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1  yielded a low serum vitamin B(12) level and macrocytosis.
2 gamma-globin mRNA expression and resulted in macrocytosis.
3 throcyte adenosine deaminase activity and/or macrocytosis.
4 a marginally significantly decreased odds of macrocytosis.
5 also noted that these children commonly show macrocytosis, abnormal platelet counts, and an increased
6 sents as a compensated hemolytic anemia with macrocytosis and abnormally shaped red blood cells (RBCs
7 scovered that Ts65Dn mice display persistent macrocytosis and develop a myeloproliferative disease (M
8 ads to the development of severe anemia with macrocytosis and monocytosis.
9                In general, the prevalence of macrocytosis and the odds of having macrocytosis did not
10  and vitamin B-12 status relative to anemia, macrocytosis, and cognitive impairment (ie, Digit Symbol
11  elevated C-reactive protein concentrations, macrocytosis, and normal serum cobalamin concentrations;
12 ortification may have led to a correction of macrocytosis associated with vitamin B-12 insufficiency.
13 ividuals with low serum vitamin B-12 without macrocytosis by sex, race, and age according to preforti
14  showed a vitamin B-12 deficit manifested by macrocytosis, circulating vitamin B-12 concentrations <1
15 lence of macrocytosis and the odds of having macrocytosis did not change significantly from 1988-1994
16 serum vitamin B-12 were likely to be without macrocytosis in the postfortification period.
17     Their morphology appeared immature, with macrocytosis, irregular shape, and large autophagolysoso
18  mutations had reduced coat pigmentation and macrocytosis of peripheral blood.
19 ia [odds ratio (OR): 2.7; 95% CI: 1.7, 4.2], macrocytosis (OR: 1.8; 95% CI: 1.01, 3.3), and cognitive
20 ed from 30% to 40% for 6 years, although the macrocytosis persists (mean corpuscular volume, 100-112
21     Children with Down syndrome (DS) display macrocytosis, thrombocytosis, and a 500-fold increased r
22 ividuals with low serum vitamin B-12 without macrocytosis (undiagnosed vitamin B-12 deficiency) has i
23 io for having low serum vitamin B-12 without macrocytosis was 3.0 (95% CI: 1.7, 5.2) in the postforti
24 subjects with low serum vitamin B-12 without macrocytosis was significantly higher in the postfortifi
25 and prevalences and likelihood of anemia and macrocytosis were determined for 26,596 adults examined
26 ends in indexes and prevalence of anemia and macrocytosis with a focus on comparison of prefortificat

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