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1 h the EGFR and a G protein-coupled receptor (lysophosphatidic acid receptor).
2 lity of crosstalk between G2A and endogenous lysophosphatidic acid receptors.
3 cells, at least in part, by associating with lysophosphatidic acid receptor 1 (LPAR1), leading to enh
4 ection of unidirectional flow; inhibition of lysophosphatidic acid receptor 1 restored the flow-depen
8 ctivation is impaired downstream of both the lysophosphatidic acid receptor, a G-protein-coupled rece
9 nduced activation of pro-apoptotic pathways (lysophosphatidic acid receptor and caspase-domain signal
11 riptionally down-regulates expression of the lysophosphatidic acid receptor EDG2 and this down-regula
12 ution, and genetics of lp(A2)/Edg4, a second lysophosphatidic acid receptor gene, we characterized it
13 Berridge [11] showed that activation of the lysophosphatidic acid receptor in a restricted region of
14 ibited the endogenous thrombin receptors and lysophosphatidic acid receptors in NIH3T3 cells, whereas
15 both GRK2 and G beta1 gamma2, activation of lysophosphatidic acid receptors leads to the rapid and t
16 The molecular cloning of three high-affinity lysophosphatidic acid receptors, LPA1, LPA2, and LPA3, p
17 tion of the epidermal growth factor (EGF) or lysophosphatidic acid receptors or by pervanadate-induce
18 n 6 modulates NADPH oxidase 2 activation via lysophosphatidic acid receptor signaling in the pulmonar
20 ere on the characterization of a third human lysophosphatidic acid receptor subtype, Edg-7, which med
21 her, our data indicate that Edg-7 is a third lysophosphatidic acid receptor that couples predominantl
22 case of the beta(2)-adrenergic receptor and lysophosphatidic acid receptor, these processes can lead
24 e retraction evoked by Galpha12/13-dependent lysophosphatidic acid receptors was augmented in R7BP-ex
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