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1  reaction specificity in HDO of furfural and m-cresol.
2 ne at the meta position, producing primarily m-cresol.
3  in comparison, produces 97% p-cresol and 3% m-cresol.
4 250 microM and produced 90% p-cresol and 10% m-cresol.
5 fferent with resorcinol than with phenol and m-cresol.
6 eniently prepared in five steps from 2-amino-m-cresol.
7 catalyzed the oxidation of catechol, 6-amino-m-cresol, 2-amino-m-cresol, and 2-amino-4-chlorophenol.
8                                     4-Chloro-m-cresol (4-CmC) is a clinically relevant activator of t
9                                     4-Chloro-m-cresol (4-CmC) is a potent and specific activator of t
10 ponse to high [K(+)], caffeine, and 4-chloro-m-cresol (4-CMC), the maximal tensions generated in Stac
11 sometric specific force followed by 4-chloro-m-cresol (4-CmC)-evoked maximal contracture force in sin
12 RyR3), are efficiently activated by 4-chloro-m-cresol (4-CmC).
13 onsistent with this interpretation, 4-chloro-m-cresol (4-CMC; 100 microm) increases the rate of Ca(2+
14 owed that the RYR-stimulating agent 4-chloro-m-cresol (4CmC) induced Ca(2+) release and thereby confi
15 ,4,6-Trichlorophenol (2) and 2,4,6-trichloro-m-cresol (5) react with calcium hypochlorite (Ca(OCl)(2)
16 +) stores, since the application of 4-chloro-m-cresol, a direct type 1 ryanodine receptor activator,
17 -loaded CD19(+) B and DAKIKI cells, 4-chloro-m-cresol, a potent activator of Ca2+ release mediated by
18 could be restored by application of 4-chloro-m-cresol, a ryanodine receptor agonist, indicating that
19 and ryanodine but not for Ca(2+) or 4-chloro-m-cresol, although they all induced Ca(2+) release.
20                                     4-Chloro-m-cresol, an activator of the skeletal muscle ryanodine
21  i.e., alcohol dehydration and alkylation of m-cresol and 2-propanol in the liquid phase, at high tem
22 ring precursors 60 and 68 were prepared from m-cresol and 3-ethylphenol, respectively.
23                                Both 4-chloro-m-cresol and halothane caused adenosine accumulation in
24 91%) and methylhydroquinone (9%), to oxidize m-cresol and p-cresol to 4-methylcatechol (100%), and to
25  cells was significantly altered by 4-chloro-m-cresol and ryanodine.
26 ation of catechol, 6-amino-m-cresol, 2-amino-m-cresol, and 2-amino-4-chlorophenol.
27 ffusion coefficients of benzene, anthracene, m-cresol, and p-nitrophenol in enhanced-fluidity liquid
28    The oxidative half-reaction of PHHY using m-cresol as a substrate is similarly affected by the mut
29 gands studied in resorcinol >> phenol > or = m-cresol as determined from their overall free energies
30           Phenolic ligands, e.g., phenol and m-cresol, bind to 2Zn(II)-insulin hexamers and induce a
31 1, they were much less sensitive to 4-chloro-m-cresol (CMC).
32 rization or RyR agonists (caffeine, 4-chloro-m-cresol) compared with wtRyR1.
33 imately 5-fold increase in the percentage of m-cresol formation relative to that of the natural isofo
34 e para-monooxygenase variant that formed 75% m-cresol from toluene and 100% m-nitrophenol from nitrob
35 was applied and validated for PCMC (4-chloro-m-cresol), household derived antimicrobial agent with no
36 roximately 0.0001), suggesting that 4-chloro-m-cresol-induced adenosine could readily distinguish bet
37 n hexamer containing two zinc ions, with two m-cresol molecules bound at each dimer-dimer interface s
38 on, near-quantitative deuterium retention in m-cresol obtained from 4-(2)H(1)-toluene, and partial lo
39  lymphocytes incubated with 0-10 mM 4-chloro-m-cresol or 0-10.7 mM halothane.
40                  Similarly, at 1 mM 4-chloro-m-cresol or 0.96 mM halothane, adenosine levels were sig
41 ,5-benzenetricarbaldehyde building blocks in m-cresol or acetic acid, named RT-COF-1 or RT-COF-1Ac/RT
42  after RyR activation (caffeine and 4-chloro-m-cresol) or beta-adrenergic stimulation (isoproterenol)
43 eceptor (10 mM caffeine, 200 microM 4-chloro-m-cresol, or 10 mM KCl).
44 rescein at pH 6.5, phenol red at pH 7.5, and m-cresol purple at pH 8.5) which permitted separation of
45 tic analyses showed areas under the 4-chloro-m-cresol receiver-operating characteristic curves near m
46                            Although 4-chloro-m-cresol receiver-operating characteristic curves reveal
47 ptible B cells treated with 0.75 mM 4-chloro-m-cresol relative to controls.
48 nodine receptor Ca channels agonist 4-chloro-m-cresol was compared in blood lymphocytes from malignan
49 -3-hexenol, acetic acid, benzyl alcohol, and m-cresol, while the addition of oxygen significantly inf
50  to probe the mechanism of the alkylation of m-cresol with isopropyl alcohol in scCO(2) using Nafion

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