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1 AML1 target gene Csf1r (c-fms), encoding the macrophage colony stimulating factor receptor.
2 including CD11b, CD11c, CD18, CD14, and the macrophage colony-stimulating factor receptor.
3 hat is driven by the increased expression of macrophage colony-stimulating factor receptor.
4 or, colony-stimulating factor 1 (also called macrophage colony-stimulating factor) receptor.
5 subunit of the interleukin 3 and granulocyte/macrophage-colony-stimulating factor receptors.
6 d interleukin (IL)-2 and GM-CSF (granulocyte/macrophage colony-stimulating factor) receptors.
7 C production of the osteoclastogenic factors macrophage colony-stimulating factor, receptor activator
8 ction, and surface expression of granulocyte-macrophage colony-stimulating factor receptor; all three
9 entiation, such as c-fes, c-pim, granulocyte-macrophage colony-stimulating factor receptor alpha chai
10 ty alpha receptor binding chain (granulocyte-macrophage colony-stimulating factor receptor alpha subu
13 of the PU.1 transcription factor such as the macrophage colony-stimulating factor receptor and PU.1 i
15 pression of the myeloid markers Gr-1, CD115 (macrophage colony-stimulating factor receptor), and F4/8
16 ters (T cell receptor beta, myeloperoxidase, macrophage colony-stimulating factor receptor, and granu
17 egulation of the IL-3, IL-5, and granulocyte-macrophage colony-stimulating factor receptor beta commo
18 ; P < 0.0001); and expression of granulocyte-macrophage colony-stimulating factor receptor beta subun
19 Gab3 has been shown to act downstream of the macrophage colony-stimulating factor receptor, c-Fms, an
20 unction of circulating leukocyte granulocyte-macrophage colony-stimulating factor receptor (CD116) me
21 stimulating factor receptor, and granulocyte macrophage colony-stimulating factor receptor) containin
26 nd binding assay demonstrated an increase in macrophage colony-stimulating factor receptor expression
27 Importantly, granulocyte/macrophage-colony stimulating factor receptor expression
28 linked to the interleukin (IL)-3/granulocyte-macrophage colony-stimulating factor receptors for growt
29 /MDS1 at the AML1 binding site of the CSF1R (macrophage-colony-stimulating factor receptor gene) prom
30 MacGreen reporter mice (mice expressing the macrophage colony-stimulating factor receptor GFP transg
32 kin-5 receptor (IL-5R) or of the granulocyte-macrophage colony-stimulating factor receptor (GM-CSFR)
33 e alpha or beta subunit of human granulocyte-macrophage colony-stimulating factor receptor (GM-CSFr)
35 Cs under the control of Flt3 and granulocyte-macrophage-colony-stimulating factor receptor (GM-CSFR)
39 lts in which PU.1, miRNA-223, NFI-A, and the macrophage colony-stimulating factor receptor (M-CSFR) a
40 /CD11b(high) cells and greater expression of macrophage colony-stimulating factor receptor (M-CSFR) i
41 sease have enhanced immunoreactivity for the macrophage colony-stimulating factor receptor (M-CSFR),
43 promote myeloid cells to differentiate into macrophage colony-stimulating factor receptor (M-CSFR)-p
44 Microglia with increased expression of the macrophage colony-stimulating factor receptor (M-CSFR; c
45 1(+) lamina propria DCs under the control of macrophage-colony-stimulating factor receptor (M-CSFR) a
46 tion (with increasing expression of CD13 and macrophage colony-stimulating factor receptor [M-CSFR])
47 els of luciferase expression directed by the macrophage colony-stimulating factor receptor (MCSFR) pr
48 on genes, such as macrophage and granulocyte macrophage colony-stimulating factor receptors (MCSFR an
49 tion of H2O2 was diminished in a granulocyte-macrophage colony-stimulating factor receptor mutant una
50 einosis without mutations in the granulocyte-macrophage colony-stimulating factor receptor or anti-gr
51 ciency to increase interleukin 3/granulocyte-macrophage colony-stimulating factor receptor signaling
53 , such as c-fms, CD11b, CD18 and granulocyte-macrophage colony-stimulating factor receptor, were not
54 The murine c-fms (Csf1r) gene encodes the macrophage colony-stimulating factor receptor, which is
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