ライフサイエンスコーパス: macrocomplex

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1 port abnormalities of proteins of the band 3 macrocomplex.

2 ges between SHP2 and the Ras.Raf.phospho-MEK macrocomplex.

3 a high dilution of sample and, thus, protein macrocomplexes.

4 4, and PKCalpha, promoting formation of NHE3 macrocomplexes.

5 of the architecture of the TCR-CD3-pMHC-CD4 macrocomplex and point to regions of high CD4-Lck + ITAM

6 ls confirming the integrated function of the macrocomplex and the importance of its role in red cell

7 l allows a complete stabilization of protein macrocomplexes and appears to be very accurate.

8 d insulin administration reconstituted these macrocomplexes and restored NHE3 expression in the BBM.

9 tool for both the protein identification in macrocomplexes and the understanding of the macrocomplex

10 t, the assembly of an eNOS-centered membrane macrocomplex, and membrane-initiated eNOS activation.

11 Since noncovalent protein macrocomplexes are implicated in many cellular functions

12 In this study, using the Hsp90/Aha1 macrocomplex as a model (where Hsp denotes a heat shock

13 h assembly of major components of the band 3 macrocomplex at an early stage during erythropoiesis.

14 with the H(+)-transporting lysosomal ATPase macrocomplex but uncovered no differences in the interac

15 Nonetheless, stabilizing these protein macrocomplexes, by introducing covalent bonds, is a prer

16 that prior to incorporation into the band 3 macrocomplex, CD47 associates with actin-binding protein

17 ults in formation of a Ras/Raf-1/phospho-MEK macrocomplex, extracellular signal-regulated protein kin

18 mbly of VG1Fs in the HA-versican-microfibril macrocomplex has not yet been elucidated.

19 The macrocomplex is involved in red cell gas exchange and re

20 ductive films of such CHIT-NAD+-GDH-GDI-CHIT macrocomplexes (MC) were prepared on glassy carbon (GC)

21 macrocomplexes and the understanding of the macrocomplexes organization.

22 arify the role of VG1Fs in the extracellular macrocomplex, specifically in mediating the recruitment

23 2 complex contacts many partners, creating a macrocomplex suitable for receptor targeting at the neur

24 ion dependent on inclusion within the band 3 macrocomplex, via protein 4.2.

25 of wildtype; other proteins of the band 3/Rh macrocomplex were also reduced.

26 ion, resulting in formation of the autophagy macrocomplex, which contains SLAMF1, beclin1, and the en

27 led that NHE3, NHERF1, IRBIT, and ezrin form macrocomplexes, which are perturbed under diabetic condi

28 odimerization mediates the formation of NHE3 macrocomplexes, which are required for the inhibition of

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