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   1 ndrome (of whom 2 eventually developed overt macrophage activation syndrome).                        
     2 e into a life-threatening condition known as macrophage activation syndrome.                         
     3  sCD163 are promising diagnostic markers for macrophage activation syndrome.                         
     4 comparable with those in patients with acute macrophage activation syndrome.                         
     5 also help identify patients with subclinical macrophage activation syndrome.                         
     6 requently associated with the development of macrophage activation syndrome.                         
     7 nd the relationship between systemic JIA and macrophage activation syndrome.                         
     8 d (5) hemophagocytic lymphohistiocytosis and macrophage activation syndrome.                         
     9 ted intravascular coagulation as features of macrophage activation syndrome.                         
    10 ses, disturbances in cytokine signaling, and macrophage activation syndromes.                        
    11 rheumatoid arthritis is the association with macrophage activation syndrome, a life-threatening compl
    12 is trials, anakinra is effective in treating macrophage activation syndrome, a similar entity with fe
    13  such dysregulation and the relation between macrophage activation syndrome and hemophagocytic lympho
    14 urrent understanding of the relation between macrophage activation syndrome and other clinically simi
    15 63 levels in sera from 7 patients with acute macrophage activation syndrome complicating systemic JIA
    16 m sIL-2Ralpha and sCD163 in diagnosing acute macrophage activation syndrome complicating systemic juv
  
    18 ospective randomized trial using features of macrophage activation syndrome for mortality risk strati
    19 tin levels (including those with subclinical macrophage activation syndrome) from those with normal o
  
    21 ut so are cytokines that are associated with macrophage activation syndrome/hemophagocytic lymphohist
  
    23 g can be a useful tool for identifying early macrophage activation syndrome in patients with systemic
    24 assification, pathogenesis and management of macrophage activation syndrome in systemic onset juvenil
    25  to further elucidate the pathophysiology of macrophage activation syndrome in systemic onset juvenil
    26 s of perforin expression may be a feature of macrophage activation syndrome in systemic-onset juvenil
  
    28 elated with clinical features of established macrophage activation syndrome, including ferritin level
  
  
  
    32 Hemophagocytic lymphohistiocytosis (HLH) and macrophage activation syndrome (MAS) are 2 similar disea
    33 hemophagocytic lymphohistiocytosis (HLH) and macrophage activation syndrome (MAS) are both characteri
  
  
  
  
  
  
  
  
  
    43  both hemophagocytic lymphohistiocytosis and macrophage activation syndrome, natural killer and cytot
  
    45 IA, 5 of whom showed evidence of subclinical macrophage activation syndrome (of whom 2 eventually dev
  
    47 an level of sIL-2Ralpha in the patients with macrophage activation syndrome was 19,646 pg/ml (interqu
    48  the median level of sCD163 in patients with macrophage activation syndrome was 23,000 ng/ml (IQR 14,
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