ライフサイエンスコーパス: m-CPP

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1 m-CPP also decreased activity in primary sensory neurons

2 m-CPP alters synaptic transmission and neuronal function

3 m-CPP elicited subtype-related differential effects amon

4 In Experiment 2, m-CPP induced a suppression of food intake in nonlesione

5 The abnormal m-CPP-induced activation-euphoria responses represent a

6 However, acute applications of p-CPA and m-CPP, followed by extensive saline washing, did not rev

7 The blunted m-CPP-induced responsiveness of the hypothalamic-pituita

8 he anterior cingulate, normally activated by m-CPP, was deactivated in patients; in contrast, the pos

9 intact rats, Fos expression was increased by m-CPP (1 mg/kg, i.p.) in the striatum and the subthalami

10 The compound m-chlorophenylpiperazine (m-CPP) is used clinically to manipulate serotonergic fun

11 agonist/antagonist m-chlorophenylpiperazine (m-CPP) was administered to male alcoholic patients who w

12 vioral response to m-chlorophenylpiperazine (m-CPP), a serotonin-specific agent.

13 c activity, and meta-chlorophenylpiperazine (m-CPP) as a probe of serotonergic activity.

14 otonergic agent meta-chlorophenylpiperazine (m-CPP) produced increases in activation and euphoria in

15 ergic stimulus, meta-chlorophenylpiperazine (m-CPP), was examined in 13 subjects with impulsive aggre

16 In the rat spinal cord, m-CPP enhances the occurrence of spontaneous excitatory

17 (3-Chlorophenyl) piperazine dihydrochloride (m-CPP), for 1 week resulted in a decreased responsivenes

18 Eight patients (31%) with PTSD experienced m-CPP-induced panic attacks and had significantly greate

19 s tended to occur in different patients from m-CPP-induced panic attacks.

20 Instead, m-CPP likely decreased sodium influx through voltage-gat

21 sion of yohimbine hydrochloride (0.4 mg/kg), m-CPP (1.0 mg/kg), or saline solution on 3 separate test

22 After nigrostriatal lesions, m-CPP-induced Fos expression remained unchanged in the s

23 l but not intrasubthalamic administration of m-CPP.

24 investigated the food-suppressive effects of m-CPP (0.5, 1.0 or 2.0 mg/kg).

25 res were recorded), followed by infusions of m-CPP (0.08 mg/kg) or placebo the next morning.

26 ngs, both peripheral and local injections of m-CPP into the subthalamic nucleus elicited oral dyskine

27 B 206553 (a 5HT2B/2C receptor antagonist) or m-CPP (a 5HT2C/1B receptor agonist) in a standard behavi

28 PP) (10 nmol), 1-(3-chlorophenyl)piperazine (m-CPP) (7.4 nmol), gepirone (70 nmol) and 2-dipropylamin

29 HT(2C) agonist 1-(m-chlorophenyl)piperazine (m-CPP) were examined.

30 In this study, we demonstrated that m-CPP did not appear to act through a 5-HT receptor in d

31 Intracellular axonal recordings showed that m-CPP reduced the amplitude of the action potentials in

32 measures were obtained before and after the m-CPP infusion.

33 ater increase in plasma ACTH response to the m-CPP infusion than the alcoholics regardless of subtype

34 re hypothesized to respond differentially to m-CPP in patients and controls.

35 ious and disorganized behavioral response to m-CPP but a normal hormonal response.

36 rtex, regional metabolic glucose response to m-CPP was entered into a group (impulsive aggressive, co

37 t anteromedial orbital cortex in response to m-CPP.

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