ライフサイエンスコーパス: macrocytic

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1 Tc1 mice do not develop leukemia, they have macrocytic anemia and increased extramedullary hematopoi

2 ications of vitamin B-12 deficiency, such as macrocytic anemia and neurological complications affecti

3 mechanism is underscored in 5q- syndrome, a macrocytic anemia caused by a large monoallelic deletion

4 Sf3b1(K700E) mice develop macrocytic anemia due to a terminal erythroid maturation

5 mitochondrial respiration and development of macrocytic anemia in mtDNA-mutator mice.

6 A 47-year-old woman with severe macrocytic anemia markedly improved during the second an

7 Macrocytic anemia occurring in patients with fatigue sug

8 Classical 5q- syndrome is an acquired macrocytic anemia of the elderly.

9 of the mtDNA polymerase, PolgA, and develop macrocytic anemia similar to that of patients with MDS.

10 In this group, the risk of macrocytic anemia was 63% lower than in the placebo grou

11 hat mutant animals develop an age-dependent, macrocytic anemia with abnormal erythroid maturation and

12 Macrocytic anemia with abnormal erythropoiesis is a comm

13 defect observed in both individuals and the macrocytic anemia with megaloblastic features of the mor

14 pc (del/+)) accelerated the development of a macrocytic anemia with monocytosis, early features of t-

15 that is deleted neonatally develop a severe macrocytic anemia with proerythroblast maturation arrest

16 se (the more severe case also presented with macrocytic anemia).

17 duced serum vitamin B12 concentrations, mild macrocytic anemia, and fecal loss of Na+ and K+, the lat

18 ad to many clinical abnormalities, including macrocytic anemia, cardiovascular diseases, birth defect

19 is characterized by red blood cell aplasia, macrocytic anemia, clinical heterogeneity, and increased

20 inal features of the 5q- syndrome, including macrocytic anemia, erythroid hypoplasia, and megakaryocy

21 , encoding the ribosomal protein S14) caused macrocytic anemia, prominent erythroid dysplasia and mon

22 in SFXN4 as a cause of mitochondriopathy and macrocytic anemia.

23 of the MDS progenitors, which corrected the macrocytic anemia.

24 neutrophils and monocytes, associated with a macrocytic anemia.

25 ia, a disorder characterized by hypoplastic, macrocytic anemia.

26 are responsible for Pearsons syndrome with a macrocytic anemia.

27 ly with multiple female individuals who have macrocytic anemia.

28 cking the heme exporter FLVCR1 have a severe macrocytic anemia; however, the mechanisms that underlie

29 inant effects; mild pigmentation defects and macrocytic, hypoplastic anemia occur in heterozygous mic

30 -deficient (PCFT(-/-)) mice developed severe macrocytic normochromic anemia and pancytopenia.

31 ctivation or inhibition of mTORC1 results in macrocytic or microcytic anemia, respectively.

32 ice develop massive splenomegaly, basophilic macrocytic red blood cells, and anemia as they age.

33 ions of myopathic exercise intolerance and a macrocytic sideroblastic anemia.

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