beta receptor on stromal cells, are implicat

92 Genetic deletion of lymphotoxin beta receptor or lymphotoxin alpha abrogated

93 lary epithelial cells of mice lacking either lymphotoxin beta receptor or the lymphotoxin alpha-chain

94 ory distress, and shows that blockade of the lymphotoxin beta receptor pathway reverses the disease.

95 analyzed death after ligation of the TNF and lymphotoxin-beta receptors, respectively.

96 mphangiogenesis in the thyroid and implicate lymphotoxin beta receptor signaling in this process.

97 Notch-dependent Esam(hi) DC subset required lymphotoxin beta receptor signaling, proliferated in sit

98 is factor superfamily member LIGHT activates lymphotoxin beta-receptor signaling, leading to the prod

99 Meanwhile, vascular smooth muscle cell lymphotoxin beta receptors (VSMC-LTbetaRs) protected aga

100 found that an agonistic antibody against the lymphotoxin beta receptor was able to facilitate liver r

101 estingly, both the tumor necrosis factor and lymphotoxin beta receptors were down-regulated by NS5A.

102 e protein binds to the cytoplasmic domain of lymphotoxin beta receptor, which is a member of tumor ne

103 nd on activated lymphocytes and binds to the lymphotoxin-beta receptor, which is generally present on